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1 isotopic composition of atmospheric nitrate (nitric acid (HNO(3)) + particulate nitrate (pNO(3)(-)))
2 ia merging a droplet of an ionic liquid with nitric acid (HNO(3)), and the coalescence of a solid par
3 is cold enough (below -15 degrees Celsius), nitric acid and ammonia can nucleate directly through an
6 t CERN, that below about +5 degrees Celsius, nitric acid and ammonia vapours can condense onto freshl
7 ations, we analyze the interface affinity of nitric acid and provide an order of magnitude for its li
8 ain insight into the properties of molecular nitric acid at the surface of liquid water (the air-wate
9 ntly by a water-mediated process (leading to nitric acid formation), while the lifetime of a similar
10 which has huge environmental relevance since nitric acid is a component of acid rain and also contrib
11 cted from the continental boundary layer and nitric acid is abundant from electrical storms(4,5).
13 ysed post hoc by baseline fractional exhaled nitric oxide (<35 and >=35 ppb) and blood eosinophil (<2
14 hing (+30%), nitrous oxide emissions (+30%), nitric oxide (+66%) emissions, and ammonia volatilizatio
16 lammation was assessed by fractional exhaled nitric oxide (FeNO) and exhaled breath condensate pH and
17 icosteroid adherence, and fractional exhaled nitric oxide (FeNO) in 17 studies for inhaled corticoste
19 t 2013 and February 2015, fractional exhaled nitric oxide (FeNO) was measured from 112 participants a
20 omuscular blockade, corticosteroids, inhaled nitric oxide (iNO), prone positioning, high-frequency os
21 b) and a 6 min inhalation of the vasodilator nitric oxide (iNO; 40 ppm in 21% O(2) ), to selectively
24 emed to be endothelium dependent, as well as nitric oxide (NO) and guanylyl cyclase, but not prostagl
25 genomic approaches, we provide evidence that nitric oxide (NO) and oxylipin signalling pathways in di
26 A subset of these TFs is targeted each by nitric oxide (NO) and the phytochrome-interacting TF PIL
27 of evidence demonstrating that scavenging of nitric oxide (NO) and the reduction of nitrate/nitrite r
28 ction, due at least in part to reductions in nitric oxide (NO) bioavailability and associated vascula
29 onths of RT on sleep quality, redox balance, nitric oxide (NO) bioavailability, inflammation profile,
32 al-mediated routes for nitrite (NO(2)(-)) to nitric oxide (NO) conversion and phenol oxidation are of
33 discovered that KSHV hijacks the citrulline-nitric oxide (NO) cycle to promote growth proliferation
35 cle, cytoglobin (Cygb) functions as a potent nitric oxide (NO) dioxygenase and regulates NO metabolis
49 o investigate how the environmental stimulus nitric oxide (NO) is linked to biofilm dispersal, focusi
52 a reduced growth and photosynthesis, altered nitric oxide (NO) level and leaf and root anatomy, inhib
54 reported concerning the effects of augmented nitric oxide (NO) on skeletal muscle force production an
55 dividual subunits, the KsNaxLS complex binds nitric oxide (NO) only at the distal heme side, forming
56 ransfer (PET) mechanism, we designed a smart nitric oxide (NO) probe, PYSNO, with high sensitivity an
60 strated with pharmacological approaches that nitric oxide (NO) produced through the citrulline-NO pat
62 ically alters the transcriptome and triggers nitric oxide (NO) production in F. graminearum We identi
66 controls pathogenesis via the regulation of nitric oxide (NO) resistance and inorganic nitrogen meta
67 y-activated tumor cells, iNOS expression and nitric oxide (NO) secretion were significantly increased
68 sorders are associated with perturbations to nitric oxide (NO) signaling and impaired glucose metabol
74 ement was abolished by pretreatment with the nitric oxide (NO) synthase inhibitor l-N (G)-nitro-l-arg
76 pecific tyrosine residues and interaction of nitric oxide (NO) with the P450 heme are necessary for N
77 mor-promoting and immunosuppressive molecule nitric oxide (NO), whereas macrophages largely express a
78 on product (APOP), myeloperoxidase (MPO) and nitric oxide (NO), while depleting levels of endogenous
79 uman cytochrome P450 (P450) CYP2B6 undergoes nitric oxide (NO)-dependent proteasomal degradation in r
