ライフサイエンスコーパス: not clear

1 ard decisions during temporal discounting is not clear.

2 th, but how these peptides enter the cell is not clear.

3 ce of ck-RNAi in distinct plant pathogens is not clear.

4 ligand recognition and activation of FPRs is not clear.

5 ps of CLEC-2-induced platelet activation are not clear.

6 een known for many years, how this occurs is not clear.

7 this predates, or is driven by, symptoms is not clear.

8 eta for substrate search and recognition are not clear.

9 in viral control and in immune pathology is not clear.

10 s defect and its impact on gametogenesis are not clear.

11 ver, the underlying molecular mechanisms are not clear.

12 by which the EsxA:B heterodimer separates is not clear.

13 echanisms underlying these abnormalities are not clear.

14 associated with alphaherpesvirus latency are not clear.

15 ct role of CD80 in HSV-1 immune pathology is not clear.

16 g entrainment of the light-mediated clock is not clear.

17 However, how NS5 engages SLA is not clear.

18 hes for this resource-intensive strategy are not clear.

19 h it intersects with the neuroimmune axis is not clear.

20 nd subsequent inflammation and cell death is not clear.

21 ed by W to regulate these host responses are not clear.

22 the link between MCs and NLR-mediated HR are not clear.

23 22 production by CD4(+) T cells and ILCs are not clear.

24 for the regulation of cardiac vagal tone are not clear.

25 r mechanism underlying the mRNA reduction is not clear.

26 ogical mechanisms underlying this effect are not clear.

27 e, but mechanisms underlying this effect are not clear.

28 n nature, but their biosynthetic origins are not clear.

29 ts different roles are coordinated are still not clear.

30 ng risk for outcomes in patients with CKD is not clear.

31 on nanoparticle diffusion dynamics is still not clear.

32 ve immunity on responses to RSV vaccines are not clear.

33 r dispersal of poultry-adapted AI viruses is not clear.

34 n to viral rebound upon ART interruption are not clear.

35 Why these patients relapse is not clear.

36 ystematic disentanglement of DNA in vivo are not clear.

37 , but its role in the replication process is not clear.

38 his disease affects alveolar bone healing is not clear.

39 pecially for the sporadic variant, are still not clear.

40 vivorship trajectory in the United States is not clear.

41 receptor but the relevant partner in vivo is not clear.

42 arget for anti-oxidant treatment in vivo are not clear.

43 ut the cellular nature of these processes is not clear.

44 pontaneous DNA damage during development are not clear.

45 eservoir conditions on salt precipitation is not clear.

46 e N-terminal extension in UFM1 activation is not clear.

47 mechanisms of HNO(3(s)) photolysis are still not clear.

48 t the mechanisms regulating polarization are not clear.

49 s the role of disorder in protein folding is not clear.

50 anisms regulating NAMPT expression are still not clear.

51 ed T cells proceed in their dead-end fate is not clear.

52 or mechanism of this metabolic aberration is not clear.

53 bal transcriptional response to infection is not clear.

54 is of diet-induced atherosclerosis are still not clear.

55 idization events in modern breeding is often not clear.

56 ans, but how HCMV avoids humoral immunity is not clear.

57 of the large and small airways in asthma is not clear.

58 l role of FGF21 during thermal challenges is not clear.

59 ubunit isoforms influence enzyme activity is not clear.

60 nce of ART on subclinical atherosclerosis is not clear.

61 he precise molecular mechanisms involved are not clear.

62 aging at the post-transcriptional level are not clear.

63 re evidence, but the mechanisms involved are not clear.

64 ctors mediating this mortality reduction are not clear.

65 , exactly how this packaging event occurs is not clear.

66 A mutations, but the underlying mechanism is not clear.

67 pulation but its role in cardiac patients is not clear.

68 h the relevance of the PSD-95 interaction is not clear.

69 hanism by which SYK affects Tau pathology is not clear.

70 lecular mechanisms of inflammation are still not clear.

71 risk of dementia in asymptomatic subjects is not clear.

