ライフサイエンスコーパス: nitrogen dioxide

1 and nitrogen oxides (e.g., peroxynitrite and nitrogen dioxide).

2 ozone, and 1.079 (95% CI: 1.065, 1.093) for nitrogen dioxide.

3 ntrolling for long-term exposure to UFPs and nitrogen dioxide.

4 hey obtained similar results for exposure to nitrogen dioxide.

5 adical, ozone, the nitrate radical (NO3) and nitrogen dioxide.

6 s nitrous and nitric acid, nitric oxide, and nitrogen dioxide.

7 ity in the lavage of mice acutely exposed to nitrogen dioxide.

8 sures to fine particulate matter, ozone, and nitrogen dioxide.

9 utants [e.g., ultrafine particles (UFPs) and nitrogen dioxide].

10 ), the ORs were 0.84 (95% CI, 0.76-0.92) for nitrogen dioxide; 0.85 (95% CI, 0.74-0.97) for ozone, 0.

11 6% per 10 parts per billion; 1.35-3.38), and nitrogen dioxide (1.70% per 10 parts per billion; 1.25-2

12 levels of air contaminants such as benzene, nitrogen dioxide, 1,3-butadiene, and particulate matter.

13 absolute decrease in prevalence of 1.8% for nitrogen dioxide, 1.7% for ozone, 2.2% for PM10, and 2.3

14 carbon monoxide: 1.048; 95% CI, 1.026-1.070; nitrogen dioxide: 1.011; 95% CI, 1.006-1.016; sulfur dio

15 n dioxide (13%), carbon monoxide (0.68%) and nitrogen dioxide (1000 ppm) in air.

16 artile-range increase in average exposure to nitrogen dioxide (13.6 parts per billion) during pregnan

17 ltiexposure model identified particulate and nitrogen dioxide air pollution inside the home, urine co

18 or copollutants (fine particulate matter and nitrogen dioxide), although some evidence of confounding

19 piratory inflammation within the population [Nitrogen Dioxide | American Lung Association].

20 sion processes (nitric oxide, nitrous oxide, nitrogen dioxide, ammonia, hydrazine, hydroxylamine, nit

21 Here we discuss the removal of nitrogen dioxide, an important toxic industrial chemical

22 t ventricular outflow tract obstructions and nitrogen dioxide and between hypoplastic left heart synd

23 secondary free radical intermediates such as nitrogen dioxide and carbonate radicals.

24 n detect aggressive oxidizing vapors such as nitrogen dioxide and chlorine at 250 and 500 ppb, respec

25 sociations were observed between exposure to nitrogen dioxide and coarctation of the aorta and pulmon

26 the association between maternal exposure to nitrogen dioxide and fine particulate matter (aerodynami

27 omine oxide and iodine oxide, as well as for nitrogen dioxide and formaldehyde.

28 In contrast, combination of nitrogen dioxide and guanine neutral radicals generated

29 rement of the blackness of PM2.5 filters and nitrogen dioxide and nitrogen oxide levels.

30 Exposures to oxides of nitrogen (nitrogen dioxide and nitrogen oxides) and particulate ma

31 ts of meteorological data and air pollution (nitrogen dioxide and ozone).

32 Regional exposure measures of nitrogen dioxide and particulate matter less than 2.5 an

33 olling for the traffic-related co-pollutants nitrogen dioxide and particulate matter with an aerodyna

34 in Southern California, decreases in ambient nitrogen dioxide and PM2.5 between 1993 and 2014 were si

35 adjustment for gaseous pollutants other than nitrogen dioxide and sulfur dioxide.

36 trous acid (HONO) yield for reaction between nitrogen dioxide and the hydroperoxyl-water complex and

37 the reaction between electronically excited nitrogen dioxide and water vapor is an important atmosph

38 ant models (with fine particulate matter and nitrogen dioxide) and effect measure modification by rac

39 less than 10 microm in aerodynamic diameter, nitrogen dioxide, and carbon monoxide, while evidence of

40 diameter less than 2.5 mum, sulfur dioxide, nitrogen dioxide, and carbon monoxide.

