ライフサイエンスコーパス: normoxia

1 however, commonly performed in ambient air (normoxia).

2 growth recovery when cells were returned to normoxia.

3 d Experiment 3) Lutein supplemented media in normoxia.

4 ced by over 2-fold under hypoxia compared to normoxia.

5 bon dioxide, electrophiles, and oxidants) in normoxia.

6 means that these afferents are active under normoxia.

7 ygen sensor that controls HIF activity under normoxia.

8 evels of l-lactate after its formation under normoxia.

9 real time are limited to areas of hypoxia or normoxia.

10 he P wave in V1 was lower in hypoxia than in normoxia.

11 occurring during neuronal regeneration under normoxia.

12 0001) and hyperoxia (P=0.0006) compared with normoxia.

13 enerally showed a more favorable outcome for normoxia.

14 tamine to lipogenesis in hypoxia, but not in normoxia.

15 Ia up-regulated the HIFalpha Ia levels under normoxia.

16 is limited by the lability of the protein in normoxia.

17 eases basal expression of GM-CSF in cells in normoxia.

18 y reduced sRBC adhesion to levels seen under normoxia.

19 ts HIFs to the proteasome for degradation in normoxia.

20 CSH rats but had no effect on ventilation in normoxia.

21 iferation of Leishmania promastigotes during normoxia.

22 ow that ERRs also stimulate glycolysis under normoxia.

23 after exposure to IH in vitro compared with normoxia.

24 m for multilevel regulation of HIF-1alpha in normoxia.

25 m mice exposed to either 24 hours hypoxia or normoxia.

26 se tissue is insufficient to maintain tissue normoxia.

27 ntrol); and (3) to the limit of tolerance in normoxia.

28 ood flow during hypoxic exercise relative to normoxia.

29 pal water content was increased, compared to normoxia.

30 knockdown of DEGS1 or DEGS2 by siRNA during normoxia.

31 ed relative to the same level of exercise in normoxia.

32 tant inhibitory effect on its activity under normoxia.

33 and oxidative function to values observed in normoxia.

34 ic factors in MDSCs regardless of hypoxia or normoxia.

35 , 8, 15 or 30 days, before being returned to normoxia.

36 ggerated hypoxic PH and failed to recover in normoxia.

37 the reduction in metabolic rate compared to normoxia.

38 PIO did not affect fetal growth under normoxia.

39 RNA that were associated with hypoxia versus normoxia.

40 BK/Kv were mostly closed at rest in normoxia.

41 posure to hypoxia, continuing upon return to normoxia.

42 ivalent relative and absolute intensities in normoxia.

43 iated with increased mortality compared with normoxia.

44 (18%), 3999 had hypoxia (63%), and 1171 had normoxia (19%).

45 3D pellets of human primary chondrocytes in normoxia (20% oxygen) and hypoxia (2.5% oxygen) and empl

46 to the limit of tolerance (T(lim)), once in normoxia (20.9% O(2); CON) and twice in hypoxia (14.5% O

47 in hypoxia (1 and 0.2% oxygen) compared with normoxia (20.9%).

48 rowth, not only when the cells were grown in normoxia (21% O(2)) but also remarkably in hypoxic condi

49 prague-Dawley rats were preexposed to either normoxia (21% O(2)) or hyperoxia (85% O(2)) for up to 21

50 Newborn rats (6 days) were subjected to normoxia (21% O(2)) or PC (8% O(2)) for 3h followed by 2

51 en pASCs cultured under hypoxia (1% O(2)) or normoxia (21% O(2)).

52 Pregnant ewes were exposed to normoxia (21% O2 ) or hypoxia (10% O2 ) from 105 to 138

53 exposed to normobaric hypoxia (12% O2 ) and normoxia (21% O2 ).

