ライフサイエンスコーパス: nitric oxide synthase

1 own by immunohistochemistry (CD31, inducible nitric oxide synthase).

2 actile inhibition via the enzyme endothelial nitric oxide synthase.

3 ormed cells, which also depends on inducible nitric oxide synthase.

4 rons ( approximately 1%) expressing neuronal nitric oxide synthase.

5 egregated population that expresses neuronal nitric oxide synthase.

6 ction, presumably by counteracting inducible nitric oxide synthase.

7 terin (BH4) is an essential cofactor for all nitric oxide synthases.

8 The role of nitric oxide synthase 1 (NOS1) as a major modulator of c

9 factor for the production of nitric oxide by nitric oxide synthase 1 (NOS1), major modulator of cardi

10 nergy balance (e.g. oxytocin (OXT), neuronal nitric oxide synthase 1 (NOS1), melanocortin 4-receptor

11 cula densa, enhancing generation of neuronal nitric oxide synthase 1 (NOS1)-dependent nitric oxide (N

12 e, including the interaction between NOS1AP (nitric oxide synthase 1 adaptor protein) and neuronal ni

13 e with macula densa-specific knockout of all nitric oxide synthase 1 isoforms (MD-NOS1KO) had a signi

14 tide 3-phosphate kinase, Akt Ser/Thr kinase, nitric oxide synthase 1, nitric oxide, soluble guanylate

15 taglomerular apparatus of wild-type (WT) and nitric oxide synthase 1alpha-knockout (NOS1alphaKO) mice

16 Immunohistochemistry was conducted to detect nitric oxide synthase 2 (iNOS) expressing M1 polarized a

17 Inbred mice that lack the gene encoding nitric oxide synthase 2 (Nos2) are susceptible to the re

18 in 6 (IL6), tumor necrosis factor (TNF), and nitric oxide synthase 2 (NOS2) were studied further.

19 infection, and testing MAIT cell priming in nitric oxide synthase 2 (NOS2)-deficient mice all failed

20 luding IL-6, VEGFA, TNFalpha, NFkappaB2, and nitric oxide synthase 2 (NOS2A).

21 that therapy requires induced expression of nitric oxide synthase 2 in monocyte-derived dendritic ce

22 d direct antitumor cytotoxic effects through nitric oxide synthase 2-mediated actions.

23 Only anti-MDA5 DM showed numerous nitric oxide synthase 2-positive muscle fibers with sarc

24 CXCL9/CXCL10), and innate immunity-related (nitric oxide synthase 2/inducible nitric oxide synthase

25 Nitric oxide synthase 2/inducible nitric oxide synthase

26 gulate expression of the inflammatory marker nitric-oxide synthase 2.

27 )) and triple-negative breast cancer (TNBC), nitric oxide synthase-2 (NOS2) and cyclooxygenase-2 (COX

28 ecrosis factor-alpha, interleukin-1beta, and nitric oxide synthase-2, highlighting the potential of V

29 ediators including cytokines, chemokines and nitric oxide synthase-2.

30 h is located at 11p14, potentially affecting nitric oxide synthase 3 (NOS3) expression, as shown by m

31 2 genes that mediate nitric oxide signaling (Nitric Oxide Synthase 3 [NOS3] and Guanylate Cyclase 1,

32 Conversely, expression of inducible nitric oxide synthase, a marker of classical activation

33 d indomethacin and inhibition of endothelial nitric oxide synthase abolished the effects of therapeut

34 nd isoform-specific endothelial and neuronal nitric oxide synthase activation and nitric oxide produc

35 ribe a new mechanism for reduced endothelial nitric oxide synthase activation during renal IR that ca

36 receptor agonist analog elicited endothelial nitric oxide synthase activation to promote endothelial

37 eptor and consequently decreased endothelial nitric oxide synthase activation.

38 r arginase level and activity, and decreased nitric oxide synthase activity.

