戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 eased proopiomelanocortin mRNA in cells from noninflamed and inflamed LN.
2                  Human gingival tissues from noninflamed and inflamed sites were also analyzed by RT-
3 tive expression of hBD-1 mRNA was similar in noninflamed and inflamed tissues obtained from each of f
4 c fusion protein administration does not, in noninflamed animals, change basal pain sensitivity nor t
5 lymphocytes already reside within the normal/noninflamed aorta before the onset atherosclerosis as a
6 he differences between inflamed and adjacent noninflamed areas in the rat model suggest a tissue grad
7 olonic mucosa revealed minimal expression in noninflamed areas, with striking upregulation under coli
8 for the recruitment of Th1 effector cells to noninflamed as well as inflamed intestine.
9  colitis and Crohn disease compared with the noninflamed bowel by 2.98-fold and 1.92-fold, respective
10 enting with aGMAb and were detectable in the noninflamed CD mucosa.
11 measured HLA-A2 binding between inflamed and noninflamed cohorts.
12                             While MPNSTs are noninflamed "cold" tumors, here, we converted MPNSTs int
13                           Inflamed (hot) and noninflamed (cold) HCC tumors and genomic signatures hav
14                                           In noninflamed colons of wild-type mice not receiving tegas
15 ubsets to enter and persist in non-lymphoid, noninflamed compartments, we examined the migration and
16 study, we demonstrate that murine LECs under noninflamed conditions actively scavenge and cross-prese
17                                        Under noninflamed conditions, C3aR and C5aR protein and mRNA w
18 l tissues such as skin and gut under normal, noninflamed conditions.
19 d UC biopsies and was the sole form found in noninflamed cont.
20 CD compared with ulcerative colitis (UC) and noninflamed control (cont) patients, and were more abund
21 rated little reactivity, as did tissues from noninflamed control brains.
22 erosing panencephalitis, but not in IgG from noninflamed control tissue.
23 A] encephalitis and multiple sclerosis), and noninflamed controls (ie, individuals with Bell palsy, n
24 f inflammatory bowel disease patients versus noninflamed controls.
25 10 weeks of protocol therapy and had stable, noninflamed corneas after 16 weeks.
26 zed dissociation to mCRP in inflamed but not noninflamed cremaster muscle.
27 ater adhesion of MB(sLex) to inflamed versus noninflamed endothelium (P = 0.0081).
28 dhesion to inflamed endothelium, relative to noninflamed endothelium, under in vitro flow conditions
29  samples) and one group of four age-matched, noninflamed enucleated globes (control samples) were use
30 otomously as having inflamed fat (n = 78) or noninflamed fat (n = 31) on the basis of the presence (+
31             In contrast, obese subjects with noninflamed fat exhibited a mixed clinical phenotype wit
32 Cr and CXCL10:Cr significantly distinguished noninflamed from inflamed biopsies (area under the curve
33    HBD-2 mRNA was also expressed in 14 of 15 noninflamed gingival tissue samples.
34  diet) as well as 18 subjects with confirmed noninflamed gut mucosa (controls, 12 presumed healthy, 5
35 0 [3.3, 13.7], P = 0.002, n = 9) compared to noninflamed histology (1.4 [0.4, 4.2], normal and inters
36 a protective role of capsule in inflamed and noninflamed hosts.
37 abel macrophages in the medullary sinuses of noninflamed human lymph node.
38                                           In noninflamed human skin during steady state, there are th
39  in a restricted subset of dermal vessels in noninflamed human skin from three different sites.
40 al vessels could serve to recruit T cells to noninflamed human skin.
41 ed gene expression signatures in inflamed vs noninflamed intestinal and rectal tissues collected from
42 ific localization of CD4 T cells into normal noninflamed islets.
43  average was lower than that in inflamed and noninflamed joints from dogs in group A.
44 rom 1.5 percentage injected dose per gram in noninflamed joints to 22.6 percentage injected dose per
45 percentage injected dose per gram (%ID/g) in noninflamed joints to 32.1 %ID/g in severely inflamed jo
46 antigens is dependent on KCs as well as on a noninflamed liver microenvironment, thereby providing me
47 as increased only in inflamed paw tissue and noninflamed LN-immune cells.
