ライフサイエンスコーパス: nothing

1 bles with constant outcome values (0.5 ml or nothing).

2 g the Nash equilibrium action, namely giving nothing).

3 e avoiding the overused approach of assuming nothing.

4 vy financial burden while arguably achieving nothing.

5 in which starvation corresponded to winning nothing.

6 onder rejects the proposal, both players get nothing.

7 The control group received nothing.

8 rable even when in vivo examination detected nothing.

9 sus the negative control PEU films releasing nothing.

10 ects swallow up whatever comes near and emit nothing.

11 and used only single antiplatelet therapy or nothing.

12 for 90 d, whereas control subjects received nothing.

13 l suppression (1%), mitigation (10%), and do nothing (80%).

14 atory reverberations create something out of nothing: A third flash where there are only two.SIGNIFIC

15 Understanding its workings, to say nothing about intervening in them, is among the hardest

16 I knew nothing and had thought nothing about parasites until 1971.

17 A few decades ago, we knew next to nothing about the behavior of our closest animal relativ

18 However, we know nothing about the endogenous control of hemocyte develop

19 We assume nothing about the Markov chain beyond reversibility and

20 anging behavioral states, yet we know almost nothing about the mechanisms of such regulation.

21 We know almost nothing about the N form preferentially transferred to t

22 thenticity of submitted items while learning nothing about them.

23 le for the clients to share a key and reveal nothing about what key has been agreed upon to the serve

24 , but not all veins, contrasting with all-or-nothing alternatives available with the single-order vei

25 I knew nothing and had thought nothing about parasites until 19

26 ing among people otherwise inclined to share nothing and increases the proportion of participants wil

27 ductive dechlorination rates; however, a "do nothing" approach that relies on indigenous diazotrophs

28 A fourth option, a 'do-nothing' approach, is wholly inconsistent with the well-

29 t steering can be detected by the all-versus-nothing argument experimentally even in the presence of

30 -Podolsky-Rosen steering based on all-versus-nothing argument, offering a strong condition to witness

31 1), popular fiction (experiments 2 to 5), or nothing at all (experiments 2 and 5).

32 to disparity in procedural approach, "leave nothing behind" or "combination therapy," and vessel siz

33 been prepared via hydrothermal synthesis in nothing but H(2) O under high-pressure and high-temperat

34 o create mirrors for space telescopes, using nothing but microscopic particles held in place by light

35 s, whereas Fourier-defined "brain waves" are nothing but poorly resolved oscillons.

36 Native assemblies are obtained using nothing but their simulated profiles as restraints and e

37 Thus, nothing can be affirmed a priori concerning the binding

38 represents distress or unmet need, but that nothing can be done to alleviate the calling out.

39 One in three survivors cited "nothing can be done" as a reason for not discussing dail

40 energy recovery scenarios compared to a "do nothing" case.

41 ic theories that allow everything but demand nothing, cases where seeing and thinking conflict, menta

42 binding triggers a highly localized, all-or-nothing change of water accessibility to the transport s

43 us recombination DNA repair is not an all-or-nothing concept, but a spectrum, and that where a tumor

44 ene guests was observed to effect the all-or-nothing cooperative templation of an S6-symmetric host s

45 ered at FLC in individual cells in an all-or-nothing (digital) manner or is continuously varying (ana

46 n, such that the group identifies itself and nothing else in a search of GenBank.

47 When nothing else is known, the base rate principle favors Jo

48 to prefer to be doing something rather than nothing, even if that something is negative.

49 h, we demonstrate that MOMP is not an all-or-nothing event.

50 bit of information to coordinate the all-or-nothing expression of early genes, but also over a longe

51 ure, cells respond autonomously in an all-or-nothing fashion, with the fraction of cells that stably

52 he complex at the cost of its normal, all-or-nothing functionality.IMPORTANCECaudovirales encode cell

53 However, nothing further is known about the activation process.

54 creases, slot machine switches and double or nothing gambles) was associated with this self-reported

55 network was associated with more 'double or nothing' gambles, weighted by connections between the su

56 sands of genes of which we understand almost nothing: genomics has failed biology.

57 elf in highly predictable environments where nothing happens.

58 e neural and muscular systems, and virtually nothing has been reported about their embryogenesis.

59 Almost nothing has been reported on longer term prognosis follo

60 Woolf is also quoted as saying "nothing has really happened until it has been described,

61 Nothing, however, is known about the molecular identity

62 If vaccine action is all-or-nothing (ie, a proportion of vaccine recipients receive

63 Disagreement primarily occurred in the "nothing in bed" versus "in-bed activity" categories beca

64 ty scale was collapsed into four categories: nothing in bed, in-bed activity, out-of-bed activity, an

65 ed in different cognitive domains often have nothing in common.