54 In contrast, normoxia (21% O2) prevented the expression of these inhi

55 IL-2 and IL-4 (T(C)2 cells) were exposed to normoxia (21% oxygen) or hypoxia (3% oxygen), and IL-13

56 Wistar rat dams underwent normoxia (21%) or hypoxia (13%) during pregnancy.

57 Wistar rat dams were randomized to either normoxia (21%) or hypoxia (13%) from day 6 post-mating u

58 walking under normal conditions (normobaric normoxia, 21% O(2)) and moderate hypoxia (13% O(2)) was

59 sulin was decreased in hypoxia compared with normoxia (225 +/- 23 vs. 128 +/- 30 nmol (kg fat free ma

60 -1); P < 0.05), but this was not the case in normoxia (289 +/- 15 ml min(-1); P = 0.33).

61 ime was reduced by 54%in hypoxia compared to normoxia (3.6 +/- 1.3 vs. 8.1 +/- 2.9 min; P<0.001).

62 n of exercise in hypoxia was greater than in normoxia (345 +/- 21 ml min(-1) vs. 297 +/- 18 ml min(-1

63 s pretreated with IH-1 (2 min hypoxia, 2 min normoxia; 8 h) or sham normoxia and allowed 16 h for rec

64 R 1.5, 95% CI 1.1-2.5, p=.01) as compared to normoxia (87/403 [23%]).

65 .001) with hypoxia (63 +/- 2%) compared with normoxia (96 +/- 0%), and was unaffected by sympathetic

66 addition of a blue light-blocking filter in normoxia, a significant increase in angiogenin levels wa

67 layed aerobic glycolysis when cultured under normoxia, accompanied by increased free NADH and NADH/NA

68 ntly, regeneration of dopamine neurons under normoxia also depends on ROS-production.

69 In normoxia, AMPA increased ventilation 25% and 50% in CON

70 In normoxia, AMPK induces PGC-1alpha, but how HIF is activa

71 wide mRNA expression arrays of HUVECs during normoxia and after 2, 8, and 16 h of hypoxia, we show us

72 compared with wild-type PASMCs, during both normoxia and after acute hypoxia.

73 (2 min hypoxia, 2 min normoxia; 8 h) or sham normoxia and allowed 16 h for recovery.

74 ore and enhance P. aeruginosa motility under normoxia and anoxia in an isolate dependent manner.

75 d facilitating P. aeruginosa dominance under normoxia and anoxia is greater than 3 kDa in size and is

76 cular density between young and aged mice in normoxia and at 2 and 3weeks of hypoxia.

77 r variable hypoxia were intermediate between normoxia and constant hypoxia, and gonad production corr

78 H23390 increased ventilation during baseline normoxia and did not affect ventilation during exposure

79 an explicit delineation between physiologic normoxia and genuine hypoxia is defined here, with impli

80 ragmatic pressure (P(di) ) of 92 cmH(2) O in normoxia and hypoxia (8% O(2) ) were performed on separa

81 tion in ECs suppressed Dll4 expression under normoxia and hypoxia and inhibited Dll4-induced Notch si

82 t multiple RTKs may regulate the HIF axis in normoxia and hypoxia and suggest that multikinase inhibi

83 were to compare ECG at moderate exercise in normoxia and hypoxia at the same heart rate, to provide

84 asurement of inner retinal OEF in rats under normoxia and hypoxia based on vascular oxygen tension (P

85 It inhibits HIF1alpha in both normoxia and hypoxia by reducing mRNA expression.

86 a cells in the presence/absence of OCs under normoxia and hypoxia conditions and did protein profilin

87 apturing RBC images from the SCD patients in normoxia and hypoxia conditions.

88 ily member in breast cancer cells under both normoxia and hypoxia conditions.

89 posure, compared to dark conditions, in both normoxia and hypoxia conditions.

90 Both the normoxia and hypoxia groups consisted of seven patients.

91 al oxygen metabolism (MO2_IR) under systemic normoxia and hypoxia in rat.

92 subjects performed maximal exercise tests in normoxia and hypoxia to determine how their altered haem

93 nner retinal OEF measurements obtained under normoxia and hypoxia were compared.