39 in capillary/arteriole density, endothelial nitric oxide synthase/Akt/vascular endothelial growth fa

40 obese type 2 diabetic mice by an endothelial nitric oxide synthase/Akt/vascular endothelial growth fa

41 tream targets, including Akt and endothelial nitric oxide synthase, although incremental activation b

42 l-MDSC contact and was mediated by inducible nitric oxide synthase and cationic amino acid transporte

43 EDH), ACh trials were repeated with combined nitric oxide synthase and cyclooxygenase inhibition.

44 pregulation of interleukin-1 beta, inducible nitric oxide synthase and cyclooxygenase-2 in the cortex

45 lance between pulmonary vascular endothelial nitric oxide synthase and endothelin receptor expression

46 lance between pulmonary vascular endothelial nitric oxide synthase and endothelin was assessed in lun

47 nucleotide phosphate-oxidase 2 and inducible nitric oxide synthase and enhanced lung concentrations o

48 SIRT1-regulated Akt, endothelial nitric oxide synthase and GLUT4 levels were also induced

49 ased glutathione levels, increased inducible nitric oxide synthase and heme-oxygenase 1 expression, a

50 ased glutathione levels, increased inducible nitric oxide synthase and heme-oxygenase 1 expression, a

51 y-related (nitric oxide synthase 2/inducible nitric oxide synthase and IL-17C) products (FDR < 0.05).

52 , inhibition of VE-PTP activated endothelial nitric oxide synthase and led to nuclear exclusion of th

53 ed distinct anti-inflammatory (low inducible nitric oxide synthase and lower tumour necrosis factor-a

54 Increased activities of inducible nitric oxide synthase and NADPH oxidase 1 enzymes at myo

55 is attributable to preservation of neuronal nitric oxide synthase and sarcoplasmic reticulum Ca(2+)

56 at the growth cone, leading to activation of nitric oxide synthase and the downstream cGMP kinase For

57 n) of Protein kinase B (Akt) and endothelial nitric oxide synthase and the phosphorylation (inhibitio

58 munoreactivity for calbindin, calretinin and nitric oxide synthase and their relationship with calbin

59 OSITE families, detected 98.89 and 92.88% of nitric oxide synthase and TRYPSIN-like enzyme sites in C

60 1; 2) suppressed the expression of inducible nitric oxide synthase and type I IFN and 3) induced auto

61 e availability of BH4 leads to uncoupling of nitric oxide synthases and production of highly oxidativ

62 levels of proinflammatory markers: inducible nitric-oxide synthase and tumor necrosis factor-alpha, w

63 gressive blockade of adenosine receptors and nitric oxide synthase, and by modeling the determinants

64 , involving c-Jun-N-terminal kinases (JNKs), nitric oxide synthase, and intracellular cGMP, exerts a

65 hways, specifically MAPK/ERK1/2, endothelial nitric oxide synthase, and mammalian target of rapamycin

66 dase, uncoupling of endothelial and neuronal nitric oxide synthase, and vascular/brain infiltration w

67 glial fibrillary acidic protein and neuronal nitric-oxide synthase, and Abeta and p-Tau(Ser-202) also

68 neurons showed increased levels of inducible nitric oxide synthase, another indicator of cell damage.

69 % of myenteric neurons stained with neuronal nitric oxide synthase antibody and approximately 33% of

70 ther IL-17/arginase I or IFN-gamma/inducible nitric oxide synthase axis as suppressor mechanisms.

71 e factor-1alpha, heme oxygenase-1, inducible nitric oxide synthase, B-cell lymphoma-extra large, and

72 by the sigma1-receptor antagonist or various nitric oxide synthase blockers.

73 ry caspase 1, interferon gamma-receptor, and nitric oxide synthase (Casp1, Ifngr, Nos2)(-/-) present

74 formed for simultaneous detection of TH with nitric oxide synthase, choline acetyltransferase, calbin

75 mitogen-activated protein kinase (MAPK), and nitric oxide synthase deactivation.

