ライフサイエンスコーパス: nociceptive response

1 AF receptors decreased the late-phase of the nociceptive response.

2 s are crucial to maintaining this prefrontal nociceptive response.

3 neurons are used as an in vitro model of the nociceptive response.

4 in higher level cognitive processes or basic nociceptive responses.

5 ed PACAP-induced CeA neuronal activation and nociceptive responses.

6 are known to inhibit dural trigeminovascular nociceptive responses.

7 sure and salt and water balance and modulate nociceptive responses.

8 melastatin-8 (TRPM8) channel without evoking nociceptive responses.

9 n injection and showed sustained increase in nociceptive responses.

10 of Sp5C, without affecting cutaneous-evoked nociceptive responses.

11 ile-pain interactions employed heat to evoke nociceptive responses.

12 of the pain modulatory system that regulates nociceptive responses.

13 This shows a motivational trade-off of nociceptive responses.

14 of nociceptive neurons, and reduced thermal nociceptive responses.

15 adiol alters injury- or inflammation-induced nociceptive responses after carrageenan administration i

16 by quantifying palpebral opening and evoked nociceptive responses after corneal application of capsa

17 n-injected mice showed significantly reduced nociceptive responses after injection of C terminus of m

18 tribution of heteromeric P2X2/3 receptors to nociceptive responses and mechanosensory transduction wi

19 aling in sensory SGCs decreased heat-induced nociceptive responses and reversed inflammation-induced

20 entral nerve cord (VNC) are required for the nociceptive response, and that multidendritic sensory no

21 In the absence of verbal report, these nociceptive responses are used as measures of pain sensa

22 GABAergic systems may contribute to aberrant nociceptive responses associated with inflammatory and n

23 n behaviors including food and water intake, nociceptive responses, breathing regulation, as well as

24 of stress exhibit larger amplitude cortical nociceptive responses, but this is not reflected in thei

25 inflammation, indicating that modulation of nociceptive responses by amygdala ERK activation is spec

26 PRL enhances nociceptive responses by rapidly modulating channels in

27 hase 1 do not contribute to the magnitude of nociceptive responses during Phase 2.

28 lesions significantly decreased inflammatory nociceptive responses during the prolonged, tonic portio

29 large, dose- and time-related reductions in nociceptive responses following administration into the

30 ncrease both stimulus-evoked and spontaneous nociceptive responses following an injury in male and fe

31 algesia after L5 ligation or on inflammatory nociceptive responses following formalin injection in th

32 odulation of cutaneous-evoked C-fiber spinal nociceptive responses from the brainstem.

33 al and/or cutaneous facial trigeminovascular nociceptive responses, from the brainstem ventrolateral

34 may block spinal NMDA receptors to attenuate nociceptive responses, however, our data cannot exclude

35 of BN 52021 decreased the late-phase of the nociceptive response in a concentration-dependent manner

36 nanoparticles (NM0127) showed a strong anti-nociceptive response in multiple assays of evoked and on

37 gnified contribution of these neurons to the nociceptive response in the ACC.

38 lacement attenuated TMJ inflammation and the nociceptive responses in a dose-dependent manner in the

39 imulation, which might modulate differential nociceptive responses in AD skin.

40 These data suggest that the enhanced nociceptive responses in AS model mice are due to loss o

41 EA-1021 showed equally potent attenuation of nociceptive responses in both animal models of pain.

42 eceptors in the generalized sensitization of nociceptive responses in caterpillars.

43 show that gentle mechanical stimuli inhibit nociceptive responses in Drosophila larvae.

44 e not only headache-associated pain but also nociceptive responses in extracranial tissues.

45 Morphine failed to significantly reduce nociceptive responses in hot plate or tail flick tests o

46 In the present study, we compared nociceptive responses in inflammatory and neuropathic mo

47 ifferences in the effects of KOR agonists on nociceptive responses in males and females.

48 -induced inflammation and pain, we evaluated nociceptive responses in mice that carry a null mutation

49 of chronic pain and nociceptive, but not non-nociceptive responses in normal animals.