94 y impaired in conidia germination, growth in normoxia and hypoxia, and displayed attenuated virulence

95 it different interaction profiles under both normoxia and hypoxia, and only breast cancer cells gain

96 Both in normoxia and hypoxia, dietary nitrate suppressed cardiac

97 und, CM-272, was examined in HCC cells under normoxia and hypoxia, human hepatic stellate cells and L

98 tochondria of these cells, shuttling between normoxia and hypoxia, maintain bioenergetic efficiency a

99 In both normoxia and hypoxia, PASMC HIF-1alpha maintains low pul

100 chondrial respiration in breast cancer under normoxia and hypoxia, which correlates with decreased mi

101 opy or healthy controls were incubated under normoxia and hypoxia, with or without glucocorticoids.

102 r for glycolysis and energy production under normoxia and hypoxia.

103 static) and HMPOS (highly metastatic), under normoxia and hypoxia.

104 sponse elements, are regulated by ERalpha in normoxia and hypoxia.

105 beta-adrenergic-stimulated lipolysis in both normoxia and hypoxia.

106 by heterogeneous palladium chemistry both in normoxia and hypoxia.

107 ndent reduction in HIF-1 activity under both normoxia and hypoxia.

108 as detected in MMP-1-transfected cells under normoxia and hypoxia.

109 argely sequestered in breast cancer cells at normoxia and hypoxia.

110 ity and decreases polyubiquitination in both normoxia and hypoxia.

111 led with decreased GSK3beta activation under normoxia and hypoxia.

112 PC-3, and HCT-116 cancer cell lines both in normoxia and hypoxia.

113 e, inducing a mortality by about 50% in both normoxia and hypoxia.

114 noma cell lines with patient sera under both normoxia and IH.

115 ort term culture under hypoxia compared with normoxia and in response to interleukin 15 (IL-15) primi

116 n both the immature and mature neurons under normoxia and in the mature neurons under hypoxic conditi

117 Inner retinal OEF was 0.46 +/- 0.13 under normoxia and increased significantly to 0.67 +/- 0.16 un

118 on yeast exhibited growth defects under both normoxia and low oxygen conditions.

119 DO2_IR was similar under normoxia and moderate hypoxia (P = 0.7), but significant

120 Likewise, MO2_IR under normoxia and moderate hypoxia was similar (P = 0.1), but

121 on early after cardiac arrest such that both normoxia and normocarbia were documented in only 25 pati

122 tion guidelines that advocate maintenance of normoxia and normoventilation after pediatric cardiac ar

123 HIF-1alpha restores the microvascular airway normoxia and prevents airway fibrosis highlight a novel

124 dipocytes exhibited lower basal lipolysis in normoxia and reduced beta-adrenergic-stimulated lipolysi

125 1) min(-)(1); P =0.03), and unchanged during normoxia and sympathetic inhibition (219 +/- 19; P =0.86

126 oxia (FIO(2) =0.21), hypoxia (FIO(2) =0.11), normoxia and sympathetic inhibition (via 48 h transderma

127 glycogen provisioning when they experienced normoxia and to decrease embryo glycogen provisioning wh

128 re compared with wild-type littermates under normoxia and with exposure to either acute or chronic hy

129 t in normoxia, during hypoxia (P<0.05 versus normoxia), and especially during exercise (P<0.05 versus

130 ) was injected into the arterial line during normoxia, and during early and late hypoxia, and their h

131 ur growth to levels similar to those seen in normoxia, and in a HIF-2alpha-specific fashion, correlat

132 ected into the vitreous after restoration to normoxia, and its effects on vascular growth were analyz

133 omotor exercise in acute hypoxia compared to normoxia, and that such change would be related to reduc

134 Comparisons of normoxia- and hyperoxia-exposed samples were made by rea

135 ioning underlies adaptation to a fluctuating normoxia-anoxia hatching environment by increasing embry

136 , populations facing irregularly fluctuating normoxia-anoxia hatching environments failed to evolve r

137 )) clamps 1 week apart, randomized to either normoxia (arterial P(O2) (P(aO2)) 111 +/- 6.3 mmHg) or h

138 tylcysteine (NAC), HBO and NAC, and control (normoxia at sea level).

