ライフサイエンスコーパス: nuclear stain

1 lon cancer cells with Hoescht 33342, a vital nuclear stain.

2 s to that obtained using flow-cytometry with nuclear stains.

3 and glia, as detected by ethidium homodimer nuclear staining.

4 his protein reproduced Purkinje and podocyte nuclear staining.

5 substitution mutants led to increased SP100 nuclear staining.

6 dy to the 16-amino acid peptide demonstrated nuclear staining.

7 l body and neurite projections, with minimal nuclear staining.

8 s were brightly fluorescent with cytoplasmic/nuclear staining.

9 d diffuse cytoplasmic staining and punctuate nuclear staining.

10 by flow cytometry by using propidium iodide nuclear staining.

11 murine MTC, while normal tissue retained RB nuclear staining.

12 acement dose (1 mg/kg), there was only light nuclear staining.

13 ',6-diamidino-2-phenylindole dihydrochloride nuclear staining.

14 O) with anti-TRF1 antibody revealed punctate nuclear staining.

15 dehydrogenase (LDH) release and trypan blue nuclear staining.

16 s, with cytoplasmic as well as dendritic and nuclear staining.

17 internalization by ECs with cytoplasmic and nuclear staining.

18 four lines that show peripheral or punctate nuclear staining.

19 diffuse cytoplasmic distribution with faint nuclear staining.

20 h PCa progression with many cells exhibiting nuclear staining.

21 phils within MGAS2221 infection sites had no nuclear staining.

22 H2AX phosphorylation as a diffuse, even, pan-nuclear staining.

23 n increase in the frequency and intensity of nuclear staining.

24 One patient showed loss of BAP1 nuclear staining.

25 ancer epithelia (14 of 126) showed mild hCG2 nuclear staining.

26 ptosis was investigated by Hoechst and TUNEL nuclear staining.

27 l death was detected by TUNEL assay and DAPI nuclear staining.

28 ion assays and 4',6-diamidino-2-phenylindole nuclear staining.

29 d but the RPE attached were stained with the nuclear stain 4',6-diamidine-2-phenylindole dihydrochlor

30 c hyperplasia (BPH) samples presented strong nuclear staining (78% of the cases).

31 The lack of TRbeta1 nuclear staining, a likely result of biallelic gene inac

32 Ancillary nuclear staining allowed cell association with the AF st

33 2, intense apical/cytoplasmic and occasional nuclear staining along the longitudinal crypt axis was o

34 Nevertheless, nuclear staining analysis revealed that both TGP1KO and

35 gmentation, ethidium bromide-acridine orange nuclear stain and TdT-mediated dUTP nick-end labeling (T

36 Nuclear staining and aggregates predominated in striatum

37 s, as indicated by phosphorylated Smad 1/5/8 nuclear staining and by elevated expression of Bmp2, Bmp

38 a dose-dependent apoptosis as determined by nuclear staining and DNA ladder assay.

39 Nuclear staining and immunofluorescence revealed that in

40 e biotools and were applied for targeted red nuclear staining and in-vivo tumor imaging.

41 Nuclear staining and MTT assays following dsRNA stimulat

42 However, a few cases displayed weak nuclear staining and strong cytoplasmic staining (possib

43 greatly diminished as both the intensity of nuclear staining and the number of cells expressing C/EB

44 f nuclear morphologic changes by Hoechst DNA nuclear staining and transmission electron microscopy.

45 eal color discrimination to exclude pigment, nuclear staining and unstained areas, the type and amoun

46 se-enriched HeLa cells demonstrated punctate nuclear staining, and AAF co-localized with proliferatin

47 mic presence of core protein with occasional nuclear staining, and both cytoplasmic and membrane expr

48 d reduced serum cytokines, reduced NF-kappaB nuclear staining, and decreased neutrophil infiltration

49 The new primary MLBCLs had prominent c-REL nuclear staining, and the MLBCL cell line exhibited high

50 confocal microscopy, the same surface label, nuclear stains, and antitubulin antibodies.

