ライフサイエンスコーパス: nodular

1 classified by MRI as infiltrative and 81 as nodular.

2 <50 years]), superficial spreading subtype (nodular, 0.5 [0.2-1.0]; lentigo maligna, 0.4 [0.2-0.7];

3 The iris tumor was nodular (16/21, 76%) or diffuse (5/21, 24%).

4 92; 15%), cutaneous (84; 14%), and pulmonary nodular (47; 8%).

5 eterogeneous, including maculopapular (82%), nodular (6%), and diffuse cutaneous (12%) mastocytosis.

6 er; however, their role in primary pigmented nodular adrenocortical disease (PPNAD) has not been inve

7 Primary pigmented nodular adrenocortical disease (PPNAD) is associated wit

8 ng PRKAR1A mutations cause primary pigmented nodular adrenocortical disease (PPNAD) or Carney complex

9 stology (hazard ratio, 0.19 for desmoplastic/nodular and 45.97 for large-cell/anaplastic medulloblast

10 Furthermore, we show that the nodular and digitate silica structures at El Tatio that

11 Nodular and macular PKDL, and VL, can be infectious to s

12 potential significance of the characteristic nodular and mm-scale digitate opaline silica structures

13 to be approximately 3-fold more abundant in nodular and tumor tissue than in control tissue.

14 Female sex,nodular and unclassified or other histologic subtypes, i

15 pes: superficial spreading, lentigo maligna, nodular, and acral lentiginous.

16 f BCC and histological subtype (superficial, nodular, and infiltrating) in the confocal mosaics.

17 l markings, consolidation, and ground-glass, nodular, and reticular opacity), distribution, and exten

18 Types of CM included superficial spreading, nodular, and unclassified in 5106 cases among 3206 men a

19 iffuse anterior in 80% (n = 28), followed by nodular anterior 11.4% (n = 4), and necrotizing in 8.6%

20 ck of corneal involvement, papillomatous and nodular appearance, microscopic multifocality, and posit

21 Eight CAN biopsies showed nodular arteriolar hyalinization and one was positive fo

22 This high prevalence of nodular autonomy usually results in a further increase i

23 id activity which, in the long term, reduces nodular autonomy.

24 cessful elimination of superficial and early nodular basal cell carcinoma (BCC) in 2 cases using RCM

25 The recurrence rate of periocular nodular basal cell carcinoma (PNBCC) following treatment

26 mod compared with surgery for superficial or nodular basal cell carcinoma at low-risk sites in our no

27 (superficial basal cell carcinoma, 6 weeks; nodular basal cell carcinoma, 12 weeks) or excisional su

28 normal morphology or, in a few cases, small nodular basal laminar deposits.

29 ent option for small low-risk superficial or nodular basal-cell carcinoma dependent on factors such a

30 s of resting hair follicles in mice, induced nodular BCC development from a small subset of cells in

31 ke tumors from interfollicular epidermis and nodular BCC-like tumors from hair follicle stem cells.

32 cases of NM, 134 of invasive non-NM, 115 of nodular benign melanocytic tumors, and 135 of nodular no

33 17 patients with treatment-naive noncavitary nodular bronchiectatic MAC lung disease.

34 atment regimen for patients with noncavitary nodular bronchiectatic MAC lung disease.

35 ded for the initial treatment of noncavitary nodular bronchiectatic Mycobacterium avium complex (MAC)

36 were low body mass index (BMI), radiographic nodular-bronchiectatic (NB) pattern, and increase in the

37 constitutively phosphorylated at Ser-380 in nodular but not superficial spreading melanoma and did n

38 were infiltrative and two of 27 tumors were nodular) but none were found in HCV patients (P = .001).

39 ; of the remaining 6 that were classified as nodular by MRI, 5 histologically demonstrated stage II o

40 ve cusps undergo retraction, stiffening, and nodular calcification.

41 g studies showed a presacral solid mass with nodular calcifications.