41 status, seven contextual covariates, ozone, nitrogen dioxide, and combined oxidative potential.

42 ate matter <2.5 mum in aerodynamic diameter, nitrogen dioxide, and nitric oxide) and woodsmoke were e

43 or equal to 2.5 mum in aerodynamic diameter, nitrogen dioxide, and nitric oxide, were estimated at ea

44 ach 25-ppb increase in average nitric oxide, nitrogen dioxide, and nitrogen oxide levels, respectivel

45 ased estimates of exposures to nitric oxide, nitrogen dioxide, and nitrogen oxides were assigned base

46 modeled estimates of levels of nitric oxide, nitrogen dioxide, and nitrogen oxides were used to asses

47 ous nitrogen oxides, including nitric oxide, nitrogen dioxide, and nitrous oxide, on carbon dioxide e

48 Measured PM2.5, nitrogen dioxide, and ozone concentrations were spatiall

49 ; ambient concentrations of carbon monoxide, nitrogen dioxide, and ozone were obtained from state mon

50 Annual mean community-level ozone, nitrogen dioxide, and particulate matter less than 10 mu

51 e of ambient exposures to ozone, acid vapor, nitrogen dioxide, and particulate matter.

52 ciations were observed with carbon monoxide, nitrogen dioxide, and PM(10).

53 Ambient fine particulate matter, nitrogen dioxide, and preterm birth in New York City.

54 ess (PC20) with ozone, carbon monoxide (CO), nitrogen dioxide, and sulfur dioxide concentrations in 1

55 mic diameter (PM10), ozone, carbon monoxide, nitrogen dioxide, and sulfur dioxide) and cardiac autono

56 </=10 mum and </=2.5 mum in diameter, ozone, nitrogen dioxide, and sulfur dioxide), derived from part

57 ter of <10 microm (PM(10)), carbon monoxide, nitrogen dioxide, and sulfur dioxide-but not ozone-were

58 late matter (PM), gaseous pollutants (ozone, nitrogen dioxide, and sulphur dioxide), and mixed traffi

59 dicals, the trioxidocarbonate radical anion, nitrogen dioxide, and the glutathionyl radical, via one-

60 -day moving average concentrations of ozone, nitrogen dioxide, and the organic carbon fraction of par

61 ed with autism during gestation (exposure to nitrogen dioxide: AOR, 1.81 [95% CI, 1.37-3.09]; exposur

62 d during the first year of life (exposure to nitrogen dioxide: AOR, 2.06 [95% CI, 1.37-3.09]; exposur

63 ect of this reagent by in situ generation of nitrogen dioxide as a radical on aromatic compounds to g

64 elieve that the observed carbon monoxide and nitrogen dioxide associations can probably be attributed

65 rals, massicot, and litharge, are exposed to nitrogen dioxide at different relative humidity.

66 re to ambient volatile organic compounds and nitrogen dioxide at relatively low concentrations is ass

67 age exposures to fine particulate matter and nitrogen dioxide at the residential census-tract level.

68 However, adjustment for nitrogen dioxide attenuated the full-pregnancy-particula

69 estimated annual concentrations of UFPs and nitrogen dioxide by means of land-use regression models

70 gents including superoxide anion radical and nitrogen dioxide can react with GXXXXGK(S/T)C motif-cont

71 2.5 microm in aerodynamic diameter (PM2.5)), nitrogen dioxide, carbon monoxide, and ozone increases r

72 ants (particles <2.5 mum in diameter, ozone, nitrogen dioxide, carbon monoxide, and sulfur dioxide).

73 ural gas production and were correlated with nitrogen-dioxide columns at a ratio that is consistent w

74 -range increase in the time-weighted average nitrogen dioxide concentration (4.8 ppb) yielded an odds

75 increased with each 10-ppb increase in mean nitrogen dioxide concentration in the first trimester (o

76 large and small fluctuations in the ambient nitrogen dioxide concentration.