66 partner could not reciprocate, and expected nothing in return.

67 h both subject groups contributing virtually nothing in the control condition.

68 for a ruthenium catalyst, but since there is nothing in the mechanism restricting it to this metal, t

69 nts might argue that we are advocating doing nothing instead, but we are not; we are only advocating

70 nion transporters has flourished from almost nothing into a rapidly maturing field of research.

71 Almost nothing is currently known about the maturation of funct

72 gradation of all these compounds, yet almost nothing is currently known about what these fungi do or

73 e cell types including conventional T cells, nothing is currently known concerning Notch function in

74 (iv) there is two-sided irreversibility; if nothing is done and the problem proves serious, the clim

75 plained by cold winters or hot summers, then nothing is explained.

76 own for its role in olfaction, but virtually nothing is known about a clade of approximately 35 membe

77 However, nothing is known about a potential functional interactio

78 Meanwhile, almost nothing is known about App's function in glia, where it

79 of established asthma in schoolchildren, but nothing is known about ASM in preschool wheezers.

80 on by bacteria is well described, but almost nothing is known about bacterial iron export even though

81 le biofilm formation is well studied, almost nothing is known about biofilm dispersal.

82 are our closest living relatives but almost nothing is known about bonobo internal anatomy.

83 nhibitors are in clinical trials, yet almost nothing is known about Brd4 function in the brain.

84 hat adults are broadly exposed to OPFRs, but nothing is known about children's exposure.

85 However, nothing is known about epigenetic regulation of this Ins

86 However, almost nothing is known about events leading to this islet auto

87 However, little or nothing is known about expression in moderately-sized (1

88 However, nothing is known about FucP conformational dynamics.

89 ted traffic to and from the plasma membrane, nothing is known about GORK, its distribution and traffi

90 s do not transfer to other tasks, and almost nothing is known about how activation in IC-related brai

91 Yet little to nothing is known about how coacervates evolve into intri

92 s of DISC1-binding proteins reported, almost nothing is known about how DISC1 interacts with other pr

93 e Gi and abrogate PI3K-Akt signals; however, nothing is known about how GIV's GEF function is regulat

94 ied camphor elicits a sensation of cool, but nothing is known about how it affects cold temperature s

95 ories on aging on the cellular scale, nearly nothing is known about how microscopic failures cascade

96 However, nothing is known about how potential posttranslational m

97 ons as a modulator of inflammation; however, nothing is known about how Rc3h1 expression is regulated

98 signaling remain incompletely understood, as nothing is known about how superficial dorsal horn neuro

99 int and the spindle assembly checkpoint, and nothing is known about how T. brucei controls its cell c

100 Almost nothing is known about how these three-dimensional topol

101 a decrease in FSAP expression but, to date, nothing is known about how this might be regulated.

102 To date, nothing is known about how wounding activates DUOX.

103 ges in vitro by a variety of mechanisms, but nothing is known about intracellular bacterial trafficki

104 However, virtually nothing is known about intracellular signaling proteins

105 d the neuroprotective properties of PEA-OXA, nothing is known about its effects on the gut-brain axis

106 lso used for wine and cider fermentation but nothing is known about its natural history.

107 muscle cell contraction in several tissues, nothing is known about its possible role in tracheal smo

108 in this process have been characterized, but nothing is known about its regulation.

109 However, nothing is known about its role in memory retrieval afte

110 or (HIF) in vitro and in cultured cells, but nothing is known about its roles in mammalian erythropoi

111 Almost nothing is known about Kv2 channel functions in medial e

112 al infections has only recently emerged, and nothing is known about MDSC function in the context of S

113 , 2 and 4 have been characterized, virtually nothing is known about members of cluster 3 (ACA12 and A

114 ptor 2 (NPFFR2) mRNA is highest in placenta, nothing is known about NPFF-NPFFR2 functions in placenta

115 However, nothing is known about SAMHD1 posttranslational modifica

116 However, almost nothing is known about strike performance of viperid sna

117 a number of biological functions, virtually nothing is known about structure-function relationships

118 However, nothing is known about the alteration and role of MEG3 i

119 extracellular electron transfer, and almost nothing is known about the associated mechanisms.

