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1 sufficient to produce subsequent deficits in novel object recognition.
2  vivo model to assess cognitive performance, novel object recognition.
3 om WT in swimming ability, cued learning and novel object recognition.
4  for a less demanding task, such as a simple novel object recognition.
5 t memory enhancing effects in a rat model of novel object recognition.
6 memory was assessed by Morris water maze and novel object recognition.
7 , increased locomotor activity, and impaired novel object recognition.
8 lidation in contextual fear conditioning and novel object recognition.
9 vivo efficacy in auditory sensory gating and novel object recognition.
10 nse to whisker deprivation, impaired texture novel object recognition and altered social behavior.
11 icacy in rodent behavioral cognition models (novel object recognition and auditory sensory gating).
12 citatory inputs and enhanced both short-term novel object recognition and cocaine-induced conditioned
13 ered faster than memory formation, impacting novel object recognition and cued fear conditioning but
14  lesion reduced long-term but not short-term novel object recognition and decreased long-term potenti
15 BI) induced lasting cognitive impairments in novel object recognition and less severe deficits in Y-m
16  prevented the ability of SKF81297 to rescue novel object recognition and long-term potentiation.
17  long-term potentiation, superior memory for novel object recognition and more persistent remote cont
18  learning and memory impairments measured by novel object recognition and Morris water maze tests, an
19 adiation, cognitive function was assessed by novel object recognition and Morris water maze.
20 learning as shown by improved performance in novel object recognition and Morris water maze.
21 tivity as well as mTBI-induced impairment in novel object recognition and object location tests.
22 ment of nonemotional cognitive tasks (in the novel object recognition and object pattern of separatio
23  puzzle box assay and impaired memory in the novel object recognition and object place recognition as
24 say, and memory performance was studied with novel object recognition and object place recognition as
25 lpha (PPT) or ERbeta (DPN) agonists enhanced novel object recognition and object placement memory in
26 n hippocampal behavioral assays, it prevents novel object recognition and placement without affecting
27                                     Impaired novel object recognition and rotarod performance were co
28             In contrast, castration impaired novel object recognition and spatial memory retention in
29    Short-term memory of mice was assessed by novel object recognition and Y-maze tests.
30  recognition and spatial memory, measured by novel object recognition and Y-maze tests.
31 rols, PV-M1 knockout mice exhibited impaired novel object recognition and, to a lesser extent, impair
32 evaluated by Y-maze spontaneous alternation, novel object recognition, and Barnes maze spatial memory
33 sts: Light Dark Latency, Elevated Plus Maze, Novel Object Recognition, and Barnes Maze.
34 luding social interaction deficits, impaired novel object recognition, and behavioral inflexibility.
35  Ts65Dn mice in the novel place recognition, novel object recognition, and contextual fear conditioni
36 ent working memory in the Morris water maze, novel object recognition, and contextual fear-conditioni
37 ry, as assayed by social recognition memory, novel object recognition, and Morris water-maze tests.
38 r function and balance, sensorimotor gating, novel object recognition, and spatial memory.
39 ion assessed on the novel place recognition, novel object recognition, and temporal order tasks was n
40 , as measured by novel location recognition, novel object recognition, and Y-maze memory tests.
41          MIA does not affect social novelty, novel object recognition, anxiety-like behavior nor sens
42                       Gamma oscillations and novel object recognition are impaired in dysbindin-1 nul
43 its in radial-arm water maze performance and novel object recognition as early as 8 months, outcrosse
44 ovement in cognitive abilities, as seen with novel object recognition as well as spatial learning and
45                Learning and memory using the novel object recognition assay also illustrated improved
46 mitigated SARS-CoV-2 induced deficits in the novel object recognition assay for learning and memory a
47 rofile with procognitive efficacy in the rat novel object recognition assay.
48 eficits as assessed by Morris water maze and novel object recognition assays, neurodegeneration, and
49 term effects were assessed by Open-field and Novel-Object-Recognition at P30 and P120.
50 in vivo potency in the place recognition and novel object recognition cognitive paradigms.
51 8 weeks using zero maze, locomotor activity, novel object recognition, cued, hidden and reduced Morri
52 tial tasks dependent on hippocampus (Y-maze, novel object recognition, dual solution cross-maze) and
53 o demonstrated dose-dependent improvement in novel object recognition following acute POM, which was
54 king memory [g = -0.56; (CI: -0.93, -0.18)], novel object recognition [g = -0.66; (CI: -0.97, -0.35)]
55 ich contains protocols for echocardiography, novel object recognition, grip strength, rotarod, glucos
56 d a borderline recognition memory deficit by novel object recognition in aged Tmprss9-/- female mice,
57                            Stress did impair novel object recognition in both sexes and social prefer
58  Multiple short training trials also rescued novel object recognition in Fmr1 KOs.
59  studies of contextual fear conditioning and novel object recognition in I-2 heterozygous mice sugges
60 sion of DPFE into perirhinal cortex restored novel object recognition in long-access meth rats.
