ライフサイエンスコーパス: nontransgenic

1 c (0.053 +/- 0.020 min(-1); n = 32 mice) and nontransgenic (0.056 +/- 0.029 min(-1); n = 17 mice) ani

2 and thus palmitoyl-coenzyme A recycling, in nontransgenic (4.5+/-2.3 micromol/min per gram dry weigh

3 to pressure overload and oxidative stress in nontransgenic adult mouse hearts.

4 rons derived from various brain regions from nontransgenic and genetically engineered mice and rats c

5 and stroma of untreated or estrogen-treated nontransgenic and HPV-transgenic mice.

6 Additionally, we fed nontransgenic and K8 overexpressing mice (K8tg) with the

7 Adult male nontransgenic and myocyte-restricted HO-1 transgenic mic

8 al doses of 10 mg/kg trichostatin A (TSA) in nontransgenic and SMA model mice resulted in increased l

9 ate-salt model of neurogenic hypertension in nontransgenic and syn-hACE2 mice overexpressing ACE2 in

10 We performed adoptive transfers of both nontransgenic and TCR-transgenic OVA(257-264)-specific (

11 age, cell turnover was reduced with aging in nontransgenic and TGF-alpha mice, indicating that some g

12 lanine, and fructose remained similar in the nontransgenic and transgenic fruits.

13 tified in isolated hearts, from normal mice (nontransgenic) and mice with cardiac-specific overexpres

14 Atrial JPH2 levels in WT-JPH2 transgenic, nontransgenic, and JPH2 knockdown mice correlated negati

15 2CTF), and describe a novel disease-relevant nontransgenic animal model of AD.

16 ew representative analogs were assessed in a nontransgenic animal model of Alzheimer's disease (AD),

17 A nontransgenic animal model that still develops hallmarks

18 ice may be used as a simple, toxin-free, and nontransgenic animal model to study PD-related nigrostri

19 gnitude higher in transgenic animals than in nontransgenic animals 2 to 4 weeks postinfection, and th

20 inhibition of these neurons in freely moving nontransgenic animals has not been possible.

21 ells accumulated in inflamed joints, even in nontransgenic animals.

22 red with genetically matched and age-matched nontransgenic animals.

23 antly reduced polyp load (60%) compared with nontransgenic Apc(Min+) littermates.

24 addition, RLK pathways are ideal targets for nontransgenic approaches, such as synthetic molecules, p

25 nced (anergic) immunoglobulin transgenic and nontransgenic B cells but not by transiently stimulated

26 , onward transmission to both transgenic and nontransgenic birds was prevented.

27 However, nontransgenic, Bt and BtHr cotton had similar yields ove

28 In nontransgenic CD4 T cells, blocking IL-4Ralpha with Abs

29 nt as well as lean control Tg-fMCD(Skel) and nontransgenic control mice were treated with Shield-1 an

30 In nontransgenic control mice, oxidative stress was coincid

31 ls of CA-IX, Glut1, and VEGF than tubules in nontransgenic control mice.

32 ischemic reserve capacity when compared with nontransgenic control mice.

33 nits were indistinguishable from age-matched nontransgenic control mice.

34 NA expression below approximately 50% of the nontransgenic control.

35 with increased macrophage uPA expression or nontransgenic controls (all apolipoprotein E-null [Apoe(

36 a a bacterial artificial chromosome (TG) and nontransgenic controls (Cont).

37 ize of MI was similar between Tg-DN-Mst1 and nontransgenic controls (NTg).

38 human APOE3 or APOE4 to the levels of their nontransgenic controls and the significant decrease of i

39 ly (P < 0.05) from nonmodified transgenic or nontransgenic controls in biomass growth or leaf morphol

40 els of iron in transgenic storage roots than nontransgenic controls in confined field trials in Puert

41 zinc levels 3-10 times higher than those in nontransgenic controls in the field.

42 maize events had a greater grain yield than nontransgenic controls under both drought stress and wel

43 Relative to nontransgenic controls, calcium dynamics were significan

44 Compared with nontransgenic controls, hAPP mice had significantly fewe

45 Compared with nontransgenic controls, neither transgenic model exhibit

46 persistently oxidized glucose compared with nontransgenic controls, while exhibiting supranormal per

47 -hiPSCs was increased 100-fold above that of nontransgenic controls.

48 pression in 10-13 month old APP-PS1 mice and nontransgenic controls.

49 uridine (BrdU), Ki-67, and c-Myc relative to nontransgenic controls.

