コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 n of iNOS and production of the free radical nitric oxide.
2 n, paralysis, prone positioning, and inhaled nitric oxide.
3 s low micromolar, or iNOS-derived, levels of nitric oxide.
4 thought to be targets of endothelium-derived nitric oxide.
5 thase expression and beta-cell production of nitric oxide.
7 hing (+30%), nitrous oxide emissions (+30%), nitric oxide (+66%) emissions, and ammonia volatilizatio
10 Here we show that the protective actions of nitric oxide against EMCV infection are selective for be
12 d an increase in the inhibition of hydroxyl, nitric oxide and alpha-amylase, as well as a decrease in
13 rved in the high baseline fractional exhaled nitric oxide and blood eosinophil subgroups (207 mL [95%
14 50] for the high baseline fractional exhaled nitric oxide and blood eosinophil subgroups, respectivel
16 le signaling processes such as production of nitric oxide and caveolae-mediated intracellular traffic
17 calmodulin nitrated at Tyr-138 produced more nitric oxide and did so more efficiently than WT calmodu
18 r that promotes vasorelaxation by increasing nitric oxide and downregulating endothelin-1 (ET-1) prod
19 view is to highlight the biologic effects of nitric oxide and hydrogen sulfide, their seemingly indis
21 ich nanoparticles, oxidants that may destroy nitric oxide and impair endothelial function, but also l
23 osation in response to exposure to both free nitric oxide and nitrosothiols (k (inact)/K(I) >= 5 m(-1
24 assessed innate immune indices (haptoglobin, nitric oxide and ovotransferrin concentrations, and haem
25 molecule generated from the reaction between nitric oxide and superoxide radicals, in impairing endot
26 ons in obese mice generated higher levels of nitric oxide and superoxide radicals, resulting in incre
29 NMDA receptors (NMDARs) play a role through nitric oxide, and the CBF increase produced by endotheli
30 data point towards haemoglobin scavenging of nitric oxide as a key regulatory factor of brachial flow
31 cterization revealed that in the presence of nitric oxide as a spin probe and oxygen surrogate, both
33 gonorrhoeae blocks exfoliation by producing nitric oxide at the bacterial-host cell interface to pro
34 al at the ovule triggers the accumulation of nitric oxide at the filiform apparatus in a process that
35 itively, release of the vasodilator molecule nitric oxide attenuated endothelial TRPV4 channel activi
36 (1-methylnicotinamide, +33%; P=6.1x10(-67)), nitric oxide bioavailability (arginine/ornithine + citru
39 CI, -32.9%, -10.2%), and fractional exhaled nitric oxide, CCL26 and SERPINB2 mRNA expression in bron
41 trogenase inactivation; and Glb1-1 modulates nitric oxide concentration during symbiosis, from the ea
43 to -0.77]) and had lower fractional exhaled nitric oxide concentrations (13.9 vs. 24.4 ppb; P < 0.00
44 sis following IGF-1 treatment, adenosine and nitric oxide contributed to hypoxia-mediated coronary va
45 rpose of this study was to determine whether nitric oxide contributes to the beta-cell response to vi
46 off axonal actin polymerization, mediated by nitric oxide-cyclic GMP signaling leading to inhibition
47 ), and at least 2 conditions associated with nitric oxide deficiency (diabetes, hypertension, obesity
48 tify glutamatergic signaling dysfunction and nitric oxide deficiency as yet-undescribed early manifes
54 Collectively, these results suggest that nitric oxide-derived oxidants may causatively link nucle
55 lobins are cytoplasmic proteins required for nitric oxide detoxification and nitrosative stress resis
57 ADP-ribosylation reactions using a chemical nitric oxide donor S-nitrosoglutathione and enzymatic AD
59 itrite, which may subsequently be reduced to nitric oxide during conditions of hypoxia and in the pre
60 reduced production of the potent vasodilator nitric oxide during glutamatergic synaptic activity.
62 f nitrate leaching, nitrous oxide emissions, nitric oxide emissions, and ammonia volatilization total
63 ctivity, characterised by the measurement of nitric oxide, endothelin-1, tissue plasminogen activator
65 lammation was assessed by fractional exhaled nitric oxide (FeNO) and exhaled breath condensate pH and
66 icosteroid adherence, and fractional exhaled nitric oxide (FeNO) in 17 studies for inhaled corticoste
68 t 2013 and February 2015, fractional exhaled nitric oxide (FeNO) was measured from 112 participants a
69 >=150/300 cells/uL and/or fractional exhaled nitric oxide [FeNO] >=25 ppb), annualized severe exacerb
70 oped iron sulfide nanoclusters that catalyse nitric oxide generation from benign sodium nitrite in th
74 unoblotting, proximity ligation as-says, and nitric oxide imaging, we report that phosphorylation of
77 y advance mechanistic studies of the role of nitric oxide in the nervous system and other organs.
