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1 or retinoid and thyroid receptor) and N-CoR (nuclear receptor corepressor).
2 cid and thyroid hormone receptor) and N-CoR (nuclear receptor corepressor).
3 mediator for retinoid and thyroid receptors/nuclear receptor corepressor.
4 Atrophin proteins represent a novel class of nuclear receptor corepressors.
5 s to repressed genes by the HDAC activity of nuclear receptor corepressors.
6 acid and thyroid hormone receptor (SMRT) and nuclear receptor corepressor 1 (N-CoR) that accumulated
11 hinge region which specifically concentrates nuclear receptor corepressor 1 (NCOR1) at the genome.
12 study, we report that deletion of intestinal nuclear receptor corepressor 1 (NCoR1) completely dimini
15 which heme binding to Rev-erbalpha recruits nuclear receptor corepressor 1 (NCoR1) into an active re
17 ction with the histone deacetylase 3 (HDAC3)-nuclear receptor corepressor 1 (NCoR1) repressor complex
20 ely ablated in skin keratinocytes for either nuclear receptor corepressor 1 (NCoR1)/silencing mediato
21 ant mice and in mice ablated selectively for nuclear receptor corepressor 1 (NCoR1)/silencing mediato
22 tes the complex mediator of transcription 13/nuclear receptor corepressor 1 axis, which in turn promo
24 e-mediated autophagy - and its co-repressor, nuclear receptor corepressor 1, resulting in changes of
25 nd the nuclear receptor-interacting protein, nuclear receptor corepressor 1, to specific cis-regulato
26 SUMOylation of LXRs, but not the presence of nuclear receptor corepressor 1, was required for repress
27 em, HDAC3 is unique in that interaction with nuclear receptor corepressors 1 and 2 (NCoR1/2) is requi
28 creased the interaction between TR-alpha and nuclear receptor corepressor 2 (NCOR2) and suppressed Pl
30 due to loss of glucose homeostasis regulator nuclear receptor corepressor 2 repression, and chromatin
31 due to loss of glucose homeostasis regulator nuclear receptor corepressor 2 repression, and chromatin
33 RF7 promoter region through interaction with nuclear receptor corepressor 2/histone deacetylase 3 for
34 l coactivator) and the dissociation of NCoR (nuclear receptor corepressor, a transcriptional corepres
35 expression in vivo and indicates a role for nuclear receptor corepressors and associated histone dea
36 h the deacetylase-activation domain (DAD) of nuclear receptor corepressors and inositol tetraphosphat
39 local chromatin condensation, recruitment of nuclear receptor corepressor, and histone deacetylase co
40 T (switching-defective protein 3, adaptor 2, nuclear receptor corepressor, and transcription factor I
44 ults suggest that atrophin-1 associates with nuclear receptor corepressor complexes and is involved i
46 stem, we identified ETO/MTG8, a component of nuclear receptor corepressor complexes, as an atrophin-1
48 To explore this possibility, we examined nuclear receptor corepressor expression in a panel of no
49 a structurally and functionally more related nuclear receptor corepressor family and suggest an addit
51 ly associated with glutathione S-transferase-nuclear receptor corepressor fragments harboring one of
52 iator retinoid and thyroid hormone receptors/nuclear receptor corepressors), has been reported as the
53 utually exclusive and sequential manner: the nuclear receptor corepressor-HDAC3 complex followed by n
54 ther, our results demonstrate involvement of nuclear receptor corepressor/histone deacetylase complex
57 e of another class of nuclear cofactors, the nuclear receptor corepressors, in modulating PPARgamma t
58 in immunoprecipitation assays indicated that nuclear receptor corepressor is present on the endogenou
61 -like nuclear export signal that resembles a nuclear receptor corepressor motif (aa 86-95) impaired t
62 oncogenic fusion proteins interact with the nuclear receptor corepressor N-CoR and, in comparison wi
64 more, we demonstrate that DAX-1 recruits the nuclear receptor corepressor N-CoR to SF-1, whereas natu
65 the binding of RXR ligands and recruits the nuclear receptor corepressor N-CoR, PPAR permits the bin
66 e we demonstrate that ETO interacts with the nuclear receptor corepressor N-CoR, the mSin3 corepresso
67 ruitment of a corepressor complex, including nuclear receptor corepressor N-CoR, which, unexpectedly,
70 nd cell-based assays to demonstrate that the nuclear receptor corepressors N-CoR and SMRT interact wi