72 he optimal timing of such an intervention is not clear.

73 , but the pathophysiological role of Pyk2 is not clear.

74 tion in nucleotide-dependent RCK domains are not clear.

75 mechanisms underlying these associations are not clear.

76 on alveolar type 2 (AT2) epithelial cells is not clear.

77 le of polyphosphate in neuronal signaling is not clear.

78 pressed Wnt ligands on tooth development are not clear.

79 o lineage-specific differentiation defect is not clear.

80 cally contributes to genome organization are not clear.

81 ks of complex and specific heart defects are not clear.

82 eam signals to regulate the Hippo pathway is not clear.

83 ecular mechanisms by which they interact are not clear.

84 inant T-helper 17 (Th17) response in PSC are not clear.

85 underlying such a duality in CD5 function is not clear.

86 the development of prediabetes/diabetes are not clear.

87 of Li enrichment in arc magmatic systems is not clear.

88 s for this dominant TH17 response in PSC are not clear.

89 sed cMyBP-C phosphorylation in the heart are not clear.

90 the rates and extents of biodegradation are not clear.

91 tion and attention to choice alternatives is not clear.

92 he individual subunits on enzyme activity is not clear.

93 role in visual perceptual decision-making is not clear.

94 ed histidine-containing dipeptides (HCDs) is not clear.

95 gence), but the role of JH in this switch is not clear.

96 ith innate responses at the tissue level are not clear.

97 y, how Cdc42p regulates the fMAPK pathway is not clear.

98 interaction during malaria transmission are not clear.

99 hat the net effect on species coexistence is not clear.

100 and the cause of the opposing conclusions is not clear.

101 e instructive roles of mechanical inputs are not clear.

102 the relationship between these variables is not clear.

103 ut the precise pro-metastatic mechanisms are not clear.

104 conserved gene in filamentous ascomycetes is not clear.

105 olling fungal responses to the plant cell is not clear.

106 se responses are coordinated by the brain is not clear.

107 tion of non-coding RNAs to complex traits is not clear.

108 expression changes, or gene duplication are not clear.

109 but the role of vitamin D in this process is not clear.

110 thelial barrier for spreading to the body is not clear.

111 channels are activated by depolarization is not clear.

112 e whose role in PEI and PEII biosynthesis is not clear.

113 etic basis of this broad adaptation is still not clear.

114 arrives in the extracellular environment is not clear.

115 nderlying this thymic positive selection are not clear.

116 ted in virulence in mice yet surprisingly is not cleared.

117 ng, whereas MGAS2221 DeltasagA infection was not cleared.

118 ot have been possible if the obstruction was not cleared.

119 f-time of 293 +/- 157 h (33 patients) or did not clear (19 patients).

120 he mechanisms that underlie this benefit are not clear(4-6).

121 Furthermore, it is not clear a priori whether experimental approaches based

122 However, accounts are usually not clear about how motivation functions during the appl

123 mismatch might be because current theory is not clear about the role of behavior in individual alloc

124 The infection was not cleared after the first treatment with convalescent

125 pendent safeguards, genome-damaged cells are not cleared, alleviating microcephaly, but paradoxically

126 ompounds with relatively short half-lives is not clear and may require either a longer deployment tim

127 events take place with different viruses is not clear and the effects of glycosylation on the foldin

128 The reasons for these disparate results are not clear and warrant further investigation of abatacept

129 Further, this role is not clear because these viruses have typically lower pac

130 in disease initiation of these cytokines is not clear, but plasmacytoid dendritic cells have been th

131 n 15 of those patients (6.6%), parasites had not cleared by 72 hours after initiation of therapy.

132 persion, but the mechanism for this is still not clear; cells grown in 3D have opportunities to devel

133 isms that allow cells to use iron safely are not clear; defects in iron management are a causative fa

134 ciated with reservoirs of CHIKV RNA that are not cleared efficiently by adaptive immune responses.

