ライフサイエンスコーパス: normal control

1 nd II plasma tested was less than 40% of our normal control.

2 nts possessing different FUS mutations and a normal control.

3 03 HD patients and 103 healthy volunteers as normal control.

4 group showed increased levels comparable to Normal-Control.

5 quire data from reference samples or matched normal controls.

6 ein, in some cases more than 50% relative to normal controls.

7 ease, had poorer quality of life compared to normal controls.

8 iety of pediatric cancer lines compared with normal controls.

9 monkeys (Macaca nemestrina) and two visually normal controls.

10 tabolic syndrome compared with metabolically normal controls.

11 men and 21 women) were age- and sex-matched normal controls.

12 nochoroidopathy and 37 eyes of 37 historical normal controls.

13 adults with poorly controlled asthma and 10 normal controls.

14 poral dementia (FTD) and matched cognitively normal controls.

15 c profiles of PD were clearly different from normal controls.

16 tic patients without retinopathy compared to normal controls.

17 ures are indistinguishable from age-matched, normal controls.

18 rment and AD dementia compared to clinically normal controls.

19 tients with acoustic neuromas compared to 24 normal controls.

20 ndemented controls compared with age-matched normal controls.

21 of seizures, and 19 age-matched, cognitively normal controls.

22 opa metabolites and biliverdin than those of normal controls.

23 nary artery vasculopathy with 35 age-matched normal controls.

24 pective study of individuals with CRSwNP and normal controls.

25 9 eyes had proliferative DR, and 4 eyes were normal controls.

26 decreased hippocampal activity, relative to normal controls.

27 GA region did not significantly deviate from normal controls.

28 te-onset Chinese AD patients and cognitively normal controls.

29 n participants with early AD and cognitively normal controls.

30 wedish AD cases, and 707 Swedish cognitively normal controls.

31 ciated Stargardt disease (STGD1) relative to normal controls.

32 ith early, diffuse SSc compared with that in normal controls.

33 epair for isolated MR, were compared with 13 normal controls.

34 ncreased in PDAC patients by comparison with normal controls.

35 : Age and sex matched healthy individuals as normal controls.

36 umented dogs, 8 with pacing-induced HF and 6 normal controls.

37 n capillaries of diabetic animals but not in normal controls.

38 d BRB (with normal fundus or initial DR) and normal controls.

39 though higher than age-matched, cognitively normal controls.

40 ls of diabetic animals (P<0.01), compared to normal controls.

41 ere no false positives in the 25 age-matched normal controls.

42 losive disorder compared with psychiatric or normal controls.

43 fficient to distinguish these specimens from normal controls.

44 y and could be completely distinguished from normal controls.

45 atients whose disease was in remission or in normal controls.

46 ic leukemia, whereas it was not expressed in normal controls.

47 sample of 200 angle closure subjects and 302 normal controls.

48 ntly decreased in pups with NEC, compared to normal controls.

49 in athletes with comparable hypertrophy and normal controls.

50 nes (DEGs) in pathologic tissues relative to normal controls.

51 23 eyes without rejection) and 9 age-matched normal controls.

52 he individual samples of 38 HCC serum and 24 normal controls.

53 enhanced PECAM-1 expression when compared to normal controls.

54 nts, mild cognitive impairment subjects, and normal controls.

55 compared with those in bronchiolar walls of normal controls.

56 he expression of all mature tRFs in CLLs vs. normal controls.

57 oliferation and tube formation compared with normal controls.

58 ferral practice with diabetes, glaucoma, and normal controls.

59 ntral striatum than did age- and sex-matched normal controls.

60 han in myopic subjects without staphyloma or normal controls.

61 sleep-onset insomnia as compared to healthy normal controls.

62 nts are down-regulated by 5-fold in CLLs vs. normal controls.

63 1% for serum from patients with DLBCL versus normal controls.

64 n their peripheral blood, in comparison with normal controls.

65 the submacular RPE on OCT when compared with normal controls.

66 ontotemporal dementia against neurologically normal controls.

67 ive CNV due to AMD, non-neovascular AMD, and normal controls.

68 ity of Pittsburgh (U. Pitt., n = 70) than in normal controls.

