ライフサイエンスコーパス: normoglycemic

1 750 IEQ (7% normoglycemic) and 1000 IEQ (30% normoglycemic).

2 ZF rats were obese, hyperlipidemic, and normoglycemic.

3 d diabetes compared with those that remained normoglycemic.

4 s of CRP at baseline than those who remained normoglycemic.

5 ns had normal pancreatic morphology and were normoglycemic.

6 eased significantly with PGA and 3/10 became normoglycemic.

7 mice receiving the vehicle control remained normoglycemic.

8 lets with PGA polymers subcutaneously became normoglycemic.

9 y higher PI/C ratios than mice that remained normoglycemic.

10 DM/IGT (aHR, 1.14 [1.00-1.30]) compared with normoglycemic.

11 diabetic by streptozotocin injection or kept normoglycemic.

12 al size and quantity, and these animals were normoglycemic.

13 peratively and a 10-fold increase at 6 mo in normoglycemics.

14 150 mg/kg extract-treated ob/ob mice became normoglycemic (137 +/- 6.7 mg/dl) and had significantly

15 Normoglycemic 2-week-old betaDKO mice manifest reduced b

16 rAd-GLP-1-treated diabetic ob/ob mice became normoglycemic 4 days after treatment, remained normoglyc

17 ecretion and insulin sensitivity in 49 white normoglycemic (4.99 +/- 0.51 vs. 4.95 +/- 0.41 mmol/l) n

18 e multicenter study (N = 248), we identified normoglycemic (48.7%), prediabetic (44.4%), and diabetic

19 ARgamma knockout mice (PANC PPARgamma(-/-)), normoglycemic 60% pancreatectomy rats (Px), normoglycemi

20 teraction in B-cell adaptation was tested in normoglycemic 60% partial pancreatectomy (Px) and hyperg

21 y patients as hyperglycemic (> 200 mg/dL) or normoglycemic (60-200 mg/dL).

22 Light and electron microscopy of normoglycemic 7-week-old ZDF rats showed thickened endot

23 from 347 deeply phenotyped individuals (174 normoglycemic, 79 prediabetic and 94 T2D).

24 ee of insulin resistance evidenced in young, normoglycemic adults with sex-specific fingerprints.

25 kers for adipose tissue and T2D incidence in normoglycemic adults.

26 IGF-I levels were highest in normoglycemic African Caribbeans and declined with age (

27 /kg to assess the anti-diabetic potential in normoglycemic, alloxan-induced and glucose-loaded diabet

28 l glycemic risk profiles, validated in 1,955 normoglycemic and 114 prediabetic individuals from an in

29 es from 207 patients with periodontitis (118 normoglycemic and 89 with type 2 DM) were analyzed.

30 Obese individuals were normoglycemic and characterized by low-grade inflammatio

31 cines were effective at both the prediabetic normoglycemic and dysglycemic stages of disease, Proinsu

32 These results suggest that under normoglycemic and hyperglycemic conditions, small human

33 effects of IGF-I on JNK and p38 kinase under normoglycemic and hyperglycemic conditions.

34 QTL genes, whose expression differed between normoglycemic and hyperglycemic individuals, siRNA of te

35 of exenatide inhibited thrombus formation in normoglycemic and hyperglycemic mice in vivo.

36 by 0.5, 1, and 3 h of recirculation in sham, normoglycemic and hyperglycemic rats.

37 normoglycemic 60% pancreatectomy rats (Px), normoglycemic and hyperglycemic Zucker fatty (ZF) rats,

38 mediated at the level of the VMH under both normoglycemic and hypoglycemic conditions.

39 rom hyperalimentation, all patients remained normoglycemic and insulin-independent 6-months posttrans

40 All recipients became normoglycemic and insulin-independent posttransplant.

41 Normoglycemic and insulin-sensitive C57BL/6J mice; hyper

42 Here we report that normoglycemic and insulinopenic 3-month-old LP progeny s

43 After 14 days of treatment, mice remained normoglycemic and islet allografts were functional for u

44 s with increased albumin excretion were both normoglycemic and normotensive (systolic/diastolic blood

45 essible scorecard form, tested the models on normoglycemic and prediabetes subcohorts, and compared t

46 RIP-M3 mice were normoglycemic and responded normally to glucose challeng

47 ated with ALS/PLNC, diabetic NOD mice become normoglycemic and tolerated minor antigen-disparate isle

48 All 1943 normoglycemic and transiently hypoglycemic infants (23-4

49 in the subcutaneous adipose tissue of obese normoglycemic and type 2 diabetic subjects compared with

50 ected cytokine-bacterial network, while both normoglycemics and T2DM subjects with periodontitis demo

51 groups that were implanted with 750 IEQ (7% normoglycemic) and 1000 IEQ (30% normoglycemic).

