ライフサイエンスコーパス: nontransformed

1 The proliferation of all nontransformed adherent cells is dependent upon the deve

2 or tyrosine kinase is found at low levels on nontransformed adult breast epithelial cells but is freq

3 Knockdown of PHB1 in murine nontransformed AML12 cells (normal mouse hepatocyte cell

4 ces chromatin structural alterations in both nontransformed and cancer cells that normally lead to AT

5 ression was evaluated in ovarian cell lines (nontransformed and cancer), 12 benign ovarian samples, a

6 de antigen were observed in extracts of both nontransformed and oncogene-transformed cell lines.

7 ry factor (IRF)-1 expression was surveyed in nontransformed and oncogene-transformed mouse fibroblast

8 bility assays were performed with a panel of nontransformed and transformed cell lines cultured at 37

9 Analysis of RTVP-1 expression in nontransformed and transformed cells further supported p

10 tion and behavior of exosomes differ between nontransformed and transformed cells is unknown.

11 difference in the need for glutamine between nontransformed and transformed cells.

12 mphopenia and have an increased incidence of nontransformed and transformed T cells with T cell recep

13 The MCF-12A cell line is immortalized, but nontransformed, and importantly, these cells fail to exp

14 site-specific copy gains (TSSGs) in primary, nontransformed, and transformed human cells.

15 M tumor cells amplify properties inherent to nontransformed astrocytes.

16 re also more sensitive to this compound than nontransformed astrocytes.

17 to a greater extent in glioma cells than in nontransformed astrocytes.

18 chain subunit of the IgM BCR of normal (ie, nontransformed) B cells is ubiquitinated.

19 not a primary mechanism of glucose action in nontransformed beta cells.

20 TS: With citrus intake, linear regression on nontransformed biomarker data resulted in the optimal ca

21 here was no increase in cytotoxicity against nontransformed blood and mesenchymal cells.

22 generate a "phenotypic series" by treating a nontransformed breast cell line with a wide variety of m

23 PDLIM2 supports the epithelial polarity of nontransformed breast cells, suggesting distinct roles i

24 -induced protection against apoptosis in the nontransformed breast epithelial cell line MCF10A.

25 We found that SLC5A8 is expressed in nontransformed breast epithelial cell lines but silenced

26 biosynthetic inhibition could be conveyed to nontransformed breast epithelial cells through transduct

27 naffected by ErbB activation in immortalized nontransformed breast epithelial cells.

28 rved with the expression of oncogenic Ras in nontransformed breast epithelial cells.

29 ate but did not yield a notable phenotype in nontransformed breast epithelial cells.

30 Our research establishes HIEs as nontransformed cell culture models to understand human i

31 xpressing human cancer cell lines but not in nontransformed cell lines and HER2/neu low-expressing hu

32 ry memory epithelial cells, and immortalized nontransformed cell lines, and we examine whether chemot

33 was indistinguishable from rAAV2 in several nontransformed cell lines, as well as in tissues (liver,

34 hat when compared to similarly proliferating nontransformed cell lines, cancer cell lines often expre

35 g to the formation of donut-shaped nuclei in nontransformed cell lines, tumor cell lines, and tissues

36 fects were observed at 39.5 degrees C in the nontransformed cell lines; however, cytolytic effects oc

37 rief exposure, whereas solid tumor cells and nontransformed cell types are less sensitive to such tre

38 death, and selectivity for cancer cells over nontransformed cell types.

39 is overactive in a cancer cell relative to a nontransformed cell.

40 or into peritumoral normal tissue subjecting nontransformed cells adjacent to the tumor edge to an ex

41 at ShcA protects the epithelial integrity of nontransformed cells against EMT by repressing TGF-beta-

42 1 activity caused more potent G2/M arrest in nontransformed cells and antagonized the response to sub

43 is a major determinant of CSC properties in nontransformed cells and in esophageal cancer cells by d

44 IR regulation of protein synthesis occurs in nontransformed cells and is lost with transformation.

45 licates in and eradicates transformed versus nontransformed cells and is thus being considered for us

46 le to bortezomib-induced apoptosis than were nontransformed cells and that bortezomib-induced apoptos

47 sor contributes to low levels of MAGE-A11 in nontransformed cells and that higher levels of MAGE-A11

48 all HER2-overexpressing tumor cells, whereas nontransformed cells and tumor cell lines with normal HE

49 ase isozymes are differentially expressed in nontransformed cells and tumor cells such that the latte

50 us particles decreased by >1,000-fold in the nontransformed cells at 39.5 degrees C.