77 or for the monitoring of ambient atmospheric nitrogen dioxide concentrations (0-200 ppb).

78 a citywide low-emission zone failed to bring nitrogen dioxide concentrations under control.

79 Increased 4-month average nitrogen dioxide concentrations were associated with red

80 Adjustment for fine particles and nitrogen dioxide did not materially change these estimat

81 ng of NO2 in macrophage cells treated with a nitrogen dioxide donor.

82 ciated with interquartile-range increases in nitrogen dioxide during cumulative lag 3 (average of the

83 suicide risk was associated with exposure to nitrogen dioxide during the spring/fall transition perio

84 ns have experienced significant increases in nitrogen dioxide emissions from new trucks equipped with

85 From these incomplete measures, long-term nitrogen dioxide exposure and its effect on health must

86 IQR increase for LUR-based nitric oxide and nitrogen dioxide exposure estimates.

87 whereas among 2,136 boys, sulfur dioxide and nitrogen dioxide exposure in utero, during infancy, and

88 e, and smoking, an interquartile increase in nitrogen dioxide exposure increased the risk of membersh

89 The effect of nitrogen dioxide exposure on various measures of lung fu

90 usted odds ratio for the highest quartile of nitrogen dioxide exposure was 1.7 (95% confidence interv

91 each 1-ppb increase in long- and short-term nitrogen dioxide exposure was associated with 3.24 (95%

92 ccurate measure of household-level long-term nitrogen dioxide exposure was available.

93 nuously over many years, but household-level nitrogen dioxide exposure was measured only during two 2

94 age at ROM associated with PM2.5 absorbance, nitrogen dioxide exposure, and nitrogen oxide exposure d

95 that long- and short-term PM2.5, ozone, and nitrogen dioxide exposures were all associated with incr

96 These ozone and nitrogen dioxide findings were fairly stable after adjus

97 cally more robust predictors of eNO than was nitrogen dioxide, for which associations were highly sen

98 A positive association with nitrogen dioxide ([Formula: see text]) was also observed

99 ne particulate matter ([Formula: see text]), nitrogen dioxide ([Formula: see text]), and ozone ([Form

100 association between time-varying exposure to nitrogen dioxide ([Formula: see text]), ozone ([Formula:

101 mated prenatal and 1-y childhood exposure to nitrogen dioxide ([Formula: see text]), particulate matt

102 re to traffic-related air pollutants such as nitrogen dioxide has been linked to cardiovascular disea

103 er (PM10), nitric oxide, sulfur dioxide, and nitrogen dioxide in different growth phases with clinica

104 examined, the quantity of carbon dioxide and nitrogen dioxide in particular were close to or even abo

105 ological particles are nitrated by ozone and nitrogen dioxide in polluted air.

106 Adjusted estimates for nitrogen dioxide indicated that for each 10-ppb increase

107 Nitrogen dioxide is a common air pollutant with growing

108 Nitrogen dioxide is a highly toxic reactive nitrogen spe

109 he conclusion that generation of the mutagen nitrogen dioxide is peculiar to cell culture systems and

110 rs examined whether air pollution at school (nitrogen dioxide) is associated with poorer child cognit

111 V s)), chlorine (K0 = 2.24 cm(2)/(V s)), and nitrogen dioxide (K0 = 2.25 cm(2)/(V s)).

112 Photoionization of nitrogen dioxide led to the formation of the nitrate ion

113 )) and 10 mum (PM(10)) or less and ozone and nitrogen dioxide levels 7 days before Feno measurement w

114 udy, Huls et al. show an association between nitrogen dioxide levels in outdoor air and number of len

115 above 1,170 m, a 10-ppb increment in modeled nitrogen dioxide levels was associated with current asth

116 llution (calibrated to a 10-ppb increment in nitrogen dioxide levels) were associated with a 2.4% dec

117 llution (calibrated to a 10-ppb increment in nitrogen dioxide levels) were associated with pulmonary

118 onceptual recall memory after adjustment for nitrogen dioxide levels.

