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1  level of </=2 IU per deciliter, or </=2% of normal levels).
2  the epithelial barrier function to a nearly normal level.
3 oxp3- effector T cells (Teffs) remained at a normal level.
4 r formation, and reduces TRPM7 expression to normal levels.
5 id not affect the growth CRC cell lines with normal levels.
6 ormed abnormal complexes but were present at normal levels.
7 s likely to have periodontitis as those with normal levels.
8 utant nodules increased nitrogen fixation to normal levels.
9 XCL13 at relapse including 3 with previously normal levels.
10 ntact time between secretion and EGJ towards normal levels.
11 lial damage (thrombomodulin) markers to near-normal levels.
12 ation patient lymphoblasts restores MeCP2 to normal levels.
13 ponse, decreasing excess spermidine pools to normal levels.
14 ors and increased expression of TGF-beta1 to normal levels.
15 stablished measure of muscle damage, to near-normal levels.
16 ly restores p70S6K activity and cell size to normal levels.
17 ted basally and were increased by insulin to normal levels.
18 ths after transplantation, but did not reach normal levels.
19  by restoring penile PDE5 gene expression to normal levels.
20 class switching from IgM to IgG1 at 5-10% of normal levels.
21 le were still present at 50% (or greater) of normal levels.
22 reas renal EPO was down-regulated to 6.7% of normal levels.
23 "liking" reactions to approximately one-half normal levels.
24 : then, a year or so later, they declined to normal levels.
25 ostnatal proliferation and spatial memory to normal levels.
26 eficient animals express slow fiber genes at normal levels.
27 o I-SceI-dependent class switching at almost normal levels.
28 tructure brought the PLP1/DM20 ratio to near normal levels.
29 main-containing inositol 5'-phosphatase 1 to normal levels.
30 pair, the extension rate recovered to nearly normal levels.
31 ry state induced by periadolescent MK-801 to normal levels.
32 cient to restore anchoring fibril density to normal levels.
33 downstream pro-fibrotic TGFbeta signaling to normal levels.
34  DRL/DCL, receptor densities had returned to normal levels.
35 TA biomarkers improved but did not return to normal levels.
36 itive valence and followed by short decay to normal levels.
37 was rapidly restored by decreasing the pH to normal levels.
38 t-induced secretion of bFGF and VEGF to near normal levels.
39 decrease in IL excitability was increased to normal levels.
40 NAT2 expression in rTg4510 tauopathy mice to normal levels.
41 tored M3R-stimulated insulin release to near normal levels.
42  thylakoid membrane complexes accumulated to normal levels.
43 c triglyceride levels were fully restored to normal levels.
44 nt with dichloroacetate restored PDH flux to normal levels (0.018 +/- 0.002 s(-1)), reversed diastoli
45 <240 U/L), albumin level, 2.6 g/dL (26 g/L) (normal level, 3.5-5.0 g/dL [35-50 g/L]), and creatinine
46    Interestingly, c-Jun protein rebounded to normal levels 4 h following U0126 exposure but not after
47 or overall survival compared with those with normal level (60.0% vs. 83.6%, log-rank test, p = 0.035)
48 ion would be necessary to maintain stress at normal level according to our results and therapeutic ap
49  drugs that might restore gene expression to normal level across the Br and T modules.
50 r1p are antiprion system components at their normal levels, acting with Hsp42.
51 n mutant (DeltassrA) colonized the host to a normal level after 24 h, it produced only 2/10 the lumin
52 sumption after 1 h of exposure, returning to normal levels after 16 h.
53 respiratory allergic patients that return to normal levels after steroid administration.
54      Post-RYGB, 49 proteins were returned to normal levels after surgery.
55 ed increased mortality that declines to near-normal levels after the first several years, but in our
56             We observed a restoration of the normal level alphaB-crystallin in the striatum of the BA
57 ibrin generation to approximately 78% of its normal level and hence improve clot formation under dilu
58 y), disrupted by IR injury, were restored to normal level and the induction of ventricular tachycardi
59 ubes boosts PI3K-AKT-mTOR signaling to supra-normal levels and fully rescues myotube formation.