69 e to TE was higher than that measured in the normal controls (1.07%).

70 In comparison with data from normal controls, 12 patients had normal (11)C-HED PET, 5

71 patients with APS (21.8%) or SLE (28.6%) or normal controls (23.3%), and have mutations at a rate si

72 2.05 mum) was significantly less compared to normal control (245 +/- 15.87 mum; P = .004).

73 tly higher in patients with chronic HCV than normal controls (65.9 % vs 28.7 %, respectively).

74 in 141 lower-limb involved ALS patients, 218 normal controls, 67 disease controls, and 32 lumbar spon

75 12 normal controls, 7 patients with primary open angle glau

76 d in 8943 US AD cases, 10 480 US cognitively normal controls, 862 Swedish AD cases, and 707 Swedish c

77 y lower (worse) PedEyeQ scores than visually normal controls, across functional vision and eye-relate

78 sed tracer retention compared to cognitively normal controls, adjusting for age as a covariate.

79 (aged 53.7 +/- 9.8 years) and 30 eyes of 15 normal controls (aged 50.7 +/- 9.8 years).

80 l RNAs are similar between sickly plants and normal controls, although a few upregulated transcripts

81 modulator (GSM) in neurons derived from both normal control and 3 PS1 mutations (A246E, H163R, and M1

82 RNA in MLL-rearranged AML compared with both normal control and non-MLL-rearranged AML.

83 Normal rats were used as normal control and were divided into NC and NR group fed

84 r thickness (CMT) were measured using OCT in normal controls and 3 months postoperatively in children

85 CD8(+) T cells, reaching levels observed in normal controls and also modestly increased the number o

86 ant difference expression (p < 0.05) between normal controls and gastric cancer patients, including 7

87 Compared to both normal controls and hypertensive heart disease patients,

88 er that accounted for the difference between normal controls and iRLS data.

89 sis had higher serum levels of IFNgamma than normal controls and patients with ET showed higher IFNga

90 icited by HDACi 4b in human fibroblasts from normal controls and patients with Huntington's disease (

91 ved from mutant Huntingtin (mHTT) embryos or normal controls and performed RNA sequencing (RNA-seq) t

92 ressed in ESCC tissues and cells compared to normal controls and that its overexpression is correlate

93 r518Met mutation was absent from 362 matched normal controls and was extremely rare in a large popula

94 Of these, 42 subjects had IED, 40 were normal controls, and 50 were clinical psychiatric contro

95 e cancer were more efferocytic compared with normal controls, and CXCL5 serum levels were higher in m

96 stinguish different disease types as well as normal controls, and highlight the importance of cell co

97 and occipital cortex compared to clinically normal controls, and in probable DLB, the uptake in thes

98 fold (P=0.005) higher in MR patients than in normal controls, and LV ejection fraction was 64+/-7% (5

99 patients with COPD compared with those from normal controls, and the levels were correlated with pul

100 re increased in FSGS glomeruli compared with normal controls, and urinary excretion of both cathepsin

101 sentation of a noise mask to the FE, as in a normal control animal.

102 differential methylation calling method when normal controls are not available.

103 ta and right carotid artery as compared with normal control arteries (mean TBR = 1.95 +/- 0.51 vs. 1.

104 ta and right carotid artery as compared with normal control arteries (mean TBR = 1.95 +/- 0.51 vs. 1.

105 umes, lower RVEF and global area strain than normal controls as expected.

106 thrombin time > 20 s compared with that of a normal control), ascites, splenomegaly, portal hypertens

107 ne tumours plus eight human and three murine normal controls, augmented with matched whole-genome, RN

108 olar I disorder in the euthymic state and 25 normal controls balanced for age and gender.

109 comparative real sample analysis of healthy normal (Control), benign and ovarian cancer patients wit

110 in idiopathic narcolepsy-cataplexy and with normal control brains.

111 th AQP4-IgG- subjects, patients with MS, and normal controls) but not in photopic ERGs.

112 je cells in 15 HD cases and 8 neurologically normal control cases.

