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1 er than expression of the latency-associated nuclear antigen.
2 kinase 2 (CDK2), CDK4 and proliferating cell nuclear antigen.
3 nd the replication clamp, proliferating cell nuclear antigen.
4 ions and by immunochemical detection of Ki67 nuclear antigen.
5 ant acid phosphatase, and proliferating cell nuclear antigen.
6 t also had high levels of proliferative cell nuclear antigen.
7 ive autoantibodies directed against distinct nuclear antigens.
8 companied by production of autoantibodies to nuclear antigens.
11 mologous proteins such as Epstein-Barr virus nuclear antigen 1 (EBNA-1) of the related gamma-herpesvi
12 associations were found between maternal EBV nuclear antigen 1 (EBNA-1), diffuse early antigen, or cy
14 s, but also efficiently present targeted EBV nuclear antigen 1 (EBNA1) and EBV-latent membrane protei
15 The viral protein Epstein-Barr virus (EBV) nuclear antigen 1 (EBNA1) binds to FR and DS to promote
18 dentical to that found in Epstein-Barr virus nuclear antigen 1 (EBNA1) that interacts with the N-term
19 IgG antibody reactivity toward ANO2 and EBV nuclear antigen 1 (EBNA1) was measured using bead-based
20 quires the interaction of Epstein-Barr virus nuclear antigen 1 (EBNA1) with the viral origin of plasm
22 d nuclear antigen 1 (LANA1) and Epstein-Barr nuclear antigen 1 (EBNA1)] necessary to maintain and rep
23 Nrf2 interacted with KSHV latency-associated nuclear antigen 1 (LANA-1) and the host transcriptional
24 in cells expressing only latency-associated nuclear antigen 1 (LANA-1) protein, and in KSHV latently
25 he expression of the KSHV latency-associated nuclear antigen 1 (LANA-1; ORF73) and LANA-1 nuclear pun
26 express nuclear antigens [latency-associated nuclear antigen 1 (LANA1) and Epstein-Barr nuclear antig
27 ciated herpesvirus (KSHV) latency associated-nuclear antigen 1 (LANA1) protein is constitutively expr
28 eterologous prime-boost vaccination with the nuclear antigen 1 of EBV (EBNA1), either targeted to the
30 cargos such as STAT1 and Epstein-Barr Virus Nuclear Antigen 1, as well as the influenza virus polyme
32 nly express the less immunogenic antigen EBV nuclear antigen-1 (EBNA-1), rendering them sensitive to
33 -LMPpoly has been generated that encodes EBV nuclear antigen-1 (EBNA1) fused to multiple CD8(+) T-cel
34 V viral capsid antigen positive Epstein-Barr nuclear antigen-1 positive serostatus at transplant (p =
35 ats (GAr) from the EBNA1 (Epstein-Barr virus nuclear antigen-1) protein, can trigger partial degradat
37 fold enrichment in host regions bound by EBV nuclear antigen 2 (EBNA2) and EBNA3 transcription factor
40 hich mimics CD40 signaling), and EBV-encoded nuclear antigen 3A (EBNA3A) and EBNA3C (which inhibit on
45 ence of NKT cells hepatic proliferating cell nuclear antigen and cyclin B1 decreased in mice injected
46 ntly (P < 0.05) inhibited proliferating cell nuclear antigen and cyclin D and caused considerable apo
47 ication factor C-Delta1N, proliferating cell nuclear antigen and DNA polymerase delta was found to re
48 d in the presence of both proliferating cell nuclear antigen and DNA, but the activity was not shut d
49 ation and associated with proliferating cell nuclear antigen and other components of the DNA replicat
50 Whereas association with proliferating cell nuclear antigen and participation in processive genome r
51 nuclear antigen 1 (EBNA1) is the EBV-encoded nuclear antigen and sequence-specific DNA binding protei
57 ly, these events promote the accumulation of nuclear antigens and activate innate sensors that drive
58 human systems, MutSalpha, proliferating cell nuclear antigen, and replication factor C activate MutLa
59 for BrdU, the mature neuron marker neuronal nuclear antigen, and the astrocytic marker glial fibrill
60 ASMC proliferation (Ki67, proliferating cell nuclear antigen, and WST1 assays) and resistance to apop
61 3-positive cells and more proliferating cell nuclear antigen- and pSMAD5-positive cells were found in