60 -DG in the regenerating fibers reaches up to normal levels and lasts for >4 weeks, but no up-regulati
61 s restored resting mean arterial pressure to normal levels and remarkably alleviated naturally occurr
62  TLR2 signaling restored the inflammation to normal levels and the ability to clear Lm in Pxr(-/-) mi
63  the protein product was expressed at nearly normal levels and was functional in the NKTCL cell lines
64 etter restoration of complete blood count to normal level, and significantly longer median survival a
65  Csf1r were expressed on the cell surface at normal levels, and bound CSF1, but were not able to sust
66 PS I content and activities were restored to normal levels, and cells again produced thylakoids that
67 cy of cell division when DamX is produced at normal levels, and inhibiting cell division when DamX is
68 ls, increased serum levels of ALT (even high-normal levels) are associated with markers of cardiovasc
69 degree of disease activity, and increased to normal levels as a result of anti-TNF-alpha therapy.
70 esponse to relative hypoxia and recovered to normal levels at the end of blood vessel formation.
71                     POT1(CP)was expressed at normal levels, bound TPP1 and telomeres, and blocked ATR
72  BEP treatment, the risk of CVD decreased to normal levels, but after 10 years, increasing risks were
73                 Restoration of p75ECD to the normal level by brain delivery of the gene encoding huma
74 KO triple mice reduced the liver size to the normal level by decreasing lipid storage in both hepatoc
75 BP1-knockdown Huh7.5 cells was restored to a normal level by downregulation of either p53 or BCCIP.
76 the peripheral T cells were repopulated to a normal level by syngeneic bone marrow transplantation.
77 01) despite recovery of touch sensitivity to normal levels by 6 months.
78     Remarkably, returning Mtor and Rictor to normal levels by deleting one allele of Mtor and one all
79  patient's fibroblasts were also restored to normal levels by exogenous expression of wild-type MRPS7
80 in MeCP2 mutant INs, which were recovered to normal levels by JQ1.
81 ine co-transport stimulation was reversed to normal levels by ketotifen.
82 y reduced endocytosis, which was restored to normal levels by PLS3 overexpression.
83   The salt tolerance of sos2 was restored to normal levels by wild-type SOS2, but not by a mutated fo
84 in cancer cell lines to approximately 50% of normal levels causes a delay in the cell cycle and accum
85 st, thymus markers were delayed but achieved normal levels, concurrent with complete loss through apo
86 t and Flt3 ligand restores cDC1 abundance to normal levels, decreases cDC1 apoptosis, and repairs cDC
87 expression pattern of these mediators toward normal levels during 4 weeks of treatment.
88 ectivity of newborn neurons were reversed to normal levels during remyelination.
89     CD3 T-cell counts gradually recovered to normal levels during this period but CD8 T cells recover
90  these circulating microRNAs decrease toward normal levels during treatment with N-acetyl cysteine (N
91    Although Est1/2 binding to TLC1 occurs at normal levels, Est1 (and hence Est3) binding is highly u
92  can be manipulated to enhance GSIS to supra-normal levels even in the face of defective mitochondria
93 group the majority of amino acids were below normal levels, except for phenylalanine.
94 mice with PPARgamma expression at 25% of the normal level exhibited high autoantibody levels and deve
95 tored dopamine neuron population activity to normal levels following CMS; LHb inactivation had no res
96 iosidosis mouse brain, which are restored to normal levels following weekly intracerebroventricular d
97                 Counts did not usually reach normal levels for age, but patients were able to clear p
98                                         This normal-level Hsp104 curing is promoted by Sti1p, Hsp90,
99 he enhanced response to PM(2.5) plus O(3) to normal levels in an established model of chronic allergi
100 al diet, the gut microbiome recovered toward normal levels in both controls and GWI agent-treated mic
101 ome that is elevated in PWID, can regress to normal levels in former injection drug users who are HCV
102  of stem cells and restored their numbers to normal levels in Iduna mutants.
103 (<1 year) T1D patients tested, but return to normal levels in individuals diabetic for >1 year.
104    IL-22 frequencies serially increased from normal levels in infants to highly significant levels in
105 s DEmiRs and target DEGs to their respective normal levels in inflammation, mitochondrial function an
106 iac hypertrophy (PCH) and restoring GDF11 to normal levels in old mice rescued PCH.
107 me activity for 6 mo at approximately 30% of normal levels in one animal, and in excess of normal lev
108 Serum thrombopoietin (TPO) was maintained at normal levels in Pf4-Cre-positive Jak2(f/f) mice, consis
109 ated in Tbx1(+/-) embryos and is restored to normal levels in Tbx1(+/-);Trp53(+/-) embryos.