79 we develop a step towards protocell-mediated nitric-oxide-induced vasodilation by constructing a new
82 omuscular blockade, corticosteroids, inhaled nitric oxide (iNO), prone positioning, high-frequency os
83 b) and a 6 min inhalation of the vasodilator nitric oxide (iNO; 40 ppm in 21% O(2) ), to selectively
84 Preclinical models have demonstrated that nitric oxide is a key component of neurovascular couplin
86 r coupling response by ~30%, indicating that nitric oxide is integral to neurovascular coupling in hu
87 d to cause cell death by virtue of releasing nitric oxide, is highly unstable in air and is rapidly o
88 odilatation and reduced resting steady-state nitric oxide levels in the blood Collectively, our data
93 ysed post hoc by baseline fractional exhaled nitric oxide (<35 and >=35 ppb) and blood eosinophil (<2
94 ed on a combination of tests including nasal nitric oxide measurement, transmission electron microsco
96 anoclusters with multimaterial fibres allows nitric oxide-mediated neuronal interrogation in vivo.
97 calization phenotype of these two mutants by nitric oxide-mediated/protein kinase G-dependent phospho
101 impact of various nitrogen oxides, including nitric oxide, nitrogen dioxide, and nitrous oxide, on ca
105 emed to be endothelium dependent, as well as nitric oxide (NO) and guanylyl cyclase, but not prostagl
106 genomic approaches, we provide evidence that nitric oxide (NO) and oxylipin signalling pathways in di
107 A subset of these TFs is targeted each by nitric oxide (NO) and the phytochrome-interacting TF PIL
108 of evidence demonstrating that scavenging of nitric oxide (NO) and the reduction of nitrate/nitrite r
109 ction, due at least in part to reductions in nitric oxide (NO) bioavailability and associated vascula
110 onths of RT on sleep quality, redox balance, nitric oxide (NO) bioavailability, inflammation profile,
113 al-mediated routes for nitrite (NO(2)(-)) to nitric oxide (NO) conversion and phenol oxidation are of
114 discovered that KSHV hijacks the citrulline-nitric oxide (NO) cycle to promote growth proliferation
116 cle, cytoglobin (Cygb) functions as a potent nitric oxide (NO) dioxygenase and regulates NO metabolis
130 o investigate how the environmental stimulus nitric oxide (NO) is linked to biofilm dispersal, focusi
133 a reduced growth and photosynthesis, altered nitric oxide (NO) level and leaf and root anatomy, inhib
135 reported concerning the effects of augmented nitric oxide (NO) on skeletal muscle force production an
136 dividual subunits, the KsNaxLS complex binds nitric oxide (NO) only at the distal heme side, forming
137 ransfer (PET) mechanism, we designed a smart nitric oxide (NO) probe, PYSNO, with high sensitivity an
141 strated with pharmacological approaches that nitric oxide (NO) produced through the citrulline-NO pat
143 ically alters the transcriptome and triggers nitric oxide (NO) production in F. graminearum We identi
147 controls pathogenesis via the regulation of nitric oxide (NO) resistance and inorganic nitrogen meta
148 Recombinant proteins were used to examine nitric oxide (NO) scavenging in vitro and transgenic pla
149 y-activated tumor cells, iNOS expression and nitric oxide (NO) secretion were significantly increased
150 sorders are associated with perturbations to nitric oxide (NO) signaling and impaired glucose metabol
156 ement was abolished by pretreatment with the nitric oxide (NO) synthase inhibitor l-N (G)-nitro-l-arg
158 pecific tyrosine residues and interaction of nitric oxide (NO) with the P450 heme are necessary for N
159 mor-promoting and immunosuppressive molecule nitric oxide (NO), whereas macrophages largely express a
160 on product (APOP), myeloperoxidase (MPO) and nitric oxide (NO), while depleting levels of endogenous
161 uman cytochrome P450 (P450) CYP2B6 undergoes nitric oxide (NO)-dependent proteasomal degradation in r
163 he transduction of endothelium-dependent and nitric oxide (NO)-mediated vasodilator activity, given i
172 ella strains induced apoptosis or stimulated nitric oxide or lactose dehydrogenase production in matu
173 e and S-nitroso-N-acetyl-d,l-penicillamine), nitric oxide, oxidized GSH, and hydrogen peroxide to pos
174 acorporeal membrane oxygenation, and inhaled nitric oxide; Pao2:Fio2 ratio measured daily from day 1
175 amate metabolism, dysfunctional arginine and nitric oxide pathways, and increased oxidative stress.