76 (AAV) expressing cKL to diabetic endothelial nitric oxide synthase-deficient mice or alphaKL-null mic

77 of ONOO(-)formed from Nox2-derived O2 ()and nitric oxide synthase-derived nitric oxide.

78 Neuregulin production, endothelial nitric oxide synthase dimerization, and Bcl-2 expression

79 opterin concentrations, ratio of endothelial nitric oxide synthase dimers/monomers, and nitric oxide

80 Inhibition of nitric oxide synthase diminished the CORM-3-mediated inc

81 n, shedding of procoagulant MPs, endothelial nitric oxide synthase downregulation, and reduced nitric

82 ng cell premature senescence and endothelial nitric oxide synthase downregulation.

83 ossibly by affecting the NF-kappaB-inducible nitric oxide synthase-endoplasmic reticulum stress pathw

84 d it was associated with blunted endothelial nitric oxide synthase (eNOS) activation in skeletal musc

85 e whether Hhcy homocysteinylates endothelial nitric oxide synthase (eNOS) and alters caveolin-1 expre

86 ime polymerase chain reaction of endothelial nitric oxide synthase (eNOS) and endothelin-1; (d) immun

87 s response and reduced levels of endothelial nitric oxide synthase (eNOS) and NO.

88 s were used, and intervened with endothelial nitric oxide synthase (eNOS) antagonist L-NNA and its ag

89 ce K(+) (IK and SK) channels and endothelial nitric oxide synthase (eNOS) are present in the endothel

90 VEGF-A(164) strikingly increased endothelial nitric oxide synthase (eNOS) expression.

91 annels in some vascular beds and endothelial nitric oxide synthase (eNOS) in others.

92 O) synthesized by the endothelial isoform of nitric oxide synthase (eNOS) is a critical determinant o

93 Nitric oxide (NO) produced by endothelial nitric oxide synthase (eNOS) is a critical mediator of v

94 Endothelial nitric oxide synthase (eNOS) is protective against kidne

95 t-translational modifications of endothelial nitric oxide synthase (eNOS) lead to impaired nitric oxi

96 ty and hypertension by impairing endothelial nitric oxide synthase (eNOS) phosphorylation and promoti

97 re suppressed insulin-stimulated endothelial nitric oxide synthase (eNOS) phosphorylation in skeletal

98 tact with blood to interact with endothelial nitric oxide synthase (eNOS) present in blood.

99 fragment of the shear-responsive endothelial nitric oxide synthase (eNOS) promoter, we tested effects

100 ndeed, RNAi of p47phox inhibited endothelial nitric oxide synthase (eNOS) serine 1179 phosphorylation

101 dysfunction, which is caused by endothelial nitric oxide synthase (eNOS) uncoupling, is an initial s

102 in (TM), a surface receptor, and endothelial nitric oxide synthase (eNOS), an enzyme that generates n

103 Endothelial nitric oxide synthase (eNOS), an enzyme that synthesizes

104 s to AMPK-mediated activation of endothelial nitric oxide synthase (eNOS), enhanced levels of reactiv

105 ated protein kinase (AMPK), Akt, endothelial nitric oxide synthase (eNOS), nuclear factor erythroid 2

106 Instead, expression of endothelial nitric oxide synthase (eNOS), which generates the potent

107 skin wound angiogenesis through endothelial nitric oxide synthase (eNOS)-dependent Src, PI3K, and MA

108 MD), which is mainly mediated by endothelial nitric oxide synthase (eNOS).

109 and increased the phosphorylated endothelial nitric oxide synthase (eNOS)/eNOS ratio in an age-depend

110 n species caused by dysregulated endothelial nitric-oxide synthase (eNOS) activity is linked to vascu

111 Endothelial nitric-oxide synthase (eNOS) and its bioactive product,

112 he stability and activity of the endothelial nitric-oxide synthase (eNOS) and that Cavin-2 knockdown

113 mbrane Ca(2+)-ATPase (PMCA), and endothelial nitric-oxide synthase (eNOS).