51 , [(3)H]thymidine incorporation, fluorescent nuclear staining, annexin V binding, DNA fragmentation a

52 Results of nuclear staining, apoptotic-specific poly(ADP-ribose) po

53 cytoplasmic in untreated cells, but intense nuclear staining appeared in 8-bromo (Br)-cGMP-treated c

54 acterized with TUNEL, DNA fragmentation, and nuclear staining assays.

55 Moreover, nucleolar stress only alters OGAse nuclear staining, but not OGT staining.

56 d biglycan immunoreactivity colocalized with nuclear staining by propidium iodide and was also seen i

57 ase in the number of tumor cells with strong nuclear staining compared to typical carcinoids.

58 0, 95%CI = 1.42-8.15, P = 0.006 for positive nuclear staining) compared to non-CRC samples (Normal or

59 In addition to nuclear staining, cytoplasmic localization of p27 was no

60 In addition to the nuclear staining, cytoplasmic staining of p27 was noted

61 Cellular morphology, time-lapse imaging, and nuclear staining demonstrated that this activity occurre

62 Nuclear staining did not correlate with sex, age, size,

63 laser scanning confocal microscopy using the nuclear staining dye Hoechst 33342, immunohistochemical

64 axel and Hoechst 33342, a membrane-permeable nuclear staining dye, were reduced to 51% and 45%, respe

65 protoplasts using the membrane-impermeable, nuclear-staining dye, YO-PRO-1, and transgenic Arabidops

66 s as assessed by morphological studies using nuclear staining, electron microscopy, and flow cytometr

67 and human tumor specimens with positive TP53 nuclear staining exhibited reduced TbetaRII and knocking

68 Intense nuclear staining for 8-OHdG was observed in central endo

69 ely 90% of transfected cells exhibit partial nuclear staining for AGL.

70 AR-driven luciferase activity and distinct nuclear staining for AR were seen in several key brain a

71 llular tumors from FGF19 transgenic mice had nuclear staining for beta-catenin.

72 pa B-alpha Within 45 minutes after wounding, nuclear staining for both NF-kappa B components was noti

73 ndrocyte markers showed up-regulation of the nuclear staining for both NF-kappaB and JNK on a large p

74 human samples revealed mixed cytoplasmic and nuclear staining for both proteins in benign nevi and su

75 and esophagus had both positive and negative nuclear staining for CDX1.

76 Nuclear staining for cyclin D and Ki67 was observed in a

77 In situ hybridization studies showed nuclear staining for Epstein-Barr virus (EBV) within the

78 d DNA laddering of genomic DNA and increased nuclear staining for fragmented DNA in neonatal cardiomy

79 These consisted of nuclear staining for huntingtin and huntingtin-containin

80 0-day-old primary gel culture, showed strong nuclear staining for incorporated 5-bromo-2'deoxyuridine

81 Nuclear staining for KLF6 protein was evident at E15.5 i

82 d cell size and in the presence of increased nuclear staining for MIB-1, a marker of proliferation.

83 these findings, we found that an absence of nuclear staining for NKX2.2 in SCLC primary tumors was a

84 Nuclear staining for p27(kip1) was greatly reduced in CE

85 l was observed in Western blot analysis, and nuclear staining for p27kip1 was greatly reduced in HCEC

86 Strong nuclear staining for Rb was present in most tumors.

87 hat it colocalizes, in certain tissues, with nuclear staining for RelA/p65 and for p300, suggesting t

88 Nuclear staining for survivin was positive in 12 of 13 a

89 ls, and T-regulatory cells was determined by nuclear staining for TBET, GATA3, and FOXP3.

90 or the origin-binding protein show a diffuse nuclear staining for UL29.

91 al microscopy revealed gamma-H2AX peripheral nuclear staining (gamma-H2AX ring) colocalizing with pho

92 ng involving all or part of their cytoplasm; nuclear staining, however, was reduced in intensity, bei

93 polyclonal antibodies demonstrated specific nuclear staining in 10 of 12 lung adenocarcinoma samples

94 s used to identify cell nucleus and quantify nuclear staining in 323 tissue cores.

95 nuclear staining in Ba/F3 cells and diffuse nuclear staining in 32D cl3 cells.

