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1 e TS (+0.19 +/- 0.08%) compared with that in nonatherosclerotic (-0.10 +/- 0.13%) and control (-0.09
2 neous coronary artery dissection (SCAD) is a nonatherosclerotic acute coronary syndrome for which opt
3 idase proteins (gp91phox, Nox1, and Nox4) in nonatherosclerotic and atherosclerotic human coronary ar
4 nal antiserum was performed on a total of 83 nonatherosclerotic and atherosclerotic human coronary ar
5 ined ACE expression immunohistochemically in nonatherosclerotic and diseased human coronary arteries.
6 diagnosis and management of a patient with a nonatherosclerotic and noncardioembolic arterial event,
7 43+/-0.02 at induction versus 0.35+/-0.02 in nonatherosclerotic animals (P=0.031) and no further incr
8 andardized uptake value mean, 0.10+/-0.01 in nonatherosclerotic animals versus 0.20+/-0.03 [P=0.002]
9           In contrast to previous studies of nonatherosclerotic animals, the results indicate that in
10 e marrow-derived progenitor cells from young nonatherosclerotic ApoE-/- mice prevents atherosclerosis
11     Fibromuscular dysplasia (FMD) is a rare, nonatherosclerotic arterial disease for which the molecu
12 or basal NFkappaB activity in SMC in vivo as nonatherosclerotic arteries did not contain nuclear p65
13 ested the hypothesis that human atheroma and nonatherosclerotic arteries express the two Cox isoforms
14                                           In nonatherosclerotic arteries, ACE immunoreactivity was fo
15 rarchical clustering of genes and miRs among nonatherosclerotic arteries, stable, and unstable plaque
16 ntimal expansion has been reduced in vivo in nonatherosclerotic balloon-injured arteries by transfer
17 ts, n = 4), whereas it only infected 2 of 14 nonatherosclerotic blood vessel explants (SV, n = 10; IM
18 e cells residing in both atherosclerotic and nonatherosclerotic blood vessels were free of active CMV
19 sease, and abdominal aortic aneurysm and (2) nonatherosclerotic cardiovascular disease phenotypes, st
20 eparately modify risk of atherosclerotic and nonatherosclerotic cardiovascular outcomes.
21 section (SCAD) is an increasingly recognized nonatherosclerotic cause of acute coronary syndrome.
22 section (SCAD) is an increasingly recognized nonatherosclerotic cause of acute myocardial infarction
23 s from injured atherosclerotic and uninjured nonatherosclerotic common femoral arteries (CFA) in chol
24 ther aberrant angiogenesis also occurs under nonatherosclerotic conditions, we performed femoral arte
25  transcript in carotid plaques compared with nonatherosclerotic control arteries, which was confirmed
26 position was seen in aortas of the wild-type nonatherosclerotic control mice.
27 as stable lesions showed only the precursor; nonatherosclerotic control segments were negative for bo
28 ild-type C57BL/6 mice served as sham-treated nonatherosclerotic controls.
29 uptake in atherosclerotic animals but not in nonatherosclerotic controls.
30 t or with premounted stents were inflated in nonatherosclerotic coronary arteries of either young dom
31   Minimal extracellular PLTP was detected in nonatherosclerotic coronary arteries, but extracellular
32                                           In nonatherosclerotic coronary arteries, superoxide was pre
33                                              Nonatherosclerotic coronary artery ring segments were un
34  of specific binding of [3H]-leukotrienes to nonatherosclerotic coronary artery.
35               In contrast, only 1 (4%) of 24 nonatherosclerotic coronary specimens showed any evidenc
36 the expression of 11 000 murine genes in the nonatherosclerotic descending aorta by Affymetrix gene c
37  strong c-FLIP immunoreactivity in SMCs from nonatherosclerotic diffuse intimal thickening and in the
38 t not all, prospective studies report higher nonatherosclerotic disease rates in people with low seru
39 ether low cholesterol is causally related to nonatherosclerotic disease risk.
40 ibromuscular dysplasia is a noninflammatory, nonatherosclerotic disease that has been reported in alm
41 bserved associations of low cholesterol with nonatherosclerotic disease.
42 binding activity in both atherosclerotic and nonatherosclerotic human coronary arteries.
43 is monoclonal expansion can also be found in nonatherosclerotic intima and media.
44                                              Nonatherosclerotic intima contained extensive deposits o
45 chemic stroke, including atherosclerotic and nonatherosclerotic ischemic stroke subtypes.
46                                   The common nonatherosclerotic, large-vessel arteriopathies affectin
47 eriopathies in the context of other systemic nonatherosclerotic, large-vessel vascular disease (e.g.
48                                Compared with nonatherosclerotic-MSCs, atherosclerotic-MSCs displayed
49 as performed on atherosclerotic (n = 87) and nonatherosclerotic (n = 51) coronary arterial segments f
50 ents (75%) due to atherosclerotic (n=12) and nonatherosclerotic (n=3) causes.
51 ere isolated from atherosclerotic (n=38) and nonatherosclerotic (n=42) donors.
52  Arterial stiffness leads to LV mass through nonatherosclerotic pathways in mice.
53 gh ABI might lead to greater LV mass through nonatherosclerotic pathways.
54 dependent and -independent vasodilation in a nonatherosclerotic peripheral conduit artery of 26 elder
55 thelial regeneration after balloon injury in nonatherosclerotic rabbits.
56                                 In VSMC from nonatherosclerotic regions, RT-PCR analysis revealed an
57                                  We analyzed nonatherosclerotic repair arteries gathered at coronary
58 s the intimal response to arterial injury in nonatherosclerotic rodents and rabbits and inhibits the
59                                              Nonatherosclerotic samples contained neither HSP.
60                                              Nonatherosclerotic SCAD patients were prospectively foll
61 ective, observational study of patients with nonatherosclerotic SCAD presenting acutely from 22 North
62              Thromboangiitis obliterans is a nonatherosclerotic segmental inflammatory disease that a
63 -rich atherosclerotic lesions and 26 normal, nonatherosclerotic segments) were exposed to heparinized
64     In contrast, GPI-PLD was not detected in nonatherosclerotic segments.
65  segments of human arteries as compared with nonatherosclerotic segments.
66                                              Nonatherosclerotic spontaneous coronary artery dissectio
67 untries have long revealed a predominance of nonatherosclerotic stroke, hypertensive heart disease, n
68 iovascular Data Registry enhanced pediatric, nonatherosclerotic structural heart disease and congenit
69 solate actively infected none of six sets of nonatherosclerotic SV explants at 5 to 7 days after inoc
70 nted in 2 or 3 main coronary arteries of 136 nonatherosclerotic swine and examined by light microscop
71  14) had 5-fold higher levels of TIMP-3 than nonatherosclerotic tissue (n= 10).
72 as detected in the endothelial cell layer of nonatherosclerotic vessels but was absent from the endot
73  reduced in human plaques when compared with nonatherosclerotic vessels.