114 osine kinase(Tek), isolectin B4, endothelial nitric oxide synthase(eNOS), von Willebrand factor(vWF),

115 l perfusion has been used to determine which nitric oxide synthase enzymes are active in discrete reg

116 Nitric oxide synthase 2/inducible nitric oxide synthase expression (determined by quantita

117 beta cells, which are mediated by inducible nitric oxide synthase expression and beta-cell productio

118 with a 2-fold increase in exosomal inducible nitric oxide synthase expression and function through TL

119 ia in cDC1s indirectly by reducing inducible nitric oxide synthase expression early after infection a

120 reased striatal NADPH oxidase-4 and neuronal nitric oxide synthase expression in the adult (7- to 9-m

121 rotyrosine formation and increased inducible nitric oxide synthase expression.

122 mulation of inflammatory cells and inducible nitric oxide synthase expression.

123 ass II (MHCII) and proinflammatory inducible nitric oxide synthase expression.

124 crosis factor alpha (TNFalpha) and inducible nitric-oxide synthase expression, indicative of enhanced

125 Organophosphate pesticide exposures, nitric oxide synthase gene variants, and gene-pesticide

126 A), an endogenous inhibitor and uncoupler of nitric oxide synthase, has gained attention as a risk fa

127 tric oxide (NO) produced by either inducible nitric oxide synthase in activated microglia/macrophages

128 PGL-1 induces nitric oxide synthase in infected macrophages, and the r

129 activated microglia/macrophages or neuronal nitric oxide synthase in neurons.

130 emodelling by over-expression of endothelial nitric oxide synthase in the fat pad of the adult rat me

131 ry cytokines and the mRNA encoding inducible nitric-oxide synthase in both LRP1-expressing and -defic

132 gain may itself be plastic because neuronal nitric oxide synthase increases when animals have tinnit

133 of pro-inflammatory cytokines and inducible nitric-oxide synthase induction in BV2 cells in a concen

134 in young adults with VVS can be corrected by nitric oxide synthase inhibition, demonstrated with our

135 ing loss of ROV and was eliminated following nitric oxide synthase inhibition.

136 The neuronal nitric oxide synthase inhibitor AAAN (N-(4S)-4-amino-5-[

137 rotocols utilizing intravenous infusion of a nitric oxide synthase inhibitor and isovolumic haemodilu

138 Importantly, infusion of the nitric oxide synthase inhibitor l-N(G)-monomethyl-l-argi

139 tric tissues and these were inhibited by the nitric oxide synthase inhibitor L-NNA.

140 (placebo) and the non-selective competitive nitric oxide synthase inhibitor N(G) -monomethyl-l-argin

141 l death in vitro, which was prevented by the nitric oxide synthase inhibitor NG-nitro-l-arginine meth

142 Intravenous infusion of a nitric oxide synthase inhibitor reduced the neurovascula

143 In 8 healthy subjects, a nitric oxide synthase inhibitor was infused to evaluate

144 etric dimethylarginine (ADMA), an endogenous nitric oxide synthase inhibitor, are associated with mit

145 mice, we found that expression of inducible nitric oxide synthase (iNos or Nos2) was elevated in TTP

146 ne metabolizing enzymes, including inducible nitric oxide synthase (iNOS) and arginase (ARG), are typ

147 Inducible nitric oxide synthase (iNOS) and B-cell lymphoma-2-assoc

148 OPE also had higher NO, inducible nitric oxide synthase (iNOS) and cyclooxygenase (COX-2)

149 ionally, the protein expression of inducible nitric oxide synthase (iNOS) and cyclooxygenase-2 (COX-2

150 PepNats representing hot loops of inducible nitric oxide synthase (iNOS) and human agouti-related pr

151 biomarkers include suppression of inducible nitric oxide synthase (iNOS) and induction of sterol reg

152 esulted from increased activity of inducible nitric oxide synthase (iNOS) and S-nitrosylation of the

153 receptor (CB1R) and activation of inducible nitric oxide synthase (iNOS) are associated with nonalco

154 complex 1 (mTORC1), HIF1alpha and inducible nitric oxide synthase (iNOS) coordinates DC metabolism a

155 r of RNS produced from arginine by inducible Nitric Oxide Synthase (iNOS) enzyme.

