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1  two-phase interface at room temperature and normal pressure.
2 are prominent in some glaucoma patients with normal pressures.
3 sed 2.2-2.5 times (28-30.5 mmHg) that of the normal pressure (12.5-14.5 mmHg) by cauterization of thr
4 ed under conventional extraction conditions (normal pressure, ambient temperature) suggests that reco
5 luid (CSF) analysis revealed clear fluid and normal pressure and biochemistry, except for elevated pr
6 ortant role in conformational fluctuation at normal pressures, and are implicated as the nucleation s
7 l Society of Hypertension have proposed that normal pressure be lower than 130 mm Hg, with an optimum
8 bject is pressed by another soft object, the normal pressure cannot be measured independently from th
9                                   A constant normal pressure, constant surface tension, and constant
10  The "open" conformation under conditions of normal pressure displays increased mobility, focused on
11 how a pressure sensor that measures only the normal pressure, even under extreme bending conditions.
12 f anti-rhodopsin antibodies in patients with normal pressure glaucoma (NPG) has been previously demon
13 els of IL-10 was higher in the patients with normal pressure glaucoma compared to age-matched control
14                   In addition, patients with normal pressure glaucoma had a higher titer of autoantib
15 eported in patients, particularly those with normal pressure glaucoma.
16  primary open-angle glaucoma [POAG]; 40 with normal-pressure glaucoma [NPG]) and 25 age-matched healt
17 ed in age-matched groups of 60 patients with normal-pressure glaucoma, 36 patients with high-pressure
18 tial effects can be quite significant at the normal pressure gradient conditions typical for espresso
19 rrhagic hydrocephalus (16.2%) and idiopathic normal pressure hydrocephalus (14.2%) in adults.
20 tasets containing 12 volumes with idiopathic normal pressure hydrocephalus (iNPH) and 30 normal volum
21                                   Idiopathic normal pressure hydrocephalus (iNPH) is a common neurolo
22                                   Idiopathic Normal Pressure Hydrocephalus (iNPH) is a neurological c
23                                   Idiopathic normal pressure hydrocephalus (iNPH) is a prevalent, yet
24                                   Idiopathic normal pressure hydrocephalus (iNPH) is an enigmatic neu
25 eral blood and ventricular CSF in idiopathic normal pressure hydrocephalus (iNPH) patients totaling 1
26 e ventricular ependymal lining of idiopathic normal pressure hydrocephalus (iNPH) patients.
27 ical outcomes, with patients with idiopathic normal pressure hydrocephalus (iNPH) serving as a contra
28 veness in individuals treated for idiopathic normal pressure hydrocephalus (iNPH), 37 patients clinic
29 ely 10% of dementia patients have idiopathic normal pressure hydrocephalus (iNPH), an expansion of th
30 inal tap test in individuals with idiopathic normal pressure hydrocephalus (iNPH), and explored if th
31 ctancy of patients suffering from idiopathic normal pressure hydrocephalus (iNPH).
32                                              Normal pressure hydrocephalus (NPH) is a neurological di
33                                              Normal pressure hydrocephalus (NPH) is a poorly understo
34                                              Normal pressure hydrocephalus (NPH) is a reversible caus
35  the hypokinetic gait disorder in idiopathic normal pressure hydrocephalus (NPH) patients from the ga
36                                   Idiopathic normal pressure hydrocephalus (NPH) remains both oversus
37 s generally been demonstrated to be lower in normal pressure hydrocephalus (NPH) than in normal contr
38  (CSF) differentiate patients with suspected normal pressure hydrocephalus (NPH) who respond to CSF d
39 widely used to evaluate CSF hydrodynamics in normal pressure hydrocephalus (NPH), Chiari type I malfo
40 s (MCI-other, n = 77; DEM-other, n = 23), or Normal Pressure Hydrocephalus (NPH, n = 57).
41  hydrocephalus, neurodegeneration in elderly normal pressure hydrocephalus and, in all age groups, in
42                                Research into normal pressure hydrocephalus has often focused on the c
43 Alzheimer disease patients and patients with normal pressure hydrocephalus may have a higher risk of
44 d controls (ie, individuals with Bell palsy, normal pressure hydrocephalus, or Tourette syndrome).
45 rol samples were obtained from patients with normal pressure hydrocephalus.
46 standing hydrocephalus as well as idiopathic normal pressure hydrocephalus.
47 ic conditions, such as Alzheimer disease and normal pressure hydrocephalus.
48 al biopsies taken during shunt placement for Normal Pressure Hydrocephalus.
49 erative disease and CNS injuries, as well as normal pressure hydrocephalus.
50  manifests as Alzheimer's disease (AD) or as normal-pressure hydrocephalus (NPH).
51 nation) in 36 shunt-responsive patients with normal-pressure hydrocephalus (NPH; mean age, 75 years;
52                                   Idiopathic normal-pressure hydrocephalus is a neurologic disorder c
53           Among participants with idiopathic normal-pressure hydrocephalus who had a response to temp
54 ad elevated pressure in vessels proximal and normal pressure in vessels distal to the coarctation, ye
55 o the fully active conformation, which under normal pressure is only populated when a G protein or a
56                                              Normal pressure is restored in salt-fed DBH-ET(B);ET(B)(
57                              Measuring small normal pressures is essential to accurately evaluate ext
58 rms in explicit lipid bilayers with constant normal pressure, lateral area, and temperature using the
59 rgy from various mechanical stimuli, such as normal pressure, lateral strain, bending, and vibration,
60 se time of approximately 20 ms), large-area, normal pressure monitoring under different, complex bend
61 R (>2-<=3WU) was lower than among those with normal pressures (mPAP <21 mmHg) and normal PVR (PVR <=
62 etermine 4 hemodynamic profiles: normal flow-normal pressure, normal flow-high pressure, low flow wit
63      Prognosis worsened from the normal flow-normal pressure profile to the low flow with right ventr
64 ressures were eventually stabilized within a normal pressure range from 18 to 38 months following Nd:
65 y remove carbon dioxide from gas mixtures at normal pressure/temperature through breakthrough experim
66 (2) at 2 K as the pressure is increased from normal pressure to 1.02 GPa.
67 ge, whereas the lung as a whole maintained a normal pressure/volume relationship.
68       Moreover, both gases were effective at normal pressures when they replaced nitrogen in a gas mi
69                     The compound 3 reacts at normal pressure with methylamine or ethylamine, forming
70 reliably workable under room temperature and normal pressure with the generation rate reaching ~617 u