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1 producing (virulent) or non-toxin-producing (nonvirulent).
2 iformly virulent, while types II and III are nonvirulent.
3 g the virulent, intermediately virulent, and nonvirulent.
4 ontrolled, comparing their expression to the nonvirulent ASFV-BA71V strain to identify key genes that
5         These findings provide evidence that nonvirulent bacteria can be exploited as targeting vehic
6 least three phases: a virulent Bvg+ phase, a nonvirulent Bvg- phase, and an intermediate Bvgi phase.
7 ning between a virulent (Bvg(+)) phase and a nonvirulent (Bvg(-)) phase, a process referred to as phe
8       Wild-type Shigella flexneri, but not a nonvirulent derivative, induced human macrophage apoptos
9  gene expression than was infection with the nonvirulent dsTE12Q strain.
10 d decreased expression in either one or both nonvirulent Entamoeba species/strains.
11 rge proportion actually carry Wolbachia in a nonvirulent form, which might affect their longevity and
12 e lipid A molecules of both the virulent and nonvirulent forms of L. interrogans serovar Icterohaemor
13 nd robustly distinguish between virulent and nonvirulent Francisella strains is desirable.
14 e-negative cases were included or not in the nonvirulent group.
15 chovirus 12 (ECV12), a wild-type, relatively nonvirulent human enterovirus, exchanged with the homolo
16                                  However the nonvirulent, hypha-defective cla4 mutant line was consid
17                                    Second, a nonvirulent mutant lacking the esaI gene adheres strongl
18                  In this study, we show that nonvirulent mycobacteria such as Mycobacterium smegmatis
19 d MV production in a variety of virulent and nonvirulent mycobacterial species, indicating that relea
20  polysialylation) were cleared as rapidly as nonvirulent organisms.
21 es (ROS) signaling drives differentiation of nonvirulent promastigotes into forms capable of infectin
22  chemotaxis to a much higher degree than the nonvirulent Rhigh strain.
23 ort the identification of enteric organisms (nonvirulent Salmonella strains) whose direct interaction
24          Two clinical isolates (virulent and nonvirulent) showed equivalent hemoglobin-mediated growt
25 ivirus system was derived from SIVmac1A11, a nonvirulent SIV strain.
26 orm similar biological roles in virulent and nonvirulent species.
27 onvirulent strain, and Entamoeba dispar is a nonvirulent species.
28 nomic evolution, had lower expression in the nonvirulent species/strains than in E. histolytica HM-1:
29 9 genes had decreased expression in both the nonvirulent species/strains than the virulent E. histoly
30 ncidence: a pathogenic strain (CVB3/M) and a nonvirulent strain (CVB3/GA).
31 ith human disease or with an equal dose of a nonvirulent strain (O86:H8) that lacks these factors.
32 ysialic acid-containing capsule, whereas the nonvirulent strain did not.
33 s with the virulent strain compared with the nonvirulent strain were shorter with viable bacteria (5
34  virulent strain, E. histolytica Rahman is a nonvirulent strain, and Entamoeba dispar is a nonvirulen
35 HAdV) may confer virulence upon an otherwise nonvirulent strain.
36                           Using virulent and nonvirulent strains of Bacillus anthracis, we have shown
37 re recognized by the immune system, but only nonvirulent strains were controlled.
38 and III consisted of moderately virulent and nonvirulent strains, which are separated from each other
39 ing active toxins can be differentiated from nonvirulent strains, which do not produce active toxins.
40 stinct from those of moderately virulent and nonvirulent strains.
41 n ROD was virulent whereas strain G622-M was nonvirulent, thus demonstrating that the presence of all
42              STEC strains, both virulent and nonvirulent to humans, are frequently isolated from heal
43                                              Nonvirulent, tumor-tropic bacteria, such as Salmonella t
44 henotype in vitro, that was not expressed by nonvirulent type II and type III strains.
45 bited a superior migratory capacity than the nonvirulent type II and type III strains.
46 ection with parasites of virulent type I and nonvirulent type II genotypes.
47 lial cell polarity during the interaction of nonvirulent (XL1-Blue) and uropathogenic (J96) strains o