52 lus infarction) was larger in hyper- than in normoglycemic animals at 2 and 4 h of recirculation.

53 ion of bcl-2 and bfl-1 and reduced damage in normoglycemic animals but failed to protect the diabetic

54 In normoglycemic animals this damage usually consists of se

55 easing density of SNN in the caudoputamen of normoglycemic animals.

56 ic rats and accelerates bone regeneration in normoglycemic animals.

57 Hyperglycemic, but not normoglycemic, animals showed pan-necrotic lesions ('inf

58 eased macrophage infiltration, compared with normoglycemic ApoE(-/-) controls.

59 Interestingly, castrated normoglycemic ApoE(-/-) mice developed larger atheroscle

60 ays), and three of the recipients were still normoglycemic at 100 days after transplantation.

61 th Asian and 53% of Nordic participants were normoglycemic at the time of measurement.

62 islets, and all were insulin independent and normoglycemic at the time of study.

63 yperglycemic nonobese diabetic (NOD) and old normoglycemic BALB/c mouse strains.

64 levels increasing approximately 20% from the normoglycemic baseline (BLI reduction approximately 70%)

65 in response to hypoglycemia (P = 0.13 versus normoglycemic baseline; P = 0.007 versus untreated hypog

66 stance, most obese humans and rodents remain normoglycemic because of compensatory hyperinsulinemia.

67 s with diagnosed diabetes had slower PS than normoglycemics (beta = -0.12; P < 0.05); diabetes durati

68 Perfusion of the sciatic nerve with the normoglycemic buffer solution did not affect withdrawal

69 ion of the insulin receptor gene (MIRKO) are normoglycemic but have increased fat mass.

70 uman muscle biopsies from patients with T2D; normoglycemic but insulin-resistant subjects with a pare

71 received ALS at a time after transplant when normoglycemic but prompted by a reduction (approximately

72 cal mAb, 100% of B-cell-depleted mice became normoglycemic by 2 days, and 70% of them maintained a st

73 o rescue islet loss, diabetic mice were made normoglycemic by islet transplantation and immunization

74 Thirteen normoglycemic C57BL/6 mice were studied.

75 a small but significant inhibition of DHT in normoglycemic cells.

76 disposal during a two-step hyperinsulinemic, normoglycemic clamp) wasn't met, hepatic insulin sensiti

77 They underwent hyperinsulinemic-normoglycemic clamps with [6,6-(2)H2]glucose to assess w

78 d fasting (8 h) blood samples from an obese, normoglycemic cohort and an obese, T2DM cohort of UAE na

79 slets of 1000 IEQ were sufficient to achieve normoglycemic condition (83%).

80 diabetes is maintenance of a long-term, near-normoglycemic condition and prevention of the onset or p

81 Pg LPS stimulated insulin secretion in the normoglycemic condition by approximately 1.5- to 3.0-fol

82 ediated epithelial-mesenchymal transition in normoglycemic condition.

83 ediated epithelial-mesenchymal transition in normoglycemic condition.

84 in cerebral cortex with (18)F-FDG PET under normoglycemic conditions (isoflurane and awake) were gen

85 Healthy control subjects were studied under normoglycemic conditions (n = 24).

86 wever, 2-DG infusion to the MAN or VMH under normoglycemic conditions had no systemic effect.

87 pairment of atherosclerosis regression under normoglycemic conditions in mice lacking PRMT2 (Prmt2(-/

88 Underestimation of rCMR(glc) by (18)F-FDG in normoglycemic conditions may be due to partial-volume ef

89 lard oil and heparin into rats for 6 h under normoglycemic conditions resulted in a marked elevation

90 PRMT2-deficient plaque CD68+ cells under normoglycemic conditions showed increased expression of

91 nd decreased insulin sensitivity, even under normoglycemic conditions.

92 ss caused by three different pro-oxidants in normoglycemic conditions.

93 proximately 3% of the filtered glucose under normoglycemic conditions.

94 divided into streptozotocin-induced DCM and normoglycemic control groups.

