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1 xpression of CD16, and the ability to reduce nitroblue tetrazoleum dye was inhibited in transfectants
2 ease (CGD), which is diagnosed by use of the nitroblue tetrazolium (NBT) and Fc-Oxyburst assays that
3 were detected spectrophotometrically using a nitroblue tetrazolium (NBT) assay in cells treated with
4 timulated isoniazid oxidation as measured by nitroblue tetrazolium (NBT) reduction and O2 consumption
8 tion and immunocompetence measured using the Nitroblue Tetrazolium (NBT) test were significantly diff
9 , superoxide production, as monitored by the nitroblue tetrazolium (NBT) test, was detected in up to
10 ion as measured by (1) the ability to reduce nitroblue tetrazolium (NBT), (2) the expression of Mac-I
12 cceptors for NADH oxidoreductase (0.3 mmol/L nitroblue tetrazolium and 0.1 mmol/L ferricyanide) inhib
13 preparation: the Cyt c assay to cytosol, the nitroblue tetrazolium assay to plasma membrane, and the
15 , HO2( *)) were detected by the reduction of nitroblue tetrazolium into formazan, and hydroxyl radica
17 4 subclones MR-2 and R4 cells as measured by nitroblue tetrazolium reduction and tRA-inducible genes
18 OD and tcvSOD recombinant proteins inhibited nitroblue tetrazolium reduction of superoxide anion gene
19 entiation, as measured by growth inhibition, nitroblue tetrazolium reduction, nuclear body relocaliza
25 eutrophil function in the proband, including nitroblue tetrazolium tests, myeloperoxidase assays, neu
28 ine dinucleotide hydrogen; substrate, 184 uM Nitroblue tetrazolium, and 1.9 uM phenazine methosulfate
29 hrome c (Cyt c), assays for the reduction of nitroblue tetrazolium, and assays for the chemiluminesce
30 on of NADH on nitroprusside was inhibited by nitroblue tetrazolium, ferricyanide, and diphenyliodoniu
31 NO release was inhibited by the presence of nitroblue tetrazolium, ferricyanide, and diphenyliodoniu