62 Amerindians and tested for HHV-8 anti-latent nuclear antigen (anti-LANA) and antilytic antibodies by
63 onegative recipients was associated with EBV nuclear antigen antibody deficiency, polymorphic disease
65 l characterization of the proliferating-cell-nuclear-antigen-associated factor p15(PAF), showing that
66 nti-double-stranded DNA and anti-extractable nuclear antigen autoantibodies following treatment with
67 leading strand, and PCNA (proliferating cell nuclear antigen) binds tightly to Pol delta and recruits
68 methyltetrazolium, Ki-67, proliferating cell nuclear antigen, bromodeoxyuridine, and caspase-Glo 3/7
69 the human clamp protein, proliferating cell nuclear antigen, by monitoring the change in the fluores
70 eration was assessed with proliferating cell nuclear antigen, CD1, and Ki67 markers and along with as
71 narily interacts with the proliferating cell nuclear antigen clamp loader replication factor C, DNA p
73 and protein expression of proliferating cell nuclear antigen, cyclin D1, E-cadherin, beta-catenin, Dv
74 inding protects Set8 from proliferating cell nuclear antigen-dependent degradation during the cell cy
75 es and is characterized by the production of nuclear antigen-directed autoantibodies (e.g., anti-dsDN
76 ere IgG seropositivity to Epstein-Barr virus nuclear antigen (EBNA) (random effects odds ratio [OR] 4
77 gramme, consisting of highly immunogenic EBV nuclear antigen (EBNA) and latent membrane proteins (LMP
79 blastoid Cell Lines (LCLs) requires four EBV nuclear antigen (EBNA) oncoproteins: EBNA2, EBNALP, EBNA
80 ent for expression of the Epstein-Barr Virus nuclear antigen (EBNA), making it particularly beneficia
81 ic cells or an adenovirus approach targeting nuclear antigen EBNA1 followed by a modified vaccinia vi
86 d Cp, resulting in the expression of six EBV nuclear antigens (EBNAs) and the viral Bcl2 homologue BH
87 EBV latent membrane proteins (LMPs) and EBV nuclear antigens (EBNAs), as well as nontranslated viral
89 oxyuridine incorporation, proliferating cell nuclear antigen expression, and histone H3 phosphorylati
90 tion-PCR and detection of latency-associated nuclear antigen expression, respectively, in cell lysate
91 sured by doublecortin and proliferating cell nuclear antigen expression, was also suppressed after ne
92 to both the EBV viral capsid antigen and EBV nuclear antigen, followed by a more rapid rise in antibo
96 , EBV latent membrane protein-1 and -2A, EBV nuclear antigen, HBV-encoded X antigen, and nonstructura
97 viral genes included the latency-associated nuclear antigen homolog ORF73 but none of the regions kn
99 cyclin D1, E, and A, and proliferating cell nuclear antigen in meningeal cells while significantly r
100 or mono-ubiquitination of proliferating-cell nuclear antigen in response to oxidative DNA damage, whi
101 creased expression of proliferating cellular nuclear antigen in Sertoli cells were observed in Ppard(
102 ignificantly prevented reduction of neuronal nuclear antigen in the infarcted area, although no impro
103 conclusion, lower IgG autoantibodies against nuclear antigens in DLE+SLE+ versus DLE-SLE+ subjects su
104 f a multitude of membranous, cytoplasmic and nuclear antigens in whole mouse organs and embryos, huma
106 tion that p12 possesses a proliferating cell nuclear antigen-interacting protein-degron (PIP-degron)
107 ic polymerase domain, the proliferating cell nuclear antigen-interacting region, the Rev1-interacting
108 ct regions, including the proliferating-cell-nuclear-antigen-interacting protein motif (PIP-box) and
110 ch compete for binding to proliferating cell nuclear antigen, is critical to prevent genomic instabil
111 rmal thickening, blocked the accumulation of nuclear antigen Ki67(+) cells in the basal and the supra
112 we show that KSHV-encoded latency-associated nuclear antigen (LANA) disrupts the association of CIITA
