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1 d independently reviewed for hepatic surface nodularity.
2 eedle biopsy if required to evaluate thyroid nodularity.
3 ith 13 (34%) of these patients having antral nodularity.
4 s between PNPLA3 rs2281135 variant and tumor nodularity.
5 (SN), lobar redistribution, and hepatic vein nodularity.
6 th International Labour Office (ILO) grade 3 nodularity, 112 patients with less severe silicosis (ILO
7 ging included multiloculation (56.9%), mural nodularity (16.5%), and biliary ductal dilatation (17.7%
8 more commonly observed in malignant lesions (nodularity: 42.9% vs 10.0%; difference, 32.9%; OR, 6.72;
9 ial (RPE) atrophy (43%), RPE loss (14%), RPE nodularity (8%), photoreceptor loss (43%), inner segment
10 Multivariate analysis demonstrated mural nodularity and atypical cytopathology were predictive of
13 spectively scored qualitative (reticulation, nodularity, and total scores) and quantitative (contrast
15 patients (43%) demonstrated hepatic surface nodularity at pretransplant imaging, none of whom had ci
18 oved with the addition of a ML parameter for nodularity, Enhanced Liver Fibrosis, platelets, aspartat
19 ividuals with medulloblastoma with extensive nodularity, four of 20 with desmoplastic/nodular medullo
21 liver surface, patients with hepatic surface nodularity had a significantly greater proportion with t
22 t, some PNPLA3 variants correlate with tumor nodularity, higher miR-122 expression, and distinct demo
26 s, including quantification of liver surface nodularity (LSN) for PSVD diagnosis has not been establi
27 quantitative method to measure liver surface nodularity (LSN) from routine computed tomographic (CT)
28 o determine whether use of the liver surface nodularity (LSN) score, a quantitative biomarker derived
29 toma (ND) and medulloblastoma with extensive nodularity (MBEN) have been associated with a more favor
30 toma (DMB) or medulloblastoma with extensive nodularity (MBEN; n = 42) had 93% progression-free survi
32 ral valve) manifested predominantly (74%) as nodularities of the aortic cusps and basal thickening of
33 ents with lung cancer, smooth enlargement or nodularity of the adrenal glands at baseline CT is not a
34 5.36; 95% confidence interval, 1.84-15.56), nodularity on endoscopy (relative risk, 3.98; 95% confid
35 uct IPMN and for branch-duct IPMN with mural nodularity or positive cytology irrespective of location
36 r developed irregular wall thickening, mural nodularity, or septations that would raise concern for m
38 scopic abnormalities, that is, ulceration or nodularity (P = 0.0002; relative risk [RR] = 7.6; 95% co
39 e, location, septation, calcification, mural nodularity, pancreatic duct involvement, and presence of
40 (smooth or nodular septal lines, subpleural nodularity, peribronchovascular thickening, satellite no
42 , and liver ultrasound parameters of surface nodularity (SN), lobar redistribution, and hepatic vein
43 ch SNP with tumor prognostic factors such as nodularity, tumor size and AFP (alpha-feto protein) leve
48 iation between pleural thickness and pleural nodularity with histopathological examination findings w