ライフサイエンスコーパス: no evidence

1 Using a multi-omics approach, we find no evidence ALKBH7 functions as a prolyl-hydroxylase, bu

2 However, there are no evidence-based benchmarks for the proportion of area

3 ther serious illnesses and cancer, there are no evidence-based structures and processes to support it

4 With the exception of azithromycin, there is no evidence-based treatment for primary ciliary dyskines

5 Surprisingly, we find no evidence for a canonical stem-cell organizing center

6 ta are linked with inflammation, but we find no evidence for a distinct microbial diagnostic signatur

7 Prior to that, there is no evidence for a distinct, rate-limiting initiation eve

8 umorigenesis and viral load and consequently no evidence for a functional role of the DNA viruses inc

9 Furthermore, nonlinear MR analyses found no evidence for a J-shaped relationship; instead confirm

10 The results provide no evidence for a life-prolonging effect of HCFD for the

11 A third subspecies pair exhibited no evidence for a migratory divide and hybridised extens

12 lysis of the genetic effect of DBP, we found no evidence for a nonlinear J- or U-shaped relationship

13 rption band at 1694 cm(-1); however, we find no evidence for a previously proposed ylide pathway.

14 ary evaluation of our national cohort showed no evidence for a severe, clinically relevant systemic i

15 The study therefore provided no evidence for adverse effects of intensive breeding on

16 king IL-17RA, Act1, or interleukin 22 showed no evidence for altered VVC susceptibility or immunopath

17 There was no evidence for an effect on coffee or tea intake.

18 We found no evidence for an impact of age or maturation.

19 ilibrium population of I(T) using NMR showed no evidence for an increased population of the I(T)-stat

20 No evidence for an often-assumed thin-brine wetting laye

21 We found no evidence for association between methylation levels a

22 There was no evidence for associations of physical activity with h

23 We found no evidence for associations with physical fitness.

24 We found no evidence for baseline amygdala or ventromedial PFC vo

25 ons behind the model's predictions, we found no evidence for bias based on topics, journals, discipli

26 We observed no evidence for carbonate affecting Fe(3+)/Fe(2+) reduct

27 Correspondingly, there is no evidence for coinfection with an HBV-related hepadnav

28 There was no evidence for confounding of these genetic analyses by

29 We found no evidence for Ct genomic variation contributing to con

30 No evidence for DDI2 acting as a de-ubiquitylating enzym

31 ation in the calcium mobilization assay gave no evidence for desensitization or receptor internalizat

32 We observe no evidence for dietary exploitation of horses prior to

33 Using our model, we find no evidence for differences in PPI in a rat model of Fra

34 signal loss of other tissue lipids and found no evidence for either, making this LPS-to-Lipid A-MSI (

35 Whereas we found no evidence for elevated levels of lipoxidation and glyc

36 However, no evidence for excessive belief updating was found.

37 By contrast, we found no evidence for functional gene transfer to the nucleus

38 No evidence for gene-environment interaction between gen

39 -associated bacterial composition, there was no evidence for high prevalence of SIBO or small intesti

40 acute rejection in the first year suggesting no evidence for immunologic memory response.

41 aseline MA-specific CD8 T cell responses but no evidence for improved functional responses to the vac

42 There was no evidence for increased risks of prostate or colorecta

43 oup of cases suggest that there is currently no evidence for intrauterine infection caused by vertica

44 However, there was no evidence for LAI at longer intervals of 100-200 ms, n

45 No evidence for ligand-specific differences in receptor

46 ified no patient-to-patient transmission and no evidence for livestock as a source of K. pneumoniae i

47 Si coordination observed at 20 GPa, we find no evidence for major structural changes occurring in th

48 s with N-(hydroxymethyl)benzamide compounds, no evidence for mechanistic variation based upon the nat

49 We found no evidence for patient antibody activity specific to au

50 ty in relying on learned contingencies, with no evidence for perseverative behavior.

51 8.27, P = .16), and the funnel plot provided no evidence for publication bias.

52 gin of the Arabian horse in the Middle East, no evidence for reduced global genetic diversity across

53 mice, however, was unaltered, and there was no evidence for reduced pH-buffering capacity in the ske

54 ified as showing strong, good, some, or weak/no evidence for reinfection.

55 ents to aldehydes or methyl ketones provided no evidence for ring-opened products that would indicate

56 There was no evidence for selection in highly estrogen-dependent c

57 tive power than polygenic hazard scores with no evidence for sex differences.