113 Compared with age-matched normal controls, CD7(+) lymphocytes, NK cells, and CD16

114 ivo xenografts in mice, but had no effect on normal control cells.

115 s obtained from asthmatic donors compared to normal control cells.

116 tween malignant hyperthermia-susceptible and normal controls cells.

117 ed with renal tubules from kidney samples of normal controls, cysts in kidney samples from patients w

118 perimental designs including with or without normal controls, different sources of normal tissue cont

119 nvasive hemodynamic data were collected from normal controls, dyssynchronous HF and CRT models.

120 ucoma suspects (6 eyes from 3 subjects), and normal control eyes (16 eyes from 9 normal subjects).

121 m(2); 6 x 6 = 1,104 mum(2)) compared to both normal control eyes (752 mum(2); P < .0001; 802 mum(2);

122 A total of 43 diabetic and 11 normal control eyes were evaluated with OCTA.

123 A cohort of diabetic and normal control eyes were imaged with a prototype SS-OCT

124 Compared to normal controls, eyes with pLPC may present a higher pro

125 A total of 38 foetuses with IUGR and 18 normal control foetuses with similar gestational age wer

126 re WM differences between the simple T2h and normal control for both preterm and full-term neonates,

127 on was assessed in HFpEF, and compared to 50 normal controls free of cardiovascular disease and to 44

128 s of 86 prediagnostic samples and 86 matched normal controls from a high-risk cohort revealed 48 prot

129 her in the group with CP (85.7%) than in the normal control group (73.3%; P <0.03).

130 myopic subjects compared with an age-matched normal control group using ultra widefield optical coher

131 Comparing with the normal control group, the GM and WM density at each leve

132 h hippocampal damage (moderate/severe) and a normal control group.

133 h "moderate" hippocampal damage (MHD), and a normal control group.

134 in myopic eyes compared with an age-matched normal control group.

135 whereas it decreased in glaucoma control and normal control groups by 2.0 mm Hg (P = .003) and 2.1 mm

136 enia, bipolar disorder, major depression and normal controls (>700 subjects).

137 UGR offspring were similar to those found in normal controls in a geriatric cohort.

138 l adenoma patients and 39 colonoscopy-proven normal controls in order to find potential circulating m

139 tagonistic signals that were even lower than normal controls in the double CAM alpha(1)A/B-AR mice fo

140 VHs exhibited reduced GM volumes relative to normal controls in the left superior temporal gyrus, fro

141 2 diabetes without clinical retinopathy and normal controls, in order to assess possible increased m

142 Normal controls included chest CTs from oncology, emerge

143 of 162 samples were analyzed, composed of 91 normal control individuals (51 male, 33 female, and 7 no

144 s with cross-sectional data from cognitively normal control individuals (age 65-89 years) who were ne

145 set representing the spectrum of cognitively normal controls, individuals with mild cognitive impairm

146 ene expression profiling was performed for 4 normal (control) livers as well as 8 background liver an

147 ophic lateral sclerosis patients compared to normal controls, measuring the rates of production of re

148 Compared with normal controls, MF skin displayed increased mRNA levels

149 unts, and lower mean corpuscular volume than normal control mice, a phenotype that becomes more evide

150 roke compared with the recolonization with a normal control microbiota.

151 In normal controls, miR-127-3p, miR-382-5p and miR-425-3p c

152 on of Bq reduced the discrepancy between the normal control model and the iRLS data by 92.1%.

153 mild bilateral disease (n = 12) and matched normal controls (n = 12) using in vivo high-resolution p

154 t sample with Parkinson disease (n = 15) and normal controls (n = 13) in the substantia nigra and put

155 nd NFKB in specimens of rosacea (n = 15) and normal controls (n = 14).

156 In this cross-sectional study, normal controls (n = 145) from the Alzheimer's Disease N

157 affected eyes were studied and compared with normal controls (n = 15).

158 y randomly assigning Wistar rats (n = 28) to normal controls (n = 4), CDAA diet with vehicles, and CD

159 inical research criteria for PSP (n = 33) to normal controls (n = 46) and patients meeting criteria f

160 from dystrophinopathy patients (n = 28) and normal controls (n = 6).