50 differ significantly between transgenic and nontransgenic controls.

51 Vgp120tg mice expressing or lacking CCR5 and nontransgenic controls.

52 reased insulin sensitivity compared to obese nontransgenic controls.

53 Transgenic cotton had higher yield than nontransgenic cotton for any given number of insecticide

54 largely because higher insecticide use with nontransgenic cotton improved control of key pests.

55 lar effects of cultivation of transgenic and nontransgenic cotton on biodiversity.

56 tical rates of fluorescence yield decline as nontransgenic cotton.

57 LUT4 gene and promoter (hGLUT4 TG) and their nontransgenic counterparts (NT) were fed either a contro

58 We demonstrate that IL-60 enables nontransgenic expression of an entire bacterial operon i

59 i-glomerular basement disease model, whereas nontransgenic FcRgamma(-/-) mice were completely protect

60 yogenic differentiation when cocultured with nontransgenic fetal cardiomyocytes (>18 000 EGFP(+) cell

61 r proteinuria and 75% lower albuminuria than nontransgenic FHH littermates.

62 ant in Bt cotton and Bt maize fields than in nontransgenic fields managed with insecticides.

63 fection with all three types of viruses than nontransgenic FVB mice.

64 Nontransgenic genome editing in regenerable protoplasts,

65 addition, our study lays the foundation for nontransgenic genome editing of citrus.

66 ls mature, a higher proportion expresses the nontransgenic H chain allele.

67 TAC induced a 25% increase in nontransgenic heart size but only a 7% increase in ssTnI

68 ation in hearts of Pim-wt mice versus 26% in nontransgenic hearts after infarction challenge.

69 iptional profiling of Galphaq transgenic and nontransgenic hearts by Illumina RNA sequencing and Affy

70 Differential mRNA expression in Galphaq and nontransgenic hearts correlated well between microarrays

71 xpression augmented TAG turnover 3-fold over nontransgenic hearts, despite similar fractions of acety

72 -energy phosphate content similar to that in nontransgenic hearts, providing evidence for greater ene

73 basal and beta-adrenergic stress compared to nontransgenic hearts, with a reduction in maximal Ca(2+)

74 mitigated in MHC-ACSL1 hearts compared with nontransgenic hearts.

75 Four weeks after ligation, nontransgenic HF mice exhibited postinfarction left vent

76 diac matrix with cardiomyocytes derived from nontransgenic human induced pluripotent stem cells and g

77 ansgenic infected mice relative to those for nontransgenic infected mice.

78 study using a wild gourd and transgenic and nontransgenic introgressives, we measured the effects of

79 transgenic mouse islets develop amyloid but nontransgenic islets do not, a broad spectrum MMP inhibi

80 oped much less severe proteinuria than their nontransgenic Lamb2-deficient littermates; the level of

81 function, and remodeling were compared with nontransgenic littermate control (NLC) and wild-type (WT

82 s of myocardial fibers from Tg-CTGF mice and nontransgenic littermate control (NLC) mice were discern

83 after TAC, swim-trained transgenic GRK5 and nontransgenic littermate control mice exhibited similar

84 KChIP2 from ventricles between p90RSK-Tg and nontransgenic littermate control mice were similar, as a

85 Cardiac-specific GRK5 transgenic mice and nontransgenic littermate control mice were subjected to

86 ter I/R in DN-RSK (0.9+/-0.2%) compared with nontransgenic littermate controls (6.2+/-2.6%).

87 reperfusion from 46.9+/-5.6% area at risk in nontransgenic littermate controls to 26.0+/-4.2% in DN-R

88 chain (alphaMHC)-BMP10 transgenic hearts and nontransgenic littermate controls using Affymetrix mouse

89 Beginning on day of life 75, TG9 mice and nontransgenic littermate controls were given a daily 10

90 ergic excitation to the same extent as their nontransgenic littermate controls, as a result of the ex

91 Compared with nontransgenic littermate controls, monocytes of TLR10 tr

92 toimmune alopecia susceptibility relative to nontransgenic littermate controls.

93 in size and macrophage content compared with nontransgenic littermate controls.

94 Nontransgenic littermate mice were not affected by this

95 pect to the control astrocytes isolated from nontransgenic littermates (NTg).