110 t, with markers of inflammation returning to normal levels in the CSF, where they were sustained at w
111 tability and levels of KCC2 and NKCC1 toward normal levels in the lumbar spinal cord.
112 enerated mice in which Mecp2 remains at near normal levels in the nervous system, but is severely dep
113 , assessed by western blot, was below 15% of normal levels in the nine mutation carriers in whom this
114 ormal levels in one animal, and in excess of normal levels in three animals.
115              Return of Dyrk1a copy number to normal levels in Ts65Dn mice rescued the appendicular bo
116                       Reducing hematocrit to normal levels in tubular Vegfa-deficient mice resulted i
117 TL target protein is unable to accumulate to normal levels in ubp mutants.
118 aneously restoring fibrinogen (to 88% of its normal level) in diluted blood can restore fibrin genera
119 s that express APP and processing enzymes at normal levels is not well understood.
120 s likely to have periodontitis as those with normal levels (&lt;/=199 mg/dL), and those with STC >/=240
121 creatinine level, 1.7 mg/dL (150.3 mumol/L) (normal level, &lt;1.2 mg/dL [<106.1 mumol/L]).
122 l level, <300 U/L); amylase level, 1435 U/L (normal level, &lt;140 U/L); lactate dehydrogenase level, 25
123  phosphatase level of 85 U/L (1.42 mukat/L) (normal level, &lt;142 U/L [2.37 mukat/L]), a serum creatini
124 0.8-2.0), a urine protein level of 15 mg/dL (normal level, &lt;20 mg/dL), a total serum calcium level of
125  U/L); lactate dehydrogenase level, 253 U/L (normal level, &lt;240 U/L), albumin level, 2.6 g/dL (26 g/L
126 ge, 1.1-1.3 mmol/L); lipase level, 2423 U/L (normal level, &lt;300 U/L); amylase level, 1435 U/L (normal
127 glutamyl transferase (77 U/L [1.28 mukat/L]; normal level, &lt;55 U/L [<0.92 mukat/L]) and C-reactive pr
128 -reactive protein (97.1 mg/L [924.8 nmol/L]; normal level, &lt;8 mg/L [<76.2 nmol/L]]) levels were chron
129 phorylase activity rose from undetectable to normal levels (median 697 nmol/h/mg protein, range 262-1
130  each of the patient groups when compared to normal levels observed in healthy local controls: uncomp
131     ABCB4(S320F) homozygosity, with half the normal level of ABCB4, is the tipping point between more
132 low 0.05 IU/mL, HBV DNA target not detected, normal level of alanine aminotransferase) after removal
133 l (HBsAg positive, HBV DNA below 2000 IU/mL, normal level of alanine aminotransferase) or functional
134 ration with densely aligned collagen fibers, normal level of cellularity, and functional restoration.
135  ERPs of a mouse line expressing 150% of the normal level of cone M-opsin with those of WT mice revea
136                                          The normal level of creatinine in the serum and its excretio
137                                 Therefore, a normal level of CtpA activity is critical for T3SS funct
138                                   However, a normal level of LTP can be generated by repetitive 100-H
139 , such as TACI(+/-) mice, expressed half the normal level of TACI on their B cells and exhibited simi
140  anti-inflammatory eicosanoids, and restored normal levels of acylcarnithins but not of sphingomyelin
141 es (OR = 0.04, 95% CI 0.005-0.49; P = 0.01), normal levels of alanine aminotransferase at the end of
142 HBV) DNA who were positive for HBeAg and had normal levels of alanine aminotransferase were randomly
143 ith chronic HBV infection, high viral loads, normal levels of alanine aminotransferase, and therapy w
144 99 IU/mL (intermediate viral load [IVL]) and normal levels of alanine aminotransferase, HBcrAg levels
145 )-positive patients with high viral load and normal levels of alanine aminotransferase.
146 rtensive patients with hyperaldosteronism or normal levels of aldosterone exhibited increased activit
147 e channels but normal surface expression and normal levels of alpha and gamma subunit-activating clea
148                Cells lacking CnrN accumulate normal levels of AprA and CfaD.
149 dispersed in axons, thereby maintaining near normal levels of ATP even in boutons lacking mitochondri
150 ation carriers were healthy and demonstrated normal levels of atrial natriuretic peptide.
151 efective in fusion activity and yet retained normal levels of attachment to cell surface heparan sulf
152 with no evidence of immunohistopathology and normal levels of autoantibody.
153 bnormalities in gait and balance and exhibit normal levels of basal locomotion.