119 formation recall memory after adjustment for nitrogen dioxide levels.

120 s, health events, and ambient air pollution (nitrogen dioxide, nitrogen monoxide, particulate matter

121 ted the associations of maternal exposure to nitrogen dioxide, nitrogen oxides, and particulate matte

122 pectral features suggesting the formation of nitrogen dioxide NO(2).

123 rrently available for the analysis of NO(*), nitrogen dioxide (NO(2) (*)), dinitrogen trioxide (N(2)O

124 haze event that rapid oxidation of SO(2) by nitrogen dioxide (NO(2)) and nitrous acid (HONO) takes p

125 l air pollution exposure scenarios targeting nitrogen dioxide (NO(2)) and particulate matter <2.5 mum

126 Street-level concentrations of nitrogen dioxide (NO(2)) and particulate matter (PM) exc

127 c-related air pollution (TRAP), specifically nitrogen dioxide (NO(2)) and particulate matter (PM), an

128 ffects of indoor particulate matter (PM) and nitrogen dioxide (NO(2)) concentrations on COPD morbidit

129 ng students' terrain-based dosage of ambient nitrogen dioxide (NO(2)) during their commute from home

130 SCR also mitigates DPF-related increases in nitrogen dioxide (NO(2)) emissions.

131 Sparse data address the effects of nitrogen dioxide (NO(2)) exposure in inner-city schools

132 ion and growth are adversely associated with nitrogen dioxide (NO(2)) exposure.

133 ynamic diameter (PM(2.5)), ozone (O(3)), and nitrogen dioxide (NO(2)) exposures at participants' resi

134 er NO yields and a comparable level of toxic nitrogen dioxide (NO(2)) formation is observed.

135 ty for the dynamic production of mixtures of nitrogen dioxide (NO(2)) in nitrogen (N(2)) based on con

136 Nitrogen dioxide (NO(2)) is a major component and common

137 (a)pyrene (BaP), sulfur dioxide (SO(2)), and nitrogen dioxide (NO(2)) over two consecutive 24-h sampl

138 loss at rates between 0 and 2.2 +/- 0.4% of nitrogen dioxide (NO(2)) photolysis, equivalent to avera

139 Nitrogen dioxide (NO(2)) remains an important traffic-re

140 We developed LUR models for nitrogen dioxide (NO(2)) using measurements conducted at

141 diacy of protein-free radicals, ferryl heme, nitrogen dioxide (NO(2)), and hydrogen peroxide (H(2)O(2

142 ynamic diameter < 2.5 microm (PM(2).(5)) and nitrogen dioxide (NO(2)), and modelled estimates of ozon

143 utants considered here include nitric oxide, nitrogen dioxide (NO(2)), carbon monoxide, formaldehyde,

144 de (CO(2)), ozone (O(3)), nitric oxide (NO), nitrogen dioxide (NO(2)), other trace gases, and submicr

145 the simultaneous measurement of atmospheric nitrogen dioxide (NO(2)), ozone (O(3)), and relative hum

146 10 (PM(10)) and less than 2.5 mum (PM(2.5)), nitrogen dioxide (NO(2)), ozone, and sulfur dioxide (SO(

147 les (PM(2.5)), inhalable particles (PM(10)), nitrogen dioxide (NO(2)), sulfur dioxide (SO(2)), ozone

148 nual exposure to five air pollutants: ozone, nitrogen dioxide (NO(2)), sulfur dioxide, particulate ma

149 ) (particulate matter >2.5-10 mum diameter), nitrogen dioxide (NO(2)), sulphur dioxide (SO(2)), carbo

150 ata sets using low-cost passive samplers for nitrogen dioxide (NO(2)), which complement data from the

151 e (NH(4)NO(3)) aerosol (at 540 degrees C) to nitrogen dioxide (NO(2)), whose mixing ratio is monitore

152 globally, especially for reactive gases like nitrogen dioxide (NO(2)).