42 phs of 50 patients with 55 confirmed primary nodular cancers (mean diameter, 20 mm) as well as 30 pat

43 computed tomographic examinations range from nodular centrilobular opacities in acute/subacute diseas

44 ibitor of metalloproteinases 1 in metastatic nodular compared with metastatic superficial spreading m

45 ma lines and metastatic tissues derived from nodular compared with superficial spreading melanoma.

46 histologically desmoplastic; the other 2 had nodular components.

47 ctive lymphoid hyperplasia (BRLH) (38 [5%]), nodular conjunctivitis (31 [4%]), dermoid (30 [4%]), and

48 pterygia (vascular component) and Salzmann's nodular degeneration (corneal component).

49 bed in more recent case series of Salzmann's nodular degeneration and diffuse keratoconjunctival prol

50 helial basement membrane dystrophy, Salzmann nodular degeneration, and pterygium, should be addressed

51 d at the periphery of discoid platelets into nodular densities consistent with clustered alphaIIbbeta

52 demonstrated sub-retinal pigment epithelial nodular deposits, some of which were confluent with over

53 Nodular desmoplastic medulloblastoma (ND) and medullobla

54 atient's postablation CT images showed focal nodular diaphragmatic thickening.

55 Twenty-four (47%) patients had nodular disease with bronchiectasis and 27 (53%) had upp

56 ocyte sedimentation rate, rheumatoid factor, nodular disease, modified Health Assessment Questionnair

57 d for total thyroidectomy for goiter, benign nodular disease, suspected thyroid cancer, or known thyr

58 s, there was cellular atypia consistent with nodular dysplasia.

59 ed States and had endoscopic evidence of non-nodular dysplastic BE </=8 cm in length.

60 he orphaned lesion known as Quilty effect or nodular endocardial infiltrates.

61 he orphaned lesion known as Quilty effect or nodular endocardial infiltrates.

62 studies revealed multiple punctate foci with nodular enhancement in the brain and multifocal cystic l

63 nvolving the cervicothoracic spinal cord and nodular enhancement of the cauda equina.

64 Lesions with initially nodular enhancement were smaller than those initially en

65 found that the variables MRI size>25mm, non-nodular enhancement, adenopathies on MRI, and p53 expres

66 MRI tumor size>25mm, non-nodular enhancement, breast edema, areola-nipple complex

67 ial response by EASL criteria and peripheral nodular enhancement, respectively.

68 ted with lower disease-free survival are non-nodular enhancement, size>25mm, and adenopathies on MRI,

69 Conclusions and Relevance: Papular and nodular eruptions with scale, as well as mucosal erosion

70 Analysis of primary clinical samples of nodular fasciitis confirmed the activation of a Jak1-STA

71 Chromosomal translocations in nodular fasciitis result in USP6 overexpression, leading

72 ne cyst (ABC), and the related benign lesion nodular fasciitis.

73 , which recapitulate key features of ABC and nodular fasciitis; however, the identity of USP6's relev

74 eosinophilic angiocentric fibrosis, reactive nodular fibrous pseudotumor, sclerosing mesenteritis, an

75 us gingivitis, is a rare condition involving nodular gingival enlargement with ulceration and periodo

76 ney involvement and histologically exhibited nodular glomerular involvement.

77 f a monoclonal immunoglobulin LC, leading to nodular glomerulosclerosis and nephrotic syndrome.

78 Morphologically, smoking-associated nodular glomerulosclerosis closely resembles diabetic ne

79 sufficient to induce Kimmelstiel-Wilson-like nodular glomerulosclerosis in mice through a process tha

80 supports that the entity known as idiopathic nodular glomerulosclerosis is not idiopathic.

81 rogates renal insufficiency and the diabetic nodular glomerulosclerosis phenotype of diabetic Sema3a(

82 use mesangial sclerosis (focally approaching nodular glomerulosclerosis), focal arteriolar hyalinosis

83 in of function in eNOS(-/-) mice resulted in nodular glomerulosclerosis, mesangiolysis, microaneurysm

84 teinuria, renal insufficiency, and extensive nodular glomerulosclerosis, mimicking advanced DN in hum

85 mesangiolysis, and focal segmental and early nodular glomerulosclerosis.