156 inhibition of 5-LOX, COX-1-2, and inducible nitric oxide synthase (iNOS) enzymes (p < 0.05).

157 used mainly by enhanced microglial inducible nitric oxide synthase (iNOS) expression and NO release,

158 s that are sufficient to stimulate inducible nitric oxide synthase (iNOS) expression and production o

159 taglandin E synthase (mPGES)-1 and inducible nitric oxide synthase (iNOS) expression.

160 of the NF-kappaB subunit p65; and inducible nitric oxide synthase (iNOS) expression.

161 by genetic deletion of inducible isoform of nitric oxide synthase (iNOS) from VCP TG mouse and by ph

162 ion of nitric oxide production and inducible nitric oxide synthase (iNOS) gene expression was evident

163 Inducible nitric oxide synthase (iNOS) generates nitric oxide (NO)

164 ported that Sal-1 targets cellular inducible nitric oxide synthase (iNOS) in a miRNA manner, leading

165 lyze the role of NADPH oxidase and inducible nitric oxide synthase (iNOS) in a murine model of A. act

166 Depletion of either ASS1 or inducible nitric oxide synthase (iNOS) in KSHV-transformed cells s

167 showed that radiotherapy increased inducible nitric oxide synthase (iNOS) in the tumor tissues.

168 Increased inflammation and inducible nitric oxide synthase (iNOS) levels characterized the M1

169 regulates prolidase, we probed the inducible nitric oxide synthase (iNOS) mediated phosphorylation of

170 interleukin (IL)-1beta, IL-6, and inducible nitric oxide synthase (iNOS) messenger (m)RNAs in the gi

171 ce nitric oxide (NO) or upregulate inducible nitric oxide synthase (iNOS) mRNA following Toll-like re

172 The levels of inducible nitric oxide synthase (iNOS), bone morphogenetic protein

173 n markers, MHC-II, CD86, IL-6, and inducible nitric oxide synthase (iNOS), but not TNF-alpha.

174 essions of interleukin (IL)-1beta, inducible nitric oxide synthase (iNOS), cyclooxygenase (COX)-2, ma

175 ith rapid induction of nitrite and inducible nitric oxide synthase (iNOS), followed by induction of T

176 ta); and the inflammatory markers: Inducible Nitric Oxide Synthase (iNOS), Interleukin 1beta (IL-1bet

177 aB p65-responsive genes, including inducible nitric oxide synthase (iNOS), interleukin-6 (IL-6), and

178 d HIF-2alpha, leading to decreased inducible-nitric oxide synthase (iNOS), NO-production, and VEGF.

179 endothelial growth factor (VEGF), inducible nitric oxide synthase (iNOS), nuclear factor-kappa B (NF

180 ro-apoptotic signaling mediated by inducible nitric oxide synthase (iNOS), transforming growth factor

181 nylate-binding proteins (GBPs) and inducible nitric oxide synthase (iNOS), which we found to inhibit

182 d corneal allograft survival in an inducible nitric oxide synthase (iNOS)-dependent manner.

183 1 (CB1R) and inhibitory effect on inducible nitric oxide synthase (iNOS).

184 proximately 60% of which expressed inducible nitric oxide synthase (iNOS).

185 teins cyclooxygenase-2 (COX-2) and inducible nitric oxide synthase (iNOS).

186 s and markedly decreased levels of inducible nitric oxide synthase (iNOS).

187 ule involved in innate immunity is inducible nitric oxide synthase (iNOS).