161 sis of very mild AD (n = 23) and cognitively normal controls (n = 64) who were enrolled in longitudin

162 AD dementia (n = 19) patients and clinically normal controls (n = 95) from an epidemiological cohort

163 ressure [PASP] >/=40 mm Hg) were compared to normal controls (n=12) and patients with primary pulmona

164 sAPPbeta between groups and with cognitively normal controls (n=77), and assessed their diagnostic ut

165 nondemented patients (10 LPD, 10 RPD) and 11 normal control (NC) adults.

166 with noncirrhotic liver disease (DC); and 88 normal control (NC) healthy volunteers.

167 intracellular calcium concentration in both normal control (NC) rats and DCP rats, but the sensitivi

168 26 normal control (NC), 37 AD, and 24 patients with PSP par

169 lated tau concentrations were compared in 47 normal controls (NC), 8 asymptomatic gene carriers (NC2)

170 rks in RIS subjects, demographically matched normal controls (NC), and relapsing-remitting (RR) MS pa

171 switching are increased compared with AR and normal controls (NC).

172 141 plasma samples (96 PD, 45 neurologically normal control [NC] individuals; 45.4% female, mean age

173 ary disorders (BHD) and 20 from age-matched "normal controls"(NC).

174 CSU patients, 10 AD patients, and 10 healthy normal controls (NCs) and measured contents of FBPs (pyr

175 d cognitive impairment (MCI), in cognitively normal controls (NCs), and in cognitively normal apolipo

176 using western blots of postmortem ACC in: 1) normal controls (NCs, n=13) vs subjects with SZ (n=25);

177 QR] = 1.6-6.7 hours) and from neurologically normal controls (NCs; N = 112).

178 Heavy drinkers (HD, N=16, 16 males) and normal controls (NM, N=24, 14 males) were tested after p

179 re cortical responses in color-anomalous and normal control observers.

180 f the basal ganglia, playing a major role in normal control of behavior and in the pathophysiology of

181 These plants retained normal control of degradation.

182 of covert attention and is necessary for the normal control of spatial attention during perceptual ju

183 ritis (CAIA) in mice; the mechanisms whereby normal control of the AP is overcome and injury develops

184 lish that somatic mutations in DP-1 uncouple normal control of the E2F pathway, and thus define a new

185 singly, the D40Y and R44H mutant RGS2 showed normal control of vasodilation.

186 inuous (mmHg) and binary ( 130/80 mmHg, i.e. normal/controlled or high/uncontrolled BP) variables.

187 patients with symptomatic AD and cognitively normal controls over time.

188 ficantly higher in patients with cHL than in normal controls (P < .0001).

189 ed MPTP hemispheres relative to those of the normal controls (P < 0.00005) but was reduced (P < 0.05)

190 aseline in 57.6% of DED patients vs.10.5% of normal controls (P < 0.0001).

191 rafts (P = 0.004) and different from that of normal controls (P = 0.001).

192 ave the dominant TLR7 rs179010-T allele than normal controls (p = 0.0435).

193 a was in the capillaries, compared to 47% in normal controls (P<0.01).

194 ; P < .001) in transgenic mice compared with normal, control pancreata of WT mice (mean intensity, 0.

195 ve mild cognitive impairment, stable MCI and Normal Control participants).

196 nd included a total of 308 participants (181 normal control participants, 65 healthy siblings, and 62

197 for differences in neural activation across normal control participants, patients with schizophrenia

198 inations, or mean deviation [MD] >-5 dB) and normal control participants.

199 encoding of novel stimuli when compared with normal control participants.

200 mosaic regularity in aniridia compared with normal control participants.

201 roke patients without diabetes compared with normal control patients, while in stroke patients with d

202 control patients with diabetes compared with normal control patients.

203 Compared with normal controls, patients with PH had a 2-fold increase

204 dhesion and spreading defects as compared to normal controls, PI3K/mTOR hyperactivation interfered wi

205 ined cell cycle markers in IBM compared with normal control, polymyositis (PM) and non-inflammatory d

206 mer disease (AD) cases and 5,096 cognitively normal controls primarily of European ancestry (EA), amo

207 tory verbal hallucinations (AVHs) to that of normal controls remain controversial.