96 tween young and old TAU58/2 mice relative to nontransgenic littermates and by comparative analysis wi

97 significantly weaker with age compared with nontransgenic littermates and exhibited typical myopathi

98 iferation in bone marrow and spleen, whereas nontransgenic littermates and hMRP8-NPM transgenic mice

99 had little to no effect on ERG responses in nontransgenic littermates and other retinal degeneration

100 developed normally in comparison with their nontransgenic littermates but had a suppressed rate of h

101 enic mice survived significantly longer than nontransgenic littermates in response to a lethal tumor

102 This phenotype was not observed in nontransgenic littermates or in mice expressing an hP23H

103 At 2 to 3 months of age, ssTnI mice or their nontransgenic littermates underwent aortic constriction

104 omatic and symptomatic PrP-SCA7-92Q mice and nontransgenic littermates was compared.

105 Age-matched nontransgenic littermates were controls.

106 o(+/-)), and the eyes of transgenic mice and nontransgenic littermates were exposed for 2.5 minutes t

107 estricted dominant-interfering p53 and their nontransgenic littermates were treated with doxorubicin

108 tablished beta-amyloid peptide pathology and nontransgenic littermates were treated with either alpha

109 Tg2576 mice and their nontransgenic littermates were treated with simvastatin

110 inked mutation in tropomyosin (Tm-E180G) and nontransgenic littermates with FTY720 or vehicle for 6 w

111 nally, infection of both transgenic mice and nontransgenic littermates with HSV-1 revealed no differe

112 diac gene transcription differs from that of nontransgenic littermates, primarily in the expression o

113 and the TMA decreased with age compared with nontransgenic littermates, ruling out the SR as a source

114 In contrast to nontransgenic littermates, such animals had B-cell-restr

115 In contrast to nontransgenic littermates, which develop lymphocytic spo

116 asmacytoid DCs (pDCs) in transgenic, but not nontransgenic littermates, without elimination of spleni

117 epared from G93A-SOD1 embryos and from their nontransgenic littermates.

118 hibited a distinct immunoprofile compared to nontransgenic littermates.

119 acquisition of the MWM task when compared to nontransgenic littermates.

120 te uptake into peripheral organs compared to nontransgenic littermates.

121 ed with wild-type ERalpha transgenic mice or nontransgenic littermates.

122 nd compared their diabetes susceptibility to nontransgenic littermates.

123 nse to azoxymethane (AOM) when compared with nontransgenic littermates.

124 duction and dilated ducts when compared with nontransgenic littermates.

125 significantly different between SA mice and nontransgenic littermates.

126 a transgenic mice when compared with that of nontransgenic littermates.

127 constitutively active Gqalpha compared with nontransgenic littermates.

128 cell population and comparing it to that of nontransgenic littermates.

129 other TCRBV gene segments when compared with nontransgenic littermates.

130 crypt foci and no tumors, in comparison with nontransgenic littermates.

131 ce, nor did it affect tumor dissemination in nontransgenic littermates.

132 um and membrane depolarization compared with nontransgenic littermates.

133 on with influenza viruses of human origin as nontransgenic littermates.

134 orebrains of Tg-Abeta+Tau mice compared with nontransgenic littermates.

135 comparable metabolic dysfunction relative to nontransgenic littermates.

136 sing HuWtSOD1, but not to cells derived from nontransgenic littermates.

137 DDC feeding induced MBs in nontransgenic livers, but MBs were rarely seen in any of

138 RGS5 expression in nontransgenic MA was induced after activation of either

139 easured by Akt phosphorylation compared with nontransgenic macrophages.

140 he male sterile phenotype, producing stable, nontransgenic male sterility.

141 l-mesenchymal transformation was frequent in nontransgenic malignancies.

142 PyroGlu-3 Abeta is further present in two nontransgenic mammalian models of cerebral amyloidosis,

143 ocal Lesions in Genomes (TILLING) provides a nontransgenic method for reverse genetics that is widely

144 ted from mice that overexpress cyclin E1 and nontransgenic mice (controls) and analyzed for tumor bur

145 icant increase in infarct size compared with nontransgenic mice (from 36.9+/-2.5% to 50.9+/-4.3%).

146 We compared data obtained from control nontransgenic mice (NTG) with a transgenic mouse model e

147 protein transgenic mice (CRPtg) compared to nontransgenic mice (NTG).

148 ACE2 activity in the cerebrospinal fluid of nontransgenic mice after deoxycorticosterone acetate-sal

149 reduction also protected both transgenic and nontransgenic mice against excitotoxicity.