51 t oncogenesis model, CSCs are generated from nontransformed cells at a specific time during the trans

52 ~0.2 muM, whereas it affected the growth of nontransformed cells at significantly higher concentrati

53 5 degrees C, E1a expression was inhibited in nontransformed cells but was still abundant in the trans

54 s of tumor cell growth and invasion, and how nontransformed cells can affect miRNA expression in adja

55 However, nontransformed cells can force Src-transformed cells to

56 Nontransformed cells can force tumor cells to assume a n

57 s dramatically enhanced by surrounding stiff nontransformed cells compared with single cells or a mon

58 that microRNA-mediated target repression in nontransformed cells depends not only on abundance of sp

59 greatly elevated in PDAC cells compared with nontransformed cells expressing endogenous levels of mut

60 The expression profile of nontransformed cells expressing PML-RARalpha was remarka

61 nd adhesive mismatch between transformed and nontransformed cells in a cell monolayer can trigger enh

62 in cancer cells without causing the death of nontransformed cells in culture.

63 ial than exosomes from isogenically matched, nontransformed cells in which mutant KRAS was eliminated

64 Instead, nontransformed cells increased the expression of serum d

65 ingly, combination of these three factors in nontransformed cells induced drug resistance of a magnit

66 gene could induce sustained dissemination of nontransformed cells into collagen I.

67 multiforme, its regulation and functions in nontransformed cells of the central nervous system are w

68 Our results show that in nontransformed cells only during quiescence, protein p53

69 Inhibition of Cdk1 did not sensitize nontransformed cells or tissues to inhibition of PARP.

70 intercellular induction of apoptosis whereby nontransformed cells selectively remove transformed cell

71 In comparison, experiments in nontransformed cells showed neither a reduction in RTK-d

72 studied in tumor cells is representative of nontransformed cells such as stem cells.

73 a layered culture system to investigate how nontransformed cells suppress the growth of neighboring

74 Moreover, nontransformed cells suppressed Pdpn expression in adjac

75 afety tests, NK-HDAC-1 was far less toxic to nontransformed cells than tumor cells and showed no sign

76 We conclude that oocytes are the only nontransformed cells that fail to launch a robust G2 pha

77 E2F-1 expression are sufficient to sensitize nontransformed cells to flavopiridol.

78 nced the sensitivity of cancer cells but not nontransformed cells to ionizing radiation.

79 We find that nontransformed cells undergo a TP53-dependent growth arr

80 ect physical contact between transformed and nontransformed cells was required for growth suppression

81 or cells that were normalized by neighboring nontransformed cells was used as an additional filter to

82 In contrast, isogenic nontransformed cells were resistant to fatty acid biosyn

83 agar colony-forming abilities, but it spares nontransformed cells where expression of 5-Lox is undete

84 ransiently induced by growth factors (GF) in nontransformed cells with delayed kinetics (4-6 hours),

85 We report that irradiation of nontransformed cells with low doses of either high linea

86 mprise a complex mixture of malignant cells, nontransformed cells, and microorganisms.

87 low concentrations to support the growth of nontransformed cells, but transformed cells require much

88 In nontransformed cells, centrosome loss triggers a p53-dep

89 gulated by HER-2 in a manner not observed in nontransformed cells, even when HER-2 is overexpressed.

90 s not similarly involved in proliferation of nontransformed cells, including diploid human IMR-90 fib

91 In nontransformed cells, IR rapidly activates the MAP kinas

92 MIF and d-DTs AMPK-activating properties in nontransformed cells, MIF and d-DT act cooperatively to

93 Similar structures exist in nontransformed cells, such as osteoclasts and dendritic

94 site of chromatin in cancer cells but not in nontransformed cells, suggesting that STAT3 binding and

95 esults in marked defects in proliferation of nontransformed cells, whereas siRNA knockdown does not,

96 wding of the stiff nuclei of the surrounding nontransformed cells, whose movements depend directly on

97 ells without reducing the growth rate of the nontransformed cells.

98 observing phenotypes after expressing it in nontransformed cells.

99 capacity for beta-oxidation, in contrast to nontransformed cells.

100 ced in the transformed cells relative to the nontransformed cells.