50 consumption and provoked apathy and reduced nociceptive responses in rats.

51 ctions of improgan were evaluated on thermal nociceptive responses in rats.

52 ecdysone signaling is sufficient to promote nociceptive responses in second instar larvae and suppre

53 ve NMDA receptor/glycine site antagonist, on nociceptive responses in the tail flick and formalin tes

54 We determined whether PN contributes to nociceptive responses in two distinct models of migraine

55 levels and consequently alters inflammatory nociceptive responses in vivo.

56 isense oligodeoxynucleotide (ODN) on bladder nociceptive responses induced by intravesical acetic aci

57 The nociceptive response is limited by enkaphalin-expressing

58 ously found that TIP39 affects several acute nociceptive responses, leading us to now investigate its

59 , injection of FcgammaRIII siRNA reduced the nociceptive response of rats with an arthritic TMJ and r

60 Neither drug altered the nociceptive responses of sham-operated controls.

61 mine whether local inhibition of FAAH alters nociceptive responses of spinal neurons in the spinal ne

62 n the potency of opioid agonists to suppress nociceptive responses of the contralateral, uninflamed h

63 st, two sequential doses of WB4101 increased nociceptive responses on both the tail-flick and foot-wi

64 us, in addition to somatotopically organized nociceptive responses, orofacial deep tissue injury also

65 hat MOR ligands directly modulate SP-induced nociceptive responses primarily at postsynaptic sites, r

66 ember 1), but the mechanisms underlying this nociceptive response remain poorly understood.

67 as potentially important to early immune and nociceptive responses.SIGNIFICANCE STATEMENT Bacterial i

68 c administration of ACEA-1021 attenuated the nociceptive responses solely in the formalin test.

69 Comparative studies assessed behavioral nociceptive responses, spinal cytokine mRNA and protein

70 thod, the mGluR(5) antagonist did not affect nociceptive responses, suggesting that thalamic receptor

71 d hot plate tests for spinal and supraspinal nociceptive responses than wild-type mice.

72 reas the contribution of PLCbeta1 to thermal nociceptive response thresholds requires a critical conc

73 eral feed-forward inhibition that attenuates nociceptive responses through a pathway involving the lo

74 tial vanilloid 1 (TRPV1), which transduces a nociceptive response to capsaicin.

75 n, constitutes an important component of the nociceptive response to CO(2).

76 ats depleted of dopamine displayed increased nociceptive responses to chemical stimulation of the fac

77 n spinal slices and in inhibiting behavioral nociceptive responses to heat.

78 in sensory neurons that ensures appropriate nociceptive responses to inflammatory mediators.

79 ally to mice, CGRP8-37-cholestanol inhibited nociceptive responses to intraplantar injection of capsa

80 man NK(1)R, aprepitant transiently inhibited nociceptive responses to intraplantar injection of capsa

81 ventrolateral periaqueductal gray modulates nociceptive responses to noxious heating of the feet by

82 at are different from those that inhibit the nociceptive responses to noxious heating of the feet.

83 t are different from those that inhibits the nociceptive responses to noxious heating of the feet.

84 e ventrolateral periaqueductal gray inhibits nociceptive responses to noxious heating of the tail by

85 e ventrolateral periaqueductal gray inhibits nociceptive responses to noxious heating of the tail by

86 at are different from those that inhibit the nociceptive responses to noxious heating of the tail.

87 Nociceptive responses to select noxious thermal and mech

88 ked excitation of spinal neurons and blunted nociceptive responses to SP.

89 stimuli and morphine fails to attenuate the nociceptive responses to thermal stimuli in these animal

90 The nociceptive response (visceromotor response [VMR]) to CR

91 sion in DRGs neurons and to evaluate whether nociceptive responses were affected in AS model mice (gl

92 d in male and female AS model mice; however, nociceptive responses were not altered by the conditiona

93 Nociceptive responses were quantitated in the rat by mea

94 Kappa opioid receptor (KOR) ligands alter nociceptive responses when applied to the rostral ventro

95 ascular neuronal tone and Adelta-fiber dural-nociceptive responses, which differs from the way the br