58 There was no evidence for significant effects of probiotics for an

59 rogeneity in bacterial adhesion, and we find no evidence for significant heterogeneity in later steps

60 At this stage we find no evidence for significantly more transmissible lineage

61 We find no evidence for stable oligomers of RHBDL2 in giant plas

62 lsification end points was found, suggesting no evidence for substantial residual confounding.

63 The LFP experiments showed no evidence for tautomerization of dG(N2-H)(.) to dG(N1-

64 revealed a dense macrophage infiltrate, but no evidence for the vasculopathy typically associated wi

65 ses varied independently among species, with no evidence for trade-offs.

66 We find no evidence for transcription factor tethering and DNA-b

67 ntial over large size scales, our work finds no evidence for transport limitations that would decreas

68 conform to normative Bayesian models: we saw no evidence for use of priors in our cognitively-complex

69 , no morphological changes were observed and no evidence for viral infection or integration could be

70 ntrasting inference with simulation, we find no evidence for viral lineages to preferentially circula

71 analyses with Bayesian statistics and found no evidence in favor of the hypothesis that language lat

72 Under the conditions employed we find no evidence in support of a barotactic response; the cel

73 Nevertheless, we found no evidence in support of GGI effects influencing T2D ri

74 However, no evidence indicated that vaccination reduced hospitali

75 described as acidic or thermally labile, but no evidence is available from direct measurements of cro

76 rapy reduced the time to decannulation, with no evidence of a between-group difference in the inciden

77 study, which had nationwide coverage, showed no evidence of a causal link between HPV vaccination and

78 hol when delivered to couples, but there was no evidence of a decrease in reported symptoms of depres

79 In conclusion, there was no evidence of a difference among the 3 treatment approa

80 t secondary mobility outcomes, but there was no evidence of a difference between groups for most othe

81 However, there was no evidence of a difference between groups for upright t

82 p: 4) due to unrelated causes, and there was no evidence of a difference between groups in fall rates

83 There was no evidence of a difference between the protocols for an

84 There was no evidence of a difference in favourable outcomes of pa

85 There was no evidence of a difference in incidence of dental pain

86 n the intention-to-treat analysis, there was no evidence of a difference in the proportion of partici

87 This study provides no evidence of a major concern in Europe for olodaterol

88 o 0.3), SF = -0.9% (95% CI -2.0 to 0.2); but no evidence of a significant effect of either interventi

89 and ingestion rates in >500 species, we find no evidence of a trade-off across species.

90 nts treated for latent tuberculosis we found no evidence of acquired drug resistance, underscoring th

91 linical resolution and viral clearance, with no evidence of acquired drug resistance.

92 There was no evidence of acute or chronic toxicity associated with

93 sessable lobes (including 10/21 [47.6%] with no evidence of acute pulmonary emboli).

94 reased MRI brain volume loss at Week 13 with no evidence of additional loss through Week 78.

95 he newcomers from South America, with almost no evidence of admixture.

96 After 14 days, no evidence of adverse effect on body weight, mortality

97 were colonized after adding social cues and no evidence of Allee effects was found.

98 Finding no evidence of allometry, we made morphological extrapol

99 We found no improvement in response and no evidence of an abscopal effect with the addition of S

100 ng 498- to 754-fold increases from baseline; no evidence of an antigen dose response was observed.

101 Conclusion There was no evidence of an association between MRI parameters tha

102 There was no evidence of an association between radiofrequency ene

103 No evidence of an association was observed for NO2 or NO

104 However, no evidence of an association was observed for suicidal

105 ty in nonobstetrical care settings, 15 found no evidence of an association with liability risk and 5

106 For patients with stable CAD, PCI shows no evidence of an effect on any of these outcomes.

107 We found no evidence of an increase in social cohesion after simu

108 There was no evidence of an increased risk of major congenital ano

109 There was no evidence of an intervention effect in the TSM group c

110 mic-level B'(III)/B"(I) site ordering (i.e., no evidence of antisite defects).

111 0 000 person-years), adjusted results showed no evidence of any association between the use of glitaz

112 They also found no evidence of any unintended increase in suicides due t

113 The analysis revealed no evidence of association between the presence of a sul

114 We found no evidence of association of HP CNV or rs200999 with fu

115 No evidence of association was observed for either women

116 etween membrane rupture and delivery) showed no evidence of association with atopic dermatitis.

117 ith a total of 18,847 participants and found no evidence of association.