113 increased numbers of KSHV latency-associated nuclear antigen (LANA) dots, as detected by immunofluore
117 ls of mRNAs encoding KSHV latency-associated nuclear antigen (LANA) in primary effusion lymphoma (PEL
118 ciated herpesvirus (KSHV) latency-associated nuclear antigen (LANA) is a 1,162-amino-acid protein tha
119 ciated herpesvirus (KSHV) latency-associated nuclear antigen (LANA) is a 1,162-amino-acid protein tha
124 g KSHV gene products, the latency-associated nuclear antigen (LANA) is absolutely required in the mai
134 erpesvirus (KSHV)-encoded latency-associated nuclear antigen (LANA) protein functions in latently inf
135 Using immunofluorescence labeling of latent nuclear antigen (LANA) protein, together with fluorescen
136 geted by the KSHV-encoded latency-associated nuclear antigen (LANA) to repress expression of the majo
137 aposi sarcoma herpesvirus latency-associated nuclear antigen (LANA)(1-23), human papillomavirus 8 E2,
140 to the host chromosome by latency associated nuclear antigen (LANA), which binds in the terminal repe
141 t of genes, including the latency-associated nuclear antigen (LANA), which mediates viral episome per
147 ion and the expression of latency-associated nuclear antigen (LANA-1) upregulates the angiogenic mult
148 on of the highly abundant latency-associated nuclear antigen, LANA, on the host genome and its impact
149 BV) are human DNA tumor viruses that express nuclear antigens [latency-associated nuclear antigen 1 (
150 The diversity of the latency protein EBV nuclear antigen leader protein (EBNA-LP) resides predomi
151 ibosomal protein L16; in humans, MYC-induced nuclear antigen (MINA53; also known as MINA) and nucleol
152 ddress the role of MuHV-4 latency-associated nuclear antigen (mLANA) E3 ligase activity in gammaherpe
154 th induced Rad18-mediated proliferating cell nuclear antigen mono-ubiquitination during G(0), G(1) an
155 NA synthesis initiated by proliferating cell nuclear antigen monoubiquitination or less well-characte
157 s of DNA damage, inducing proliferating cell nuclear antigen monoubiquitination, and suppressing muta
158 hrenia and 38 control subjects) for neuronal nuclear antigen (NeuN+) and 65/67 kDa isoform of glutami
159 nt reduction in levels of proliferating cell nuclear antigen, NF-kappabeta/p50, cyclooxygenase-2, and
160 polymerase sliding clamp, proliferating cell nuclear antigen or PCNA, is a ring-shaped protein comple
161 We found that fungal proliferating cell nuclear antigen-partner interaction networks diverged in
162 n, which binds DNA-loaded proliferating cell nuclear antigen (PCNA(DNA)) and recruits CRL4(Cdt2).
164 During DNA replication, proliferating cell nuclear antigen (PCNA) adopts a ring-shaped structure to
166 expression of cyclins and proliferating cell nuclear antigen (PCNA) and evidence for DNA replication
167 sed platinum drug-induced proliferating cell nuclear antigen (PCNA) and FANCD2 monoubiquitinations (s
169 53 increased the level of proliferating cell nuclear antigen (PCNA) and minichromosome maintenance 4
170 red box protein 7 (Pax7), proliferating cell nuclear antigen (PCNA) and nicotinamide phosphoribosyltr
171 on factories by retaining proliferating cell nuclear antigen (PCNA) and other replisome proteins on t
172 tone substrates including proliferating cell nuclear antigen (PCNA) and promotes carcinogenesis by de
173 ctions with two proteins, Proliferating Cell Nuclear Antigen (PCNA) and Replication Protein A (RPA),
174 ication accessory protein proliferating cell nuclear antigen (PCNA) and the scaffold protein Rev1.
175 nary complexes containing proliferating cell nuclear antigen (PCNA) and two non-classical DNA polymer
176 on factor C (RFC) and the proliferating cell nuclear antigen (PCNA) are additional components of the
177 C (RFC) and sliding clamp proliferating cell nuclear antigen (PCNA) are both essential and play criti
178 study, we identified the proliferating cell nuclear antigen (PCNA) as a nIGF-1R-binding partner.