95 et diabetes (diagnosed at <35 years) and 292 normoglycemic control subjects of French origin.

96 a family history of type 2 diabetes and 372 normoglycemic control subjects without a family history

97 hort of 327 type 2 diabetic subjects and 357 normoglycemic control subjects, the H611 allele and the

98 etic subjects compared with that of nonobese normoglycemic control subjects.

99 gion in 859 subjects with diabetes and 1,151 normoglycemic control subjects.

100 d inhibition of glucose output relative to a normoglycemic control.

101 of patients with T2D (n = 102) compared with normoglycemic controls (n = 87).

102 ines and proliferated more than T cells from normoglycemic controls after anti-CD3e or Ag stimulation

103 Hypoglycemic rats allowed to feed and normoglycemic controls both showed sparse Fos-positive (

104 induced by streptozotocin (STZ) injection or normoglycemic controls injected with citrate buffer alon

105 that are critical for healing compared with normoglycemic controls.

106 and increased immune pathology compared with normoglycemic controls.

107 s between the two solutions in the number of normoglycemic days (IGL-1: 11.5 +/- 6.2 versus UW: 8.5 +

108 eiving insulin implants and determined to be normoglycemic (DB-IN group), and 3) normal, nondiabetic

109 model of maternal low-protein (LP) diet are normoglycemic despite collapsed insulin secretion.

110 ndividuals will become diabetic; many remain normoglycemic despite profound obesity.

111 d were transplanted into abdominal cavity of normoglycemic (empty capsules) or streptozotocin induced

112 ion, thus contributing to the restoration of normoglycemic equilibrium.

113 All recipients were normoglycemic (fasting glucose: autograft recipients, 5.

114 glycemic female NOD mice than in age-matched normoglycemic female NOD mice.

115 7% (67/77) of patients receiving placebo had normoglycemic findings.

116 d RBMEC were challenged with 1 h of normoxic-normoglycemic flow cessation (NNFC) followed by reperfus

117 eived transplants of porcine islets remained normoglycemic for 1 year with progressive weight gain.

118 h CT25 when glycemia was <500 mg/dL remained normoglycemic for 100 days after alloislet transplantati

119 10(9) infectious units) rAAV-IL-10 remained normoglycemic for at least 117 days, whereas diabetes re

120 Treated mice were healthy and normoglycemic for the duration of the experiment (>120 d

121 treatment, participants were categorized as normoglycemic (FPG <5.6 mmol/L), prediabetic (FPG 5.6-6.

122 The remaining patients are normoglycemic (glucose 92 +/- 23 mg/dl) and have a creat

123 .6+/-7.2, 54.0+/-7.8, 105.0+/-15.0 s for the normoglycemic group (n=7), and 49.2+/-33.0, 116.4+/-2.4,

124 Corresponding time intervals for the normoglycemic group were 1.86 and 1.14 min, respectively

125 In the normoglycemic group, ADCw declined to the same value as

126 re reduced in the diabetic compared with the normoglycemic group, both in shallow and in deep sites.

127 a peak at 3h after ischemia relative to the normoglycemic group.

128 of donor-type (WF) islets became permanently normoglycemic (>100 days) while the third-party (BN) gra

129 splant, and all were insulin-independent and normoglycemic (HbA(1c), 5.8 +/- 0.2%).

130 The kidneys in normoglycemic humans filter 160-180 g of glucose per day

131 translocated, in soleus muscles of both (1) normoglycemic hyperinsulinemic obese/aged rats and (2) m

132 Compared with normoglycemic individuals (70-110 mg/dL), adjusted odds

133 Total vasodilator capacity was similar in normoglycemic individuals (IS, IR, and IGT), whereas it

134 ence interval [CI], 1.28-1.55) compared with normoglycemic individuals across all cancer types.

135 Among 2,422 normoglycemic individuals followed for 12 years, 201 dev

136 We leveraged information from 952 normoglycemic individuals for whom genome-wide genotypin

137 The study of these T2D risk variants in normoglycemic individuals has revealed that a significan

138 low-fat and high-carbohydrate diet, whereas normoglycemic individuals lost a mean of 0.43 kg (95% CI

139 ordic Diet than on the control diet, whereas normoglycemic individuals lost a mean of 2.20 kg (95% CI

140 than on the low-glycemic load diet, whereas normoglycemic individuals regained a mean of 1.44 kg (95

141 ese correlations were also evident when only normoglycemic individuals were included in the analyses

142 ulation at low genetic risk for disease, 181 normoglycemic individuals with no family history of diab

143 stroke (per 100 person-years) were: 1.14 in normoglycemic individuals, 1.40 in those with pre-diabet

144 In a subset of 287 normoglycemic individuals, acute insulin secretion was m

145 In 12 normoglycemic individuals, reduced-function variants in

146 In a subset of normoglycemic individuals, we did not observe significan

147 etes experience worse fracture outcomes than normoglycemic individuals.