101 a cells while minimizing the toxicity toward nontransformed cells.

102 24 expression promotes nonanchored growth of nontransformed cells.

103 tivation of endogenous HIF-1alpha targets in nontransformed cells.

104 osis in established tumor cell lines but not nontransformed cells.

105 a preferential effect on transformed versus nontransformed cells.

106 d HDAC activity in these cells compared with nontransformed cells.

107 morphology can be normalized by contact with nontransformed cells.

108 tic response to JNK activity as it occurs in nontransformed cells.

109 upled to adhesion to extracellular matrix in nontransformed cells.

110 ovarian cancer cell lines, but absent in the nontransformed cells.

111 cell lines overexpressed FAK compared to the nontransformed cells.

112 s was not directly inhibited by contact with nontransformed cells.

113 y block in viral replication occurred in the nontransformed cells.

114 ansiently arrest RNA transcription in normal nontransformed cells.

115 he contrast between images of metastatic and nontransformed cells.

116 on, but had less cytotoxic effects on normal nontransformed cells.

117 g from the drug treatment of transformed and nontransformed cells.

118 n recovered to levels comparable to those in nontransformed cells.

119 transformed with CdCl(2) when compared with nontransformed cells.

120 metastatic cells while minimizing damage to nontransformed cells.

121 its accumulation in cancer cells but not in nontransformed cells.

122 ways that lead to the expression of B7-H6 in nontransformed cells.

123 ied neuroblastoma cell lines, in contrast to nontransformed cells.

124 ufficient to enhance PDGFRbeta activation in nontransformed cells.

125 receptors and increased their activation in nontransformed cells.

126 R-193a is lower in transformed cells than in nontransformed cells.

127 sient increase in translation, IR-sensitive (nontransformed) cells inhibit cap-dependent protein synt

128 t contain not only neoplastic cells but also nontransformed cellular elements such as stromal cells,

129 rse array of somatic cell types in a normal, nontransformed cellular milieu.

130 gy of a malignant cell is inherited from its nontransformed cellular progenitor-GC centroblasts-aberr

131 me, we showed that E(2) alters the growth of nontransformed colonocytes in vitro and that, through an

132 de and AMPK activation, but had no effect in nontransformed control cells.

133 tly reduced in transgenic plants compared to nontransformed controls (65%-89% reduction at high oxali

134 oliferate at an increased rate compared with nontransformed controls and show elevated levels of PI3K

135 Compared with nontransformed controls, v-Jun transfectants show enhanc

136 of aerobic glycolysis in comparison to their nontransformed counterparts, although the molecular basi

137 s greater in transformed cells compared with nontransformed counterparts, an effect independent of ce

138 and low malignant potential alongside their nontransformed counterparts, we identify an HSF1-regulat

139 significantly higher in melanomas than their nontransformed counterparts.

140 intain proliferation and survival than their nontransformed counterparts.

141 Using primary and immortalized, nontransformed cultures of human mammary epithelial cell

142 report that a fusion event involving normal, nontransformed, cytogenetically stable epithelial cells

143 e complex (pre-RC) protein Cdc6 causes human nontransformed diploid cells to arrest nonlethally in G1

144 uch greater requirement for Plk1 than normal nontransformed diploid cells.

145 Our work demonstrates that nontransformed ECs respond differently to excess centros

146 These findings correlate with retention of a nontransformed endocardial sheet and lack of invasion.

147 When overexpressed in the nontransformed EpH4 mammary epithelial cell line, BCL-6

148 alignant cells but not against monolayers of nontransformed EphA2-positive cells except at the edges

149 When nontransformed epithelia are injured, activation of the

150 IT2 is sufficient to induce migration of the nontransformed epithelial cell line MCF10A.

151 (TGF-beta) induces cell cycle arrest of most nontransformed epithelial cell lines.

152 particular is well known as an inhibitor of nontransformed epithelial cell proliferation.

153 interference-mediated silencing of ARID1A in nontransformed epithelial cells is sufficient to enhance

154 d that the stable intercellular junctions of nontransformed epithelial cells occlude the binding site

155 intestine as a result of stromal invasion of nontransformed epithelial cells.

156 malignant cells, which are not available on nontransformed epithelial cells.