81 he transduction of endothelium-dependent and nitric oxide (NO)-mediated vasodilator activity, given i
88 >=150/300 cells/uL and/or fractional exhaled nitric oxide [FeNO] >=25 ppb), annualized severe exacerb
90 Here we show that the protective actions of nitric oxide against EMCV infection are selective for be
91 d an increase in the inhibition of hydroxyl, nitric oxide and alpha-amylase, as well as a decrease in
92 rved in the high baseline fractional exhaled nitric oxide and blood eosinophil subgroups (207 mL [95%
93 50] for the high baseline fractional exhaled nitric oxide and blood eosinophil subgroups, respectivel
94 le signaling processes such as production of nitric oxide and caveolae-mediated intracellular traffic
95 calmodulin nitrated at Tyr-138 produced more nitric oxide and did so more efficiently than WT calmodu
96 r that promotes vasorelaxation by increasing nitric oxide and downregulating endothelin-1 (ET-1) prod
98 ich nanoparticles, oxidants that may destroy nitric oxide and impair endothelial function, but also l
100 osation in response to exposure to both free nitric oxide and nitrosothiols (k (inact)/K(I) >= 5 m(-1
101 assessed innate immune indices (haptoglobin, nitric oxide and ovotransferrin concentrations, and haem
102 molecule generated from the reaction between nitric oxide and superoxide radicals, in impairing endot
103 ons in obese mice generated higher levels of nitric oxide and superoxide radicals, resulting in incre
104 data point towards haemoglobin scavenging of nitric oxide as a key regulatory factor of brachial flow
105 cterization revealed that in the presence of nitric oxide as a spin probe and oxygen surrogate, both
107 gonorrhoeae blocks exfoliation by producing nitric oxide at the bacterial-host cell interface to pro
108 al at the ovule triggers the accumulation of nitric oxide at the filiform apparatus in a process that
109 itively, release of the vasodilator molecule nitric oxide attenuated endothelial TRPV4 channel activi
110 (1-methylnicotinamide, +33%; P=6.1x10(-67)), nitric oxide bioavailability (arginine/ornithine + citru
112 trogenase inactivation; and Glb1-1 modulates nitric oxide concentration during symbiosis, from the ea
114 to -0.77]) and had lower fractional exhaled nitric oxide concentrations (13.9 vs. 24.4 ppb; P < 0.00
115 sis following IGF-1 treatment, adenosine and nitric oxide contributed to hypoxia-mediated coronary va
116 rpose of this study was to determine whether nitric oxide contributes to the beta-cell response to vi
117 ), and at least 2 conditions associated with nitric oxide deficiency (diabetes, hypertension, obesity
118 tify glutamatergic signaling dysfunction and nitric oxide deficiency as yet-undescribed early manifes
121 lobins are cytoplasmic proteins required for nitric oxide detoxification and nitrosative stress resis
123 ADP-ribosylation reactions using a chemical nitric oxide donor S-nitrosoglutathione and enzymatic AD
125 itrite, which may subsequently be reduced to nitric oxide during conditions of hypoxia and in the pre
126 reduced production of the potent vasodilator nitric oxide during glutamatergic synaptic activity.
128 f nitrate leaching, nitrous oxide emissions, nitric oxide emissions, and ammonia volatilization total
129 oped iron sulfide nanoclusters that catalyse nitric oxide generation from benign sodium nitrite in th
132 unoblotting, proximity ligation as-says, and nitric oxide imaging, we report that phosphorylation of
135 y advance mechanistic studies of the role of nitric oxide in the nervous system and other organs.
139 Preclinical models have demonstrated that nitric oxide is a key component of neurovascular couplin
141 r coupling response by ~30%, indicating that nitric oxide is integral to neurovascular coupling in hu
142 odilatation and reduced resting steady-state nitric oxide levels in the blood Collectively, our data
147 ed on a combination of tests including nasal nitric oxide measurement, transmission electron microsco
152 ella strains induced apoptosis or stimulated nitric oxide or lactose dehydrogenase production in matu
153 activated the JAK/STAT1/IRF1 axis, inducing nitric oxide production and driving caspase-8/FADD-media
154 suring mechanosensing, which is essential to nitric oxide production and flow-induced vasodilation.