120 characterized to a limited extent and almost nothing is known about the behavior of these materials u

121 Curiously, nothing is known about the behavior of this cellular mac

122 Nothing is known about the binding mode, however.

123 ence suggests that IscS and Fdx interact but nothing is known about the binding site and the role of

124 nt biochemical function between GNC and GNL, nothing is known about the biological role of the four o

125 However, nothing is known about the cell surface dynamics of Kv7.

126 In contrast, almost nothing is known about the circuitry or laminar features

127 ains with short incubation periods; however, nothing is known about the conversion of the long-incuba

128 Nothing is known about the effects of AHLs on the earlie

129 Virtually nothing is known about the effects on fetal physiology o

130 n numerous nonerythroid cells, but virtually nothing is known about the expression and potential sign

131 expression directly or indirectly, virtually nothing is known about the extent to which CodY exerts i

132 However, nothing is known about the factors that ensure effective

133 Yet, almost nothing is known about the fate of stalled replication i

134 Almost nothing is known about the final (completion) step becau

135 nello import studies and in silico analyses, nothing is known about the function and properties of pr

136 rane domains have been analyzed extensively, nothing is known about the function of CAR-D2 and the ex

137 Nothing is known about the function of hTim8a in neurona

138 e been investigated extensively, practically nothing is known about the function of NRG2, the closest

139 Essentially nothing is known about the functions of SEIPIN homologs

140 However, almost nothing is known about the heterochromatin's structural

141 However, essentially nothing is known about the identity of these inhibitory

142 ) triggers prominent motivation deficits but nothing is known about the impact of this exposure in th

143 y to deletion in P. falciparum in vitro, but nothing is known about the in vivo functions of PAT in P

144 However, nothing is known about the influence of Jak/Stat-signali

145 o-inflammation and -degeneration, but almost nothing is known about the interactions between the peri

146 known about epigenetic signaling in glia and nothing is known about the intersection of glial epigene

147 produce nitrous acid, HONO, but essentially nothing is known about the isomeric nitrosyl-O-hydroxide

148 nd ability to transmit vertically, virtually nothing is known about the life history of these endobac

149 ortant to achieving high toxicity, virtually nothing is known about the matrix that binds the toxin i

150 Almost nothing is known about the mechanism by which a magnetic

151 However, nothing is known about the mechanism of hepcidin binding

152 Nothing is known about the mechanisms by which increased

153 However, nothing is known about the mechanisms controlling chromo

154 Almost nothing is known about the mechanisms of dissimilatory m

155 However, almost nothing is known about the mechanisms of these polymeras

156 ntially important physiological role, almost nothing is known about the mechanisms responsible for th

157 chwann cell development during this process, nothing is known about the mechanisms that mediate the m

158 Despite the importance of DGs, nothing is known about the mechanisms underlying their t

159 ealth of information about archaeal biology, nothing is known about the molecular basis of DNA segreg

160 However, for most of these mutations, nothing is known about the molecular mechanism linking K

161 core management strategy for this pathogen, nothing is known about the nature of resistance.

162 o known as hypocretin) signaling, but almost nothing is known about the neural mechanisms through whi

163 spite these far-reaching behavioral effects, nothing is known about the neural processes impacted by

164 Nothing is known about the pathways that regulate gameto

165 ssion has been observed in cancer cells, but nothing is known about the piRNA partners or the functio

166 lease contributes to the pathogenesis of AF, nothing is known about the potential role of JPH2 in atr

167 ed vaccine (LAV) to protect against measles, nothing is known about the primary cells in which the vi

168 linked to the flagellar basal body; however, nothing is known about the proteins encoding the collar

169 ngage a pseudo-ELR and an N-like loop motif, nothing is known about the regions of CXCR4 and MIF that

170 However, almost nothing is known about the regulation of the central gen

171 Virtually nothing is known about the relevance of glycolysis in pa

172 However, nothing is known about the role of histone modifications

173 However, virtually nothing is known about the role of MDSC in the regulatio

174 Virtually nothing is known about the role of mechanotransductive s

175 the other post-translational modifications, nothing is known about the role of SUMO conjugation to h

176 However, nothing is known about the role of SUMOylation in regula

177 Virtually nothing is known about the role of these nonproteolytic

178 However, nothing is known about the role or importance of CtpA.

179 However, nothing is known about the roles of HPATs in basal plant

180 However, nothing is known about the single T. brucei CDS gene (Tb

181 ion in other environmental compartments, and nothing is known about the stability of high-molecular-w

182 Almost nothing is known about the structural factors driving th

183 Apart from their identification, almost nothing is known about the structure and function of PRO

184 multimeric channel complex, although almost nothing is known about the structure or subunit composit

185 To our knowledge, nothing is known about the survival of plasmacytoid dend

186 ell-established clinical syndrome, virtually nothing is known about the tumor cells responsible for t

187 er for sufferers and their families, and yet nothing is known about the underlying mechanism.

