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1 including rapamycin, 17-alpha-estradiol, and nordihydroguaiaretic acid.
2 lfonamide dihydrochloride], indomethacin, or nordihydroguaiaretic acid.
3 y inhibited by caffeic acid, resveratrol and nordihydroguaiaretic acid.
4 Similarly, inhibition of lipoxygenase with nordihydroguaiaretic acid (1 micromol/L), eicosatetrayno
5 Treatment with the lipoxygenase inhibitor nordihydroguaiaretic acid (10 microM) did not affect tum
6 by AA or DHA was unaffected by extracellular nordihydroguaiaretic acid (10 microM), indomethacin (10
7 of the AT1 receptor-subtype of AngII, and by nordihydroguaiaretic acid (50 microM), which at this con
13 ed the effects of 5-lipoxygenase inhibitors (nordihydroguaiaretic acid and AA861) on IL-1 beta-induce
14 clusion of the characteristic LOX inhibitors nordihydroguaiaretic acid and eicosatetraynoic acid redu
15 nked lignans, whose representatives, such as nordihydroguaiaretic acid and its congeners, have potent
17 e-1-oxide, the antioxidants desferrioxamine, nordihydroguaiaretic acid, and Amytal, and by the enhanc
19 ffects were not prevented by indomethacin or nordihydroguaiaretic acid, blockers of cyclooxygenase an
21 ne expression is inhibited by tetra-O-methyl nordihydroguaiaretic acid (M(4)N) and that M(4)N is like
22 The transcription inhibitor tetra-O-methyl nordihydroguaiaretic acid (M4N) was found to arrest the
25 Our labs have synthesized and investigated nordihydroguaiaretic acid (NDGA) derivatives and have es
26 of neurons with 8-bromo-cAMP (8-Br-cAMP) or nordihydroguaiaretic acid (NDGA) had no effect on the re
27 gents such as chloroquine (CQ), NH(4)Cl, and nordihydroguaiaretic acid (NDGA) that alter discrete ste
30 agent that does not inhibit COX enzymes; and nordihydroguaiaretic acid (NDGA), a lipoxygenase inhibit
31 ha-synuclein aggregation by novel analogs of nordihydroguaiaretic acid (NDGA), a phenolic dibenzenedi
32 when the 13-LOX activity was inhibited using nordihydroguaiaretic acid (NDGA), a specific inhibitor o
33 the metabolism of AA by cyclo-oxygenase, nor nordihydroguaiaretic acid (NDGA), an inhibitor of the li
35 igated the effects of a hypolipidemic agent, nordihydroguaiaretic acid (NDGA), on host lipid/fatty ac
36 (Dex), mepacrine (Mep), indomethacin (Indo), nordihydroguaiaretic acid (Ndga), phenylephrine (Pe), so
38 ere treated with the lipoxygenase inhibitor, nordihydroguaiaretic acid (NDGA), the 5-lipoxygenase inh
40 se inhibitors 5,8,11-eicosatrienoic acid and nordihydroguaiaretic acid partially prevented MIN6 cell
41 trathecal (IT) delivery of the LOX inhibitor nordihydroguaiaretic acid prevented the carrageenan-evok
42 ng enzymes, only the lipoxygenase inhibitor, nordihydroguaiaretic acid reduced EGF-mediated ROS accum
43 S, pyrrolidine dithiocarbamate, pyruvate, or nordihydroguaiaretic acid, reduced both cellular oxidant
44 n diet-associated constituents, curcumin and nordihydroguaiaretic acid, reversibly targets UGTs causi
45 t with the antioxidants N-acetylcysteine and nordihydroguaiaretic acid, suggesting a role for oxidati
48 ause incubation with 5-LO inhibitors (AA861, nordihydroguaiaretic acid, zileuton) but not a flavoenzy