118 There was no evidence of associations of genetically predicted alc

119 unostaining in undamaged DDH cartilage, with no evidence of augmented cell death by activation of cas

120 rths vs FBH+ 25.4 per 1000 livebirths), with no evidence of between-site heterogeneity (I(2)=0.7%; p=

121 NAs and were more methylated than genes with no evidence of capture.

122 The younger patient had no evidence of cardiac disease, but met possible ARVC di

123 y related with coronary artery disease, with no evidence of causal effects on other diabetes complica

124 However, we found no evidence of CB2 in the brain using either [3H] CP-55,

125 s of human cirrhotic liver tissue, there was no evidence of chromosome-level copy number variations.

126 mAb MIL77 starting 3 days postexposure show no evidence of clinical illness and survive challenge.

127 We found no evidence of clinically meaningful benefits of serum u

128 d Kv3.3 mRNA and protein were measured, with no evidence of compensation by Kv3.2 or Kv3.4 in the res

129 ioned SCR during threat acquisition, despite no evidence of compromised SCR to unconditioned stimulus

130 We found no evidence of confounding by or interaction with indivi

131 However, there is no evidence of cross-reactivity, which explains this obs

132 a broad population of PFC neurons, we found no evidence of current-goal-specific memory in any previ

133 L [interquartile range, 638-3,735 ng/mL] for no evidence of deep venous thrombosis; p < 0.0001).

134 There was minimal evidence of abuse, and no evidence of dependence or opioid withdrawal by AEs or

135 These findings indicate no evidence of difference in incidence of red blood cell

136 We found no evidence of differences in behavioral risk factors, i

137 No evidence of differences in effectiveness were found i

138 In 24-h urine samples, there was no evidence of differences in sodium levels (mean differ

139 Despite showing no evidence of direct cellular toxicity, this anchoring

140 Currently, there is no evidence of direct interactions between mosquitoes an

141 s with evidence of disease activity (EDA) or no evidence of disease activity (NEDA; occurrence of rel

142 aged 18-80 years with stage IV melanoma with no evidence of disease after surgery or radiotherapy.

143 xcludes patients with stage IV melanoma with no evidence of disease.

144 cebo in patients with stage IV melanoma with no evidence of disease.

145 d no evidence of neoplastic features in BPH: no evidence of driver genomic alterations, including low

146 We found no evidence of drought-buffered routes.

147 ificant age-related cataracts, but there was no evidence of early onset of age-related cataracts in D

148 In this large well-conducted trial, we found no evidence of effect of a plausible very brief pedomete

149 However, there is still no evidence of effects longer than a month after stimula

150 t on cooling (pH decrease of ~1.0 unit), and no evidence of either buffer salt crystallization or pro

151 There was no evidence of enlarged axillary lymph nodes.

152 only found in lung cancer were observed with no evidence of enrichment in any mutation following cana

153 No evidence of genotype selection was observed seven yea

154 While we found no evidence of global differences in GI between domestic

155 or pH-dependent channel formation, there was no evidence of H-L-H involvement in either pH gating or

156 ic disease should have a diverse diet, given no evidence of harm and some potential association of be

157 tion fraction and fractional shortening, but no evidence of heart failure.

158 ion for SK-MEL-147 when using nude mice with no evidence of hepatotoxicity.

159 Lastly, there was no evidence of heterogeneity in effect estimates by pati

160 There is no evidence of heterogeneity in PRS performance in Chine

161 attern reported for other flatfishes in that no evidence of higher order periodicity was present.

162 Additionally, no evidence of higher trophic level bioaccumulation or t

163 Furthermore, we found no evidence of hypoxia gene expression in HBV de novo in

164 Notably, we found no evidence of immune-enhanced disease after viral chall

165 There was no evidence of increased inflammation or histologic inju

166 We found no evidence of increased proximal humeral (HR, 1.06; 99%

167 enarios, including as-treated analyses, with no evidence of increased risk over time with PTXDs.

168 There is no evidence of increased toxicity or CBC events from rad

169 No evidence of innervation by excitatory, cholinergic mo

170 There was no evidence of interaction between MRD status and condit

171 ad CT findings (not shown) were normal, with no evidence of intracranial hemorrhage or edema.

172 ad CT findings (not shown) were normal, with no evidence of intracranial hemorrhage or edema.Her subs

173 There was no evidence of large reductions in SFA, but we are unabl

174 covery sequences and native T1 mapping, with no evidence of late gadolinium enhancement suggestive of

175 sied and multi-segment tumor tissues reveals no evidence of less proliferative diploid/2N lesions in

176 There was no evidence of macrolide-resistance in this population.

177 on and exhibit long-lasting patterning, with no evidence of malformations or angiomas.

