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1                           Anti-Sm B cells in nonautoimmune 2-12H-transgenic (Tg) mice are functional,
2                                       In the nonautoimmune 3H9H/56R BALB/c, most B cells generated do
3 lenic and peritoneal anti-Sm B cells of both nonautoimmune and autoimmune genetic backgrounds indicat
4 sm with lower doses of donor BM cells in old nonautoimmune and autoimmune lupus mice.
5 compare the regulation of anti-Sm B cells in nonautoimmune and autoimmune MRL/Mp-lpr/lpr (MRL/lpr) an
6 crimal tissue, or aqueous tear deficiency in nonautoimmune animals and humans.
7 mice can stimulate autoantibody synthesis in nonautoimmune anti-snRNP Tg mice.
8  backcross mice, inheritance of H2b from the nonautoimmune B10 strain was strongly linked with both a
9 , and NZB/W F(1) mouse strains compared with nonautoimmune B10.BR mice.
10 eleted as efficiently as in Dbl-Tg mice of a nonautoimmune B10.D2 genetic background.
11 n vivo, transgenic mice were outcrossed onto nonautoimmune B6 and SLE-prone MRL backgrounds and expos
12 c modifier of the CD45 E613R mutation on the nonautoimmune B6 background to understand how complex su
13 gent anti-CD45RB induces HSC mobilization in nonautoimmune B6 mice but not in diabetes-prone NOD mice
14 nd anti-single-stranded (ss)DNA B cells in a nonautoimmune background by generating mice carrying imm
15  chain knockin (3H9.KI) transgenic mice on a nonautoimmune background resulted in specific activation
16                       Taken together, on the nonautoimmune background, Sle3/5 affected V(H)DJ(H) junc
17 omoter induces nondestructive insulitis on a nonautoimmune background.
18 milar to other CfH-deficient mouse models on nonautoimmune backgrounds, immunofluorescence staining s
19 Fas (Fas(lpr)), on both autoimmune (MRL) and nonautoimmune backgrounds.
20                              Immunization of nonautoimmune BALB/c mice with multimeric DWEYSVWLSN, a
21 Anti-dsDNA B cells are actively tolerized in nonautoimmune BALB/c mice, as manifested by their develo
22 een used to track anti-dsDNA B cells in both nonautoimmune BALB/c mice, in which autoreactive B cells
23 mic lupus erythematosus-like syndrome in the nonautoimmune BALB/c mouse strain.
24  99D.7E, an IgG2b monoclonal antibody from a nonautoimmune BALB/c mouse that is cross-reactive with b
25 from the lupus-prone MRL-lpr/lpr mouse and a nonautoimmune BALB/c mouse with the pathogenic anti-DNA
26    In this backcross, contributions from the nonautoimmune BALB/c strain were also apparent.
27 mmune (MRL+/+ and lpr/lpr) mice with that in nonautoimmune (BALB/c) mice.
28 at are not seen in similar preparations from nonautoimmune C3H animals.
29 pr/lpr mice compared with MRL-fas/fas and/or nonautoimmune C3H/Hej and BALB/c mice.
30                   In heterozygous mice, on a nonautoimmune C57BL/6 background, the transgene allotype
31 pared with the Th2-type response observed in nonautoimmune C57BL/6 mice.
32 ted to the heavy and light chain loci of the nonautoimmune C57BL/6 mouse strain.
33 requency in the periphery of NOD mice versus nonautoimmune C57BL/6 VH125Tg mice; however, the ontogen
34    Dissimilar to reports of Bcl3 ablation in nonautoimmune C57BL/6-background mice, we found that spl
35 iagnosis and a deliberate attempt to exclude nonautoimmune causes of thrombocytopenia and secondary I
36 mice in comparison with age- and sex-matched nonautoimmune CD1 mice.
37 re significantly greater than those found in nonautoimmune CD40L-deficient animals.
38 venting islet allograft destruction in other nonautoimmune combinations often fail in NOD recipients.
39 Ag (BCMA) is a receptor for BAFF that, under nonautoimmune conditions, is important for sustaining en
40  cells have been found in organs affected by nonautoimmune conditions.
41 res of bone marrow derived from B6.NZMc1 and nonautoimmune congenic B6 mice carrying allelic T and B
42 lupus-prone MRL mice are more resistant than nonautoimmune control cells to anergy induction.
43 allele frequencies were compared to those in nonautoimmune controls.