177 activated the JAK/STAT1/IRF1 axis, inducing nitric oxide production and driving caspase-8/FADD-media
178 suring mechanosensing, which is essential to nitric oxide production and flow-induced vasodilation.
179 ortance of the simultaneous consideration of nitric oxide production and inactivation when investigat
182 factor alpha (TNF-alpha) mRNA and increased nitric oxide production during T. parva lethal infection
183 vessel wall shear stress and, consequently, nitric oxide production may contribute to heightened sys
184 , regulates permeability, leukocyte traffic, nitric oxide production, and coagulation, and harbors di
185 factor-kappaB (NF-kappaB) subunit p65, lower nitric oxide production, and diminished CHOP expression
186 ctivator of transcription (STAT) pathway and nitric oxide production, as well as the promotion of ade
187 ency prevents NMDA receptors from triggering nitric oxide production, thereby attenuating the flow in
188 Tyr-99 revealed that this nitration reduces nitric-oxide production and increases eNOS decoupling co
190 The reduction of NO to N(2)O by flavodiiron nitric oxide reductases (FNORs) is related to the disrup
193 sis revealed that endogenous accumulation of nitric oxide resulted in up-regulation of genes involved
194 The activity is critical in initiating a nitric oxide/S-nitrosylation-dependent signal transducti
197 Locally generated nitric oxide activates the nitric oxide-sensitive cation channel, transient recepto
198 rescued F508del-CFTR by arginine-dependent, nitric oxide signaling through inhibition of endogenous
203 Akt Ser/Thr kinase, nitric oxide synthase 1, nitric oxide, soluble guanylate cyclase, cyclic GMP (cGM
204 in regulating cell growth and responding to nitric oxide stress in M. tuberculosis, but its underlyi
205 ime polymerase chain reaction of endothelial nitric oxide synthase (eNOS) and endothelin-1; (d) immun
206 ce K(+) (IK and SK) channels and endothelial nitric oxide synthase (eNOS) are present in the endothel
208 Nitric oxide (NO) produced by endothelial nitric oxide synthase (eNOS) is a critical mediator of v
209 fragment of the shear-responsive endothelial nitric oxide synthase (eNOS) promoter, we tested effects
210 in (TM), a surface receptor, and endothelial nitric oxide synthase (eNOS), an enzyme that generates n
211 ated protein kinase (AMPK), Akt, endothelial nitric oxide synthase (eNOS), nuclear factor erythroid 2
212 skin wound angiogenesis through endothelial nitric oxide synthase (eNOS)-dependent Src, PI3K, and MA
213 and increased the phosphorylated endothelial nitric oxide synthase (eNOS)/eNOS ratio in an age-depend
214 PepNats representing hot loops of inducible nitric oxide synthase (iNOS) and human agouti-related pr
215 biomarkers include suppression of inducible nitric oxide synthase (iNOS) and induction of sterol reg
216 receptor (CB1R) and activation of inducible nitric oxide synthase (iNOS) are associated with nonalco
218 s that are sufficient to stimulate inducible nitric oxide synthase (iNOS) expression and production o
220 essions of interleukin (IL)-1beta, inducible nitric oxide synthase (iNOS), cyclooxygenase (COX)-2, ma
221 ta); and the inflammatory markers: Inducible Nitric Oxide Synthase (iNOS), Interleukin 1beta (IL-1bet
222 ro-apoptotic signaling mediated by inducible nitric oxide synthase (iNOS), transforming growth factor
223 nylate-binding proteins (GBPs) and inducible nitric oxide synthase (iNOS), which we found to inhibit
225 erythroid-derived 2)-like 2 (Nrf2), neuronal Nitric Oxide Synthase (nNOS) expression and nitrergic re
226 olves a tau-induced dissociation of neuronal nitric oxide synthase (nNOS) from postsynaptic density 9
228 In the course of our studies on neuronal nitric oxide synthase (nNOS), a client of the Hsp90 and
230 ide synthase 1 adaptor protein) and neuronal nitric oxide synthase (nNOS), nNOS enzymatic activity, a
233 od flow (FBF) response to acetylcholine, and nitric oxide synthase (NOS) activity was defined as the
234 erized further by triple labeling with CGRP, nitric oxide synthase (NOS) and calretinin (CALR) antibo
236 -like 2 (Nrf2)/tetrahydrobiopterin (BH(4) )/ nitric oxide synthase (NOS) expression in primary human
237 phorase is used as a histochemical marker of nitric oxide synthase (NOS) in aldehyde-treated tissues.