188 872, P < 0.0005) and expression of inducible nitric oxide synthase (iNOS; r = 0.9986, P < 0.0001).

189 ewly designed compounds complexed to various nitric oxide synthase isoforms have been determined.

190 and improve vascular function in endothelial nitric oxide synthase knockout (eNOS(-/-) ) mice.

191 st, affects development of DN in endothelial nitric oxide synthase-knockout db/db mice.

192 Mice lacking a functional endothelial nitric oxide synthase (KO, NOS3(-/-) ) and wild-type mic

193 SC were treated with inhibitors of inducible nitric oxide synthase (L-NAME and NCX-4016).

194 dichlorofluorescein diacetate dye, inducible nitric oxide synthase levels determined by Western blot,

195 6) and decreased the expression of inducible nitric oxide synthase (M1 marker).

196 ated signalling through Akt, the endothelial nitric oxide synthase mediated pathway, regulates anabol

197 triggers calmodulin-dependent activation of nitric oxide synthase, nitric oxide (NO) production, NO-

198 el of human enteric neuropathy, the neuronal nitric oxide synthase (nNOS(-/-)) deficient mouse model,

199 Blockers of neuronal nitric oxide synthase (nNOS) and of NMDA receptors block

200 erythroid-derived 2)-like 2 (Nrf2), neuronal Nitric Oxide Synthase (nNOS) expression and nitrergic re

201 olves a tau-induced dissociation of neuronal nitric oxide synthase (nNOS) from postsynaptic density 9

202 he nitric oxide synthesizing enzyme neuronal nitric oxide synthase (nNOS) in nerve fibers of the muri

203 Neuronal nitric oxide synthase (nNOS) in skeletal muscle is a key

204 Neuronal nitric oxide synthase (nNOS) inhibition is a promising s

205 ation related to BBB penetration of neuronal nitric oxide synthase (nNOS) inhibitors toward the devel

206 pic glutamate receptor5 (mGluR5) on neuronal nitric oxide synthase (nNOS) interneurons.

207 Inhibition of neuronal nitric oxide synthase (nNOS) is a promising therapeutic

208 Neuronal nitric oxide synthase (nNOS) is a target for development

209 se in the dimer to monomer ratio of neuronal nitric oxide synthase (nNOS) protein and lowered NOS act

210 Neuronal nitric oxide synthase (nNOS) regulates muscle atrophy in

211 In the course of our studies on neuronal nitric oxide synthase (nNOS), a client of the Hsp90 and

212 Inhibition of neuronal nitric oxide synthase (nNOS), an enzyme implicated in ne

213 ide synthase 1 adaptor protein) and neuronal nitric oxide synthase (nNOS), nNOS enzymatic activity, a

214 SIGNIFICANCE STATEMENT We show that neuronal nitric oxide synthase (nNOS), the enzyme that synthesize

215 , as they are long-range inhibitory neuronal nitric oxide synthase (nNOS)-expressing cells.

216 es calcium signals in pyramidal and neuronal nitric oxide synthase (nNOS)-expressing neurons.

217 smitter synthesized in the brain by neuronal nitric oxide synthase (nNOS).

218 tion that depended on activation of neuronal nitric oxide synthase (nNOS).

219 rvalbumin, calbindin, calretinin, and neural nitric oxide synthase (nNOS).

220 subpopulations of WMICs containing neuronal nitric oxide synthase (nNOS, ~7 million in number, with

221 scle is the muscle-specific form of neuronal nitric oxide synthase (nNOSmu) produced by the NOS1 gene

222 tuberculosis esxL induced the expression of nitric-oxide synthase, NO production, and p38 MAPK pathw

223 od flow (FBF) response to acetylcholine, and nitric oxide synthase (NOS) activity was defined as the

224 utaneous vasodilatation and sweating through nitric oxide synthase (NOS) and calcium-activated potass

225 erized further by triple labeling with CGRP, nitric oxide synthase (NOS) and calretinin (CALR) antibo