139 3.5% higher (P < 0.01) during hyperoxia than normoxia at steady state during the clamp (28.2 +/- 0.15

140 lyzed VHL-R167Q proteostasis and function at normoxia, at hypoxia with different oxygen pressure, and

141 increased levels under hypoxia compared with normoxia both in vitro and in vivo.

142 sue oxygen tension less than 20 mm Hg versus normoxia (brain tissue oxygen tension > 20 mm Hg).

143 mplex for driving RTA expression not only in normoxia but also in hypoxia.

144 pensable for muscle stem cell function under normoxia but are required for maintaining satellite cell

145 , which express F77 antigen moderately under normoxia but at an elevated level under hypoxia.

146 and von Hippel-Lindau protein (pVHL) during normoxia but not in hypoxia.

147 H(2)S decreased in the presence of NO under normoxia but not under anoxia indicating that H(2)S does

148 conclusion, CYGB revealed TSG properties in normoxia but promoted tumourigenic potential of the cell

149 to be critical in cardiac hypertrophy under normoxia, but its role in the heart under hypoxia is poo

150 ation of sodium nitrite had little effect in normoxia, but produced significant vasodilation and incr

151 C1 neurons have low activity under normoxia, but their activation is important to BP stabil

152 etabolic adaptation under hypoxia as well as normoxia, but whether HIF2alpha contributes to the contr

153 as a suppressor of tumor angiogenesis under normoxia by simultaneously down-regulating potent pro-an

154 e activity of these kinases is stimulated in normoxia by the oxygen-sensing prolyl hydroxylase PHD1 (

155 ow that chondrocytes cultured in hypoxia and normoxia can be differentiated by their lipid profiles.

156 In breast cancer cells under normoxia, CHD4 enrichment at HIF target gene promoters i

157 e and body weight were reduced compared with normoxia, cIH induced systemic insulin resistance in a h

158 Compared with normoxia, CO increased approximately 30% phagocytosis of

159 To test this, we treated rats held in normoxia (CON) or 10% O2 (CSH) for 7 days and measured v

160 t in liver mitochondria at a low level under normoxia conditions.

161 te hypoxia; (2) for the same duration but in normoxia (control); and (3) to the limit of tolerance in

162 ed among seven treatments after oviposition; normoxia (control; 21% O(2)), or hypoxia (1% O(2)) for 3

163 binding of other transcription factors under normoxia, control cell-type-specific hypoxia responses i

164 of normal blood vessel maturation similar to normoxia controls.

165 Bifurcated BACH2 controls during hypoxia and normoxia coordinate not only MCL tumor dispersal but als

166 gher in the hyperoxia group as compared with normoxia (crude odds ratio 1.7 [95% CI 1.3-2.1]; p < 0.0

167 cantly during chronic hypoxia but not during normoxia (Delta: 4.8 +/- 1.6 vs. 0.5 +/- 1.4 mumol l(-1)

168 od flow after 2 hours of hypoxia (hypoxia vs normoxia: Delta148ml/min(-1) , 95% confidence interval [

169 and near-physiological concentrations via a normoxia-dependent mechanism that is associated with cGM

170 to men, whereas the magnitude of fatigue in normoxia did not differ between sexes.

171 rmation (venous>arterial; P<0.05) at rest in normoxia, during hypoxia (P<0.05 versus normoxia), and e

172 hat ERF-VII N-terminal cysteine oxidation in normoxia enables arginylation followed by proteasomal de

173 cultured for 24 h as follows: Experiment 1) Normoxia, Experiment 2) Hypoxia, and Experiment 3) Lutei

174 ts of i.v. vasodilators in Sugen5416/hypoxia/normoxia-exposed PAH rats.

175 a very late stage of the Sugen 5416/hypoxia/normoxia-exposed rat is accompanied by the formation of

176 on, and DNA synthesis compared to cells from normoxia-exposed rats.

177 (means +/- SEM)), in a random order, during normoxia (FIO(2) =0.21), hypoxia (FIO(2) =0.11), normoxi

178 then either sampled (embryo) or returned to normoxia for 2 years (juvenile).

179 ays in comparison to both mild hyperoxia and normoxia for all metrics except for the worst PaO2.