153 mpliant with legal air quality standards for nitrogen dioxide (NO(2)).

154 try resulted in elevated levels of gas-phase nitrogen dioxide (NO(2)).

155 ation-weighted concentration of ground-level nitrogen dioxide (NO(2): 60% with 95% CI 48 to 72%), and

156 Models included regional pollutants (nitrogen dioxide [NO(2)] or particulate matter with an a

157 tant exposures of nitrogen oxides (including nitrogen dioxide [NO(2)]) and particulate matter with a

158 k carbon, total nitrogen oxides [NO(X)], and nitrogen dioxide [NO(2)]) were measured at 10 school sit

159 have previously shown that both NO/O(2) (via nitrogen dioxide, (*)NO(2)) and superoxide radical anion

160 llutants such as particulate matter (PM) and nitrogen dioxide (NO2 ) are independently associated wit

161 A new type of nitrogen dioxide (NO2 ) gas sensor based on copper phtha

162 mic diameter <2.5microm (PM2.5) (1999-2004), nitrogen dioxide (NO2) (2006), and ozone (O3) (2002-2004

163 Exposure to traffic related nitrogen dioxide (NO2) air pollution is associated with

164 ation systems contributed to satellite-based nitrogen dioxide (NO2) and fine particulate matter (</=

165 sia to evaluate associations with changes in nitrogen dioxide (NO2) and fine particulate matter (PM2.

166 s via three different channels, forming: (1) nitrogen dioxide (NO2) and hydroxyl radical ((*)OH), (2)

167 lso calculated photolysis rate constants for nitrogen dioxide (NO2) and nitrate radicals (NO3) in the

168 (LUR) model estimates of nitric oxide (NO), nitrogen dioxide (NO2) and nitrogen oxides (NOx).

169 s was PM2.5 absorbance and concentrations of nitrogen dioxide (NO2) and nitrogen oxides.

170 ta-analyses of studies examining exposure to nitrogen dioxide (NO2) and NOx and its association with

171 ions of average pollutant concentrations for nitrogen dioxide (NO2) and NOX in GMCs of 6.4 and 21.7 p

172 fleet was also observed in on-road ratios of nitrogen dioxide (NO2) and NOX.

173 O3) with nitrogen monoxide (NO) resulting in nitrogen dioxide (NO2) and oxygen (O2).

174 in the development of exposure surfaces for nitrogen dioxide (NO2) and ozone (O3).

175 Our aim was to evaluate LUR models for nitrogen dioxide (NO2) and particulate matter (PM) compo

176 ether exposure to elevated concentrations of nitrogen dioxide (NO2) and particulate matter with aerod

177 trument (OMI) were used to examine trends in nitrogen dioxide (NO2) and sulfur dioxide (SO2) over a l

178 fy key controls on concentrations of ambient nitrogen dioxide (NO2) at a local-scale within a central

179 anuary and July) localized concentrations of nitrogen dioxide (NO2) by 28% (-2.33 ppbV) in highway gr

180 Ambient and home exposure to nitrogen dioxide (NO2) causes asthma symptoms and decrea

181 We analyzed satellite observations of nitrogen dioxide (NO2) columns by the Ozone Monitoring I

182 PM2.5, PMcoarse, and PM10, respectively) and nitrogen dioxide (NO2) concentrations at place of reside

183 d function and estimated residential outdoor nitrogen dioxide (NO2) concentrations from the Multi-eth

184 PM) concentrations, ultrafine particles, and nitrogen dioxide (NO2) concentrations were measured.

185 essment of airborne nicotine, endotoxin, and nitrogen dioxide (NO2) concentrations.

186 osures to ambient fine particles (PM2.5) and nitrogen dioxide (NO2) during the first, second, and cum

187 chniques to examine the long-term effects of nitrogen dioxide (NO2) exposure on children's lung funct

188 lso had more asthma symptoms associated with nitrogen dioxide (NO2) exposure than normal-weight parti

189 Mean annual residential nitrogen dioxide (NO2) exposure was estimated for curren

190 Including satellite observations of nitrogen dioxide (NO2) in land-use regression (LUR) mode

191 (PM10) and < 2.5 mum in diameter (PM2.5) and nitrogen dioxide (NO2) in relation to PD risk.