226 nt expression of NMDA receptors (NMDARs) and nitric oxide synthase (NOS) and is therefore much more s

227 While NO production by nitric oxide synthase (NOS) enzymes in the eye has been

228 oduction from the amino acid l-arginine, via nitric oxide synthase (NOS) enzymes, research in recent

229 -like 2 (Nrf2)/tetrahydrobiopterin (BH(4) )/ nitric oxide synthase (NOS) expression in primary human

230 nvestigated the nitrergic system with neural nitric oxide synthase (NOS) immunohistochemistry and NAD

231 phorase is used as a histochemical marker of nitric oxide synthase (NOS) in aldehyde-treated tissues.

232 We investigated the role of nitric oxide synthase (NOS) in mediating blood-brain bar

233 ytotoxic effects, which are inhibited by the nitric oxide synthase (NOS) inhibitor L-NIO, and genetic

234 Nitric oxide synthase (NOS) inhibitors rescue this patho

235 e dystrophin-sarcoglycan complex delocalizes nitric oxide synthase (NOS) to alter its signaling, and

236 Oxidative stress and nitric oxide synthase (NOS) uncoupling are thought to co

237 This imbalance leads to nitric oxide synthase (NOS) uncoupling with reduced nitr

238 ermined by lactate dehydrogenase release and nitric oxide synthase (NOS)-dependent cGMP production.

239 aneous vasodilatation and sweating through a nitric oxide synthase (NOS)-dependent mechanism.

240 s by mitochondria, NADPH oxidase, and type 2 nitric-oxide synthase (NOS-2) and resulted in S-nitrosyl

241 We identify the nitric oxide synthase (Nos1 to 3) gene family as the clo

242 HG-induced increases of phosphor-Erk1/2 and nitric oxide synthase (NOS1) expressions as well as hype

243 hat protein kinase Cbeta (PKCbeta) and brain nitric oxide synthase (NOS1), both identified by proteom

244 ed STAT1 and its downstream target inducible nitric oxide synthase (NOS2) as being upregulated in the

245 the metalloproteinase ADAMTS1 and inducible nitric oxide synthase (NOS2) as therapeutic targets in i

246 Rs) on the host cell, it activates inducible nitric oxide synthase (NOS2) which instantly fills the p

247 racellular adhesion molecule 1 (ICAM-1), and nitric oxide synthase (NOS2), developed leukostasis and

248 ells are phenotypically similar to inducible nitric oxide synthase (NOS2)- and tumor necrosis factor

249 FNgamma-dependent genes, including inducible nitric oxide synthase (Nos2).

250 The endothelial nitric oxide synthase (NOS3) system is crucial for vascu

251 hyperoxia-induced BPD and PH via endothelial nitric oxide synthase (NOS3).

252 Nitric oxide synthases (NOSs) are heme-based monooxygena

253 ochromes P450, the buildup of I435 occurs in nitric oxide synthases (NOSs) upon their reaction with e

254 e improves insulin resistance in endothelial nitric oxide synthase null mice, and multiple studies ha

255 del, STZ-induced diabetes in the endothelial nitric oxide synthase-null (eNOS(-/-)) mice.

256 eam signaling pathways involving endothelial nitric oxide synthase or prostanoid production (indometh

257 nd small percentages of these also contained nitric oxide synthase or vasoactive intestinal polypepti

258 crosis factor-alpha (P = 0.04) and inducible nitric oxide synthase (P = 0.02) in skin biopsy specimen

259 anifested by decreased levels of endothelial nitric oxide synthase (P< .005) and Kruppel-like factor

260 In contrast, inhibition of nitric-oxide synthase, p38 MAPK, and G9a abrogated H3K9m

261 hosphatidylinositol 3-kinase/Akt/endothelial nitric oxide synthase pathway in protocol 3.

262 by siRNA significantly increased endothelial nitric oxide synthase phosphorylation at threonine 495 l

263 ng with reduced VEGFR3, Akt, and endothelial nitric oxide synthase phosphorylation.