180 oxia-exposed mice were allowed to recover in normoxia for an additional 56 days.

181 ted from pregnant mice exposed to hypoxia or normoxia from gestational day 14.5 to 18.5.

182 were exposed to repeated hypoxia or repeated normoxia from P1 to P3.

183 IV 4) and mature (DIV 20) neurons; 3) during normoxia GABA, glycine and taurine decreased GABA(A)Ralp

184 e interval {CI}, 60%-66%]) compared with the normoxia group (532/1171 [45%; 95% CI, 43%-48%]; proport

185 Hg (95% CI, -0.54 to 0.18; p < 0.001) in the normoxia group, but no such relation was demonstrable in

186 In the normoxia group, the mean brain tissue oxygen tension, ju

187 e small intestine and ascending colon of the normoxia group.

188 sh injected intraperitoneally with FLX under normoxia had resting cardiovascular and ventilatory para

189 Compared with normoxia, hypoxia significantly increased palmitate-indu

190 reased ventilation of EH hamsters exposed to normoxia, hypoxia, and hypercapnia.

191 hibited lower ventilation during exposure to normoxia, hypoxia, or hypercapnia, but comparable ventil

192 h activation (10 s) in conscious rats during normoxia, hypoxia, or hyperoxia.

193 Under normoxia, IGF activates the Akt-mTOR, p38, and Erk1/2 MA

194 creased ventilation in acute hypoxia but not normoxia in CH mice.

195 rols; i.e. acute hypoxia in CON and CSH, and normoxia in CSH.

196 1alpha (HIF-1alpha), which accumulates under normoxia in LKB1-deficient cells and is antagonized by i

197 ption factor to drive HK2 gene expression in normoxia in these cells.

198 AGO1 knockdown increased angiogenesis under normoxia in vivo.

199 allyl glycine, a stabilizer of HIF-1alpha at normoxia, increased IL-22 expression.

200 , the PHD inhibitors stabilise HIF-1alpha in normoxia, induce autophagy, and protect cells from a sub

201 s as a sensor of glucose availability during normoxia, inducing apoptosis in response to glucose depl

202 ia-inducible factor (HIF) target genes under normoxia is a prerequisite for HIF-mediated transactivat

203 g for proinflammatory cytokine production in normoxia is not obvious, and effects of 2-DG on cytokine

204 Hypoxia favors stem cell quiescence, whereas normoxia is required for stem cell activation, but wheth

205 sts that culturing cells in ambient air, or "normoxia," is far from physiological or "normal." In fac

206 IGF promotes myoblast differentiation under normoxia, it stimulates proliferation under hypoxia.

207 ry in four groups of ewes: normoxic control, normoxia + ketamine, hypoxic control and hypoxia + ketam

208 Recent studies suggest that atmospheric normoxia may constitute a cellular hyperoxia in mitochon

209 ate that the higher cholesterol levels under normoxia might regulate fibroblast growth factor 1 (FGF-

210 In normoxia, miR-574-3p, acting as a decoy, binds cytoplasm

211 we report that cytosolic acidification under normoxia moderately elevated 2-HG in cells, and boosting

212 80%, and were exposed to 13% O2 (hypoxia) or normoxia (n = 10 per group) for 14 days.

213 s of inspired oxygen (FiO2) to induce either normoxia (n = 10), moderate hypoxia (n = 14), or severe

214 rying male singleton fetuses were exposed to normoxia (n = 6) or hypoxia (10% inspired O(2) , n = 9)

215 centa from near-term C57BL/6J mice housed in normoxia (n = 8) or hypoxia (10% oxygen, n = 7-9) from d

216 baseline; PETO2 : 48 +/- 3 mmHg) and 20 s of normoxia (n = 9), or a 40 min air-breathing control (n =

217 Upon return to normoxia, Ndufs4 KO mice die within days.

218 In normoxia, neither lateral ventricular volume (R(2) = 0.0

219 l infarction, exosomes derived from neonatal normoxia, neonatal hypoxia, infant hypoxia, and child hy

220 cise at 20% maximal voluntary contraction in normoxia (NormEx) and isocapnic hypoxia (HypEx; O2 satur

221 uential exposure to hypoxia/hypoglycemia and normoxia/normoglycemia (H/H-N/N).