96 toplasmic staining was more intense than the nuclear staining in 35 to 49% of carcinomas, depending o

97 c cells showed a similar pattern of punctate nuclear staining in all cell lines tested using confocal

98 al analysis of human EOC tumors demonstrates nuclear staining in all histotypes.

99 Nuclear staining in all layers of human epidermis and bo

100 uorescence revealed zinc-inducible speckled, nuclear staining in Ba/F3 cells and diffuse nuclear stai

101 yzed, 73% were positive for KLF5 (defined as nuclear staining in more than 5% of tumor cells).

102 se antibodies designated 1H6 exhibits strong nuclear staining in most human and murine cells.

103 Fluorescence microscopy also demonstrated nuclear staining in mouse oocytes at the germinal vesicl

104 ker staining of IFI16 compared with positive nuclear staining in normal mammary duct epithelium.

105 Positive nuclear staining in normal, advanced adenoma and CRC was

106 3 to the cytoplasm of most resting FLS, with nuclear staining in only 10.7 +/- 2.4%.

107 s, prothymosin alpha, demonstrated prominent nuclear staining in SPEM and gastritis cystica profunda.

108 However, intruder stress increased p-Smad1 nuclear staining in the control rats only: no further in

109 epidermis and as either grainy or punctuate nuclear staining in the cultured keratinocytes.

110 NFBD1 exhibited diffuse nuclear staining in the majority of untreated cells anal

111 activated c-Rel is reflected in their strong nuclear staining in the proliferating compartment of pri

112 tissues than normal tissues, and its higher nuclear staining in tumors correlated with poorer surviv

113 At all time points, p16ink4a showed reduced nuclear staining in ZD esophagi compared with that in ZS

114 aB p65 phosphorylation, as well as NF-kappaB nuclear staining, in Waldenstrom's macroglobulinemia cel

115 Nuclear staining included rod-like crystalline structure

116 determine the immediate metabolic effects of nuclear stains, including SYTO, DAPI dilactate, Hoechst

117 However, the nuclear staining increases from G1 to S phase and become

118 ed cells and exhibited a punctate pattern of nuclear staining instead of the diffuse pattern observed

119 Sytox Green, a nuclear stain, is excluded by live spores but penetrates

120 Left-sided colon neoplasms showed nuclear staining more frequently than those right-sided

121 A particular cell showed diffuse nuclear staining, multiple nuclear foci, or one or two l

122 NF-kappaB nuclear staining, neutrophil accumulation, and localizat

123 Cyclin D1 nuclear staining occurred in 30% of adenocarcinomas and

124 ociated with marked increases in trypan blue nuclear staining of A549 monolayers.

125 PTHrP also increased the nuclear staining of beta-catenin and, similar to activat

126 We detected weak cytoplasmic and no nuclear staining of beta-catenin in 18 cases of cervical

127 We also detected increased cytoplasmic and nuclear staining of beta-catenin in invasive cervical ca

128 stochemistry analysis demonstrated increased nuclear staining of C/EBPbeta in 10 of 13 GC and at leas

129 by F-I on MDA-MB-435 was confirmed by Tunnel nuclear staining of cells with apoptotic DNA fragmentati

130 d that A-C/EBP prevented the normal punctate nuclear staining of centromeres, an indicator of C/EBPbe

131 PC nuclear staining of cyclin D1 was only observed in patie

132 Intense nuclear staining of DNA damage foci was observed in nucl

133 parakeratotic skin disorders, which exhibit nuclear staining of granular and/or cornified layers.

134 ytes and B cells in the subepithelium showed nuclear staining of multiple NF-kappaB subunits, suggest

135 lecule selective inhibitor of AURKA, reduced nuclear staining of nuclear factor-kappaB (NF-kappaB) p6

136 ring prostate epithelium, moderate to strong nuclear staining of p27Kip1 was present in greater than

137 mor samples from FMTC patients showed strong nuclear staining of phosphorylated ERK1/2 and Ser(727) S

138 zeta, as well as dysplasia-specific punctate nuclear staining of PKC mu.