157 Unlike nontransformed epithelium lacking ERBB3, intestinal tumo

158 red our finding on HeLa cells with those for nontransformed fibroblasts to help distinguish the regul

159 However, when studying nontransformed fibroblasts, we found that Ras GTPases ar

160 effectively spread beneficial genes through nontransformed field populations, the core requirement o

161 However, the cells isolated from WT, nontransformed G-/- and SOM-/- gastric tissue did not fo

162 here a simple method yielding self-renewing, nontransformed, GM-CSF/signal transducer and activator o

163 els of beta-catenin activity than either the nontransformed GMP or the transformed nonGMP, both in cu

164 Surprisingly, nontransformed hematopoietic cells can grow and prolifer

165 icient to promote the growth and survival of nontransformed hematopoietic cells.

166 y is sufficient to promote leukemogenesis in nontransformed hematopoietic precursors and maintenance

167 und to type I TGF-beta receptor (TbetaRI) in nontransformed, HER2-transformed, and HER2-negative brea

168 the D239Y variant expressed in immortal but nontransformed human and mouse mammary epithelial cells.

169 biased retroviral loss-of-function screen in nontransformed human astrocytes, we demonstrate that mit

170 te the possibility of distinguishing between nontransformed human breast epithelial cells (MCF-10A) a

171 hen exogenously expressed in cultured MCF10A nontransformed human breast epithelial cells than in bre

172 ates of protein kinase Calpha (PKC-alpha) in nontransformed human breast MCF-10A cells.

173 bility of IF-MoS2\INT-WS2, we demonstrate in nontransformed human bronchial cells (NL-20) relative lo

174 Mixture of these 2 readily available, nontransformed human cell types provides a practical app

175 ray technology (Illumina), we examined GE in nontransformed human cells after 4 h exposure to DBPs at

176 es reduplicate consistently in cancerous and nontransformed human cells during G2 arrests and that th

177 ession in all situations analyzed, including nontransformed human cells, normal mouse intestinal epit

178 ndividual kinetochores throughout mitosis in nontransformed human cells.

179 fically activated in tumor cells, but not in nontransformed human cells.

180 s-GTP to stimulate Ras-induced senescence in nontransformed human cells.

181 PGC1beta and ERRalpha are not detectable in nontransformed human colon epithelial cells, and depleti

182 ere stably transfected into NCM 460 cells, a nontransformed human colonic epithelial cell line.

183 emonstrate that silencing TLR5 expression in nontransformed human colonic epithelial cells blocks fla

184 way and IL-8 gene expression was assessed in nontransformed human colonic epithelial NCM460 cells.

185 ys including p21(WAF1/CIP1) were assessed in nontransformed human colonic NCM460 epithelial cells exp

186 Nontransformed human corneal epithelial cells (HCECs) at

187 Human Caco-2BBE cells and nontransformed human enterocytes (HIPEC) were subjected

188 on the establishment of a culture system of nontransformed human fetal hepatocytes that supports hep

189 lone of HCV (genotype 1a, H77 strain) in the nontransformed human hepatocyte line cell HH4 using the

190 system may be used to study the responses of nontransformed human hepatocytes to HCV infection, to an

191 We infected nontransformed human intestinal enteroid cultures from m

192 strains in enterocytes in stem cell-derived, nontransformed human intestinal enteroid monolayer cultu

193 Toxicogenomic analysis was conducted with a nontransformed human intestinal epithelial cell line (FH

194 itive deformation stimulates transformed and nontransformed human intestinal epithelial proliferation

195 ll lines, including, HeLa, CaCo-2 cells, and nontransformed human keratinocytes and bronchial epithel

196 sed RNA for ULBP1 and other NKG2D ligands in nontransformed human keratinocytes.

197 cripts by comparison with mRNA-Seq data from nontransformed human mammary epithelial cell cultures pl

198 reast tumor-derived cell lines, immortalized nontransformed human mammary epithelial cells, and norma

199 activation of matriptase on the surfaces of nontransformed human mammary epithelial cells.

200 In this study, we utilized nontransformed human MCF10A mammary epithelial cells and

201 To test this hypothesis, we generated nontransformed human MCF10A mammary epithelial cells sta

202 s and TNF-induced beta-catenin activation in nontransformed human NCM460 cells (TOPFlash) and mice (T

203 a panel of pancreatic cancer cell lines and nontransformed human pancreatic ductal epithelial cells

204 mitosis is prolonged by approximately 40% in nontransformed human RPE-1, approximately 80% in PtK2 (r

205 lterations to global gene expression (GE) in nontransformed human small intestine epithelial cells (F

206 To test this directly, we followed nontransformed human telomerase immortalized human retin

207 ling 10-fold fewer false-positive fusions in nontransformed human tissues.