155 ortance of the simultaneous consideration of nitric oxide production and inactivation when investigat
158 factor alpha (TNF-alpha) mRNA and increased nitric oxide production during T. parva lethal infection
159 vessel wall shear stress and, consequently, nitric oxide production may contribute to heightened sys
160 , regulates permeability, leukocyte traffic, nitric oxide production, and coagulation, and harbors di
161 factor-kappaB (NF-kappaB) subunit p65, lower nitric oxide production, and diminished CHOP expression
162 ctivator of transcription (STAT) pathway and nitric oxide production, as well as the promotion of ade
163 ency prevents NMDA receptors from triggering nitric oxide production, thereby attenuating the flow in
165 The reduction of NO to N(2)O by flavodiiron nitric oxide reductases (FNORs) is related to the disrup
167 sis revealed that endogenous accumulation of nitric oxide resulted in up-regulation of genes involved
169 rescued F508del-CFTR by arginine-dependent, nitric oxide signaling through inhibition of endogenous
174 in regulating cell growth and responding to nitric oxide stress in M. tuberculosis, but its underlyi
175 ime polymerase chain reaction of endothelial nitric oxide synthase (eNOS) and endothelin-1; (d) immun
176 ce K(+) (IK and SK) channels and endothelial nitric oxide synthase (eNOS) are present in the endothel
178 Nitric oxide (NO) produced by endothelial nitric oxide synthase (eNOS) is a critical mediator of v
179 fragment of the shear-responsive endothelial nitric oxide synthase (eNOS) promoter, we tested effects
180 in (TM), a surface receptor, and endothelial nitric oxide synthase (eNOS), an enzyme that generates n
181 ated protein kinase (AMPK), Akt, endothelial nitric oxide synthase (eNOS), nuclear factor erythroid 2
182 skin wound angiogenesis through endothelial nitric oxide synthase (eNOS)-dependent Src, PI3K, and MA
183 PepNats representing hot loops of inducible nitric oxide synthase (iNOS) and human agouti-related pr
184 biomarkers include suppression of inducible nitric oxide synthase (iNOS) and induction of sterol reg
185 receptor (CB1R) and activation of inducible nitric oxide synthase (iNOS) are associated with nonalco
187 s that are sufficient to stimulate inducible nitric oxide synthase (iNOS) expression and production o
189 essions of interleukin (IL)-1beta, inducible nitric oxide synthase (iNOS), cyclooxygenase (COX)-2, ma
190 ta); and the inflammatory markers: Inducible Nitric Oxide Synthase (iNOS), Interleukin 1beta (IL-1bet
191 ro-apoptotic signaling mediated by inducible nitric oxide synthase (iNOS), transforming growth factor
192 nylate-binding proteins (GBPs) and inducible nitric oxide synthase (iNOS), which we found to inhibit
194 erythroid-derived 2)-like 2 (Nrf2), neuronal Nitric Oxide Synthase (nNOS) expression and nitrergic re
195 olves a tau-induced dissociation of neuronal nitric oxide synthase (nNOS) from postsynaptic density 9
197 In the course of our studies on neuronal nitric oxide synthase (nNOS), a client of the Hsp90 and
199 ide synthase 1 adaptor protein) and neuronal nitric oxide synthase (nNOS), nNOS enzymatic activity, a
202 od flow (FBF) response to acetylcholine, and nitric oxide synthase (NOS) activity was defined as the
203 erized further by triple labeling with CGRP, nitric oxide synthase (NOS) and calretinin (CALR) antibo
205 -like 2 (Nrf2)/tetrahydrobiopterin (BH(4) )/ nitric oxide synthase (NOS) expression in primary human
206 phorase is used as a histochemical marker of nitric oxide synthase (NOS) in aldehyde-treated tissues.
209 ermined by lactate dehydrogenase release and nitric oxide synthase (NOS)-dependent cGMP production.
210 ed STAT1 and its downstream target inducible nitric oxide synthase (NOS2) as being upregulated in the
211 Rs) on the host cell, it activates inducible nitric oxide synthase (NOS2) which instantly fills the p
215 nergy balance (e.g. oxytocin (OXT), neuronal nitric oxide synthase 1 (NOS1), melanocortin 4-receptor
216 e, including the interaction between NOS1AP (nitric oxide synthase 1 adaptor protein) and neuronal ni
217 tide 3-phosphate kinase, Akt Ser/Thr kinase, nitric oxide synthase 1, nitric oxide, soluble guanylate
218 Inbred mice that lack the gene encoding nitric oxide synthase 2 (Nos2) are susceptible to the re
219 infection, and testing MAIT cell priming in nitric oxide synthase 2 (NOS2)-deficient mice all failed
220 CXCL9/CXCL10), and innate immunity-related (nitric oxide synthase 2/inducible nitric oxide synthase
222 receptor agonist analog elicited endothelial nitric oxide synthase activation to promote endothelial
224 y-related (nitric oxide synthase 2/inducible nitric oxide synthase and IL-17C) products (FDR < 0.05).