86 sis, such as Graves' disease and functioning nodular goiters, there are more than 20 less common caus

87 ts in an increase in the prevalence of toxic nodular goitre and hyperthyroidism in populations.

88 gs are not generally used long term in toxic nodular goitre, because of the high relapse rate of thyr

89 oidism is Graves' disease, followed by toxic nodular goitre.

90 s characterized by either the development of nodular granulomatous lesions in the peribronchial regio

91 at replaced the hepatic lobule or histologic nodular growth in the portal triad that effaced adjacent

92 urther promoted the development of MITF-high nodular growth melanomas.

93 patterns and 1 histologically demonstrated a nodular growth pattern.

94 classified as exhibiting an infiltrative or nodular growth pattern.

95 growth in the sinusoidal spaces, whereas MRI nodular growth patterns corresponded to stage II/III his

96 tients [90.5% males, 89.2% cirrhotics, 89.2% nodular HCC, median age 63 (34-84) years] underwent 111

97 ia with megalencephaly (20), periventricular nodular heterotopia (61), and pachygyria (47).

98 DCX and LIS1), persons with periventricular nodular heterotopia (FLNA), and persons with pachygyria

99 f origin and form persistent periventricular nodular heterotopia (PH).

100 developmental disorders with periventricular nodular heterotopia (PNH) are etiologically heterogeneou

101 is possible in patients with periventricular nodular heterotopia (PVNH) to detect abnormal fiber proj

102 lformations, polymicrogyria, periventricular nodular heterotopia and diffuse megalencephaly without c

103 dles syndrome, with X-linked periventricular nodular heterotopia and FG syndrome (Omim, 305450).

104 patients with epilepsy from periventricular nodular heterotopia and matched healthy controls.

105 unction of human FLNA causes periventricular nodular heterotopia in females and is generally lethal (

106 pathies A, affecting brain (peri-ventricular nodular heterotopia), heart (valve defect), skeleton, ga

107 gration disorders, including periventricular nodular heterotopia, subcortical band heterotopia and li

108 izencephaly, and 15 cases of periventricular nodular heterotopia.

109 elected subgroup of patients with unilateral nodular heterotopia.

110 mpassed cysts (70), hemangiomata (37), focal nodular hyperplasia (FNH) (23), adenomata (47), and 20 l

111 Focal nodular hyperplasia (FNH) and hepatocellular adenoma (HC

112 Follicular nodular hyperplasia (FNH) is a common benign liver tumor

113 Nonoperative management of focal nodular hyperplasia (FNH) is an accepted paradigm in adu

114 Focal nodular hyperplasia (FNH), hepatocellular adenoma (HCA),

115 h nodular regenerative hyperplasia and focal nodular hyperplasia (FNH), which finally evolved to a gi

116 logically indeterminate for adenoma or focal nodular hyperplasia (FNH): (1) continue to observe with

117 Benign focal nodular hyperplasia and hepatocellular adenoma could be

118 s (42.2%) had adenomas, 29 (19.7%) had focal nodular hyperplasia, 25 (17.0%) had hemangiomas, 11 (7.5

119 ding a rate-limiting step between simple and nodular hyperplasia, the latter of which is marked by th

120 Similarly, in nodular hyperplasia, which is the most severe form of SH

121 ve thrombosis, and lack of visibility; focal nodular hyperplasia-like nodules (six [14%] of 42 vs 0 [

122 evels in H-HCA, IHCA, b-HCA, UHCA, and focal nodular hyperplasia.

123 ves' disease and the inability to cure toxic nodular hyperthyroidism with antithyroid drugs alone, ra

124 Other important causes include toxic nodular hyperthyroidism, due to the presence of one or m

125 y, becoming nodular if small (82%) or nearly nodular if larger (18%).

126 ike lesions enhanced centripetally, becoming nodular if small (82%) or nearly nodular if larger (18%)

127 Pulmonary nodules and nodular infiltrates occur frequently during treatment of

128 nsolidation, tree-in-bud pattern, upper lobe nodular infiltration and cavitation.