208 L-7Ralpha expression and function in normal (nontransformed) human thymocytes, and human CD19(+) B-li

209 more pronounced in v-Fos-transformed versus nontransformed immortalized rat cells, and this radiosen

210 Using nontransformed, immortalized normal human fibroblasts, w

211 UCMSCs are a unique human, easily available, nontransformed, in vitro model of HBV infection that cou

212 x1 expression reduced the proliferation of a nontransformed intestinal cell line.

213 Manipulation of PKCalpha activity in IEC-18 nontransformed intestinal crypt cells determined that PK

214 a program of cell cycle withdrawal in IEC-18 nontransformed intestinal crypt cells, involving rapid d

215 We tested the hypothesis that exposure of nontransformed intestinal epithelial cells (IEC-18) to t

216 that the inducible expression of kRasV12 in nontransformed intestinal epithelial cells significantly

217 We demonstrate that treatment of nontransformed intestinal epithelial cells with TGF-beta

218 derived cell lines but was also a feature of nontransformed keratinocytes and lung fibroblasts.

219 ormed mosquitoes and compared results to the nontransformed laboratory strain.

220 In nontransformed liver cells and cultured primary liver ce

221 o examine the biological effects of K-Ras in nontransformed lung epithelial cells, stable transfectan

222 It has been reported that nontransformed mammalian cells become arrested during G1

223 me the limitations inherent in synchronizing nontransformed mammalian cells.

224 Transient expression of FOXC1 in nontransformed mammary epithelial cell lines resulted in

225 factor (Tcf/Lef) transcriptional activity in nontransformed mammary epithelial cells (MCF-10A) and th

226 inflammatory signals that induce the EMT in nontransformed mammary epithelial cells and in ZR75.1 br

227 analyzed maspin function in cell adhesion in nontransformed mammary epithelial cells and investigated

228 Since experimental Brk expression in nontransformed mammary epithelial cells enhances their m

229 HC11 nontransformed mammary epithelial cells express PIAS3, P

230 zation of ESE-1 protein induces apoptosis in nontransformed mammary epithelial cells via a transcript

231 cient to induce tumor formation by otherwise nontransformed mammary epithelial cells, and that the in

232 In nontransformed mammary epithelial cells, overexpression

233 EMT in this breast cancer cell model and in nontransformed mammary epithelial cells.

234 ability, when overexpressed in immortalized, nontransformed mammary epithelial MCF10A cells.

235 R-cad is expressed in nontransformed mammary epithelium but absent from tumori

236 ely on narrow fiber-like dimensions, whereas nontransformed MCF-10A mammary epithelial cells require

237 d increased levels of mesenchymal markers in nontransformed MCF10A breast epithelial cells, consisten

238 Nicastrin overexpression in nontransformed MCF10A cells caused an induction of epith

239 Further, expression of Amot80 induces nontransformed MCF10A cells to overgrow as disorganized

240 Conditional activation of FGFR1 in the nontransformed MCF10A human mammary cell line, MCF10A, r

241 Nontransformed mean (SD) days per week decreased for pla

242 d the replication of HAV in immortalized and nontransformed MMH-D3 mouse liver cells, which require g

243 erythroblasts (ESREs), a well-characterized, nontransformed model of erythroid maturation.

244 ent induction of autophagy was observed when nontransformed mouse embryo fibroblasts were treated.

245 Skp2 knock down inhibits S phase entry in nontransformed mouse embryonic fibroblasts but not in hu

246 Immortalized human and nontransformed murine colonic epithelial cells, along wi

247 Furthermore, TGF-beta induction of EMT in nontransformed murine mammary gland epithelial cells res

248 by primary murine bone marrow B cells and a nontransformed murine pro/pre-B cell line (BU-11).

249 normal murine promyelocyte-enriched samples, nontransformed murine promyelocytes expressing human pro

250 We made a stable Mirk-inducible subline from nontransformed Mv1Lu lung epithelial cells and now demon

251 Transient overexpression of Mirk in nontransformed Mv1Lu lung epithelial cells blocked cells

252 For the in vitro experiments, human nontransformed NCM460 colonocytes stably transfected wit

253 GE2 production and COX-2 expression in human nontransformed NCM460 colonocytes stably transfected wit

254 Within the brain, glioma cells migrate like nontransformed, neural progenitor cells-extending a prom

255 gregation, aneuploidy, and transformation in nontransformed NIH 3T3 cells.