148 from exercise, we studied eight young, lean, normoglycemic insulin-resistant (IR) offspring of indivi

149 ed normal islets under the kidney capsule of normoglycemic insulin-resistant mice with two different

150 nce that occur in the muscle of young, lean, normoglycemic, insulin-resistant offspring of parents wi

151 Compared to rats with normoglycemic ischemia, numbers of beta-APP-immunopositi

152 ile a mild increase of cyt c was observed in normoglycemic ischemic animals after 1 and 3 h of reperf

153 row transplantation from diabetic mice into (normoglycemic) Ldlr(-/)(-) mice increased aortic root at

154 e with (n=9) or without (n=6) diabetes into (normoglycemic) Ldlr(-/)(-) mice was used to assess its f

155 glycemic status in 5 groups of individuals: normoglycemic lean and obese individuals with (a) normal

156 cation sites did not differ among cohorts of normoglycemic lean or obese and type 2 diabetes mellitus

157 45+/-12 years) into 1 of 4 groups: (1) lean normoglycemic (lean), (2) overweight and obese normoglyc

158 luded 112 participants across 4 groups: lean-normoglycemic (Lean-NG), obese-normoglycemic (OB-NG), ob

159 onset of hyperglycemia and were not seen in normoglycemic, leptin receptor-deficient db/db mice.

160 reased sorbitol production can also occur at normoglycemic levels via rapid increases in aldose reduc

161 ne expression, and reduced ROS production to normoglycemic levels, both in vitro and in vivo.

162 n type-2 Zucker diabetic fatty (ZDF) rat and normoglycemic littermates, we investigated whether diabe

163 that islet-infiltrating B cells in long-term normoglycemic (Lnglc) NOD, which are naturally protected

164 he five dogs treated with pravastatin became normoglycemic (<150 mg/dL) and maintained this level dur

165 Eleven normoglycemic males with overweight or obesity (BMI 31.6

166 d three of six IT islet recipients, remained normoglycemic (mean FBG< or =250 mg%) immediately posttr

167 (9 mug/mL) days of hyperglycemia compared to normoglycemic mice (P < 0.001).

168 and caspase-3 activity in both diabetic and normoglycemic mice (P < 0.05).

169 3 activity by almost 50% in diabetic but not normoglycemic mice (P < 0.05).

170 T marker) were found in their wounds than in normoglycemic mice and healing was delayed.

171 However, there were no normoglycemic mice and no insulin-positive cells in the

172 By day 3, the levels were reduced in normoglycemic mice but remained significantly higher in

173 Diabetes could also be induced in normoglycemic mice expressing low levels of CCL2 by incr

174 All normoglycemic mice maintained graft function for 100 day

175 bral glucose metabolic rates obtained from 4 normoglycemic mice were 21.5 +/- 4.3 mumol/min/100 g (me

176 nfiltrate was noted in both the diabetic and normoglycemic mice.

177 n elimination t(1/2) of approximately 7 h in normoglycemic mice.

178 lowing transfer of T cells from HG mice into normoglycemic mice.

179 -A, which had no effect on beta cell mass in normoglycemic mice.

180 al gingiva of type 1 and type 2 diabetic and normoglycemic mice.

181 s in the time course of ADCw decline between normoglycemic (n = 8) and hyperglycemic (n = 6) groups.

182 rdiac mesenchymal cells (CMSC) obtained from normoglycemic (ND-CMSC) and type 2 diabetic patients (D-

183 fourth-grade achievement tests compared with normoglycemic newborns.

184 dult female C57BL/6 J mice were incubated in normoglycemic (NG, 5 mM) or hyperglycemic (30 mM or 50 m

185 Rats were divided into normoglycemic (NG, n = 20) and streptozotocin-induced di

186 S/HRMS DIA proteomic analysis of muscle from normoglycemic (NGT) and prediabetic (IGT) subjects after

187 bral intracellular pH (pHi) were measured in normoglycemic (NM), acute hyperglycemic (AH), and chroni

188 during the study period, no diabetic became normoglycemic, no patient decreased their antihypertensi

189 serum-free conditions, and transplanted into normoglycemic NOD/SCID mice.