154  (BBB), has an essential role in maintaining normal levels of brain BCAAs.
155                 The [URE3] variants cured by normal levels of Btn2p and Cur1p all have low seed numbe
156 n2 cur1 double mutant are cured by restoring normal levels of Btn2p and Cur1p, with both proteins nee
157 arrhythmogenicity, while helping to maintain normal levels of Ca(2+) transients and heart function.
158 rranged T cell receptor alphabeta transgene, normal levels of CD4 single-positive cells were produced
159 SCM) and CD4(+) central memory T cells, with normal levels of CD4(+) T(SCM) proliferation, and lack o
160 press a low level of total CD79b protein but normal levels of CD79a and IgM protein.
161              B cell-deficient mice developed normal levels of CD8(+) effector T cell responses early
162 and Sidekick is itself necessary to maintain normal levels of cell bond tension.
163 ivo, Fib(AEK) mice were generated that carry normal levels of circulating fibrinogen but lack the cap
164      Ablation of Erfe in Th3/+ mice restored normal levels of circulating hepcidin at 6 weeks of age,
165                                  Higher-than-normal levels of circulating triglycerides are a risk fa
166 eficient B cells lack the ability to undergo normal levels of class switch recombination and somatic
167 ally, our model predicted that restoring the normal levels of clotting factors II, IX, and X while si
168  We find that MFSD12 is required to maintain normal levels of cystine-the oxidized dimer of cysteine-
169  reveals that both proteins are required for normal levels of cytosine methylation and hydroxymethyla
170 s that dictate the ability of DAT to sustain normal levels of DA clearance, we pursued a forward gene
171         Fin1 is required for the assembly of normal levels of Dam1 and Ndc80 submodules.
172               Whole brain homogenates showed normal levels of DISC1 protein, but decreased binding of
173  meiosis and that CDKG1 is also required for normal levels of DNA damage-induced homologous recombina
174  by 4-fold in Elmo1(-/-) lymphocytes despite normal levels of Dock2 mRNA.
175 or apoptosis and atrophy and maintained near-normal levels of electroretinographic responses.
176      The heterozygous PME-1 KO mice produced normal levels of endogenous Abeta and exhibited normal e
177 ynchronization as measures that characterize normal levels of excitability and quantify any deviation
178                         We conclude that, at normal levels of expression during bacterial infection,
179  lacking DdPpk1 accumulate approximately 50% normal levels of extracellular polyphosphate, suggesting
180  directed cell migration and for maintaining normal levels of F-actin [8-10].
181                       Patients with MPGN had normal levels of factor H, and structural analysis of th
182 t reduced risk of AIS compared to those with normal levels of factor XI.
183 n certain rare genetic contexts, maintaining normal levels of FGFR2 signaling is important for human
184 roid-stimulating hormone, or TSH) level with normal levels of free thyroxine (FT4) affects up to 10%
185  cell lines, and an AML cell line expressing normal levels of GATA2, we found that inhibition of GATA
186 ATA2, but not in an AML cell line expressing normal levels of GATA2.
187 females presented abnormal levels of Ga2 but normal levels of Gb3, supporting the importance of analy
188          Although Enpp1 (-/-) mice generated normal levels of germinal center B cells and plasmablast
189 wever, MEK1DD-expressing organoids exhibited normal levels of growth and retained apicobasal polarity
190  reperfusion injury, even in the presence of normal levels of GSH.
191                                              Normal levels of H3 T118ph are important because it is r
192 ut containing intact H2Bub enzymes that have normal levels of H3K4me3, suggesting that monoubiquitina
193 t with the GNAT proactivator SPV106 restored normal levels of H3K9Ac and H3K14Ac, reduced DNA CpG hyp
194                                              Normal levels of HDL-C are not an independent risk facto
195 ceiving cognitive behaviour therapy achieved normal levels of health anxiety compared with those in t
196                             Knockin mice had normal levels of hepatic fat on a chow diet, but when ch
197 NA production and therefore fails to secrete normal levels of Hla at early phases of growth.
198  HIF1, but not HIF2 target genes in spite of normal levels of hypoxia-inducible transcription factor
199 nd that IFN-lambdaR1-deficient mice produced normal levels of IFN, robust NK cell responses, but grea
200 d production of normal levels of virions and normal levels of infectivity in the complete absence of
201                  Although Il18(-/-) mice had normal levels of inflammatory monocytes, their NK cells
202    Titrating ventilatory support to maintain normal levels of inspiratory effort may prevent changes
203 defects, SMA Schwann cells failed to express normal levels of key extracellular matrix proteins, incl
204 uring mitosis and that p150N is required for normal levels of Ki-67 accumulation on the PCL.