256 we have asked whether the nuclei of normal, nontransformed, nondividing, and terminally differentiat

257 tion affects proliferation and metabolism in nontransformed, nonimmortalized PKM2-expressing primary

258 enhanced water use efficiency compared with nontransformed or 35S:PYL4 plants and partial up-regulat

259 ght and dehydration resistance compared with nontransformed or 35S:PYL4 plants.

260 irus replication in PDA cells over that in a nontransformed pancreatic ductal cell line.

261 mportantly, IAVs did not induce apoptosis in nontransformed pancreatic ductal HPDE6 cells.

262 expression was reduced in PDAC compared with nontransformed pancreatic tissues and cell lines, sugges

263 erexpression of H-Ras(V12) oncogene to their nontransformed parental cells and found that the maligna

264 cotrienols, which were absent from leaves of nontransformed plants, and a 10- to 15-fold increase in

265 We hypothesized that nontransformed plasma cells are also hypersensitive to H

266 t different cell types arising from a common nontransformed population.

267 e models of NF1-deficient MPNSTs, but not in nontransformed precursor cells.

268 nd induces loss of the mitotic checkpoint in nontransformed preleukemic thymocytes.

269 Paradoxically, expression of EWS/FLI1 in nontransformed primary cells results in apoptosis, but t

270 l antiapoptotic factor in both neopastic and nontransformed primary human mast cells through the regu

271 n and increased P-glycoprotein expression in nontransformed, primary cultures of human microvascular

272 As demonstrated previously with a nontransformed pro-/pre-B cell line, primary pro-B cells

273 Stable transfection of SOX4 into nontransformed prostate cells enabled colony formation i

274 Expression of TRAP-1 in nontransformed prostatic epithelial BPH-1 cells inhibite

275 kinetics in cell culture similar to those of nontransformed R. conorii.

276 In a nontransformed rat hepatocyte cell line, TNFalpha-induce

277 alpha-induced activation of 4E-BP1 in IEC-18 nontransformed rat ileal crypt cells.

278 mes involved in prostacyclin biosynthesis in nontransformed rat intestinal epithelial cells (IECs).

279 COX-2 on the oncogenic potential of MEK1 in nontransformed rat intestinal epithelial cells.

280 sogenic clones derived from immortalized and nontransformed rat liver epithelial cells that expressed

281 PTK6 sensitizes the nontransformed Rat1a fibroblast cell line to apoptotic s

282 ll MPNST cells and tumor samples compared to nontransformed Schwann cells.

283 signaling enhances cell-cycle progression of nontransformed SMCs in vitro and in response to vascular

284 siRNA-mediated RNAi gene silencing in normal nontransformed somatic mammalian lymphocytes.

285 genes cooperate in the establishment of the nontransformed state.

286 MCF10A cells, a spontaneously immortalized, nontransformed strain of human mammary epithelial cells

287 lk between tumor cells and their surrounding nontransformed stroma.

288 of anti-Fas antibody than were immortalized, nontransformed T29 cells, associated with less activatio

289 Hemizygous loss of ID4 in nontransformed TCL1-positive B cells enhances cell proli

290 entify TGF-beta-induced genes in VACO 330, a nontransformed TGF-beta-sensitive cell line derived from

291 We evaluated these nontransformed, three-dimensional HIE cultures as models

292 ncreased RBP-Jkappa expression compared with nontransformed thymocytes, suggesting there is no select

293 I3K) activation caused by PTEN deficiency in nontransformed thyrocytes results in a global downregula

294 Its roles in normal nontransformed tissues are not clear.

295 Its role in normal, nontransformed tissues is not clear.

296 ty of WNT metabolism research has focused on nontransformed tissues, the role of WNT in cancer metabo

297 ere up-regulated compared with corresponding nontransformed tissues.

298 ignal can initiate an epigenetic switch from nontransformed to cancer cells via a positive feedback l

299 ess the TP-beta isoform, unlike immortalized nontransformed urothelial cells (SV-HUC) that express on

300 UHRF1 in both mammalian cancer cells and in nontransformed zebrafish cells, but not in zebrafish bea