71 ze that corepressor complexes containing the nuclear receptor corepressor (N-CoR) are key factors in
73 Several lines of evidence indicate that the nuclear receptor corepressor (N-CoR) complex imposes lig
75 he tamoxifen-mediated association of ER with nuclear receptor corepressor (N-CoR) in the antiestrogen
76 Reduced H4 acetylation and increased HDAC1/nuclear receptor corepressor (N-CoR) occupancy at some T
78 tivated ERalpha, HDAC inhibitor (HDAC1), and nuclear receptor corepressor (N-CoR) that bound the slug
79 knowledge, that the HDAC3 complex, including nuclear receptor corepressor (N-CoR), transducin-beta-li
82 TR) actively represses transcription via the nuclear receptor corepressor (N-CoR)/histone deacetylase
85 erving to mediate a required exchange of the nuclear receptor corepressors, N-CoR and SMRT, for coact
86 on requires signal-dependent turnover of the nuclear receptor corepressor NCoR from target promoters,
87 alytic activity by missense mutations in the nuclear receptor corepressor (NCOR or SMRT) does not cau
88 Mutations abolishing interactions with the nuclear receptor corepressor (NCOR or SMRT) render HDAC3
89 athway suppressor 2 (GPS2), a subunit of the nuclear receptor corepressor (NCOR) and histone deacetyl
90 irect DNA binding of the receptor along with nuclear receptor corepressor (NCoR) and silencing mediat
92 ance AR recruitment of corepressor proteins [nuclear receptor corepressor (NCoR) and silencing mediat
93 eptor interacting motifs of the corepressors nuclear receptor corepressor (NCoR) and silencing mediat
95 using a mammalian two-hybrid assay, that the nuclear receptor corepressor (NCoR) and the silencing me
97 LXRs are dependent on interactions with the nuclear receptor corepressor (NCoR) and the silencing me
98 and thyroid hormone receptor (SMRT) and the nuclear receptor corepressor (NCoR) are negative regulat
100 ted the binding of the histone deacetylase 3-nuclear receptor corepressor (NCoR) complex to the COX-2
102 )-dependent derepression of AP-1 by removing nuclear receptor corepressor (NCoR) from the chemokine p
106 ory proteins in HeLa S3 cells, including the nuclear receptor corepressor (NCoR), TIF1beta/KAP-1, HDA
107 t of two epigenetic modifying complexes, the nuclear receptor corepressor (NCoR)-histone deacetylase
108 -binding domain, which targets PPAR-gamma to nuclear receptor corepressor (NCoR)-histone deacetylase-
110 the interaction of the MeCP2 with DNA or the nuclear receptor corepressor (NCoR)/silencing mediator o
111 om repressive to activating functions of the nuclear receptor corepressor (NCoR)/silencing mediator o
112 on of PPRE-bound PV with corepressors [e.g., nuclear receptor corepressor (NCoR)] that led to transcr
113 n in human breast cancer cells by recruiting nuclear receptor corepressors (NCoR and SMRT) and histon
116 its derived mouse model harboring a mutated nuclear receptor corepressor (NCOR1DeltaID) (Thra1(PV/+)
119 d the formation of a functional complex with nuclear receptor corepressors (NCORs) were critical in r
121 noid and thyroid hormone receptor (SMRT) and nuclear receptor corepressor protein (NCoR) are corepres
122 iptional activity through the recruitment of nuclear receptor corepressor protein and silencing media
123 f retinoid and thyroid receptors) and N-CoR (nuclear receptor corepressor) recruit histone deacetylas
124 Results from these studies indicate that nuclear receptor corepressor recruitment is a key featur
127 using the conserved carboxyl terminus of the nuclear receptor corepressor SMRT as a bait led to the i
128 Histone deacetylase 3 (HDAC3) requires the nuclear receptor corepressor SMRT for HDAC enzyme activi
130 ne deacetylases (HDAC)5 and 7 along with the nuclear receptor corepressors SMRT (silencing mediator f
132 t SMRTER, an insect analog of the vertebrate nuclear receptor corepressors SMRT and N-CoR, interacts
135 well as its molecular interactions with the nuclear receptor corepressor, SMRT, and nuclear receptor
136 highlight an unexpected new function of the nuclear receptor corepressor SMRTe for its role in regul
138 expression, Hr likely defines a new class of nuclear receptor corepressors that serve a more speciali
139 n serve as an adapter molecule that recruits nuclear receptor corepressors to DNA-bound nuclear recep
140 repressor that we call SUN-CoR (Small Unique Nuclear receptor CoRepressor), which shows no homology t