150 TLR7 agonist administration to nontransgenic mice also drove type I IFN-dependent emerg

151 own to induce alphaS pathology in the CNS of nontransgenic mice and alphaS transgenic mice, albeit wi

152 ansgenic male mice at 6 months compared with nontransgenic mice and female hIAPP transgenic mice.

153 Nontransgenic mice and mice with cardiomyocyte-restricte

154 fibrosis in K18 R89C as compared with WT and nontransgenic mice and resulted in increased messenger R

155 d activity were found to decline with age in nontransgenic mice as well as in hIAPP transgenic female

156 When expressed in nontransgenic mice at levels sufficient to drive Abeta42

157 gnaling was inhibited in doxorubicin-treated nontransgenic mice but not in doxorubicin-treated MHC-CB

158 d neuronal alterations could be simulated in nontransgenic mice by excitotoxin challenge and prevente

159 ich are predisposed to MDB formation, and in nontransgenic mice by feeding the porphyrinogenic compou

160 Nontransgenic mice exhibited reduced left ventricular sy

161 0 mice had similar abnormalities relative to nontransgenic mice in spatial and nonspatial learning an

162 disease in K14E5 mice differed from that in nontransgenic mice in that benign tumors converted from

163 urdens were not significantly different from nontransgenic mice infected with either Ed N-522D or par

164 eoxycorticosterone acetate-salt treatment in nontransgenic mice led to significant increases in blood

165 Analysis in nontransgenic mice showed that thioacetamide and CCl(4)

166 r, these autoantibodies were not observed in nontransgenic mice subjected to 4 chronic injury models.

167 rphosphorylation of endogenous mouse MAPT in nontransgenic mice that is further enhanced in mice lack

168 We also observed age-matched nontransgenic mice to study normal effects of aging on s

169 ontransgenic mice, whereas neither K14E5 nor nontransgenic mice treated with the promoting agent 12-O

170 lls and pericytes from brain microregions of nontransgenic mice using CD31 and CD13 as surface marker

171 Nontransgenic mice with acquired iron deficiency anemia

172 man tau strains by intracerebrally injecting nontransgenic mice with pathological tau enriched from h

173 ly inhibited (7% in transgenic versus 40% in nontransgenic mice).

174 Here, we found that in wild-type nontransgenic mice, a single intrastriatal inoculation o

175 stic cervical disease than similarly treated nontransgenic mice, although no frank cancers were detec

176 Compared to control nontransgenic mice, as well as transgenic mice expressin

177 e serum of TLR10 transgenic mice compared to nontransgenic mice, but did not affect mouse survival in

178 eased heart weight to tibia length by 46% in nontransgenic mice, but only 26% in MHC-ACSL1 mice, wher

179 Compared with nontransgenic mice, HO-1 transgenic HF mice exhibited si

180 ervical carcinogenesis compared with that in nontransgenic mice, indicating that activities of E7 bes

181 transgenic mice vs. approximately 15.5 h in nontransgenic mice, indicating that huGYPA on mature RBC

182 cdk5 inhibitor was administered to very old, nontransgenic mice, inhibition of cdk5 reduced Abeta lev

183 In nontransgenic mice, knockdown of EphB2 mediated by short

184 Nontransgenic mice, or mice that over express either hum

185 ed in mice expressing wild-type human PS1 or nontransgenic mice, respectively.

186 e in hypertrophy in both Tg-GSK-3beta-DN and nontransgenic mice, Tg-GSK-3beta-DN exhibited better lef

187 Compared with nontransgenic mice, the CNS of AstroCD24TG mice had high

188 Compared to nontransgenic mice, transgenic mouse hearts showed signi

189 developed more papillomas than like-treated nontransgenic mice, whereas neither K14E5 nor nontransge

190 r inclusions in K8-overexpressing but not in nontransgenic mice, without causing liver injury.

191 ness and collagen accumulation compared with nontransgenic mice, yet IL-5 intestine transgenic mice d

192 nic mice consume excess calories relative to nontransgenic mice, yet they weigh less.

193 e Treg lineage in TCR transgenic mice and in nontransgenic mice.

194 The uptake into brain was minimal in nontransgenic mice.

195 03) were significantly reduced compared with nontransgenic mice.

196 sis and less lymphocyte infiltration than in nontransgenic mice.

197 iac-specific overexpression of Sirt1 than in nontransgenic mice.

198 of CNS cell type-specific genes expressed in nontransgenic mice.

199 can be used to differentiate vessel types in nontransgenic mice.

200 the production of endogenous murine Abeta in nontransgenic mice.