190 49 treatment did not lower glucose levels in normoglycemic, nondiabetic mice.

191 s in insulin-signaling events are present in normoglycemic, nonobese subjects with a strong family hi

192 Injection of the hyperimmune sera (IgG) into normoglycemic nude mice bearing porcine islets for > 70

193 groups: lean-normoglycemic (Lean-NG), obese-normoglycemic (OB-NG), obese-glucose intolerant (OB-GI),

194 Obese T2DM patients and normoglycemic obese and lean individuals (n = 48) were s

195 ponsible for some of the hyperinsulinemia in normoglycemic obese subjects; and 3) NA had no direct ef

196 Obese T2DM patients and normoglycemic obese versus lean subjects showed increase

197 rmoglycemic (lean), (2) overweight and obese normoglycemic (obese), (3) impaired glucose tolerance, a

198 no family history of diabetes (FH-) and 150 normoglycemic offspring of two type 2 diabetic parents (

199 In normoglycemic offspring of two type 2 diabetic parents,

200 ivalent human immune system development in a normoglycemic or chronically hyperglycemic environment,

201 rmoglycemic 4 days after treatment, remained normoglycemic over 60 days, and had reduced body weight

202 was conducted on stool and saliva samples of normoglycemic participants and individuals with prediabe

203 d 188 propensity-matched controls from 2,422 normoglycemic participants followed for 12 years in the

204 We studied 43 SNPs in 4,654 normoglycemic participants from the Finnish population-b

205 betes have poorer cognitive performance than normoglycemics, particularly in PS.

206 tions of red complex pathogens than those of normoglycemic patients (P < 0.05).

207 2-4 times more likely to suffer a stroke as normoglycemic patients and they also have worsened neuro

208 dysbiotic subgingival microbial profile than normoglycemic patients, including lower levels/proportio

209 y to identify the focus of infection than in normoglycemic patients.

210 with pre-diabetes, those with diabetes, and normoglycemic patients.

211 When stable and near-normoglycemic, patients with "A-B+" ketosis-prone diabet

212 with the regular-size capsules, although the normoglycemic period was comparable between two groups o

213 th an increased risk of stroke compared with normoglycemic persons (adjusted hazard ratio [adjHR]: 1.

214 Compared with normoglycemic persons, the adjusted relative risks for i

215 o incretins may contribute to the unexpected normoglycemic phenotype of Sur1KO mice versus the pronou

216 contrast to OLETF rats, possesses a lean and normoglycemic phenotype.

217 Under normoglycemic physiological conditions, insulin carries

218 associated with reduced insulin secretion in normoglycemic Pima Indians.

219 While L-SKO mice were normoglycemic, plasma glucose in beta-SKO animals was si

220 notable, as, in this unselected and largely normoglycemic population, external influences on beta-ce

221 er the total dataset or when restricted to a normoglycemic population.

222 In 70-90-day-old normoglycemic (prediabetic) female NOD TLR4(+/+) and NOD

223 abundance in the IgA-Biome profiles between normoglycemic, prediabetic, or diabetic samples distinct

224 tered to hyperglycemic (therapeutic mode) or normoglycemic (prophylactic mode) NOD mice.

225 P-1 decreased diabetic glucose levels to the normoglycemic range with significant weight reduction in

226 sk factors, blood glucose levels outside the normoglycemic range, higher mean daily delta blood gluco

227 ed at regulating blood glucose levels in the normoglycemic range.

228 mice lowered blood glucose levels toward the normoglycemic range.

229 therapy to maintain blood glucose levels in normoglycemic ranges to prevent associated morbidity and

230 Brains of normoglycemic rats (n=11), by contrast, showed only weak

231 Accelerated bone formation in normoglycemic rats caused by SA-PAE/bone graft treatment

232 0) or allogeneic (n=12) islet equivalents or normoglycemic rats with 5000 xenogeneic human islet equi

233 % of the hyperglycemic rats, but none of the normoglycemic rats, developed tonic-clonic seizures with

234 thodontic forces that was similar to that of normoglycemic rats.