135 of LSPR-induced chemical reactions is still not clear, even for H(2) dissociation on metal nanoparti

136 form of N and with the ribonucleoprotein is not clear for HMPV or other major human pathogens, inclu

137 Moreover, it is not clear from past studies whether anxiety can be susta

138 king and alcohol in head and neck cancer are not clear, given the strong association between these ri

139 Paradoxically, it is not clear how a neuronal dynamical system running in the

140 However, it is not clear how AAG might accommodate eG in an active site

141 It is not clear how active and passive forces are integrated t

142 Moreover, it is not clear how aging affects the metacontrol of decision

143 increased risk of severe COVID-19, but it is not clear how cigarette smoke (CS) exposure affects SARS

144 It is not clear how consumption of beneficial commensal microb

145 It is not clear how estrogen promotes hepatic growth.

146 e bees may support DWV replication but it is not clear how infection could occur under natural enviro

147 It is not clear how inhibitors of cytokines, such as antagonis

148 However, it is not clear how insulin self-antigen controls the selectio

149 nt tuberculosis (MDR-TB) is common and it is not clear how it affects interim and treatment outcomes.

150 ssed in the intestinal epithelium, but it is not clear how its activation affects ISCs and regenerati

151 It is not clear how LACC1 balances defenses against intestinal

152 However, it was not clear how LITAF might impact cardiac excitation.

153 andard clinical endpoints, since it is often not clear how much benefit each individual patient recei

154 However, it is not clear how Nav1.8 disease mutations induce sensory ne

155 underway in patients with cancer, but it is not clear how neutrophils contribute to colon tumorigene

156 In particular, it is not clear how olfactory circuits achieve rapid, data eff

157 It is not clear how pancreatic cancer stem cells (CSCs) are re

158 r diagnosing bacteremic illnesses, yet it is not clear how physicians in many low- and middle-income

159 It is not clear how regulation of T-cell function is altered d

160 However, it is not clear how RNA polymerase initially recognizes such s

161 nicate mainly via discrete spikes, and it is not clear how spikes give rise to dynamics that can be u

162 It is not clear how spontaneous DNA double-strand breaks (DSBs

163 It is not clear how such complex examples of host manipulation

164 It is not clear how the auditory system deals with this proble

165 However, it is not clear how the brain acquires predictive representati

166 ave reported heterogeneous results and it is not clear how the clinical heterogeneity is related to t

167 al-to-mesenchymal transition; however, it is not clear how the epicardium contributes to disease prog

168 odulate bacterial physiology; however, it is not clear how the indole signal is perceived and respond

169 It is not clear how the N protein mediates such distinct funct

170 In particular, it is not clear how the positions of the catalytic dwells infl

171 cal validation is largely lacking, and it is not clear how the power of deep learning should be used

172 le dissociation kinetics in vitro, and it is not clear how the rapid turnover of APC/C-activator comp

173 ese differences are widely recognized, it is not clear how the transition from fetal to adult identit

174 ne reaction and a repair response, but it is not clear how these are coordinated.

175 However, it is not clear how these changes within the microenvironment

176 However, it is not clear how these extensions affect the chemomechanica

177 as a strong predictor of relapse, but it is not clear how these findings apply to patients who test

178 can occur for any sensory domain, but it is not clear how these separate surprise signals are integr

179 are fragile early in development, and it is not clear how they adjust to support breathing at birth.

180 larger than the mesh size of the ECM, it is not clear how they can travel through the dense ECM.

181 Pol II) passage through chromatin, yet it is not clear how they cooperate mechanistically.

182 at the lysosome is well characterized, it is not clear how this complex is regulated at other subcell

183 However, it is often not clear how to combine ontologies or how to assess or

184 reads to ETRs remains an open problem, it is not clear how to polish draft ETR assemblies.