222 2) to fraction of inspired oxygen <300); and normoxia, not classified as hyperoxia or hypoxia.

223 O2/FIO2 ratio less than or equal to 300, and normoxia, not defined as hyperoxia or hypoxia.

224 g (7.99 kPa) or PaO2/FiO2 ratio </= 300, and normoxia, not defined as hyperoxia or hypoxia.

225 Hg (7.99 kPa) or PaO2/FiO2 ratio </=300 and normoxia, not defined as hyperoxia or hypoxia.

226 cardiopulmonary resuscitation compared with normoxia (odds ratio, 1.77; 95% CI, 1.03-3.30).

227 Alligator eggs were reared under normoxia or 10% hypoxia, then either sampled (embryo) or

228 e 2 diabetic rats were housed for 3 weeks in normoxia or 11% oxygen.

229 en or vehicle followed by exposure to either normoxia or chronic hypoxia (10% O2) for 30 days before

230 ppm) diet for 14 weeks and were continued in normoxia or exposed to hypoxia (8% O2) for the last 4 we

231 eurons contribute little to resting BP under normoxia or hypercapnia, C1 neuron discharge is restrain

232 r -deficient (ADM(+/-)) mice were exposed to normoxia or hyperoxia through postnatal days (PNDs) 1 to

233 After 7 d, mice were exposed to normoxia or hypoxia (10% O(2)) for the remainder of the

234 eterized sheep carrying singletons underwent normoxia or hypoxia (10% oxygen [O2]) +/- vitamin C trea

235 Chicken embryos were incubated under normoxia or hypoxia (14% O(2) ) from day 1 +/- pravastat

236 embryos (n = 11 per group) were incubated in normoxia or hypoxia (14% O2 ) from day 1 and treated wit

237 ncy, rats (n = 20 per group) were exposed to normoxia or hypoxia +/- vitamin C.

238 Cold exposure in normoxia or hypoxia increased mitochondrial leak respira

239 Exposure to cold in both normoxia or hypoxia increased the activities of citrate

240 In normoxia or hypoxia, RTN inhibition produced a more sust

241 th in-hospital death as compared with either normoxia or hypoxia.

242 CF-7 extract prepared from cells grown under normoxia or hypoxia.

243 F4E is the dominant cap-binding protein (21% normoxia or standard cell culture conditions), where eIF

244 RASF) under hypoxic conditions but not under normoxia or TNF-alpha treatment.

245 tes HIF-1alpha protein levels under aerobic (normoxia) or anaerobic (hypoxia) conditions.

246 m hypoxia (12 kPa O(2) ), cold (5 degrees C) normoxia, or cold hypoxia.

247 (1) Under normoxia, overexpression of N-end-rule-insensitive Delta

248 (2) decreased significantly as compared with normoxia (P < 0.001; n = 10).

249 creased V(E) from 1.1 to 1.5 mL/min/g during normoxia (P < 0.01) and from 3.6 to 4.7 mL/min/g during

250 creased V(E) from 1.1 to 1.3 mL/min/g during normoxia (P < 0.05) and from 2.8 to 3.2 mL/min/g during

251 ionships between hypoxemia (PaO2 < 60mm Hg), normoxia (PaO2 60-100mm Hg), moderate hyperoxia (PaO2 10

252 tion during anoxia alone or by NO2(-) during normoxia places constraints on how S-nitrosation occurs

253 RBC ATP release during normoxia ( PO2 ~112 mmHg) and hypoxia ( PO2 ~20 mmHg) wa

254 ned implants remained cartilaginous, whereas normoxia-preconditioned implants readily underwent calci

255 tors antimycin A and potassium cyanide under normoxia promotes transient MPK6 and MPK3 activation.

256 staurin reversed PH in the Sugen5416/hypoxia/normoxia rat model, by improving right ventricular systo

257 ed a high incidence of mortality compared to normoxia rats.