264 alpha(+)]), and effector molecule (inducible nitric oxide synthase-positive [iNOS(+)]) responses were

265 nels (K(Ca)3.1 and K(Ca)2.3) and endothelial nitric oxide synthase, produces additive tone, which is

266 mbers of nitrergic neurons, reduced neuronal nitric oxide synthase production, and reduced colonic ne

267 ty to use N via the ornithine-urea cycle and nitric oxide synthase production.

268 of capon (C-terminal PDZ ligand of neuronal nitric-oxide synthase protein), apoptosis-associated spe

269 helial growth factor), and eNOS (endothelial nitric oxide synthase) protein contents.

270 Staphylococcus aureus nitric oxide synthase (saNOS) is a major contributor to

271 dimerization and phosphorylation of neuronal nitric oxide synthase, sarcoplasmic reticulum Ca(2+) rel

272 actility, tetrahydrobiopterin, the dimers of nitric oxide synthase, sarcoplasmic reticulum Ca(2+) rel

273 e molecules, including arginase 1, inducible nitric oxide synthase, signal transducer and activator o

274 activation by fluvoxamine triggered the Akt-nitric oxide synthase signaling pathway, resulting in ti

275 capillaries via the NMDA receptors-neuronal nitric oxide synthase signaling pathway.

276 trulline (L-Cit) is converted to endothelial nitric oxide synthase substrate, L-arginine.

277 uM(vgat/vglut2)) and another also expressing nitric oxide synthase (SuM(Nos1/Vglut2)).

278 volves the activation of the Akt-endothelial nitric oxide synthase survival pathway, and the inhibiti

279 ry cytokine tumor necrosis factor, inducible nitric oxide synthase, the M1 activator interferon gamma

280 effects alter the expression of endothelial nitric oxide synthase, the stability of atherosclerotic

281 Intriguingly, eNOS activity is regulated by nitric-oxide synthase trafficking inducer (NOSTRIN), whi

282 ssion of proinflammatory proteins (inducible nitric oxide synthase, tumor necrosis factor-alpha, and

283 pro-inflammatory proteins such as inducible nitric oxide synthase, tumor necrosis factor-alpha, inte

284 d investigated the role of 12,13-diHOME in a nitric oxide synthase type 1 deficient (NOS1(-/-)) mouse

285 t radiation disrupted BH4, which resulted in nitric oxide synthases uncoupling and augmented radiatio

286 inal protein kinase, serine phosphatases, or nitric oxide synthase, unlike homosynaptic SC depotentia

287 Neuronal nitric-oxide synthase, unlike its endothelial and induci

288 high-fat diet and inhibition of constitutive nitric oxide synthase using N(omega)-nitro-L-arginine me

289 relevant changes in the vascular endothelial nitric oxide synthase vasodilatory response, which is a

290 tion that could be restored by inhibition of nitric oxide synthase via the inhibitor N-nitro-l-methyl

291 Notably, plasma arginine use by nitric oxide synthase was decreased in infected mice.

292 allografts, whereas expression of inducible nitric oxide synthase was decreased.

293 ression of Nos2, the gene encoding inducible nitric oxide synthase, was elevated in the absence of PP

294 e dismutase were increased, whereas those of nitric oxide synthase were decreased, in 7- to 10-week-o

295 of acetylcholine, which acts via endothelial nitric oxide synthase were unaffected, suggesting a nonv

296 adhesion molecule-1 (VCAM-1) and endothelial nitric oxide synthase, whereas PDEs had significantly hi

297 activation of a phagosomal enzyme, inducible nitric oxide synthase, which is necessary for NO product

298 was used for detection of CD31 and inducible nitric oxide synthase while iron deposition was shown wi

299 c contractions persisted until inhibition of nitric oxide synthase with N(omega) -nitro-l-arginine me

300 howed higher levels of phosphorylated neural nitric oxide synthase within neural stem cells (NSCs).