235 hown by a significantly higher percentage of normoglycemic recipients and higher porcine C-peptide le

236 nd Flt-1 was enhanced at 3 and 5 days in the normoglycemic recipients, while in the diabetic recipien

237 eous muscle biopsies were performed in eight normoglycemic relatives of type 2 diabetic patients (FH(

238 ion and intensive outpatient treatment, near-normoglycemic remission (fasting plasma glucose 6.1 +/-

239 retion have proven useful in predicting near-normoglycemic remission and long-term insulin dependence

240 ation of insulin therapy, the period of near-normoglycemic remission may last for a few months to sev

241 Near-normoglycemic remission may occur in up to 30% of black

242 ized the natural history of spontaneous near-normoglycemic remission off of antidiabetic medication i

243 , reversible beta-cell dysfunction, and near-normoglycemic remission.

244 I diabetes providing an insulin-independent, normoglycemic state.

245 diabetic polyneuropathy by restoration of a normoglycemic state.

246 ts of individuals with T2D, prediabetes, and normoglycemic status in the United States, Europe, Israe

247 ere measured in a respiratory chamber in 112 normoglycemic subjects (83 Pima Indians and 29 whites; 6

248 2 diabetes and impaired insulin secretion in normoglycemic subjects (P = 0.006 and 0.0001 for type 2

249 tolerance test and the minimal model in 138 normoglycemic subjects ages 53-61 years.

250 first-degree relative (controls), 39 healthy normoglycemic subjects with a history of type 2 diabetes

251 Normoglycemic subjects with a strong family history of t

252 ur age and sex comparable groups: 30 healthy normoglycemic subjects with no history of type 2 diabete

253 In conclusion, in normoglycemic subjects, insulin resistance (low SI) was

254 educe the oxidative susceptibility of LDL in normoglycemic subjects; however, there are few studies i

255 n the brain of insulin-resistant relative to normoglycemic Tg2576 mice.

256 f illness, autophagy was better preserved in normoglycemic than in hyperglycemic rabbits, which corre

257 In those animals that became normoglycemic, the glucose tolerance of the hyperoxicall

258 In normoglycemics, the nodes anchored by interleukin (IL)-4

259 mpaired fasting glucose, and 1811 women were normoglycemic; the 2029 women without diabetes were foll

260 RIPCreER-EYFP (+TM) mice were normoglycemic throughout the study, and their glucose to

261 Most 1L-4Ralpha-/- mice remained normoglycemic to 36 weeks of age.

262 sion to levels similar to those of non-obese normoglycemic transgenic mice.

263 Normoglycemic transplanted rats and age-matched controls

264 ic hypoglycemia of infancy, but the mice are normoglycemic unless stressed.

265 POD 58 and remained insulin independent and normoglycemic until POD 264.

266 These islet-engrafted mice remained normoglycemic until removal of the graft-containing kidn

267 DCM vehicle rats had larger infarcts than normoglycemic vehicle-treated animals at a comparable ar

268 d recipients (n = 4, fifth recipient remains normoglycemic) versus 10.2 +/- 2.6 days for controls (n

269 00 IEQ of human, porcine, or NHP islets (75% normoglycemic) versus groups that were implanted with 75

270 ng resorption was 2.4- to 2.9-fold higher in normoglycemic vs. diabetic rats (P < 0.05).

271 ent cerebral ischemia when carried out under normoglycemic vs. hyperglycemic conditions.

272 In normoglycemic wild-type mice, hepatic expression of Ad36

273 ntent, or insulin mRNA levels in islets from normoglycemic Wistar rats.

274 The Goto-Kakizaki (GK) rat developed from normoglycemic Wistar-Kyoto (WKY) rat is a model for type

275 n-induced diabetic FVB/NJ mice were rendered normoglycemic with a therapeutic mass of syngeneic islet

276 c (diabetic), and STZ-diabetic kept hypo- or normoglycemic with insulin pellets (diabetic-normalized)

277 of the 400 islets (n = 5) recipients became normoglycemic within 8 days.

278 Whereas normoglycemic women at baseline had a decrease in glucos

279 ncreased risk for each outcome compared with normoglycemic women.

280 s, accelerated wound healing in diabetic and normoglycemic WT mice.

281 nitric oxide-mediated relaxation compared to normoglycemic WT or hyperglycemic MBL-null mice.

282 Islets from obese normoglycemic ZF rats had twofold increased PPARgamma an