178 The fourth patient showed no evidence of malignancy 8 years after diagnosis.

179 Thermoplasmatota genomes/MAGs found no evidence of mcrA homologues outside of the Methanomas

180 oportion mediated: 12%, 95% CI = 4-20%), but no evidence of mediation was found for average blood glu

181 ter 30 days, though these communities showed no evidence of methanotrophy after nearly 8 months of en

182 No evidence of MMR infections was found, but 8 (0.7%) SO

183 Additionally, using RNA sequencing, we find no evidence of mRNA cleavage following stress.

184 allow us to draw causal conclusions, we find no evidence of negative health consequences associated w

185 There was no evidence of negative selection observed for synonymou

186 We find no evidence of neoplastic features in BPH: no evidence o

187 in the post-treatment follow-up period, with no evidence of new late side-effects.

188 he alleles at this locus were modified, with no evidence of off-target editing.

189 rctic, these degraders seem to be absent and no evidence of other wood-degrading organisms has been r

190 t infection, in the surviving mice, we found no evidence of parasite DNA, cysts, or inflammation in t

191 mble paternally mtDNA heteroplasmy, but find no evidence of paternal transmission of mtDNA in humans.

192 Although no evidence of pathogenicity of P. homopolare in Condors

193 required to have full documentation in which no evidence of peri-implantitis was not indicated.

194 The awn shows no evidence of photosynthesis.

195 Both scores showed no evidence of poor calibration.

196 tion and telemetry studies, our results show no evidence of population structure (non-significant F(S

197 he Indo-West Pacific, yet their bindins show no evidence of positive selection, possibly because the

198 Furthermore, there was no evidence of potassium wasting (P=0.20) or renal dysfu

199 There was no evidence of pre-ictal change in the spike spatial dis

200 No evidence of publication bias was found.

201 There was no evidence of rebleeding at follow-up MRI.

202 had no relevant medical or surgical history; no evidence of recent pregnancy, abortion, or breastfeed

203 However, no evidence of reducing microbial translocation was obse

204 ighly cytotoxic and proliferative states and no evidence of regulatory T cell depletion.

205 amination showed an inferior bullous RD with no evidence of retinal breaks, while a large RPE tear wa

206 on DFE or fundus photography at 3 months and no evidence of SD-exudative activity requiring retreatme

207 There was no evidence of shifts in symptoms of mild or severe depr

208 In our in-vitro environment, we found no evidence of signal drifts or fluctuations associated

209 Results: Statistical analyses revealed no evidence of significant differences in regional (11)C

210 specific cyclin (SOC2), although we obtained no evidence of SOC2 cycling by transcription, translatio

211 CRISPR/Cas9-mediated gene editing, there is no evidence of success in genetic alteration of Ag-exper

212 nance of arsenite in control experiments and no evidence of surface-bound thioarsenates at pH 7.0 sug

213 There was no evidence of systemic fetal hypoxia, or changes in fet

214 There was no evidence of target-mediated clearance or accumulation

215 Twenty-nine samples from individuals with no evidence of TB infection by TST and no known exposure

216 rs between octahedral Mn(3+) and Mn(4+) with no evidence of tetrahedral Mn(5+) or Mn(7+).

217 bservations to test this hypothesis, finding no evidence of the 3.5-keV line emission from the Milky

218 However, so far there has been no evidence of the cold dense molecular phase (10 to 100

219 overlap in nesting areas among predators and no evidence of their expansion in the absence of owls, s

220 tudied here were nearly poikilothermic, with no evidence of thermoregulation of T(leaf) towards a hom

221 n in 29% of patients with PEX and identified no evidence of this among those in the control group (P

222 noise and the incidence of AMI and CHF with no evidence of threshold values.

223 changes in any safety parameters, including no evidence of thromboembolic events.

224 No evidence of thyroid hormone-mediated neurobehavioral

225 No evidence of tissue tropism was observed, and there wa

226 It also shows no evidence of tolerance, which has been a problem with

227 pear to be linked to increased staffing with no evidence of tools providing a more efficient or effec

228 animals succumbing after treatment revealed no evidence of treatment-associated toxicity.

229 reased to 20/32 without recurrence of RD and no evidence of tumor activity.

230 al examination by electron microscopy showed no evidence of viral particles in the biopsy samples.