188 icrobial functions of porcine cathelicidins, nothing is known about their ability to promote pDC acti

189 eptors have immunomodulatory properties, but nothing is known about their antiviral effects.

190 ined unprecedented popularity, but virtually nothing is known about their cardiovascular risks.

191 ave been identified in the zebrafish genome, nothing is known about their expression pattern.

192 found in all members of this phylum, almost nothing is known about their identity, location and inte

193 (11-42) make up one fifth of plaque load yet nothing is known about their interaction with full-lengt

194 lity, cell trafficking/adhesion, and cancer, nothing is known about their involvement in liver pathol

195 water bodies is relatively well studied, but nothing is known about their presence in most of the com

196 cosylated chondroitin sulfate and fucoidan), nothing is known about their protein-linked glycosylatio

197 ls in neurodegenerative disease risk, almost nothing is known about their regulation.

198 are rapidly spreading in Southeast Asia, yet nothing is known about their transmission.

199 hyll (Chl)-Chl interactions, although almost nothing is known about these states.

200 ociated factor 2 (TRAF2); however, virtually nothing is known about TRAF2 signaling in the adult mamm

201 nt arterialization of endothelial cells, yet nothing is known about what regulates COUP-TFII expressi

202 ough the RST is present postnatally in cats, nothing is known about when rubrospinal projections coul

203 hance neuronal form and function, but almost nothing is known about whether and how it alters the bra

204 However, nothing is known about whether this equivalence extends

205 Even for sets of proteins for which nothing is known beyond unannotated sequences and struct

206 corridors impact animal movement, virtually nothing is known for species dispersed by wind, which ar

207 Without an accurate classification, nothing is known of diagnosis, prognosis, or treatment b

208 sing and consuming foodstuffs, but virtually nothing is known of how early pots were used.

209 Since nothing is known of how mRNA structural elements factor

210 in secretion, and social behavior in adults, nothing is known of its role during embryonic developmen

211 gical role is not completely understood, and nothing is known of the biology of these enzymes in Arch

212 ith the exception of one inconclusive study, nothing is known of the genetic basis of this phenomenon

213 l interactions, and in particular, virtually nothing is known of the molecular mechanisms that contro

214 eral inflammatory response to sepsis, almost nothing is known of the neuronal changes that cause asso

215 However, almost nothing is known of the skeleton's physiology, and myria

216 acaques on the binding of human (hu)IgG, and nothing is known of this interaction in the pig-tailed m

217 Alas, virtually nothing is known on how the gustatory thalamus (VPMpc) p

218 ctors of hypersensitivity to betalactam, but nothing is known on the influence of NOD genes, despite

219 ell types such as atrial myocytes, virtually nothing is known regarding endosomal transport.

220 However, nothing is known regarding the mechanism of langerin ass

221 Currently, nothing is known regarding the structural basis or molec

222 ated with deleterious clinical outcomes, but nothing is known to date about tissue protein carbamylat

223 ry nonverbal information, about which almost nothing is known.

224 question in cell biology about which almost nothing is known.

225 he cellular function of C9ORF72, about which nothing is known.

226 el applicability to unobserved environments."Nothing is less real than realism.

227 Nothing is more intuitive, yet more complex, than the co

228 However, virtually nothing is presently known about the post-translational

229 t important sources of human arsenic intake, nothing is published about uptake, toxicity, or toleranc

230 Hence, nothing is yet known of how amygdala subregional network

231 ions of TNF mediated by engagement of TNFR1, nothing is yet known of their role in CD40-mediated even

232 sues was then poorly understood, with almost nothing known about cancer drivers and stem cells.

233 this circuit to decision making, and almost nothing known about the whether any contribution is via

234 Currently, there is nothing known about transcriptional regulation of the SL

235 As a result, Mars's growth is stunted, and nothing large in the asteroid belt can accumulate.

236 explain the experimentally observed 'all-or-nothing' LAX3 spatial expression pattern in cortical cel

237 ticated catalytic centre shows that there is nothing magical about the catalytic activities or mechan

238 eceptors are thought to operate in an all-or-nothing manner, existing in an immunologically active or

239 l displacement threshold governing an all-or-nothing mechanically-induced calcium response.