192 is highly reactive and rapidly converted to nitrogen dioxide (NO2) in the presence of ozone, making

193 Chronic exposure to indoor nitrogen dioxide (NO2) is a public health concern.

194 Nitrogen dioxide (NO2) is an environmental air pollutant

195 lobal exposure estimates we created a global nitrogen dioxide (NO2) land use regression model for 201

196 ial NRP exposure and regional ozone (O3) and nitrogen dioxide (NO2) levels in Los Angeles County.

197 many of which involve chemistry that reduces nitrogen dioxide (NO2) on some time scale.

198 erodynamic diameter </= 10 mum (PM10) and to nitrogen dioxide (NO2) on specific airway resistance (sR

199 lysis was used on 11-year time series of all nitrogen dioxide (NO2) pixels from the Ozone Monitoring

200 Nitrogen dioxide (NO2) released primarily from combustio

201 tellite-based measurements of urban form and nitrogen dioxide (NO2) to explore relationships between

202 diameter (PM2.5 and PM10, respectively) and nitrogen dioxide (NO2) using land-use regression for 47,

203 r </= 2.5 mum (PM2.5), black carbon (BC), or nitrogen dioxide (NO2) variations that were independent

204 Levels of nitrogen dioxide (NO2) were estimated at subject residen

205 r (PM10; </=10 mum in aerodynamic diameter), nitrogen dioxide (NO2), and carbon monoxide (CO) and ris

206 amic diameter (PM2.5), sulfur dioxide (SO2), nitrogen dioxide (NO2), and carbon monoxide (CO) concent

207 with a diameter < 2.5 mug/m3 (PM2.5), PM10, nitrogen dioxide (NO2), and nitrogen oxides (NOx) at the

208 atter less than 2.5 mum in diameter (PM2.5), nitrogen dioxide (NO2), and ozone (O3) in the "moderate"

209 ly mean levels of particulate matter (PM10), nitrogen dioxide (NO2), and ozone (O3) were obtained fro

210 nts [PM10, ozone (O3), carbon monoxide (CO), nitrogen dioxide (NO2), and sulfur dioxide (SO2)] in sin

211 with aerodynamic diameter <= 10 mum (PM10), nitrogen dioxide (NO2), and sulphur dioxide (SO2) with a

212 idence intervals, for long-term exposures to nitrogen dioxide (NO2), black smoke (BS), PM2.5 (particu

213 te associations between maternal exposure to nitrogen dioxide (NO2), dietary intake of methyl nutrien

214 mmunity-level regional air pollutants [e.g., nitrogen dioxide (NO2), elemental carbon (EC), and fine

215 Indoor air pollutants [sulfur dioxide (SO2), nitrogen dioxide (NO2), hydrogen sulfide (H2S), formalde

216 nitrite ion and its first oxidation product, nitrogen dioxide (NO2), in AN solution proceeds via the

217 s of a transportation-related air pollutant, nitrogen dioxide (NO2), in the United States.

218 However, environmental pollutants, including nitrogen dioxide (NO2), may promote adaptive immune resp

219 aerodynamic diameter (PM2.5), formaldehyde, nitrogen dioxide (NO2), nicotine, carbon dioxide (CO2),

220 ations of traffic related pollutants such as nitrogen dioxide (NO2), nitrogen oxides (NOx) and partic

221 poral models estimated residence-based daily nitrogen dioxide (NO2), ozone, fine particulate, and bla

222 ers to provide site-specific measurements of nitrogen dioxide (NO2), sulfur dioxide (SO2) and volatil

223 PM2.5), particulate matter </=10 mum (PM10), nitrogen dioxide (NO2), sulfur dioxide (SO2), and ozone

224 evel concentrations of carbon monoxide (CO), nitrogen dioxide (NO2), sulfur dioxide (SO2), ozone (O3)

225 ts in removing O3 and one of its precursors, nitrogen dioxide (NO2), the cost effectiveness of using

226 ctance of PM2.5), nitrogen oxides (NOx), and nitrogen dioxide (NO2).

