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1 two-phase interface at room temperature and normal pressure.
2 are prominent in some glaucoma patients with normal pressures.
3 sed 2.2-2.5 times (28-30.5 mmHg) that of the normal pressure (12.5-14.5 mmHg) by cauterization of thr
4 ed under conventional extraction conditions (normal pressure, ambient temperature) suggests that reco
5 luid (CSF) analysis revealed clear fluid and normal pressure and biochemistry, except for elevated pr
6 ortant role in conformational fluctuation at normal pressures, and are implicated as the nucleation s
7 l Society of Hypertension have proposed that normal pressure be lower than 130 mm Hg, with an optimum
8 bject is pressed by another soft object, the normal pressure cannot be measured independently from th
10 The "open" conformation under conditions of normal pressure displays increased mobility, focused on
11 how a pressure sensor that measures only the normal pressure, even under extreme bending conditions.
12 f anti-rhodopsin antibodies in patients with normal pressure glaucoma (NPG) has been previously demon
13 els of IL-10 was higher in the patients with normal pressure glaucoma compared to age-matched control
16 primary open-angle glaucoma [POAG]; 40 with normal-pressure glaucoma [NPG]) and 25 age-matched healt
17 ed in age-matched groups of 60 patients with normal-pressure glaucoma, 36 patients with high-pressure
18 tial effects can be quite significant at the normal pressure gradient conditions typical for espresso
20 tasets containing 12 volumes with idiopathic normal pressure hydrocephalus (iNPH) and 30 normal volum
25 eral blood and ventricular CSF in idiopathic normal pressure hydrocephalus (iNPH) patients totaling 1
27 ical outcomes, with patients with idiopathic normal pressure hydrocephalus (iNPH) serving as a contra
28 veness in individuals treated for idiopathic normal pressure hydrocephalus (iNPH), 37 patients clinic
29 ely 10% of dementia patients have idiopathic normal pressure hydrocephalus (iNPH), an expansion of th
30 inal tap test in individuals with idiopathic normal pressure hydrocephalus (iNPH), and explored if th
35 the hypokinetic gait disorder in idiopathic normal pressure hydrocephalus (NPH) patients from the ga
37 s generally been demonstrated to be lower in normal pressure hydrocephalus (NPH) than in normal contr
38 (CSF) differentiate patients with suspected normal pressure hydrocephalus (NPH) who respond to CSF d
39 widely used to evaluate CSF hydrodynamics in normal pressure hydrocephalus (NPH), Chiari type I malfo
41 hydrocephalus, neurodegeneration in elderly normal pressure hydrocephalus and, in all age groups, in
43 Alzheimer disease patients and patients with normal pressure hydrocephalus may have a higher risk of
44 d controls (ie, individuals with Bell palsy, normal pressure hydrocephalus, or Tourette syndrome).
51 nation) in 36 shunt-responsive patients with normal-pressure hydrocephalus (NPH; mean age, 75 years;
54 ad elevated pressure in vessels proximal and normal pressure in vessels distal to the coarctation, ye
55 o the fully active conformation, which under normal pressure is only populated when a G protein or a
58 rms in explicit lipid bilayers with constant normal pressure, lateral area, and temperature using the
59 rgy from various mechanical stimuli, such as normal pressure, lateral strain, bending, and vibration,
60 se time of approximately 20 ms), large-area, normal pressure monitoring under different, complex bend
61 R (>2-<=3WU) was lower than among those with normal pressures (mPAP <21 mmHg) and normal PVR (PVR <=
62 etermine 4 hemodynamic profiles: normal flow-normal pressure, normal flow-high pressure, low flow wit
64 ressures were eventually stabilized within a normal pressure range from 18 to 38 months following Nd:
65 y remove carbon dioxide from gas mixtures at normal pressure/temperature through breakthrough experim
70 reliably workable under room temperature and normal pressure with the generation rate reaching ~617 u