240 ion of CasX specificity, revealing an all-or-nothing mechanism where mismatches can be bound, but not

241 RP promoters to provide more than an all-or-nothing mode of transcriptional regulation.

242 vital in animals that reproduce in an all-or-nothing mode, such as bristle worms: females committed t

243 for DNA loop formation are one-step, all-or-nothing models with a looped state and an unlooped state

244 The majority of cases require nothing more than a combination of symptomatic care and

245 Sign was once viewed as nothing more than a system of pictorial gestures without

246 h vigor and imagination, our pinnacle may be nothing more than an inflection point leading to decline

247 appears premature; populations may entertain nothing more than shared adaptiveness.

248 a row), what's at work is random variation, nothing more.

249 Here, we show that this all-or-nothing neuronal differentiation results from Hippo path

250 t has been done will be done again; there is nothing new under the sun.

251 To date however, we know nothing of a role for Ketamine in modulating both struct

252 knew much about Darwin, whereas Darwin knew nothing of Mendel.

253 The Netherlands, against the base case, "do-nothing" option.

254 Nothing or little is known about how eukaryotic mariner

255 r participation costs and allowed for all-or-nothing orders to avoid fragmented share portfolios.

256 applied to intentionally exposed dentin with nothing over it in vivo, whether it affects microleakage

257 We predict that while there appears to be nothing particularly special about water with regard to

258 alue of virus-based vectors is that they add nothing permanently to the environment.

259 mologous recombination (HR) is not an all-or-nothing phenomenon, but that HR competency comes on a sp

260 6:1 at the branches, we observed the "all-or-nothing" phenomenon with plasma only entering the low fl

261 ctive and matures through a symmetric all-or-nothing process.

262 In this work, we show that the all-versus-nothing proof of Einstein-Podolsky-Rosen steering can be

263 There is nothing quite like the excitement of discovery in scienc

264 Although initially considered an all-or-nothing reflex [1], numerous studies on freely diving ma

265 reater control of the emergence of an all-or-nothing response of a cell.

266 izable lactose analogues generates an all-or-nothing response, where some cells express the lac genes

267 ., nitazoxanide consistently shows an all-or-nothing response.

268 rse responses, ranging from graded to all-or-nothing responses and combinations thereof.

269 In a do nothing scenario, we estimated 146 996 excess deaths wit

270 The past 50 years has been nothing short of revolutionary as key findings have move

271 s, such as ELISA and immunofluorescence, are nothing short of ubiquitous in biotechnology and medical

272 Nothing similar to the ferromagnetic-like hysteresis loo

273 ore shared memory computers and large shared-nothing Spark clusters.

274 its the important neural functions of all-or-nothing spiking with signal gain and diverse periodic sp

275 properties of a neuron, including the all or nothing spiking, the threshold driven spiking of the act

276 w cooperative individuals to adopt an all or nothing strategy to tie strengthening based on the well-

277 ce pathway enabled transition from an all-or-nothing survival strategy to a hormone-modulated, compet

278 l genes and act as long-range enhancers, yet nothing that we know about their function explains the o

279 The remaining authors have nothing to disclose.

280 6 to 15 minutes in a room by themselves with nothing to do but think, that they enjoyed doing mundane

281 hould be an alternative explanation that has nothing to do with genetic continuity, but stresses the

282 and one-off ivory sales in 1999 and 2008 did nothing to halt elephant poaching.

283 d location of that item, despite there being nothing to look at and location memory never explicitly

284 ng DSEK to as many patients as possible (and nothing to others), rather than providing PK.

285 body to the 2F5 ELDKWA epitope but little or nothing to the 4E10 determinant.

286 e loading of a cofactor represents an all-or-nothing transition in regard to the enzymatic function a

287 ation are often assumed to involve an all-or-nothing two-state dissociation pathway, but deviations f

288 This all-or-nothing two-state solution is a hallmark of positive coo

289 Almost nothing was known about it.

290 However, nothing was known about the difference between these two

291 However, nothing was known about the mechanism by which PVR and D

292 Until relatively recently, almost nothing was known about the molecular pathogenesis of ei

293 lt to believe that in about 1960 practically nothing was known about the thymus and some of its produ

294 a possible intermediate group, and virtually nothing was known about their genetics.

295 nventional pathway enrichment tests detected nothing, while our approach discovered biological proces

296 Doing nothing will accelerate extinction.

297 ies are measured, and reported on regularly, nothing will be done explicitly to reduce them.

298 , which delivered either double the wager or nothing with 50:50 odds.

299 To do nothing would allow thousands of pregnant women to expos

300 ask that predicted whether reward, shock, or nothing would be delivered to the rat being recorded fro