231 Together, these findings indicate no evidence of widespread inappropriate methods to impro

232 th vessel area and tortuosity, together with no evidence of within-person-between-eye differences in

233 There was no evidence of within-person-between-eye differences in

234 amyotrophic lateral sclerosis, but there is no evidence regarding whether a high-caloric diet increa

235 We found no evidence supporting a relationship between alcohol co

236 No evidence supports an involvement of activated protein

237 No evidence supports that WHR and WHtR are more suitable

238 Our result provide no evidence that a reminder before extinction can preven

239 Using chimeric Fls2/Fls3 proteins, we found no evidence that a single receptor domain is responsible

240 However, there was no evidence that ACE inhibitors or ARBs affected the ris

241 We found no evidence that an increased risk for depression was co

242 Despite this, we found no evidence that aphid population growth rates were adve

243 There was no evidence that baseline Hypothyroid Symptoms score or

244 e its effect on almond production, there was no evidence that bee visitation affected almond nutritio

245 There is currently no evidence that cats or dogs play a significant role in

246 There was no evidence that clip use moderated the effects of the l

247 When compared to control periods, we found no evidence that colony-wide escape (i.e., flight) behav

248 There was no evidence that concussion or vision symptom trajectori

249 There is no evidence that domestic water softeners improve object

250 ph modelling of the social networks provided no evidence that either species was dominant over the ot

251 Although there is no evidence that elevated rates of cesarean sections (CS

252 ased on 270 events, the meta-analysis showed no evidence that event-free survival was improved with a

253 We found no evidence that formation of micrometer(s)-sized diamon

254 stent with an E*-E equilibrium and providing no evidence that free thrombin is zymogen-like.

255 We found no evidence that health expenditures changed for middle-

256 In contrast with our hypotheses, there was no evidence that IE altered cognitive biases in this stu

257 We also found no evidence that individuals sampled at the same site we

258 es between the two HVDAS-affected groups and no evidence that individuals with more widespread methyl

259 strips reduced visits to canola, but we saw no evidence that infection treatment shifted foraging pr

260 ing Bayesian regression tree models, we find no evidence that interaction with IRA accounts substanti

261 lower primary auditory cortex thickness, but no evidence that it predicted in vivo beta-amyloid depos

262 tion appears to be relatively safe, there is no evidence that it reduces esophageal eosinophil counts

263 etion inhibited cell proliferation, we found no evidence that it specifically interacts with RNA-bind

264 However, there is no evidence that masks result in significant physiologic

265 However, we found no evidence that mindfulness training is able to buffer

266 cs and single-cell gene expression, we found no evidence that nevus cells are senescent, either compa

267 te more than a century of scrutiny, there is no evidence that opsins function in chemosensation.

268 sition differed among locations but we found no evidence that primary remnants provide critical habit

269 There was no evidence that S44819 improved clinical outcome in pat

270 We found no evidence that sleep duration, sleep duration variabil

271 There was no evidence that slow-stroke massage (mixed care activit

272 nd obesity, 1.4 (0.9-2.2, p = 0.13) We found no evidence that socioeconomic factors modified the asso

273 al synapses as a metric of strength, we find no evidence that strong synapses have a predominant role

274 We found no evidence that subterranean termites (Reticulitermes)

275 We found no evidence that such hybrids form in trans and in a Rad

276 ed by A581G-bearing parasites, but there was no evidence that the adverse effects of these parasites

277 We find no evidence that the distribution of neurons differs bet

278 There is, however, no evidence that the experience of cognition reveals the

279 Moreover, there is no evidence that the loops undergo significant gating mo

280 We found no evidence that the putative protective threshold or AA

281 However, we find no evidence that these ACE2-affinity-enhancing mutations

282 ural and biophysical analyses, we have found no evidence that these inhibitors bind to the proposed a

283 in the intensive treatment group, there was no evidence that they modified or attenuated the effect

284 However, we found no evidence that they take the perspective or the knowle

285 Multivariate models show no evidence that veterinary antibiotic use increased the

286 There was no evidence that volume of or amount of sugar in purchas

287 10(10) unique sequences per microliter with no evidence that we have begun to approach complexity li

288 However, there is no evidence to explicitly support an infusion time of 30

289 e tracked to a final source, though we found no evidence to suggest that he had the ability to initia

290 correlate with hair cortisol, and there was no evidence to suggest that individual differences in hi

291 tion-based historical cohort study there was no evidence to suggest that seasonal influenza vaccine w

292 ng reduces phytate levels, however, there is no evidence to support or refute these claims for nuts.

293 There is no evidence to support the choice of any particular tool

294 We found no evidence to support the utility of risk stratifying C

295 etect ribosome-puromycin complexes, we found no evidence to support this assumption.

296 chanically ventilated patients, but there is no evidence, using nonvolitional, objective measurements

297 No evidence was found for genetic association between CF

298 No evidence was found for the interaction between photos

299 Over a long period of observation, no evidence was found that a specific AHM drug class was

300 mple sizes and sensitivity analyses, we find no evidence with Mendelian randomisation techniques that