205 from some female strains, were necessary for normal levels of male investigatory behavior of female s
206           Notably, TDP2 is also required for normal levels of many gene transcripts in developing mou
207  cells, tend to be multinucleate, accumulate normal levels of mass and protein per nucleus, and form
208 ls displayed normal morphology and contained normal levels of mast cell proteases before and after de
209 expressing the human MECP2 gene at twice the normal levels of MeCP2 (Tg1; Collins et al., 2004).
210 therapeutic approaches must restore close to normal levels of MECP2.
211 C8 in cooperative relationships that promote normal levels of meiotic DSBs at recombination hotspots
212     GLMP is essential for the maintenance of normal levels of MFSD1 in lysosomes and vice versa.
213  female mice developed normally and produced normal levels of milk protein, suggesting a defect in de
214 NA deficient oocytes but not in oocytes with normal levels of mitochondrial DNA.
215 e born to an iron-deficient mother contained normal levels of mitochondrial iron and no ferritin; the
216 t from the mother contained low ferritin and normal levels of mitochondrial iron.
217 ts contained less ferritin than controls but normal levels of mitochondrial iron.
218 Bazooka/Par-3, which in turn is required for normal levels of mobile E-cadherin.
219 ultured fibroblasts from the siblings showed normal levels of mtDNA and mitochondrial transcripts, an
220                   CS cells, in contrast, had normal levels of mutagenesis despite their TCR deficienc
221 fertile adults, with the capacity to produce normal levels of myeloid and lymphoid lineage cells.
222 ophil granules lacked lactoferrin but showed normal levels of myeloperoxidase.
223     Chronic DSS colitis was characterized by normal levels of neurogenesis but with deficits in the m
224 te with brain-adjacent stem cells to promote normal levels of neurogenesis.
225 g factors is required for the maintenance of normal levels of overlapping mRNA transcripts.
226 h-affinity haemoglobin, the model shows that normal levels of oxygen consumption can be achieved at r
227                                              Normal levels of PE can decline with age in the brain.
228 lopment of oxidative stress, and resulted in normal levels of peroxisome proliferation, whereas Pjvk
229 and showed that DVAP is required to maintain normal levels of phosphoinositides.
230 igmented cells (strain 531Ad) and cells with normal levels of pigmentation (strain 531Ac), respective
231 rved residue in the RacGEF domain results in normal levels of PIX-1 protein, but a reduced level of a
232 ve determined that this gene is required for normal levels of PME activity and homogalacturonan methy
233 vity had often fully recovered, or overshot, normal levels of productivity in both high- and low-dive
234 rmed, with transduced cells exhibiting supra-normal levels of protein expression and ex vivo migratio
235 disease (CKD) have inability to maintain the normal levels of protein metabolism products, blood pres
236                                              Normal levels of RDS and the unglycosylated RDS binding
237 able to generate variants despite expressing normal levels of RecA.
238                  Excitatory neurons regained normal levels of response, while narrow-spiking (inhibit
239 on and retinoid isomerase activity, and near-normal levels of retinal and visual functions.
240 genesis defect, allowing them to reestablish normal levels of ribosome production and cell proliferat
241 lar and pulmonary arterial pressures, toward normal levels of right-side pressure (about 20 mmHg) mea
242  with IBS (with visceral hypersensitivity or normal levels of sensitivity).
243 s reduced their visceral hypersensitivity to normal levels of sensitivity.
244 exogenous TGF-beta in vitro, they maintained normal levels of Smad2 phosphorylation under steady-stat
245      As a reciprocal experiment, we restored normal levels of SMN in the muscle with low SMN levels i
246 we used a mouse mutant engineered to express normal levels of SNAP-25 but only SNAP-25a.
247            These findings suggest that supra-normal levels of sociability and verbal functioning may
248                         Scn2b null atria had normal levels of sodium current density compared with wi
249 variant that had enhanced Sse expression and normal levels of SpeB production was identified (the var
250  compared to the dystonic patients or to the normal levels of striatal activity reported in healthy a
251 h, and such glial-specific expression resets normal levels of Sty and MAPK signaling and modulates ta
252 smitter release onto these neurons maintains normal levels of synaptic activity.