240 ermined by lactate dehydrogenase release and nitric oxide synthase (NOS)-dependent cGMP production.
241 ed STAT1 and its downstream target inducible nitric oxide synthase (NOS2) as being upregulated in the
242 Rs) on the host cell, it activates inducible nitric oxide synthase (NOS2) which instantly fills the p
246 nergy balance (e.g. oxytocin (OXT), neuronal nitric oxide synthase 1 (NOS1), melanocortin 4-receptor
247 e, including the interaction between NOS1AP (nitric oxide synthase 1 adaptor protein) and neuronal ni
248 tide 3-phosphate kinase, Akt Ser/Thr kinase, nitric oxide synthase 1, nitric oxide, soluble guanylate
249 Inbred mice that lack the gene encoding nitric oxide synthase 2 (Nos2) are susceptible to the re
250 infection, and testing MAIT cell priming in nitric oxide synthase 2 (NOS2)-deficient mice all failed
251 CXCL9/CXCL10), and innate immunity-related (nitric oxide synthase 2/inducible nitric oxide synthase
253 receptor agonist analog elicited endothelial nitric oxide synthase activation to promote endothelial
255 y-related (nitric oxide synthase 2/inducible nitric oxide synthase and IL-17C) products (FDR < 0.05).
256 ed distinct anti-inflammatory (low inducible nitric oxide synthase and lower tumour necrosis factor-a
258 % of myenteric neurons stained with neuronal nitric oxide synthase antibody and approximately 33% of
260 l perfusion has been used to determine which nitric oxide synthase enzymes are active in discrete reg
262 beta cells, which are mediated by inducible nitric oxide synthase expression and beta-cell productio
263 rotocols utilizing intravenous infusion of a nitric oxide synthase inhibitor and isovolumic haemodilu
264 (placebo) and the non-selective competitive nitric oxide synthase inhibitor N(G) -monomethyl-l-argin
267 mbers of nitrergic neurons, reduced neuronal nitric oxide synthase production, and reduced colonic ne
268 d investigated the role of 12,13-diHOME in a nitric oxide synthase type 1 deficient (NOS1(-/-)) mouse
269 osine kinase(Tek), isolectin B4, endothelial nitric oxide synthase(eNOS), von Willebrand factor(vWF),
271 gressive blockade of adenosine receptors and nitric oxide synthase, and by modeling the determinants
272 hways, specifically MAPK/ERK1/2, endothelial nitric oxide synthase, and mammalian target of rapamycin
273 dase, uncoupling of endothelial and neuronal nitric oxide synthase, and vascular/brain infiltration w
274 neurons showed increased levels of inducible nitric oxide synthase, another indicator of cell damage.
275 A), an endogenous inhibitor and uncoupler of nitric oxide synthase, has gained attention as a risk fa
276 e molecules, including arginase 1, inducible nitric oxide synthase, signal transducer and activator o
277 ecrosis factor-alpha, interleukin-1beta, and nitric oxide synthase-2, highlighting the potential of V
278 ossibly by affecting the NF-kappaB-inducible nitric oxide synthase-endoplasmic reticulum stress pathw
279 alpha(+)]), and effector molecule (inducible nitric oxide synthase-positive [iNOS(+)]) responses were
282 n species caused by dysregulated endothelial nitric-oxide synthase (eNOS) activity is linked to vascu
284 levels of proinflammatory markers: inducible nitric-oxide synthase and tumor necrosis factor-alpha, w
285 ression revealed significant upregulation of nitric oxide synthetase (NOS1 and NOS3) and neuroprotect
286 lective manner, these findings indicate that nitric oxide targets the same metabolic pathways necessa
288 yurea generates a protocell-mediated flux of nitric oxide that we exploit for in vitro and in vivo bl
289 (hemoglobin) and oxygen, carbon dioxide, and nitric oxide-the three-gas respiratory cycle-that insure
290 stress stimuli and haemoglobin scavenging of nitric oxide; these two regulatory factors have not been
291 paminergic neurons release both dopamine and nitric oxide to increase the flexibility of olfactory me
295 r of exosomal nitric oxide (NO) synthase and nitric oxide was measured by using ELISAs and NO activit
297 acula densa cells triggers the production of nitric oxide, which also contributes to glomerular hyper
298 truncated NOS2 protein that did not produce nitric oxide, which determined that the patient had auto
299 signals from activated T cells by releasing nitric oxide, which inhibits T cell proliferation and re
300 s novel insight into the mechanisms by which nitric oxide, which is produced in hepatocytes in respon