179 hat the sliding DNA clamp proliferating cell nuclear antigen (PCNA) associates with the C-terminal do
180 and polyubiquitination of proliferating cell nuclear antigen (PCNA) by regulating the recruitment of
181 he DNA replication factor proliferating cell nuclear antigen (PCNA) can be conjugated to either the s
182 t events in the reaction: proliferating cell nuclear antigen (PCNA) clamp binding/opening/closure/rel
183 C) complex loads circular proliferating cell nuclear antigen (PCNA) clamps onto DNA where they serve
184 ubunit complex that loads proliferating cell nuclear antigen (PCNA) clamps onto primer-template DNA (
185 Saccharomyces cerevisiae proliferating cell nuclear antigen (PCNA) clamps using single-molecule appr
186 cle actin (alpha-SMA) and proliferating cell nuclear antigen (PCNA) compared with 2- and 3-copy mice.
187 yuridine (BrdU) labeling, proliferating cell nuclear antigen (PCNA) expression and mitotic index incr
188 bp1), a key in regulating proliferating cell nuclear antigen (PCNA) expression and ribosomal RNA (rRN
190 ough its interaction with proliferating cell nuclear antigen (PCNA) for nucleosome assembly, particip
194 eling as well as Ki67 and proliferating cell nuclear antigen (PCNA) immunofluorescence, we determined
195 Saccharomyces cerevisiae proliferating cell nuclear antigen (PCNA) in replication factor C (RFC)-cat
203 The sliding clamp protein proliferating cell nuclear antigen (PCNA) is situated at the core of the eu
206 erturbation that supports proliferating cell nuclear antigen (PCNA) loading by replication factor C,
207 se and during DNA repair, proliferating cell nuclear antigen (PCNA) loading onto DNA (PCNA(DNA)) trig
208 omotrimeric sliding clamp proliferating cell nuclear antigen (PCNA) mediates Okazaki fragment maturat
209 se RAD18 is necessary for proliferating cell nuclear antigen (PCNA) monoubiquitination and TLS polyme
210 7 promoted the UV-induced proliferating cell nuclear antigen (PCNA) monoubiquitination in Poleta-prof
212 to damaged DNA relies on proliferating cell nuclear antigen (PCNA) monoubiquitylation and is regulat
213 ependent kinase (CDK) and proliferating cell nuclear antigen (PCNA) onto chromatin, as well as initia
216 wn that monoubiquitinated proliferating cell nuclear antigen (PCNA) plays an important role in recrui
217 raction between RECQ5 and proliferating cell nuclear antigen (PCNA) promotes RAD18-dependent PCNA ubi
219 (Pol delta) bound to the proliferating cell nuclear antigen (PCNA) replicates the lagging strand and
220 mplate and anchors to the proliferating cell nuclear antigen (PCNA) sliding clamp to form a holoenzym
221 ive monoubiquitination of proliferating cell nuclear antigen (PCNA) sliding clamps encircling damaged
223 evels of doublecortin and Proliferating Cell Nuclear Antigen (PCNA) suggested increased neurogenesis.
225 ainst an oncology target, proliferating cell nuclear antigen (PCNA) to elicit rapid and robust PCNA d
227 KT-mediated modulation of Proliferating Cell Nuclear Antigen (PCNA) ubiquitylation after UV requires
228 titutively interacts with proliferating cell nuclear antigen (PCNA) via a highly conserved PIP box mo
229 brain mantle and express proliferating cell nuclear antigen (PCNA), a cell cycling marker, indicate
230 ymerase alpha holoenzyme, proliferating cell nuclear antigen (PCNA), a homotrimeric DNA sliding clamp
231 RV2) and the host-encoded proliferating cell nuclear antigen (PCNA), a key DNA replication protein in
232 ow that monoubiquitinated Proliferating Cell Nuclear Antigen (PCNA), a marker of stalled replication
233 cently described that the proliferating cell nuclear antigen (PCNA), a nuclear factor involved in DNA
234 previously described that proliferating cell nuclear antigen (PCNA), a nuclear scaffolding protein pi
236 that described for human proliferating cell nuclear antigen (PCNA), a small ubiquitin-like modifier
237 acidophilum interact with proliferating cell nuclear antigen (PCNA), an essential co-factor for DNA p
238 the recruitment of Cdc45, proliferating cell nuclear antigen (PCNA), and polymerase delta, but not OR
240 Msh2-Msh6 (or Msh2-Msh3), proliferating cell nuclear antigen (PCNA), and replication factor C (RFC) a