201 ced mortality, compared to >30% mortality in nontransgenic mice.

202 and brain activity of 5b were established in nontransgenic mice.

203 similar to those seen in doxorubicin-treated nontransgenic mice.

204 display a baseline phenotype consistent with nontransgenic mice.

205 arcomas have not been observed previously in nontransgenic mice.

206 unction and less fibrosis and apoptosis than nontransgenic mice.

207 pared with age-matched transgenic female and nontransgenic mice.

208 e of Pseudomonas from the lung compared with nontransgenic mice.

209 inoma progression compared with like-treated nontransgenic mice.

210 ronment of HPV-transgenic mice compared with nontransgenic mice.

211 but also enhanced learning and memory in the nontransgenic mice.

212 when compared with IRS2(WT)-beta mice or to nontransgenic mice.

213 s in anatomically connected brain regions in nontransgenic mice.

214 -AD mice to the level of that in age-matched nontransgenic mice.

215 gy to bidirectionally control nociceptors of nontransgenic mice.

216 rain homogenates from human tauopathies into nontransgenic mice.

217 tion of TSPO ligands Ro5-4864 and PK11195 in nontransgenic mice.

218 mutant, decreased protein import by >50% in nontransgenic mitochondria from spinal cord, but not fro

219 had no noticeable fitness load compared with nontransgenic mosquitoes when fed on noninfected mice.

220 fecundity and lower mortality) than sibling nontransgenic mosquitoes.

221 t mosquitoes have a selective advantage over nontransgenic mosquitoes.

222 s or co-delivered with zinc-deficient SOD to nontransgenic motor neurons.

223 We now report that nontransgenic mouse AHNPCs transduced with retroviruses

224 -RTN3 mice would ever naturally occur in the nontransgenic mouse brain, we targeted our examination t

225 orCS1:SorL1, and SorCS1:Vps35 complexes from nontransgenic mouse brain.

226 exes during size exclusion chromatography of nontransgenic mouse lens extracts prepared without chemi

227 Nontransgenic mouse lens proteins incubated with purifie

228 Here, we present a nontransgenic mouse model in which spontaneous metastase

229 blood mononuclear cell culture system and a nontransgenic mouse model in which the impact of stimula

230 In nontransgenic mouse myofibrils, the Ca(2+) sensitivity o

231 ad a 2.4-fold higher Ca(2+) sensitivity than nontransgenic mouse myofibrils.

232 EMD57033 was indistinguishable from that of nontransgenic myofibrils.

233 We describe two routes to generate nontransgenic naive human ES cells (hESCs).

234 In contrast, nontransgenic neonatal CD8(+) T cells when transferred i

235 roinsulin II from a transgene in K cells and nontransgenic NOD mice (controls); pancreas and duodenum

236 creased selection of Tregs was also found in nontransgenic NOD mice in fetal through adult stages.

237 sion of beta-cell autoantigens compared with nontransgenic NOD TLOs found in inflamed islets.

238 As controls, nontransgenic NOD.H2(h4) mice similarly injected with in

239 AD mouse model of AD, compared with those in nontransgenic (non-Tg) controls, at 6, 8, and 14 mo of a

240 old transgenic (TgCRND8) (Tg) mice and their nontransgenic (non-Tg) littermates were entered in the s

241 nsmission of different tau strains occurs in nontransgenic (non-Tg) mice, and to investigate whether

242 ncrease of NAD(P)H levels and redox state in nontransgenic (non-Tg) neurons until middle age, followe

243 wo-photon imaging to study Ca2+ signaling in nontransgenic (NonTg) and several AD mouse models (PS1KI

244 hysiological recordings in young 3xTg-AD and nontransgenic (NonTg) hippocampal slices, we show that i

245 opagate neuronal and glial tau aggregates in nontransgenic (nonTg) mouse brain.

246 c Alzheimer's mice (3xTg-AD) and age-matched nontransgenic (nonTg).

247 AM14 site-directed BCR transgenic mice into nontransgenic normal recipients and elicited an EF respo

248 row of humanized NSG-SGM3 mice compared with nontransgenic NSG recipients.

249 MCK-PGC1alpha mice and nontransgenic (NT) littermates were fed a high-fat diet

250 analysis of brains from transgenic (TG) and nontransgenic (NT) mice 5 days after infection identifie

251 proteomics comparing brain-derived EVs from nontransgenic (NTg) and a transgenic amyotrophic lateral

252 y or excessive memory generalization in both nontransgenic (NTG) and Tg2576 mice.