185 It is not clear how Vgamma9Vdelta2 T cells react toward the su

186 icit approaches are now available, but it is not clear how we can best draw on this expanded modellin

187 It is not clear however, whether the limited linguistic abilit

188 So far it is not clear i) to which extent DON is degraded during baki

189 However, it is not clear if altered fear memory can also modify risk fo

190 However, it is not clear if and how the SnRK1-mediated sugar signaling

191 It is thus not clear if functional TF and pathway analysis tools es

192 It is not clear if other lipids related to serine-glycine lipi

193 In plants, it is not clear if palindromic repeats play a role in maintain

194 Equally, it is not clear if suppression of the cytotoxic program by ThP

195 However, it is not clear if the brain can actually take advantage of th

196 apabilities at the cellular level, but it is not clear if the different onset of stem cell aging betw

197 However, it is not clear if these results predict the ecosystem consequ

198 It is not clear if this also applies to diverse new cell produ

199 tis tends to be higher in women, although is not clear if this is related to a sex/gender effect or t

200 d intellectual disability in boys, but it is not clear if this is through changes in the O-GlcNAc pro

201 ludes ursodeoxycholic acid (UDCA), but it is not clear if UDCA protects the fetus.

202 contributions of the ANS and SAN to HRV are not clear, impeding effective treatment.

203 and structural changes of the materials are not clear, in part due to the difficulty in identificati

204 wever, what shapes microbiota composition is not clear, in particular the role of genetic factors.

205 chanism of APOL1-mediated podocyte damage is not clear, including APOL1's subcellular localization, t

206 The nature of the imprint is not clear: it is either a nick or a ribonucleotide inser

207 patic and extrahepatic tissue extraction are not clear.METHODSA hyperinsulinemic-euglycemic clamp and

208 the effect of weight loss on hepatic DNL are not clear.METHODSHepatic DNL, 24-hour integrated plasma

209 and circulating inflammatory monocytes, did not clear microglial debris.

210 y life acetaminophen on asthma risk is still not clear, potential interactions with glutathione S-tra

211 ould eliminate representations, hence, it is not clear that Brette's appeal to it is going to solve t

212 ness and the scale of competition, and it is not clear that these assumptions reflect the natural set

213 microbial community, but the transplants did not clear the M. abscessus infections and both patients

214 .028) compared with DP; and patients who did not clear their initial parasitaemia within 2 days (AHR

215 Although the direction of causality is not clear, this association strengthens the role for die

216 However, it is not clear to what extent these treatments have unique an

217 r adult patients undergoing hemodialysis are not clear, we aimed to identify the extent and patterns

218 The function of this circuit is not clear; we previously suggested based on computationa

219 However, it is not clear what and where neural responses in the auditor

220 It is not clear what cargo these vesicles contain and how they

221 However, it is not clear what constitutes an R0.

222 It is not clear what drives this differential progression rate

223 ADAs arise inconsistently, and it is not clear what factors determine their formation.

224 However, it is not clear what might be the diagnostic benefit and accur

225 However, it is not clear what promotes transition of ISCs to progenitor

226 However, it still is not clear what the active ingredients of many successful

227 However, it is not clear what tips the balance of protein homeostasis a

228 In addition, it is not clear whether a common set of regions are involved i

229 It is not clear whether active surveillance for thyroid cancer

230 (HCV) infection in the United States, it is not clear whether all affected persons have been diagnos

231 However, it is not clear whether all forms of proteinuria are damaging.

232 It is not clear whether alterations in the intestinal microbio

233 ed TDP-43 protein moves between cells, it is not clear whether and how this movement propagates the d

234 utonomic nervous system (ANS) changes, it is not clear whether ANS markers, if recorded from a wearab

235 eated with anti-VEGF therapy; however, it is not clear whether anti-VEGF treatment is causative of at

236 It was not clear whether any of the other 29 genes in the 1.3-M

237 It is not clear whether aromaticity has a size limit, or wheth

238 eptor signaling in native systems, and it is not clear whether biased M(1) PAMs display differences i

239 From LTEM imaging alone, it is not clear whether biskyrmions are surface-confined objec

240 However, it is not clear whether cardiac troponin T (cTnT) and troponin

241 It is not clear whether closure of mucosal defects with clips

242 It is also not clear whether cognitive deficits in Gambling Disorde

243 It is not clear whether compounds that ended up being selected

244 ch as IFN-gamma) induce LGC formation, it is not clear whether cytokines that trigger macrophage diff