61 e applied, including tasks to assess memory (novel object recognition in open field and V-maze paradi
62 ssary for 17beta-estradiol (E(2)) to enhance novel object recognition in young ovariectomized mice.
63                   However, both lesions left novel object recognition intact.
64                                 Furthermore, novel object recognition is facilitated in TgF344-AD rat
65 se inhibition, contextual fear conditioning, novel object recognition, locomotor, and social choice p
66 /kg, p.o.) and MK-801 induced disruptions of novel object recognition (MED = 10 mg/kg p.o.), thus pro
67 oral studies, RGFP966 increased subthreshold novel object recognition memory and cocaine place prefer
68 normalized elevated basal protein synthesis, novel object recognition memory and social behavior in F
69 16)1Yey mice treated prenatally had improved novel object recognition memory but do not show improvem
70 lted in normal adult synaptic plasticity and novel object recognition memory in mice exposed to ethan
71 C5/irisin impairs long-term potentiation and novel object recognition memory in mice.
72 se inhibition, improved social behavior, and novel object recognition memory in NMDA receptor hypofun
73      In the present study, rats were given a novel object recognition memory task in which initial en
74 on, it induced cognitive improvements in the novel object recognition memory test in NR1-KD animals,
75  tasks (e.g., contextual fear memory but not novel object recognition memory), and motor deficits.
76 tion in the open field, restore PPI, improve novel object recognition memory, partially normalize soc
77 n by D(3)R blockade as revealed in a test of novel object recognition memory.
78           At three doses tested in the mouse novel object recognition model (1, 3, and 10 mg/kg s.c.)
79 havioral tests including locomotor activity, novel object recognition, Morris water maze (cued, hidde
80 decline in the inhibitor-treated group using novel object recognition (NOR) and fear conditioning (FC
81          C57BL/6J mice were tested using the novel object recognition (NOR) assay at various time poi
82 ing two classic memory paradigms, Y-maze and novel object recognition (NOR) in mice and rats, respect
83 yclidine (PCP), induces enduring deficits in novel object recognition (NOR) in rodents.
84 d access meth self-administration results in novel object recognition (NOR) memory deficits in rats.
85 n reversing the effect of scopolamine in the novel object recognition (NOR) paradigm with a minimum e
86                                          The novel object recognition (NOR) task has been used widely
87 enting sleep after the learning phase of the novel object recognition (NOR) task significantly decrea
88                                          The novel object recognition (NOR) task was performed 30 and
89 se hippocampus at distinct stages during the novel object recognition (NOR) task: during object memor
90                                    Using the novel object recognition (NOR) test to evaluate cognitiv
91       Memory function was assessed using the novel object recognition (NOR) test, administered 3 days
92                                              Novel object recognition (NOR) testing paired with eithe
93 n (SND), which relies on olfactory cues, and novel object recognition (NOR), a visual-recognition tas
94 t Box (DLB), Y-Maze Spontaneous Alternation, Novel Object Recognition (NOR), and contextual and cued
95  Y-maze spontaneous alternation, open field, novel object recognition (NOR), and tail suspension test
96 sessment began 6-weeks post-irradiation with novel object recognition (NOR), egocentric learning, all
97 gnitive flexibility, social interaction, and novel object recognition (NOR).
98 e acetyltransferase, in long-term memory for novel object recognition (NOR).
99 ning, cued learning, locomotor activity, and novel object recognition (NOR).
100 oligomer-dose-dependent anxiety and impaired novel object recognition (NOR).
101                                We employed a novel-object recognition (NOR) task, using object explor
102 ssed via the novel object location [NOL] and novel object recognition [NOR] test).
103 (T-maze) and a hippocampus-independent task (Novel Object Recognition, NOR).
104 n activity, circadian rhythms and cognition (novel object recognition-NOR).
105 al testing revealed a significant deficit in novel object recognition, novel location recognition and
106 r memory in tests of cued fear conditioning, novel object recognition, object location recognition, c
107 nce of mice lacking this receptor (PRKO), in novel object recognition, object placement, Y-maze alter
108   Nonsocial behaviors or memories, including novel object recognition or fear conditioning, were not
109 hibit impairment of learning and memory in a novel object recognition paradigm.
