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1 echanism that may contribute directly to the nonadrenergic component of postvagal tachycardia.
2 udies were designed to determine whether the nonadrenergic inotropic and vasodilator drug amrinone, w
3 epinephrine released from these neurons, and nonadrenergic neurons, in which hetero-a(2a)ARs respond
4 or inhibitory neurotransmitter that mediates nonadrenergic noncholinergic (NANC) signaling.
5 ads to a significant decrease in an airway's nonadrenergic noncholinergic inhibitory (NANCi) response
6                                              Nonadrenergic noncholinergic relaxation and cyclic guano
7 bitors and HO inhibitors partially inhibited nonadrenergic noncholinergic relaxation.
8                                 Responses to nonadrenergic, noncholinergic (NANC) nerve stimulation b
9 he exact role of carbon monoxide (CO) in the nonadrenergic, noncholinergic (NANC) relaxation is not k
10                        Likewise, CO-mediated nonadrenergic, noncholinergic (NANC) relaxation of the i
11                          We investigated the nonadrenergic, noncholinergic (NANC) relaxation, nitric
12          To test this hypothesis, we studied nonadrenergic, noncholinergic (NANC) relaxation, NOS act
13  to electrical field stimulation (EFS) under nonadrenergic, noncholinergic conditions.
14 sphincter (IAS) are controlled by inhibitory nonadrenergic, noncholinergic innervation (i-NANC).
15 ha 1 levels during development act through a nonadrenergic, noncholinergic mechanism.
16 tensions in response to different agents and nonadrenergic, noncholinergic nerve stimulation by elect
17                         Maximum NO-dependent nonadrenergic, noncholinergic nerve-mediated relaxations
18 ia adrenergic mechanisms and could occur via nonadrenergic, noncholinergic nitrergic neural and/or en
19          The hypothesis that cholinergic and nonadrenergic, noncholinergic parasympathetic nerves inn
20 hanisms of action, and indicating that other nonadrenergic, noncholinergic relaxant neurotransmitters
21  demonstrate defects in gastric emptying and nonadrenergic, noncholinergic relaxation of pyloric musc
22 ation of hexamethonium significantly reduced nonadrenergic, noncholinergic relaxation, the catalytic
23 thmatic patients may reflect upregulation of nonadrenergic, noncholinergic, NO-producing nerves in ai
24 duced by neuronal NO synthase (nNOS) mediate nonadrenergic/noncholinergic (NANC) intestinal relaxatio
25  Calcitonin gene-related peptide (CGRP) is a nonadrenergic/noncholinergic (NANC) peptide with vasodil
26 -sensitive cardiac contractile modulation by nonadrenergic/noncholinergic peptide signaling.
27 trations, the P2X1 antagonists abolished the nonadrenergic, purinergic component of neurogenic contra
28 thetic innervation undergoes a switch from a nonadrenergic to cholinergic and peptidergic phenotype.
29 utcome as compared with other adrenergic and nonadrenergic vasoactive drugs.
30           However, little is known regarding nonadrenergic vasoconstriction during exercise.
31      Accordingly, we compared the effects of nonadrenergic vasopressin with those of epinephrine on c
32 s compared with the more prolonged effect of nonadrenergic vasopressin.