ライフサイエンスコーパス: nodular sclerosis

1 In all cases of nodular sclerosis (132), mixed cellularity (34), lymphoc

2 iatric cases (two lymphocyte-predominant, 25 nodular sclerosis, 16 mixed cellularity, 5 lymphocyte de

3 -rich (81-fold; 95% CI, 30- to 177-fold) and nodular sclerosis (4.6-fold; 95% CI, 2.9- to 7.0-fold) a

4 RS cells expressed BCL-2 in 359 (65%) of 551 nodular sclerosis, 67 (47%) of 143 mixed cellularity, an

5 vealed that the majority of the patients had nodular sclerosis (80.7%) and were classified as stage I

6 rall, 6 (3 T and 3 null) of 49 ALCL and 3 (2 nodular sclerosis and 1 mixed cellularity) of 72 HD show

7 In glomeruli where advanced nodular sclerosis and global sclerosis were dominant, IL

8 These associations were significant for the nodular sclerosis and mixed cellularity subtypes but wer

9 Similar associations were found for nodular sclerosis and mixed cellularity subtypes.

10 Nodular sclerosis and mixed-cellularity subtypes had sim

11 Risk was decreased for both nodular sclerosis and other subtypes.

12 tinct molecular signature that overlaps with nodular sclerosis classic Hodgkin lymphoma (cHL).

13 phocyte-rich/mixed cell/lymphocyte depleted, nodular sclerosis, classical HL, not otherwise specified

14 hat HLA class II loci predispose to sporadic nodular sclerosis HD (NSHD).

15 Bulk disease, "B" symptoms, and nodular sclerosis histology were risk factors for inferi

16 thyroidism (thyroid radiation dose, sex, and nodular sclerosis histology), the risk of hypothyroidism

17 logical subtypes (odds ratio [OR] for MC vs. nodular sclerosis histology, 3.2; 95% confidence interva

18 Nodular sclerosis Hodgkin lymphoma (NSHL) and primary me

19 loci within the HLA region are observed for nodular sclerosis Hodgkin lymphoma (rs9269081, HLA-DPB1*

20 th Reed-Sternberg cells in 30 of 35 cases of nodular-sclerosis, lymphocyte-depletion, and mixed-cellu

21 on into one of the four types of classic HL (nodular sclerosis, mixed cellularity, lymphocyte-deplete

22 Histologic distribution was as follows: nodular sclerosis (n = 64), lymphocyte predominant (n =

23 pecific mortality but only for patients with nodular sclerosis (NS) histologic subtype (hazard ratio

24 low stage (stage I/II) at presentation with nodular sclerosis (NS) histology predominating in 80% of

25 ession of eotaxin and MDC was highest in the nodular sclerosis (NS) subtype.

26 B2M-deficient cases encompassed most of the nodular sclerosis subtype cases and only a minority of m

27 ologic features, it clinically resembles the nodular sclerosis subtype of classical Hodgkin lymphoma

28 mainly confined to the mixed cellularity and nodular sclerosis subtypes.

29 wo of three lymphocyte-predominant, 26 of 39 nodular, sclerosis, two of two mixed cellularity, and tw

30 ssumed dendritic shapes, particularly in the nodular sclerosis type, producing an interdigitating mes

31 astic cells of Hodgkin's disease, notably in nodular sclerosis type.

32 the development cohort was 33 (18-65) years; nodular sclerosis was the most common histology.