129 aled that cavity, tree-in-bud and upper lobe nodular infiltration has significant association with sm

130 lymphadenopathy, consolidation, collapse and nodular infiltration in the upper lobe.

131 Cavity, tree- in-bud pattern and upper lobe nodular infiltration were highly associated with smear p

132 ologic findings of 6 patients with CVID with nodular/infiltrative lung disease who had biopsy specime

133 nd neck (29.4% vs 8.7%; P < .001) and of the nodular, lentigo maligna, or acral lentiginous histologi

134 Additionally, we demonstrated nodular leptomeningeal enhancement in 32.3% of participa

135 We hypothesized that nodular lesions abolish otolith-perceptual integration,

136 All initially nodular lesions enhanced centrifugally, whereas initiall

137 A clinical examination revealed nodular lesions in the ears and a lump in the subcutaneo

138 considered in the differential diagnosis of nodular lesions or sinus tracts present in the axillae,

139 at of the initial therapy; visible recurrent nodular lesions require endoscopic resection, whereas fl

140 Nodular lesions were present in 3 patients.

141 of the three cases had unilateral, multiple nodular lesions with smooth borders accompanied by a hyd

142 In a multivariable model, parasite load, nodular lesions, and positive skin microscopy were signi

143 nimals infected by 10(4) parasites presented nodular lesions, while those infected with 10(6) parasit

144 Twenty-five patients had benign nodular lesions.

145 rular lesions that include mesangiolysis and nodular lesions.

146 t MPNSTs purely occur multifocally as mostly nodular lesions.

147 pattern of BA, BASM, presence of ascites and nodular liver appearance at KPE, and early postoperative

148 ), ascites > 20 mL (HR: = 1.90, P = 0.0230), nodular liver appearance compared to firm (HR: = 1.61, P

149 haracterized by the emergence of spontaneous nodular liver lesions in approximately 50% of male mice

150 All 34 radiographs showed a nodular lung cancer that was apparent in retrospect but

151 Nodular lymphocyte Hodgkin lymphoma (NLPHL) is a rare di

152 Nodular lymphocyte predominant Hodgkin lymphoma (NLPHL)

153 Nodular lymphocyte predominant Hodgkin lymphoma (NLPHL)

154 dominant (LP) cells from tissues involved by nodular lymphocyte predominant Hodgkin lymphoma (NLPHL).

155 which account for the majority of cases, or nodular lymphocyte-predominant HL.

156 The optimal treatment of stage IA nodular lymphocyte-predominant Hodgkin lymphoma (NLPHL)

157 sal expression of CD20 by malignant cells in nodular lymphocyte-predominant Hodgkin lymphoma (NLPHL)

158 Nodular lymphocyte-predominant Hodgkin lymphoma (NLPHL)

159 Nodular lymphocyte-predominant Hodgkin lymphoma (NLPHL)

160 res and clinical data from 423 patients with nodular lymphocyte-predominant Hodgkin lymphoma (NLPHL)

161 Nodular lymphocyte-predominant Hodgkin lymphoma (NLPHL)

162 Nodular lymphocyte-predominant Hodgkin lymphoma (NLPHL)

163 The optimal treatment of newly diagnosed nodular lymphocyte-predominant Hodgkin lymphoma (NLPHL)

164 Nodular lymphocyte-predominant Hodgkin lymphoma (NLPHL)

165 number of reports have shown a propensity of nodular lymphocyte-predominant Hodgkin lymphoma (NLPHL)

166 and outcome for patients with advanced-stage nodular lymphocyte-predominant Hodgkin lymphoma (NLPHL).

167 Neoplastic cells of nodular lymphocyte-predominant Hodgkin lymphoma were neg

168 before treatment initiation owing to having nodular, lymphocyte-predominant Hodgkin's lymphoma and t

169 h sinopulmonary infections, lymphadenopathy, nodular lymphoid hyperplasia and viremia due to cytomega

170 A model that resembles nodular MAC disease was established in C57 (bg+/bg+) mic

171 s in vivo, including solid acinar, and solid nodular malignancies as well as cystic hyperplasia.