208 ts served as controls (Steatotic-Control and Normal-Control, respectively; n=20 for each group).

209 We found that in contrast to the normal controls, respiration in Fontan patients had a si

210 In contrast, Betz cells of AD patients and normal controls retain cellular integrity in the motor c

211 t (13 individuals with PKD1 mutations and 18 normal controls) revealed that of 2008 ELV proteins, 9 (

212 ytokines in patients with HCC, compared with normal controls, revealing a network of potential mechan

213 r patient specimens compared with the paired normal control samples.

214 hippocampal subfields, CA1 and CA3, from 32 normal controls showed that distinct co-expression modul

215 alamic volumes of the patients with AVHs and normal controls showed that the area under the curve was

216 Subjects with MCI, compared with normal controls, showed differential associations of olf

217 GF-beta1 by these lymphocytes in contrast to normal control specimens.

218 Participants (n = 42 normal controls) spent 3 consecutive nights in the labor

219 24) than in age- and sex-matched cognitively normal control subjects ( n = 24).

220 19-54 years; mean age, 41.2 years) and eight normal control subjects (age range, 22-56 years; mean ag

221 tive impairment (n = 13), and 21 cognitively normal control subjects (ages 41-76, nine male).

222 with PXG compared with patients with PXS and normal control subjects (all P < .001) without a differe

223 LVH (mean age, 56.41 +/- 2.78 years), and 12 normal control subjects (mean age, 55.67 +/- 3.08 years)

224 h congenital AS (median age 16 years) and 27 normal control subjects (median age 16 years) were evalu

225 We compared normal control subjects (n = 52), a septic shock no cort

226 impairment (MCI; n = 75), or neurologically normal control subjects (NC; n = 137) at the University

227 olecularly confirmed BVMD were compared with normal control subjects (NCs).

228 acuities of 20/40 or better, and 10 visually normal control subjects aged 22 to 65 years.

229 pants; the discovery phase study enrolled 51 normal control subjects and 183 HF patients, and the val

230 were explored in an independent sample of 36 normal control subjects and 35 unaffected siblings durin

231 and bronchoalveolar lavage (BAL) cells from normal control subjects and untreated HIV-1-infected ind

232 age) with an eye condition and 104 visually normal control subjects completed the Child PedEyeQ (fun

233 ns are actively involved when neurologically normal control subjects name visually presented objects,

234 ozygous subjects (28 +/- 7 years), and eight normal control subjects pair-matched to the heterozygous

235 h asthma (19 with and 15 without EIB) and 10 normal control subjects to examine in vivo differences i

236 The PD control cases and normal control subjects were matched first for age at de

237 iment, 17 patients with schizophrenia and 24 normal control subjects were presented in 3 T magnetic r

238 fficient cystic fibrosis (PS-CF), PI-CF, and normal control subjects, all with normal glucose toleran

239 n was lower in PI-CF compared with PS-CF and normal control subjects, and glucagon-like peptide 1 and

240 the PFC in schizophrenia, compared with the normal control subjects, but not in bipolar disorder.

241 d lipid-raft membrane domains in tissue from normal control subjects, depressed suicides, and depress

242 d differentiate gastric cancer patients from normal control subjects, dramatically (p < 0.05).

243 d glucagon responses compared with PS-CF and normal control subjects, indicating reduced beta-cell se

244 In normal control subjects, Nt of SST-IR neurons varied acc

245 ary motor cortices isolated from post-mortem normal control subjects, patients with familial ALS (fAL

246 analyses as well as exclusion testing of 500 normal control subjects, we demonstrated that this genet

247 n, glucose was higher in PI-CF compared with normal control subjects.

248 date and putamen of nine TS and nine matched normal control subjects.

249 h MPS IV, and 6 patients with MPS VI) and 20 normal control subjects.

250 losive disorder compared with psychiatric or normal control subjects.

251 Noncontrast studies were completed in the normal control subjects.

252 al pathway was used by our 24 neurologically normal control subjects.

253 ILD but not subjects without lung disease or normal control subjects.

254 ted in a separate group of schizophrenia and normal control subjects.