44 treatment are no more lymphopenic than their nonautoimmune counterparts, but they show reduced thymop
45 turity-onset diabetes of the young (MODY) or nonautoimmune diabetes in mid-adult life, and to explore
46 e that PCBD1 mutations can cause early-onset nonautoimmune diabetes with features similar to dominant
47             Mutations in several genes cause nonautoimmune diabetes, but numerous patients still have
48 ly and genetically heterogeneous subgroup of nonautoimmune diabetes, constituting 1-2% of all diabete
49 group of diabetic patients with early-onset, nonautoimmune diabetes, who respond well to diabetes tre
50  three strains of mice that develop type II (nonautoimmune) diabetes.
51 yngeneic mouse islets were transplanted into nonautoimmune diabetic hosts and islet function was anal
52 nduction of mixed chimerism with MHC-matched nonautoimmune donor BM transplants did not prevent T1D i
53 ematopoietic cell transplantation (HCT) from nonautoimmune donors could prevent or reverse type 1 dia
54 nocytes or by transplantation of islets from nonautoimmune donors.
55 ands with unexplained MODY, 95 patients with nonautoimmune early-onset diabetes (diagnosed at <35 yea
56 ansplant (+5% [95% CI, 9%-2%]) per year, and nonautoimmune etiology of liver disease (OR, 1.0 for aut
57 odified 2-way avoidance task and compared to nonautoimmune female NZW mice.
58 -fold greater levels of serum IL-21 than the nonautoimmune group.
59 ed the extent of autoreactivity arising in a nonautoimmune host during the response to a foreign anti
60 rmline mutations have been found in familial nonautoimmune hyperthyroidism and sporadic congenital hy
61 g an intercross between MRL/lpr mice and the nonautoimmune inbred strain BALB/cJ.
62 e-stranded (ds)DNA antibodies (Abs), whereas nonautoimmune individuals have negligible levels of thes
63 unomodulatory effects, the high frequency of nonautoimmune inflammation suggests other mechanisms.
64 r results from both insulin resistance and a nonautoimmune insulin secretory defect.
65 ice deficient for both E2F1 and E2F2 develop nonautoimmune, insulin-dependent diabetes with high pene
66 ulthood, especially in males associated with nonautoimmune insulinopenia and reversed alphabeta cell
67 ith alpha 1-antitrypsin (AAT) in a syngeneic nonautoimmune islet graft model.
68  who had undergone liver transplantation for nonautoimmune liver disease at median age of 1.3 years w
69 sulin-dependent diabetes mellitus, caused by nonautoimmune loss of beta cells, and neurological dysfu
70 ration of ANA(+) IgG(+) plasma cells in both nonautoimmune mice and healthy human subjects.
71 Sm pre-PCs also occur at a high frequency in nonautoimmune mice and show additional phenotypic charac
72                                      Because nonautoimmune mice are not affected by raltegravir, we c
73 ke anti-dsDNA reactivity can be generated in nonautoimmune mice by immunization with a peptide antige
74 c graft-vs-host (cGVH) disease is induced in nonautoimmune mice by the transfer of alloreactive T cel
75                             It is induced in nonautoimmune mice by the transfer of alloreactive T cel
76    Pristane induces a lupus-like syndrome in nonautoimmune mice characterized by the development of g
77 2 mouse were injected intraperitoneally into nonautoimmune mice for evaluation of pathogenicity of ea
78 bitory T cells may play a role in protecting nonautoimmune mice from developing chronic autoimmunity.
79 emonstrated that anti-snRNP B cell anergy in nonautoimmune mice may be reversed by appropriate T cell
80                  Thus, inhibitory T cells in nonautoimmune mice may normally inhibit T-dependent acti
81                   Even without immunization, nonautoimmune mice possess T cells that can inhibit auto
82 wk-old female BALB/c mice (and several other nonautoimmune mice strains) with alpha-actinin in adjuva
83                  Intraperitoneal exposure of nonautoimmune mice to 2,6,10,14-tetramethylpentadecane (
84 lerance and induce a lupus-like phenotype in nonautoimmune mice transgenic for the heavy chain of a p
85 e representative mAbs to induce nephritis in nonautoimmune mice using established adoptive transfer p
86 , the abortive response can be reproduced in nonautoimmune mice when CD4+CD25+ T regulatory cells are
87  diffuse proliferative glomerulonephritis in nonautoimmune mice whereas another anti-ssDNA with appar
88                              Immunization of nonautoimmune mice with DNA-containing amyloid fibrils i
89 ogenic anti-DNA Ab, and that immunization of nonautoimmune mice with this peptide induces autoantibod
90 ossess 11-fold fewer Ly-49(+) CD8 Tregs than nonautoimmune mice, a deficiency that worsens as NOD mic
91               As conventional transgenics in nonautoimmune mice, anti-DNA B cells have been shown to
92                                           In nonautoimmune mice, B cells bearing immunoglobulin site-
93               In this study, we show that in nonautoimmune mice, dsDNA-specific autoreactive B cells
94         The breakdown of B cell tolerance in nonautoimmune mice, however, was short-lived: anti-DNA A
95                      However, in contrast to nonautoimmune mice, there are few if any peritoneal anti
96 actions between proteins from autoimmune and nonautoimmune mice, we demonstrate interactions between
97 e mechanisms involved in their regulation in nonautoimmune mice, we have used Ig transgenes (tgs).