172 onography (EUS) that showed a 3- x 2-cm flat nodular mass with an 8-mm ulcer in the angularis.

173 pear as a rapidly growing cervical, painless nodular mass.

174 nd routine chest roentgenogram shows pleural nodular masses.

175 taneous mastocytosis/plaque mastocytosis, 22 nodular mastocytosis, and nine diffuse cutaneous mastocy

176 present in regions of decreased apoptosis in nodular medulloblastoma.

177 tic differentiation similar to that in human nodular medulloblastomas with activated Shh signaling.

178 ive nodularity, four of 20 with desmoplastic/nodular medulloblastomas, and one of 108 with other subt

179 Superficial spreading melanoma (SSM) and nodular melanoma (NM) are believed to represent sequenti

180 Nodular melanoma (NM) is a rapidly progressing potential

181 es, superficial spreading melanoma (SSM) and nodular melanoma (NM).

182 entional melanoma, superficial spreading and nodular melanoma account for most cases, with risk facto

183 ole for activated RSK1 in the progression of nodular melanoma and suggest that melanoma originating f

184 cal vigilance is warranted for patients with nodular melanoma and those with the thickest tumors.

185 Nodular melanoma cells were more sensitive to RSK1 inhib

186 Nodular melanoma occurred more frequently as amelanotic/

187 An association was seen with the nodular melanoma subtype (vs superficial spreading [refe

188 stologic subtypes, superficial spreading and nodular melanomas, differ in their speed of dermal invas

189 The nodular mesenchyme expresses the related bHLH factors Ha

190 n (P = .026), depressed areas (P < .001), or nodular mixed type (P < .001) affected accuracy of ident

191 oth 89% (95% CI, 67 to 100) for desmoplastic/nodular (n = 11), 61% (95% CI, 51 to 71) and 75% (95% CI

192 odular benign melanocytic tumors, and 135 of nodular nonmelanocytic tumors were scored for dermoscopy

193 tive correlating features of pigmented NM vs nodular nonmelanoma were peripheral black dots/globules,

194 and metastatic brain tumours, classified as nodular on the basis of their growth pattern, exert soli

195 ass opacity, consolidation, air bronchogram, nodular opacities and pleural effusion.

196 of the chest revealed multiple thick-walled nodular opacities throughout both lungs.

197 Nodular opacities, reticular opacities, pleural effusion

198 The MRI growth patterns were classified as nodular or diffuse.

199 if they had histologically confirmed primary nodular or superficial basal-cell carcinoma at low-risk

200 : thyroiditis (OR = 0.58, p = 1.4 x 10(-5)), nodular (OR = 0.76, p = 3.1 x 10(-5)) and multinodular (

201 daily for 6 weeks (superficial) or 12 weeks (nodular), or surgical excision with a 4 mm margin.

202 DPO was defined as 10 or more bilateral nodular ossifications (definition 1) or as one or more l

203 ne or more lobes with five or more bilateral nodular ossifications (definition 2).

204 PNSTs purely occurred multifocally as mostly nodular (p < 0.001), multilobulated, or ovoid lesions.

205 e describe P aeruginosa-induced locoregional nodular panniculitis as a distinct entity.

206 3 new cases of P aeruginosa-induced multiple nodular panniculitis without septicemia and describe com

207 nas aeruginosa-induced locoregional multiple nodular panniculitis without septicemia is an underrepor

208 classes of anthelminthic drugs in the swine nodular parasite Oesophagostomum dentatum.

209 ate was 95%, with complete remission in 72%, nodular partial remission in 10%, partial remission due

210 7%) achieved a complete response (CR), one a nodular partial remission, and 10 patients a partial rem

211 6%, including 12% complete responses and 12% nodular partial remissions.

212 ients who achieved complete response (CR) or nodular partial response (nPR).

213 a complete response, one patient (3%) had a nodular partial response, and 17 (55%) patients had a pa

214 omplete response with residual cytopenia, 7% nodular partial response, and 43% partial response.