227 they are highly selective in reactions with nitrogen dioxide (NO2).

228 road traffic day-evening-night noise (Lden); nitrogen dioxide (NO2); and particulate matter (PM) with

229 Matter, 2.5 micrometers or less (PM2.5) and nitrogen dioxide (NO2)] predictors, we conducted a nonli

230 xture of benzene (0.6 ppm), toluene (1 ppm), nitrogen dioxide (NO2; 2 ppm) and sulfur dioxide (SO2; 5

231 -based reaction of the GDP guanine base with nitrogen dioxide (*NO2).

232 d ring core), effecting O-O cleavage, giving nitrogen dioxide (.NO2) plus a ferryl compound [(P(Im))F

233 ated air pollutant exposures (represented by nitrogen dioxide; NO2) from a dispersion model, using re

234 , associations were strongest with ozone and nitrogen dioxide; observed associations of asthma/wheeze

235 NFA -308G>A modified the action of ozone and nitrogen dioxide on lung function, asthma risk, and symp

236 After adjustment for levels nitrogen dioxide or particulate matter with an aerodynam

237 overy cohort; for example, 5-day averages of nitrogen dioxide (OR = 0.68; 95 % CI: 0.53-0.88), and pa

238 l (CI): 0.99, 1.13) and a 10-ppb increase in nitrogen dioxide (OR = 1.08, 95% CI: 1.03, 1.13) during

239 rticle filters, but little change is seen in nitrogen dioxide over the period from 1995 to 2015.

240 oach for predicting fine particulate matter, nitrogen dioxide, oxides of nitrogen, and black carbon (

241 paramagnetic molecules such as nitric oxide, nitrogen dioxide, oxygen, and the hydroxyl/hydroperoxyl

242 PM2.5), black carbon (BC), nitrogen oxides, nitrogen dioxide, ozone (O3), and carbon monoxide measur

243 n gaseous (carbon monoxide, sulphur dioxide, nitrogen dioxide, ozone) and particulate (diameter <2.5

244 m or 2.5 mum (PM10 and PM2.5, respectively), nitrogen dioxide, ozone, and black carbon.

245 y maximum concentrations of carbon monoxide, nitrogen dioxide, ozone, and sulfur dioxide and 24-hr me

246 al components, PM10, PM2.5, carbon monoxide, nitrogen dioxide, ozone, and sulfur dioxide for births i

247 Average concentrations of nitrogen dioxide, ozone, particulate matter with an aero

248 exposure to air pollutants, carbon monoxide, nitrogen dioxide, ozone, sulfur dioxide, and particulate

249 FVC were associated with declining levels of nitrogen dioxide (P<0.001 for FEV1 and FVC) and of parti

250 Associations between nitrogen dioxide, particulate matter 2.5 (PM2.5) mass, P

251 Short-term effects of carbon monoxide, nitrogen dioxide, particulate matter less than 10 mum in

252 m ozone and 24-hour average carbon monoxide, nitrogen dioxide, particulate matter with an average aer

253 associations between short-term exposure to nitrogen dioxide, particulate matter, and sulfur dioxide

254 traffic-related pollutants (carbon monoxide, nitrogen dioxide, PM2.5 elemental carbon) were associate

255 Point estimates for associations between nitrogen dioxide, PM2.5 mass, and PM2.5 absorbance with

256 Exposure to traffic-related air pollution, nitrogen dioxide, PM2.5, and PM10 during pregnancy and d

257 isomerization of fatty acids is possible by nitrogen dioxide radical, a product of NO and nitrite ox

258 he protein nitrating agents peroxynitrite or nitrogen dioxide radicals significantly decreased comple

259 ns in DNA associated with the combination of nitrogen dioxide radicals with 8-oxo-7,8-dihydroguanine

260 The combination of nitrogen dioxide radicals with the 8-oxoGua and G radica

261 on originates from water molecules, not from nitrogen dioxide radicals.