98    To investigate B cell tolerance to GPI in nonautoimmune mice, we increased the GPI-reactive B cell
99 that sera from autoimmune mice, but not from nonautoimmune mice, were able to induce the proliferatio
100 poptotic thymocytes in MRL/MpJ compared with nonautoimmune mice.
101  analyzed single B cells from these and from nonautoimmune mice.
102 A B cells have been shown to be tolerized in nonautoimmune mice.
103 nal anti-Sm B cells in the spleen similar to nonautoimmune mice.
104 (reg) engraftment and therapeutic benefit in nonautoimmune models required ablative host conditioning
105 (PD-L1) immunotherapy in islet allografts in nonautoimmune models.
106 e rearranged V(H) domain of an IgM Ab from a nonautoimmune mouse immunized with a DNA-protein complex
107     To this end, we used the toxin-mediated, nonautoimmune MS animal model of cuprizone (Cup) intoxic
108 phased immunosuppression withdrawal (ISW) in nonautoimmune, nonviral adult liver transplant recipient
109 x NZW F1 (B/W) hybrid mice was compared with nonautoimmune NZW control mice on 3 anxiety tasks: the e
110 bone marrow from lupus patients but not from nonautoimmune patients undergoing myeloablation, where t
111                               Vaccination of nonautoimmune prone mice with syngeneic dendritic cells
112                  However, in contrast to the nonautoimmune-prone background, B cells recognizing solu
113                                       On the nonautoimmune-prone C57BL/6 genetic background, TSAd def
114  induce pathological autoimmunity we exposed nonautoimmune-prone galactose-alpha1-3-galactose-deficie
115 ese autoantibodies are normally regulated in nonautoimmune-prone mice but are induced to secrete Abs
116                                     NOD- and nonautoimmune-prone mice did not differ in ability to de
117                  It has been evident that in nonautoimmune-prone mice, dendritic cells (DCs) present
118 ent to generate classic lupus nephritis in a nonautoimmune-prone strain.
119 e to insulinopenia on alloimmunity using the nonautoimmune, spontaneously diabetic H-2(b)-expressing
120                   Costimulation delivered by nonautoimmune strain APCs normalized NOD CD4 T cell divi
121 eptide Ag in lupus-prone MRL/MpJ mice with a nonautoimmune strain using an MHC class II-restricted TC
122 n of the lupus-susceptibility gene from this nonautoimmune strain.
123 pe 1 diabetes-prone NOD strain relative to a nonautoimmune strain.
124 iscent of that of MRL mice can be induced in nonautoimmune strains by pristane, which causes a nonspe
125 B cell tolerance mechanisms in NOD mice with nonautoimmune strains by using the IgHEL and Ig3-83 tran
126                       Strikingly, none of 13 nonautoimmune strains tested exhibited this defect.
127 KT cells in young BWF1 mice as compared with nonautoimmune strains, implies a regulatory role of CD1d
128  II major histocompatibility complex-matched nonautoimmune strains, including BALB/c, (BALB/cxNZW)F1
129 When compared with homologous IgL genes from nonautoimmune strains, Vkappa genes from NOD mice are po
130 e lower in young wild-type BWF1 mice than in nonautoimmune strains.
131 reas, we find that NOD mice closely resemble nonautoimmune strains.
132         In contrast, autoreactive T cells of nonautoimmune Tg mice are tolerized by Tg B cells in the
133  individuals (2.1%, P < .001), patients with nonautoimmune thrombosis (0%, P = .017), or patients wit
134 sus without thrombosis, 62 with APS, 21 with nonautoimmune thrombosis, and 145 healthy individuals) w
135 pecific B cells are rare in the periphery of nonautoimmune VH125Tg/C57BL/6 mice and WT/NOD autoimmune
136 e, but not young mice from these strains, or nonautoimmune young and old BALB/c, MRL/MpJ/++, and C3H/

 
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