215 e response (CR) rate, defined as complete or nodular partial response, was significantly greater with

216 26 (41%) complete responses (CRs), 14 (22%) nodular partial responses (nodular PRs), and 18 (28%) pa

217 %, with 20% complete responses (CRs) and 20% nodular partial responses.

218 ancement, and two lesions showed a prevalent nodular pattern.

219 ic gave a high sensitivity (>98.0%) for both nodular pigmented and nonnodular pigmented melanoma but

220 Compared to VL, nodular PKDL was more likely and macular PKDL less likel

221 opsies from 3 patients with maculopapular or nodular post-kala-azar dermal leishmaniasis (PKDL).

222 ieved in 70%, partial remission (PR) in 18%, nodular PR in 3%, for an overall response of 92%.

223 melanoma and that Tiam1 may activate Rac in nodular presentations.

224 uding 30 partial responses (PRs; 47%), three nodular PRs (5%), and one complete response (1.6%).

225 s (CRs), 14 (22%) nodular partial responses (nodular PRs), and 18 (28%) partial responses (PRs).

226 Repeated episodes can result in nodular pulmonary infiltrates and suspected nonspecific

227 flammation to extreme hepatocellular injury, nodular regeneration, and bile duct proliferation.

228 use has been hampered due to concerns about nodular regenerative hyperplasia (NRH) of the liver.

229 Nodular regenerative hyperplasia (NRH) was seen in 9 pat

230 lar remodeling; Notch1 knockout mice develop nodular regenerative hyperplasia (NRH).

231 tion and tetralogy of Fallot associated with nodular regenerative hyperplasia and focal nodular hyper

232 is, liver fibrosis, portal hypertension, and nodular regenerative hyperplasia are discussed in this r

233 in 6 patients, there were varying findings: nodular regenerative hyperplasia, steatohepatitis, hemos

234 stopathological analysis of liver revealed a nodular regenerative hyperplasia.

235 rtension due to periportal liver fibrosis or nodular regenerative hyperplasia.

236 One CFLD patient had nodular regenerative hyperplasia.

237 ng along the sinusoids in PIR-B(-/-) mice vs nodular restricted localization in WT mice.

238 le without underlying disease presented with nodular scleritis and keratouveitis with multiple radial

239 Ds was associated with idiopathic diffuse or nodular scleritis with a high degree of scleral inflamma

240 Patients with idiopathic diffuse or nodular scleritis with a high degree of scleral inflamma

241 Ds was associated with idiopathic diffuse or nodular scleritis with a low degree of scleral inflammat

242 Patients with idiopathic diffuse or nodular scleritis with a low degree of scleral inflammat

243 ment with IMT was associated with diffuse or nodular scleritis with associated systemic disease (OR =

244 ent with BRMs was associated with diffuse or nodular scleritis with associated systemic disease (OR =

245 Patients with diffuse or nodular scleritis with associated systemic disease may r

246 9, P < 0.001) and with idiopathic diffuse or nodular scleritis without ocular complications (OR = 3.1

247 nce decreased with declining CD4 counts, but nodular sclerosing decreased more precipitously than mix

248 failure, and kidney biopsy analysis showed a nodular sclerosing GN with extensive focal global glomer

249 histological examination diagnosed Hodgkin's nodular sclerosing histological subtype disease has been

250 With more severe immunosuppression, nodular sclerosing HL becomes infrequent, explaining the

251 ligand/9p24.1 amplification is restricted to nodular sclerosing HL, the cHL subtype most closely rela

252 Nodular sclerosing Hodgkin lymphoma (NSHL) is a distinct

253 LBCL and, in fact, closely resemble those of nodular sclerosing Hodgkin lymphoma (NSHL).

254 e-B-cell lymphoma that is closely related to nodular sclerosing Hodgkin's lymphoma.

255 lastic syndrome caused by stage 2A, grade I, nodular sclerosing Hodgkin's lymphoma.

256 better outcome compared with the more common nodular-sclerosing type.