262 t are trapped by nitrate anions thus forming nitrogen dioxide radicals.

263 L showed reactivity toward both hydroxyl and nitrogen-dioxide radicals.

264 ndoor culturable Penicillium or total fungi, nitrogen dioxide, rodents (nonoccupational), feather/dow

265 Exposure to nitrogen dioxide showed little association with leukemia

266 mic diameter </=2.5 mum, or PM(2.5)), ozone, nitrogen dioxide, sulfur dioxide, and carbon monoxide in

267 Nitrogen dioxide, sulfur dioxide, and particulate matter

268 Daily concentrations of ozone, nitrogen dioxide, sulfur dioxide, carbon monoxide, and p

269 Exposures to ozone, nitrogen dioxide, sulfur dioxide, carbon monoxide, and p

270 sure (air pollution, ozone, carbon monoxide, nitrogen dioxide, sulfur dioxide, PM(10), and PM(2.5)) a

271 as a far greater impact on concentrations of nitrogen dioxide than of PM2.5.

272 ectroscopy (XPS) shows that upon exposure to nitrogen dioxide the surface of PbO particles reacts to

273 For nitrogen dioxide, the incidence rate ratio (IRR) for ast

274 For nitrogen dioxide, the odds ratio (OR) for bronchitic sym

275 Nitrogen dioxide, the proximal nitrating species formed

276 nty of 1.4% (coverage factor k = 2) based on nitrogen dioxide titration values.

277 ive reactions, via the homolytic addition of nitrogen dioxide to a double bond or via the formation o

278 th diesel particle filters; raising the mean nitrogen dioxide to oxides of nitrogen ratios from less

279 models additionally adjusted for PM(2.5) and nitrogen dioxide, UFPs exposure during the second trimes

280 lture studies results from the generation of nitrogen dioxide via the autoxidation of nitric oxide, a

281 Exposure to nitrogen dioxide was also independently associated with

282 er </= 2.5 mum (PM2.5), carbon monoxide, and nitrogen dioxide was associated with a 1-5% decrease in

283 Nitrogen dioxide was associated with HOMA-IR, glucose, i

284 Exposure to benzene, hydrocarbons, and nitrogen dioxide was estimated using land-use regression

285 Rather, community-level nitrogen dioxide was measured continuously over many yea

286 For nitrogen dioxide, we found significant associations with

287 onoxide and a 20-parts-per-billion change in nitrogen dioxide were associated with symptom odds ratio

288 /=2.5 microm (PM2.5), black carbon (BC), and nitrogen dioxide were interpolated for each mother's res

289 Lags in carbon monoxide and nitrogen dioxide were positively associated with both me

290 The associations with carbon monoxide and nitrogen dioxide were the most robust in two-pollutant m

291 culate matter, 48-hr black carbon, and 96-hr nitrogen dioxide) were collected at each school for 16 w

292 Nitrogen dioxide, which could form from nitrite under th

293 engers, indicating the likely involvement of nitrogen dioxide, which forms when nitrite reacts with h

294 than or equal to 2.5 mum (PM2.5), ozone, and nitrogen dioxide with all-cause mortality on an additive

295 ed the association of particulate matter and nitrogen dioxide with asthma.

296 h aerodynamic diameter less than 2.5 mum and nitrogen dioxide with birth weight, restricting the popu

297 associations of chronic exposure to UFPs and nitrogen dioxide with incident congestive heart failure

298 zard ratios for the associations of UFPs and nitrogen dioxide with incident CVD using random-effects

299 Heterogeneous chemistry of nitrogen dioxide with lead-containing particles is inves

300 that the reaction of electronically excited nitrogen dioxide with water can be an important source o