242 complex monoubiquitinates proliferating cell nuclear antigen (PCNA), but the basis for recruitment of
243 age and monoubiquitinates proliferating cell nuclear antigen (PCNA), facilitating engagement of Polet
244 nding capability of human proliferating cell nuclear antigen (PCNA), identified the lysine residue in
245 ability to interact with proliferating cell nuclear antigen (PCNA), it enhances its interaction with
247 s, Poliota interacts with proliferating cell nuclear antigen (PCNA), Rev1, ubiquitin and ubiquitinate
248 Increased expression of proliferating cell nuclear antigen (PCNA), TGF-beta, and p-AKT and decrease
249 the proliferation marker proliferating cell nuclear antigen (PCNA), the anti-inflammatory cytokine i
250 1 inhibits recruitment of proliferating cell nuclear antigen (PCNA), the platform for assembly of the
252 mbly might be governed by proliferating cell nuclear antigen (PCNA), the processivity factor of repli
253 The expression levels of proliferating cell nuclear antigen (PCNA), vascular endothelial growth fact
254 Lack of SIM, but not proliferating cell nuclear antigen (PCNA)-interacting motif (PIM), leads to
255 tic figures, and BrdU and proliferating cell nuclear antigen (PCNA)-reactive cells showed that, in co
269 ll molecule inhibitors of proliferating cell nuclear antigen (PCNA)/PCNA interacting protein box (PIP
270 ocessivity clamps such as proliferating cell nuclear antigen (PCNA); however, the exact mechanism of
271 e heterotrimeric PCNA123 [proliferating cell nuclear antigen (PCNA)] clamp onto DNA that includes a r
272 replication foci and the proliferating cell nuclear antigen(PCNA) protein have a high level of proxi
273 e-positive cells and more proliferating cell nuclear antigen-positive cells in all treatment groups r
275 (P < 0.05) the number of proliferating cell nuclear antigen-positive tubular epithelial cells at 24
276 raction between XEco2 and proliferating cell nuclear antigen prevents cohesion establishment while ha
277 report an ancient family of GCNA (germ cell nuclear antigen) proteins that arose in the earliest euk
278 on polyubiquitylation of proliferating cell nuclear antigen provides a backup mechanism for accurate
279 e observations show that fecal IgA features, nuclear antigen reactivity particularly, at preclinical
280 tive DNA polymerase delta/proliferating cell nuclear antigen/replication factor C complex on telomeri
281 ell cycle (e.g. P. patens proliferating cell nuclear antigen, ribonucleotide reductase, and minichrom
282 lation of chromatin-bound proliferating cell nuclear antigen, slowed cell division, and increased gen
284 ial for cccDNA formation: proliferating cell nuclear antigen, the replication factor C complex, DNA p
285 replication factor C and proliferating cell nuclear antigen to perform efficient DNA synthesis in vi
286 stimation; apoptosis with proliferating cell nuclear antigen, TUNEL, and caspase assays; and gene exp
287 tions and perform IHC for proliferating cell nuclear antigen, upstream binding factor, RNA polymerase
288 , HDAC1, and HDAC2) together with EBV latent nuclear antigens using next-generation sequencing (NGS)
289 n by ubiquitinating PCNA (proliferating cell nuclear antigen) using the RAD6-RAD18 and UBC13-MMS2-RAD
290 estasis, but unexpectedly proliferating cell nuclear antigen was down-regulated at 12 days after chol
291 LR assessed by Ki-67 and proliferating cell nuclear antigen was markedly decreased in Itpr2(-/-) mic
292 lls harboring fluorescent proliferating cell nuclear antigen, we confirmed that 8a significantly and
293 ce, whereas cyclin D1 and proliferating cell nuclear antigen were decreased to reduce cell proliferat
294 ant acid phosphatase, and proliferating cell nuclear antigen were evaluated by histochemical and immu
295 of hepatic cyclin B1 and proliferating cell nuclear antigen were evaluated by Western Blot and liver
297 cated by co-staining with proliferating cell nuclear antigen, whereas Notch3 was expressed throughout
298 NANCE 2-7 gene family and PROLIFERATING CELL NUCLEAR ANTIGEN, which encode essential DNA replication
300 ractions of S. cerevisiae Proliferating Cell Nuclear Antigen (yPCNA) with modified DNA sequences and