257 -rich (81-fold; 95% CI, 30- to 177-fold) and nodular sclerosis (4.6-fold; 95% CI, 2.9- to 7.0-fold) a

258 low stage (stage I/II) at presentation with nodular sclerosis (NS) histology predominating in 80% of

259 Similar associations were found for nodular sclerosis and mixed cellularity subtypes.

260 Nodular sclerosis and mixed-cellularity subtypes had sim

261 Bulk disease, "B" symptoms, and nodular sclerosis histology were risk factors for inferi

262 thyroidism (thyroid radiation dose, sex, and nodular sclerosis histology), the risk of hypothyroidism

263 Nodular sclerosis Hodgkin lymphoma (NSHL) and primary me

264 loci within the HLA region are observed for nodular sclerosis Hodgkin lymphoma (rs9269081, HLA-DPB1*

265 B2M-deficient cases encompassed most of the nodular sclerosis subtype cases and only a minority of m

266 on into one of the four types of classic HL (nodular sclerosis, mixed cellularity, lymphocyte-deplete

267 ent a discovered phenomenon of titanium nano-nodular self-assembly that occurs during physical vapor

268 The discovered titanium nano-nodular self-structuring has been proven feasible on bio

269 nce of signs favoring PLC on HRCT (smooth or nodular septal lines, subpleural nodularity, peribroncho

270 ns), eczematous, urticarial, papular, and/or nodular skin lesions were seen.

271 chronic pruritic skin disease with multiple nodular skin lesions.

272 from eczematous or urticarial to papular or nodular skin lesions.

273 scence, and eyes with superficial ODD showed nodular staining.

274 PAVICs grown in OST+TGF-beta1 produced nodular structures staining positive for calcium content

275 r), stiffness, and formation of macroscopic, nodular structures with calcification in the VIC-laden h

276 Analysis showed that the desmoplastic/nodular subtype was a favorable factor in predicting sur

277 derly were more common in men, mostly of the nodular subtype, and located in the head and neck region

278 ay N-fixation deficiency (25%) and increased nodular superoxide content.

279 nt IV (27 [64%] of 42 vs 10 [24%] of 42) and nodular surface (37 [88%] of 42 vs seven [17%] of 42) we

280 caused by C. burnetii: vegetation, valvular nodular thickening, rupture of chorda tendinae, and valv

281 Formation and remodeling of the nodular thickenings at the closure points of the leaflet

282 ludes condensation, elongation, formation of nodular thickenings, and remodeling of tension-resistant

283 e able to reduce the number of patients with nodular thyroid disease undergoing this invasive procedu

284 apping reveals abnormal fiber projections in nodular tissue suggestive of abnormal organization of wh

285 to the presence of abnormal fiber tracks in nodular tissue.

286 red the pattern of pulmonary metastases from nodular to diffuse and facilitated disease progression.

287 (desmoplastic/nodular versus nondesmoplastic/nodular) to stratify patients for therapy by risk of rel

288 with well-defined tumor margins (P = .013), nodular tumor enhancement (P = .021), and gross appearan

289 Acral lentiginous and nodular tumors, male sex, tumor site on the scalp or nec

290 le tumors developed into more advanced multi-nodular tumors, whereas the female tumors remain uniform

291 lloblastomas (23 classic, eight desmoplastic-nodular, two large cell, one anaplastic), 17 ependymomas

292 chioradial pruritus), and chronic prurigo of nodular type, the latter as a model for chronic scratchi

293 All other HCCs were of a nodular type, with similar nodule sizes in the two group

294 endothelial transmigration, MMP16 supported nodular-type growth of adhesive collagen-surrounded mela

295 ological subtypes, including superficial and nodular variants, raising the possibility that morpholog

296 ill use histopathologic typing (desmoplastic/nodular versus nondesmoplastic/nodular) to stratify pati

297 Differences among patients with nodular versus smooth liver surfaces in the proportion w

298 and lentiviral overexpression of Cic in the nodular zone accelerated both aberrant Purkinje neuron s

299 nd to degenerate earlier than those from the nodular zone, and this early degeneration was associated

300 I-VII (central zone, CZ) and posterior IX-X (nodular zone, NZ).