48                                 Normal (from noninflamed lung), Th1-type (induced by the pulmonary em
49 onlymphoid peripheral site (i.e., the normal/noninflamed lung).
50  naive and activated CD8+ T cells within the noninflamed lungs and quantitated the partitioning of ad
51                     Twelve patients also had noninflamed meibomian gland dysfunction (MGD).
52  already present in the adventitia of normal/noninflamed mouse aortas.
53 rophages contribute to tissue homeostasis in noninflamed mucosa through profound down-regulation of p
54  subclasses); however, anti-TNP responses in noninflamed mucosal tissues of mice with colitis exhibit
55  further observed to roll continuously along noninflamed murine dermal endothelium in vivo.
56                      In contrast, cells from noninflamed muscle contained negligible amounts of CCL19
57 tissue from patients with juvenile DM and in noninflamed muscle tissue from control subjects were exa
58                        Both the inflamed and noninflamed muscle tissue had equivalent levels of CXCL1
59                              In contrast, in noninflamed muscle, plasmacytoid DCs were scarce and did
60                  Biopsies were classified as noninflamed (n=21), inflamed (borderline changes or abov
61           Lymphocyte trafficking into normal/noninflamed or atherosclerosis-prone aortas was partiall
62 paired behavioral profile than children with noninflamed or non-T cell activated immune profiles.
63 rption, and hemoglobin response to SF) among noninflamed, outpatient populations.
64                                              Noninflamed patients in BMI Q5 formed the reference grou
65 nt analyses, the all-cause mortality risk in noninflamed patients was higher only in the lowest BMI q
66 lamed patients, this effect was mitigated in noninflamed patients.
67  was associated with higher BMI quintiles in noninflamed patients.
68  the pulmonary vascular compartment into the noninflamed pulmonary interstitium.
69 immunohistochemical analyses of inflamed and noninflamed regions of pancreatic tissue from patients w
70 rylated RelA and IkappaBalpha in inflamed vs noninflamed regions.
71 smooth muscle cells in colitic compared with noninflamed regions.
72 ncreased in regions of colitis compared with noninflamed regions.
73     Six-month urinary CCL2: Cr distinguished noninflamed renal tissue (normal, fibrosis) from IF+i wi
74             We used genetic mouse models and noninflamed samples from patients with inflammatory bowe
75 mucosa of inflamed IBD specimens compared to noninflamed sections from the same patient, establishing
76                          They also travel to noninflamed skin and peripheral LNs and remain in elevat
77 chanisms directing trafficking of T cells to noninflamed skin are less well characterized.
78 akly expressed or absent in keratinocytes of noninflamed skin but induced during acute inflammation.
79 ssion of these molecules on blood vessels in noninflamed skin provides the basis for a model of cutan
80             There are T cells within normal, noninflamed skin that most likely conduct immunosurveill
81                                              Noninflamed skin venules support constitutive leukocyte
82 cimens and additional controls consisting of noninflamed skin.
83  novel in vivo method, we report that in the noninflamed state, most transfused RBCs were consumed by
84 lymphocyte migration to the intestine in the noninflamed steady state, their role during inflammation
85 CC) with either an inflamed T cell-rich or a noninflamed T cell-deprived tumor microenvironment (TME)
86 L-1beta released significantly more END from noninflamed than from inflamed LN-immune cells.
87 DP47GS accumulation ranged from 1.6 %ID/g in noninflamed tissue to 12.0 %ID/g in severely inflamed jo
88                                           In noninflamed tissue, IL-8 expression is low but can be ra
89  reduced compared with those from autologous noninflamed tissue.
90                  However, resting Tregs from noninflamed tissues exhibit little suppressor activity,
91 trate that dermal fibroblasts and vessels of noninflamed tissues express SDF-1.
92 terized by CX(3)CR1-dependent recruitment to noninflamed tissues.
93 wn to direct blood cell trafficking to other noninflamed tissues.
94 , primary and secondary lymphoid organs, and noninflamed tissues.
95   Tumor-derived EPO autonomously generates a noninflamed TME by interacting with its cognate receptor
96 lic support for regulatory T (Treg) cells in noninflamed tumors is not well understood.