253 ce of each chaperone were cured by restoring normal levels of that protein.
254 schizophrenia, ChC cartridge boutons contain normal levels of the 67 kDa isoform of glutamic acid dec
255 body wall muscle, but approximately half the normal levels of the giant UNC-89 isoforms, dis-organiza
256      AADC deficient patients fail to produce normal levels of the monoamine neurotransmitters dopamin
257 the absence of Siw14p are cured by restoring normal levels of the protein.
258 re, in vivo, CTNNBL1 is required to maintain normal levels of the Prp19 complex and to facilitate the
259 se-mediated mRNA decay pathway, resulting in normal levels of the truncated plakoglobin.
260 60M) strain are cured by restoration of just normal levels of the WT Hsp104.
261 ing that LCMT-1 is important for maintaining normal levels of these subunits.
262                            MT mice expressed normal levels of total SNAP-25, Syntaxin 1A and SNAP-47
263      Genetic disruption of PGC1alpha rescued normal levels of type I fiber markers MyH7 and myoglobin
264  synaptic function relative to controls with normal levels of TYROBP.
265                                  Concomitant normal levels of urinary albumin, IgM and RBP identified
266 glycosylation, we demonstrated production of normal levels of virions and normal levels of infectivit
267 tments can be restricted to individuals with normal levels of white blood cells, principally in order
268  each of the five Arabidopsis CSLC genes had normal levels of XyG.
269  mice that express inactive plasmin (Pm) but normal levels of zymogen Pg (PAI-1(-/-)/Pg(S743A/S743A))
270 of neutrophils in sputum, or the absence (or normal levels) of eosinophils or other T2 markers in spu
271 egulation on Day 5 and Day 9 but returned to normal level on Day 13 and Day 17.
272                 For drug concentrations at a normal level or above, the sample can be diluted up to 1
273 nteracted such that methylation returns to a normal level over four generations.
274                   Restoring Aurora B to near-normal levels rescues mitotic phenotypes in cells lackin
275 GFP construct increased RetGC levels to near normal levels, restored dark-adapted photoresponses, and
276 hment of GILZ expression in patients' DCs to normal levels restores their capacity to activate allerg
277 pression of two Msh3 polymorphic variants at normal levels showed no detectable change in expansions,
278 phase even if stratospheric ozone returns to normal levels, so that climate conditions conducive to w
279 e than 20% gamma'-Fg, the upper limit of the normal level, the delay in lysis was magnified.
280  and cytoskeletal proteins remained at their normal levels, the major peripheral membrane proteins sp
281 er, the median CD4 cell count remained below normal levels throughout follow-up period and the propor
282 s convective O(2) delivery was maintained at normal levels under both conditions by pump-driven blood
283  on the AhR ligand-free diet but returned to normal levels upon dietary supplementation with I3C.
284 ectivity and functional imbalances revert to normal levels upon epichaperome inhibition.
285 so had increased IL-19, which was reduced to normal levels upon ixekizumab treatment, correlating wit
286     BGL was dichotomized for elevated versus normal levels using a cutoff value of >7.0 mmol/l.
287 trials targeting glycated hemoglobin to near-normal levels using intensive therapy have failed to pre
288 nockdown (by approximately 85% compared with normal levels), using lentiviral shRNA, dramatically att
289 xia (AIH) restores phrenic nerve activity to normal levels via enhanced phrenic long-term facilitatio
290                Although viral DNA is made at normal levels, viral capsid accumulation in the nucleus
291        An ATPase-defective Msh3 expressed at normal levels was as defective in expansions as Msh3-/-
292                                        Lower normal levels were associated with faster clinical progr
293 e methyltransferase Set1 is restored to near normal levels when SIN3 is also reduced.
294        The early ART group (n = 33) achieved normal levels, whereas the later ART group (n = 30) cont
295 ns in the lung are increased to greater than normal levels, which can occur during viral exacerbation
296 ried in isolation from one-half to two-times normal levels while subjects with simulated amputation w
297 mic pressure gradient (PSG) was decreased to normal level, while total bilirubin (TBIL) and liver fun
298 n led to rapid, sustained IL-19 decreases to normal levels, with decreases at 2-weeks correlating wit
299 cardiomyocyte proliferation and recovered to normal levels within 1-week time.
300 ium cyclosilicate reduced serum potassium to normal levels within 48 hours; compared with placebo, al

 
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