ライフサイエンスコーパス: normal birth

1 Their levels increased significantly after normal birth.

2 c development that peaked a few weeks before normal birth.

3 erformed in three independent white European normal birth cohorts.

4 hromosomally normal losses and chromosomally normal births combined.

5 Phenotypically, the patient had normal birth height and weight, had normal psychomotor d

6 en whose index pregnancy was a chromosomally normal birth (n=226).

7 for age-matched controls with chromosomally normal births (n = 388).

8 mTOR activity preserves ovarian function and normal birth numbers.

9 Subsequently, until the time of normal birth, the principal development is a proliferati

10 ss gestation comparing adverse outcomes with normal births using mixed-effects models.

11 t (>4 kg) participants were more likely than normal birth weight (>/=2.5-</=4 kg) participants to bec

12 w birth weight (<2,500 g), 41% in those with normal birth weight (2,500-4,499 g), and 64% in those wi

13 respectively) versus mothers of newborns of normal birth weight (2500-4000 g).

14 ed with increased rates of SLE compared with normal birth weight (7-8.5 pounds; rate ratio [RR] 2.7,

15 for N, P = .009) compared with newborns with normal birth weight (77 116 [20.1%] for S and 19 872 [5.

16 Young, healthy men with LBW (n = 55) and normal birth weight (NBW) (n = 65) were examined includi

17 alifornia (the DARLING Study), 2) infants of normal birth weight (NBW) but from low-income families i

18 ne expression, and methylation in LBW versus normal birth weight (NBW) female piglets.

19 58.05, SE = 20.00 microg/ml) compared to the normal birth weight (NBW) group (mean 13.45, SE = 3.92 m

20 lth risk compared to birth at full term with normal birth weight (NBW).

21 men born with LBW and 13 controls born with normal birth weight (NBW).

22 vs 2.3%; P<.01) compared with those born at normal birth weight (NBW).

23 ed with 21 healthy control participants with normal birth weight (NBW).

24 birth weight (MLBW, 1,500-2,499 g); and 794 normal birth weight (NBW, > or = 2,500 g).

25 th weight (MLBW: 1,500-2,499 g; n = 553) and normal birth weight (NBW: > or = 2,500 g; n = 555) singl

26 the pandemic on more than 20 participants of normal birth weight aged 6 to 17 years.

27 In children with normal birth weight and a high prevalence of obesity at

28 Affected individuals presented with low-to-normal birth weight and infantile feeding difficulties b

29 presentative samples of low birth weight and normal birth weight children from the City of Detroit (u

30 sed risk of neuroblastoma compared with term/normal birth weight delivery among infants (OR = 6.99, 9

31 Controls were normal birth weight infants (NBW).

32 die before their first birthday compared to normal birth weight infants.

33 ntrols were 304 women who delivered term and normal birth weight infants.

34 s, only 2.1% of low birth weight and 0.6% of normal birth weight neonates were affected, respectively

35 BW newborns, and 195 who delivered full-term normal birth weight newborns as the controls.

36 Girls with LBW and normal birth weight with no adversities had no adolescen

37 dmfs was 1.86 between children with low and normal birth weight, and 1.66 between children of smokin

38 dence interval 0.72-7.2) that in subjects of normal birth weight, and the odds in subjects of high bi

39 Infants with TGA had normal birth weight, but lesser head volume relative to

40 egenerative disorder characterized by low to normal birth weight, growth failure, brain dysmyelinatio

41 Clinical manifestations included normal birth weight, late onset of hypoglycemia, diazoxi

42 In comparison with mothers of newborns with normal birth weight, mothers of newborns with low birth

43 In children of normal birth weight, obesity is positively associated wi

44 al: 1.50, 2.14)) outcomes in comparison with normal birth weight.

45 D status at 4 y in 323 Chilean children with normal birth weight.

46 CI, 5.2%-13.3%) among adolescent girls with normal birth weight.

47 of in girls with LBW more than in girls with normal birth weight.

48 is associated with risk of CP in children of normal birth weight.

49 marked increase in risk of CP in infants of normal birth weight.

50 egenerative disorder characterized by low-to-normal birth weight; growth failure; brain dysmyelinatio

51 prevalence of U between low-birth weight and normal-birth weight children.

52 The role of genes in normal birth-weight variation is poorly understood, and

53 on gene variants can substantially influence normal birth-weight variation.

54 hose of low to moderate parity who delivered normal-birth-weight babies (adjusted odds ratio = 3.53,

55 5% CI, 0.6-6.3 mum; P = .02) thinner than in normal-birth-weight children after adjustment for all va

56 ever, the consistency of this association in normal-birth-weight children and its potential mediators

57 ognitive scores between low-birth-weight and normal-birth-weight children was large in the NCDS [-0.3

58 0 g) in their mid-20s was similar to that of normal-birth-weight controls (>2500g), there was uncerta

59 cases (n = 33) had infants weighing <2500 g; normal-birth-weight controls (n = 390) had infants weigh

60 t women, were significantly less likely than normal-birth-weight controls to be enrolled in postsecon

61 ug use and had lower rates of pregnancy than normal-birth-weight controls; these differences persiste

62 Four control pregnancies resulting in normal-birth-weight infants (> or =2500 g) were randomly

63 12, there were 43.0 NICU admissions per 1000 normal-birth-weight infants (2500-3999 g), while the adm

64 t age 2 months, there were 173373 full-term, normal-birth-weight infants enrolled as controls; at age

65 re than 2500 g, and 65% to 76% of full-term, normal-birth-weight infants.

66 A total of 7012 healthy normal-birth-weight neonates were randomized to BCG only

67 , and 89 of 144 sociodemographically matched normal-birth-weight term controls (61.8%) recruited at a

68 low-birth-weight survivors and 89 (33 males) normal-birth-weight term controls.

69 ewer very-low-birth-weight young adults than normal-birth-weight young adults had graduated from high

70 r intelligence quotient (IQ) than those with normal birth weights (NBW, >/=2500 g).

71 studied a control group of 43 children with normal birth weights.

72 rom the same population in Cleveland who had normal birth weights.

73 orted malformations, and 68% of neonates had normal birth weights.