245 However, it is not clear whether decreased levels of hepatic BRD7 are d

246 With small cohorts of KS2 patients, it is not clear whether differences exist in clinical manifest

247 Thus, it is not clear whether differences found in comparative studi

248 It is not clear whether distinct mechanisms of action can be a

249 However, it is not clear whether endogenous ANG is essential for produc

250 chemical signal is not understood, and it is not clear whether FAK is activated directly by force or

251 However, it is not clear whether integrating the opposing sides under a

252 ociated with Crohn's disease (CD), but it is not clear whether it is a cause or result of the disease

253 It is not clear whether network degeneracy impacts network fun

254 It is not clear whether or how neutrophils enhance recovery fr

255 esity-related diseases is ongoing, but it is not clear whether plasma and serum can be used interchan

256 It is also not clear whether smaller tRFs are generated from the tR

257 It is not clear whether sound information that reaches the str

258 However, it is still not clear whether spatial maps in sensorimotor regions k

259 However, it is not clear whether supplementary gene transfer can rescue

260 sed for the treatment of epilepsy, but it is not clear whether targeting distinct GABA(A) receptor su

261 It is not clear whether the associations with red and processe

262 Nonetheless, it is not clear whether the continued activity of these pionee

263 It is not clear whether the developmental impacts of NMDAR dys

264 l of these species have parental care, it is not clear whether the early preference for faces is a me

265 However, it is not clear whether the enhanced nuclear uptake of [Ru(bpy

266 However, it is not clear whether the ER is remodeled during motile even

267 It is not clear whether the kinetic parameters that determine

268 It is not clear whether the presence of cognitive impairment c

269 It is not clear whether the same phenomenon occurs at conventi

270 to facilitate information processing, it is not clear whether the value of task-irrelevant distracto

271 sicians compared to non-musicians, but it is not clear whether these differences are robustly linked

272 However, it is not clear whether these drugs are effective for long-ter

273 It is not clear whether these effects are distinct or related

274 otions such as "anger" and "fear," yet it is not clear whether these emotions have similar meanings a

275 MTR10p and Kap120p in yeast, although it is not clear whether these import factors are independent o

276 However, it is not clear whether these infections are more severe than

277 It is not clear whether these methods can significantly improv

278 utation detection from DNA sequencing, it is not clear whether these methods work for scRNA-seq data.

279 in patients with type 2 diabetes, but it is not clear whether these occur via the same mechanisms.

280 However, it is not clear whether this is done through a graded coding,

281 pite promises of increased efficiency, it is not clear whether this paradigm shift will change how we

282 ges in genetic ancestry over time, but it is not clear whether those changes in ancestry are associat

283 It is not clear whether treatment with direct-acting antiviral

284 Despite this it is still not clear whether, and if so how, schooling fish coordin

285 It is still not clear which are the most important prognostic factor

286 y known MAPs inhibit branch formation, it is not clear which MAP is responsible for regulating microt

287 es in microRNA (miRNA) processing, but it is not clear which miRNAs regulate this process.

288 However, it is not clear which of the clinically identified RNF43 mutat

289 Unfortunately, it is not clear which of these low Model for End-Stage Liver D

290 evelopment and progression of IBD, but it is not clear which populations of microbes are involved or

291 as a wide repertoire of motor actions, it is not clear which set of movements and postures represent

292 te previous evidence for heritability, it is not clear which specific genetic loci are implicated in

293 Yet, it is not clear which specific WM processes are affected in th

294 YNGAP1 in several cognitive disorders, it is not clear which SynGAP isoforms contribute to disease.

295 However, it is not clear which types of features are best for optimizin

296 However, it is not clear why cleavage is required for Rb degradation.

297 It is not clear why some brain regions are impaired and others

298 a fraction of all protein contacts and it is not clear why some contacts are favoured over others.

299 llular carcinoma (HCC) than women, but it is not clear why.

300 However, it is not clear yet what determines the exciton transport alon