255 metrics were compared between subgroups and normal control subjects.

256 patients with diabetes without stroke and 30 normal control subjects.

257 Subjects were divided into NAFLD group and normal control (subjects number = 564 in each group) fol

258 rent in cerebrospinal fluid (CSF) from young normal controls, suggesting that the amount of apoE isof

259 r lavage (BAL) fluid of 10 asthmatics and 15 normal controls taken before, at day four during and 6 w

260 ur adult PTSD patients and 23 trauma-exposed normal controls (TENC) underwent 4T MRS of the left and

261 lity of life of 307 glaucoma patients and 76 normal controls that were frequency matched to the age a

262 splantation in Steatotic-Control (P=0.06 vs. Normal-Control), the VSOP-NO group showed increased leve

263 Compared with the epithelial thickness in normal control, the epithelial thickness in LSCD patient

264 consistantly greater in HCC when compared to normal control tissue (p-value = 0.06) and, in general,

265 to adjacent liver tissue (p-value =0.08) and normal control tissue (p-value =0.02).

266 f the larger tumors as well as from adjacent normal control tissue and microsatellite instability (MS

267 ered abundance in MYC lymphoma compared with normal control tissue by statistical analysis with a fal

268 emethylated in patient-derived compared with normal control tissue-derived epithelial cells.

269 obtained from allergic tissue compared with normal control tissue.

270 py was a logarithmic function of ROI area in normal control tissues (aorta, psoas) and in mathematica

271 of this variant in AD cases and cognitively normal controls to determine whether this variant will s

272 ts in cancer studies, they are often used as normal controls to identify genes differentially express

273 ging eyelid severity levels, ranging from 1 (normal control) to 4 (severe sagging).

274 h the ASDs relative to 12,544 neurologically normal controls, to find potentially druggable genetic t

275 Furthermore, in normal controls, TRNT1 protein levels are 10-fold lower

276 derived from young, non-autopsy, cognitively normal controls used as a standard for tau negativity.

277 vena cava (IVC) of 14 Fontan patients and 11 normal controls using a novel approach ("physio-matrix")

278 False-positive rate in 52 normal controls was 2%.

279 patients with symptomatic AD and cognitively normal controls was performed.

280 and should have been 'possible', 'Autologous normal controls' was listed as 'Unknown' and should have

281 a large cohort of 1,098 HCC patients and 835 normal controls, we constructed a diagnostic prediction

282 gton State and 644 unrelated, neurologically normal controls, we examined whether PD was associated w

283 c cholestasis as diseased controls and seven normal controls, we identified 15 genes uniquely express

284 Patients from a dry eye clinic and normal controls were assessed by Schirmer's test for tea

285 Cognitively normal controls were classified using the longitudinal c

286 rwise specified (FSGS-NOS) or cFSGS and from normal controls were distinguished and quantified using

287 cal, and corneal conditions) and 59 visually normal controls were enrolled at 2 centers.

288 the eyes of amblyopes compared with visually normal controls were found.

289 Twenty-nine CRAO cases at acute phase and 33 normal controls were included.

290 set depression (LOD), 32 LOD patients and 39 normal controls were recruited and underwent resting-sta

291 nsic connectivity disruption, as compared to normal controls, were obtained for 27 patients subsequen

292 ents with high myopia without staphyloma and normal controls, when staphyloma was present, there was

293 nature that differentiated IPF patients from normal controls, which may allow for accurate diagnosis

294 osis of early symptomatic AD and cognitively normal controls who were enrolled in longitudinal studie

295 Cognitively normal controls whose CSF VILIP-1, tau, or p-tau181 leve

296 ) relative to that observed in myotubes from normal control (wild-type and/or heterozygous) myotubes.

297 ied 112 subjects: 92 patients with PH and 20 normal controls with 3D wall motion tracking for RV end-

298 , 14 females, 64.8 +/- 10.1 years; versus 13 normal controls with similar age/sex distributions); and

299 rrent treatment except glasses), 48 visually normal controls without glasses, and 19 controls wearing

300 red in the follicular fluid between PCOS and normal control women, correlating with age, FAI, inflamm