110                                              Novel object recognition, passive avoidance test and Mor
111 e mice were tested several weeks later using novel object recognition (recognition memory) and 5-choi
112 and aged (26 months) mice were tested in the novel object recognition task (NORT).
113 imulation of LC-NE enhanced performance in a novel object recognition task and reduced hyperactivity
114 o procognitive activity (1 mg/kg, ip) in the novel object recognition task as a model of memory defic
115   The IDUA(-/-) mice performed normally in a novel object recognition task at younger ages until 8 mo
116                  Here we report a new serial novel object recognition task designed to measure memory
117 , we used different strength of training for novel object recognition task in mice.
118 t-term (STM) and long-term memory (LTM) in a novel object recognition task, but exhibit impairments d
119                When animals were tested in a novel object recognition task, MCT2 down-regulated anima
120           Mutant mice showed deficits in the novel object recognition task, suggesting hippocampal dy
121        Object recognition was tested using a novel object recognition task.
122  SD, precipitating cognitive deficits in the novel object recognition task.
123  and displays procognitive properties in the novel object recognition task.
124 abituation and alterations in the two-object novel object recognition task.
125 ognitive alterations, including defects in a novel object recognition task.
126 ted plus maze, holeboard, light-dark box and novel object recognition task.
127 .2% lower object discrimination score in the novel object recognition task.
128 only mice outperformed wild-type mice in the novel object recognition task.
129                                  We used the novel object-recognition task as a model of nonemotional
130 adian modulation of performance in a delayed novel-object recognition task.
131  short-, but not the long-term memory in the novel-object recognition task.
132  the first experiment, mice performed both a novel-object-recognition task identical to that performe
133                                              Novel object recognition tasks are commonly used to asse
134                                   Y-maze and Novel Object Recognition tasks indicated that mTBI mice
135 Bl/6J mice were tested in the water maze and novel object recognition tasks.
136 ificant alterations in an open field test, a novel object recognition test and in a T-maze task.
137 ain and both the discrimination index in the novel object recognition test and indoxyl sulfate concen
138 uctions of Object Recognition Index (ORI) in novel object recognition test and Recognition Index (RI)
139               We assessed cognition with the novel object recognition test and stained for amyloid pr
140 ice demonstrated improved performance in the novel object recognition test at 3 dpi, and in the T-maz
141 gnitive profile as it improved memory in the novel object recognition test but had no antidepressant
142 scopolamine-induced learning deficits in the novel object recognition test in rats.
143 ognition tests, and their memory loss in the novel object recognition test is associated with high le
144 ral assessments with open field, Y maze, and novel object recognition test or for electrophysiology r
145 light-dark box test) and cognitive function (novel object recognition test).
146 dopamine-lesioned mice were subjected to the novel object recognition test, and long-term potentiatio
147 ith cognitive impairment, as assessed by the novel object recognition test, but not signs of brain in
148 m-free mice shows that memory, assessed by a novel object recognition test, is a transmissible trait.
149 D rats, we found cognitive impairment in the novel object recognition test, the object location task,
150 he water maze, Y-maze reversal task, and the novel object recognition test.
151 nd contextual memory deficits measured using novel object recognition test.
152 copolamine-induced cognitive deficits in the novel object recognition test.
153  in SST neurons did not affect memory in the novel object recognition test.
154               Memory was not impaired in the novel object recognition test.
155 m a decline in recognition memory during the novel object recognition test.
156 r cognitive impairment when examined using a novel object recognition test.
157 -term memories were robustly improved in the novel-object recognition test and Morris water-maze spat
158 ced-swim, Y-maze spontaneous alternation and novel-object recognition test performance that developed
159                                 Notably, the novel-object recognition test showed that the treatment
160  regression to a large published data set on novel object recognition testing in rats exposed to mult
161 tle and prepulse inhibition, open field, and novel object recognition tests to evaluate behavior in f
162 he memory retention in passive avoidance and novel object recognition tests, and their memory loss in
163 al fear-conditioning, Morris water maze, and novel object recognition tests.
164 e in fear conditioning, object location, and novel object recognition tests.
165 d improved memory performance of CES rats in novel-object recognition tests and in the Morris water m
166                                              Novel object recognition was not impaired in R192Q mice;
167 locomotor activity, pre-pulse inhibition and novel object recognition were not impaired.
168  hippocampus (vHipp) and social approach and novel object recognition were tested.
169 noise were modestly or minimally impaired in novel object recognition, whereas similar-duration multi
170 memory enhancing effects in a mouse model of novel object recognition with improved tolerability and
171 ases principal cell firing rates and impairs novel object recognition with reduced dimensionality of

 
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