258 der the noradrenaline curve (relative to the normoxia response) was increased with hypoxia (137 +/- 1

259 c hearts that were metabolically abnormal in normoxia, resulting in glycolytic rates 30% lower, and f

260 ption, and neither hypoxia nor atpenin A5 in normoxia robustly stabilizes hypoxia-inducible factor (H

261 4 atm) for 21 days and allowed to recover at normoxia (room air) for up to 21 days.

262 s(-1)) on a level gradient under normobaric normoxia (room air, 21% O2), moderate hypoxia (15% O2),

263 Under normoxia, SCD mice display bone loss and bone impairment

264 ecular oxygen and 2-oxoglutarate that, under normoxia, selectively hydroxylate proline residues of HI

265 In vivo studies confirm normoxia silencing, hypoxic induction, and cell specific

266 rformed on cancer cells grown in hypoxia and normoxia strongly suggest that DNQ undergoes bioreductio

267 1) expression were reduced compared with the normoxia/SU5416 control group.

268 ic subunits caused cardiac restriction under normoxia that are further aggravated under hypoxia.

269 Compared with normoxia, the breathing frequency reduction (Deltaf(R))

270 Compared to normoxia, the controls' alveolar-to-arterial oxygen grad

271 At normoxia, the levels are kept low as a consequence of th

272 In normoxia, the magnitude of reduction in P(di,tw) post-PT

273 Under normoxia, the oxygen-sensitive alpha subunit of HIF1 is

274 In normoxia, they use ubiquinone (UQ), but in anaerobic con

275 In normoxia, three isoHbs predominated (Hbalpha-3.1, -3.2,

276 table during hypoxia but destabilized during normoxia through their targeting to the N-end rule pathw

277 dazole increased from 0.44% +/- 0.17% during normoxia to 2.24% +/- 0.08% during hypoxia.

278 A shorter protocol with normoxia to hyperoxia was also performed (five levels of

279 The mean change in RBC ATP release from normoxia to hypoxia in control conditions was significan

280 erfusion pressure (p = 0.004) when comparing normoxia to hypoxia.

281 response to hypoxic gradients extending from normoxia to severe hypoxia, and therapy responsiveness,

282 |), and "sensitivity" score (|z(Hypoxia) - z(Normoxia)) to quantitatively evaluate the proteome shift

283 Whereas normoxia-treated KO mice die from neurodegeneration at a

284 delivery female offspring were maintained in normoxia until 4 months of age.

285 urally delivered offspring were raised under normoxia until early adulthood (9 months).

286 hypoxia inducible factor (HIF) pathway under normoxia using a prolyl-4-hydroxylase inhibitor, dimethy

287 under physiologically normal oxygen levels (normoxia) vaccinia virus (VACV) infection leads to a rap

288 9.6 +/- 43.2% vs. 296.0 +/- 43.9% LF/HFHRV , normoxia vs. hypercapnia, respectively), incidence of ca

289 lly exposed as adults to warm (25 degrees C) normoxia, warm hypoxia (12 kPa O(2) ), cold (5 degrees C

290 Normoxia was documented in 34% and hypoxia in 22% of pat

291 urements of inner retinal OEF obtained under normoxia were compared between nasal and temporal retina

292 as ventilation and blood gases at rest under normoxia were normal.

293 ntributes to the low sensory activity during normoxia, whereas H2S is excitatory and mediates sensory

294 n diminished white matter vascular growth in normoxia, whereas loss of Wnt7a/b function blunted the a

295 awake, during normal ventilation, and during normoxia, which contributed to hypertension and organ da

296 n of downstream protein-coding regions under normoxia, which was further modulated by hypoxia.

297 zation of hypoxia-inducible factor-1alpha in normoxia, which was required for increased mitochondrial

298 d flow and femoral blood flow (P<0.05 versus normoxia) with further, more pronounced increases observ

299 agmatic work did not differ between sexes in normoxia (women: 12,653 +/- 1796 cmH(2) O s(-1) , men: 1

300 In normoxia, women and men display a comparable magnitude o