225 ed distinct anti-inflammatory (low inducible nitric oxide synthase and lower tumour necrosis factor-a
227 % of myenteric neurons stained with neuronal nitric oxide synthase antibody and approximately 33% of
228 l perfusion has been used to determine which nitric oxide synthase enzymes are active in discrete reg
230 beta cells, which are mediated by inducible nitric oxide synthase expression and beta-cell productio
231 rotocols utilizing intravenous infusion of a nitric oxide synthase inhibitor and isovolumic haemodilu
232 (placebo) and the non-selective competitive nitric oxide synthase inhibitor N(G) -monomethyl-l-argin
235 mbers of nitrergic neurons, reduced neuronal nitric oxide synthase production, and reduced colonic ne
236 d investigated the role of 12,13-diHOME in a nitric oxide synthase type 1 deficient (NOS1(-/-)) mouse
237 osine kinase(Tek), isolectin B4, endothelial nitric oxide synthase(eNOS), von Willebrand factor(vWF),
239 gressive blockade of adenosine receptors and nitric oxide synthase, and by modeling the determinants
240 hways, specifically MAPK/ERK1/2, endothelial nitric oxide synthase, and mammalian target of rapamycin
241 dase, uncoupling of endothelial and neuronal nitric oxide synthase, and vascular/brain infiltration w
242 neurons showed increased levels of inducible nitric oxide synthase, another indicator of cell damage.
243 A), an endogenous inhibitor and uncoupler of nitric oxide synthase, has gained attention as a risk fa
244 e molecules, including arginase 1, inducible nitric oxide synthase, signal transducer and activator o
245 ecrosis factor-alpha, interleukin-1beta, and nitric oxide synthase-2, highlighting the potential of V
246 ossibly by affecting the NF-kappaB-inducible nitric oxide synthase-endoplasmic reticulum stress pathw
247 alpha(+)]), and effector molecule (inducible nitric oxide synthase-positive [iNOS(+)]) responses were
250 ression revealed significant upregulation of nitric oxide synthetase (NOS1 and NOS3) and neuroprotect
251 lective manner, these findings indicate that nitric oxide targets the same metabolic pathways necessa
253 yurea generates a protocell-mediated flux of nitric oxide that we exploit for in vitro and in vivo bl
254 paminergic neurons release both dopamine and nitric oxide to increase the flexibility of olfactory me
258 r of exosomal nitric oxide (NO) synthase and nitric oxide was measured by using ELISAs and NO activit
264 NMDA receptors (NMDARs) play a role through nitric oxide, and the CBF increase produced by endotheli
265 CI, -32.9%, -10.2%), and fractional exhaled nitric oxide, CCL26 and SERPINB2 mRNA expression in bron
266 ctivity, characterised by the measurement of nitric oxide, endothelin-1, tissue plasminogen activator
267 e involved in redox signalling, for instance nitric oxide, hydrogen sulfide and oxidized lipids.
268 d to cause cell death by virtue of releasing nitric oxide, is highly unstable in air and is rapidly o
269 impact of various nitrogen oxides, including nitric oxide, nitrogen dioxide, and nitrous oxide, on ca
270 e and S-nitroso-N-acetyl-d,l-penicillamine), nitric oxide, oxidized GSH, and hydrogen peroxide to pos
273 Akt Ser/Thr kinase, nitric oxide synthase 1, nitric oxide, soluble guanylate cyclase, cyclic GMP (cGM
274 acula densa cells triggers the production of nitric oxide, which also contributes to glomerular hyper
275 truncated NOS2 protein that did not produce nitric oxide, which determined that the patient had auto
276 s novel insight into the mechanisms by which nitric oxide, which is produced in hepatocytes in respon
277 off axonal actin polymerization, mediated by nitric oxide-cyclic GMP signaling leading to inhibition
280 Collectively, these results suggest that nitric oxide-derived oxidants may causatively link nucle
282 anoclusters with multimaterial fibres allows nitric oxide-mediated neuronal interrogation in vivo.
285 Locally generated nitric oxide activates the nitric oxide-sensitive cation channel, transient recepto
286 (hemoglobin) and oxygen, carbon dioxide, and nitric oxide-the three-gas respiratory cycle-that insure
293 The activity is critical in initiating a nitric oxide/S-nitrosylation-dependent signal transducti
294 acorporeal membrane oxygenation, and inhaled nitric oxide; Pao2:Fio2 ratio measured daily from day 1
295 stress stimuli and haemoglobin scavenging of nitric oxide; these two regulatory factors have not been
296 Tyr-99 revealed that this nitration reduces nitric-oxide production and increases eNOS decoupling co
297 n species caused by dysregulated endothelial nitric-oxide synthase (eNOS) activity is linked to vascu
299 levels of proinflammatory markers: inducible nitric-oxide synthase and tumor necrosis factor-alpha, w
300 we develop a step towards protocell-mediated nitric-oxide-induced vasodilation by constructing a new