253 itutively active (CA) PKN (Tg-CAPKN) than in nontransgenic (NTg) mice (15+/-5 versus 38+/-5%, P<0.01)

254 e was similarly accelerated by Iso in Tg and nontransgenic (Ntg) myocytes.

255 ardial strips of the following mouse models: nontransgenic (NTG), effective null for cMyBP-C (t/t), w

256 cific BisANS incorporation sites on GAPDH in nontransgenic (NTg), G93A, and H46R/H48Q mice using liqu

257 y labeled single actin filaments compared to nontransgenic or transgenic wild-type (Tg-WT) control mi

258 umerous (6 +/- 3 transgenic versus 2 +/- 1.5 nontransgenic papillomas per mouse), yet they were more

259 single-positive thymocytes was also seen in nontransgenic PKCtheta(-/-) mice.

260 In situ hybridizations of tissue from nontransgenic plants revealed that the expression of all

261 nsertion flowered significantly earlier than nontransgenic plants, supporting the identity between Ta

262 he RNAi plants had fewer oil bodies than the nontransgenic plants.

263 rrelates with b1 silencing in transgenic and nontransgenic plants.

264 tion of RTBV RNA and viral DNA compared with nontransgenic plants.

265 the genome and generate precisely modified "nontransgenic" plants.

266 In this study, by using a nontransgenic rat model of sporadic AD generated by intr

267 In summary, we provide a novel nontransgenic rat model with high endogenous ghrelin pla

268 transgenic and control (HLA-B7-transgenic or nontransgenic) rats.

269 s were adoptively transferred into syngeneic nontransgenic recipients and their fate in the periphera

270 tected by immunoblot and by inoculation into nontransgenic recipients.

271 In contrast, if tumors were implanted in nontransgenic SCID mice adjacent to a patch of transplan

272 ls and stroma, tumor cells were implanted in nontransgenic SCID mice that received a bone marrow tran

273 ter retransplantation at 14 days to control (nontransgenic) secondary BALB/c recipients, with or with

274 ing CPCs, with 36% of the CPCs versus 73% in nontransgenic sections.

275 tly increased adult survivorship relative to nontransgenic sibling controls.

276 genic kernels weighed the same on average as nontransgenic siblings, with normal endosperm starch and

277 branching and spikelet density compared with nontransgenic siblings.

278 icant difference in survivorship relative to nontransgenic siblings.

279 script accumulation in response to culturing nontransgenic soybean explants on the medium used to ind

280 ssed responses by naive TCR75 T cells and by nontransgenic spleen cells stimulated with anti-CD3.

281 and function of orphan genes, and identify a nontransgenic strategy for modulating protein levels in

282 ChR alpha-chain peptide that dominated young nontransgenic T cell responses in vitro.

283 data indicate that TCR transgenic as well as nontransgenic T(EM) differentiate into T(CM) in the abse

284 ess mature and turned over more rapidly than nontransgenic T2 cells, exhibiting neither conventional

285 Nontransgenic TAC developed diastolic dysfunction (65% i

286 ting the increase in anaplerosis observed in nontransgenic TAC hearts.

287 Isolated perfused hearts from nontransgenic TAC mice showed reduced cardiac function a

288 Contrasting nontransgenic TAC, ssTnI TAC significantly increased glu

289 r ejection fraction (ACSL1 TAC: 65.8+/-7.5%; nontransgenic TAC: 45.9+/-7.3) and improvement in diasto

290 was significantly greater in Tg-Nox4 than in nontransgenic, the LV weight/tibial length was not signi

291 Five mouse lines were used: nontransgenic, those that overexpress wild-type K18 or t

292 lants (0 d in culture) was found to resemble nontransgenic tissue that had been induced for SE by bei

293 d species, but given the 'y' number of other nontransgenic traits that breeders also need to assemble

294 gs were made with wild-type (T-cell receptor nontransgenic) Tregs, and (iii) the B29 human homolog is

295 The nontransgenic w(1118) Drosophila line was used as a cont

296 ts, transgenic mosquitoes gradually replaced nontransgenics when mosquitoes were maintained on mice i

297 es in keratin expression, respectively, with nontransgenic wild-type (WT) mice.

298 Compared with nontransgenic wild-type mice, mice with neuronal Mfn2 ov

299 had twofold increase in superoxide levels in nontransgenic (wild-type [WT]) diabetic mice compared wi

300 (+) regulatory T cells (Tregs) compared with nontransgenic WT mice.