139 Importantly, nuclear staining of pSTAT3-Y705 identified at the tumor

140 Using reporter mice and nuclear staining of Sox17 and beta-catenin, we report th

141 es was associated with sustained increase in nuclear staining of the cyclin-dependent kinase (Cdk) in

142 ning this mutation does not replicate and no nuclear staining of the Gag protein is found in transfec

143 Increased immunohistochemical nuclear staining of the HNF3alpha protein was detected i

144 TSO increased nuclear staining of the transcription factor Nrf2 in liv

145 nin IHC showed consistent diffuse and strong nuclear staining of the tumor cells in all six SN-HPCs.

146 There were no differences in the nuclear staining of these transcription factors between

147 Immunohistochemical analysis revealed nuclear staining of tumor cells with antibodies against

148 Moderate to strong nuclear staining of YAP1 and TAZ was detected in 53% and

149 and animal tissues, and stained with various nuclear stains or fluorescent protein-based nuclear repo

150 ected in 19 tumors negative for beta-catenin nuclear staining (P < 0.05).

151 ntly longer for patients with tumors lacking nuclear staining (P = 0.017).

152 Some corneal epithelial basal cells showed nuclear staining, particularly for cyclin D and Ki67, in

153 ancer phenotype that showed a characteristic nuclear staining pattern and prognostic value.

154 ction of NIH3T3 cells with PIC1 results in a nuclear staining pattern coincident with that of endogen

155 st RRV-positive cells showed a fine punctate nuclear staining pattern consistent with latent infectio

156 within the nucleus, as revealed by a unique nuclear staining pattern of receptors possessing a point

157 positive human sera, resulting in a punctate nuclear staining pattern reminiscent of LANA in BCBL-1 c

158 ge in C/EBPbeta localization from a punctate nuclear staining pattern to diffuse nuclear distribution

159 hile p53-/- cells displayed a faint cyclin G nuclear staining pattern, there was no increased express

160 nd displayed a detergent-resistant, punctate nuclear staining pattern.

161 Immunofluorescence localization shows a nuclear staining pattern.

162 ention of a speckled 14-3-3zeta mitotic cell nuclear-staining pattern that usually becomes diffuse du

163 PIR1 exhibited a nuclear-staining pattern that was sensitive to RNase A,

164 enetic Landscapes (MIEL), which captures the nuclear staining patterns of epigenetic marks and employ

165 epitope-tagged proteins produced distinctive nuclear staining patterns.

166 Nuclear staining percentage has a fair diagnostic abilit

167 s stronger in intensity for het patients and nuclear staining predominant in hom cases.

168 dUTP-digoxigenin nick end-labeling assay and nuclear staining, respectively (P < 0.001).

169 clear NF1 label eliminates cell fixation and nuclear staining resulting in efficient screening.

170 t dose of CORT (5 or 10 mg) after ADX, dense nuclear staining returned, but with a low replacement do

171 Strikingly, nuclear staining revealed a close association of BRAF35/

172 Nuclear staining reveals that a large nuclear deformatio

173 kness was measured from fluorescence-labeled nuclear-stained sections.

174 Immunohistochemistry and nuclear staining showed that nearly all rodent and human

175 a G4 DNA-specific helicase, showed stronger nuclear staining than controls.

176 coid receptor in the intact rat demonstrated nuclear staining throughout several amygdala regions, in

177 Exposure to hydrogen peroxide for increased nuclear staining to 70.7 +/- 12.8% after 8 hours (P = 0.

178 field images to identify cell boundaries and nuclear staining to automatically segment cells in two d

179 carcinoma tumors displayed an absence of p16 nuclear staining using immunohistochemistry.

180 an survival for patients with tumors lacking nuclear staining was 8.5 months and 6.9 months for patie

181 Nuclear staining was also evident.

182 ing was primarily observed in the cytoplasm, nuclear staining was also present in some RNA and DNA vi

183 cimens, weak cytoplasmic staining and strong nuclear staining was an independent predictor of poor ou

184 In the lens epithelium, nuclear staining was detected at P0, P7, and P11.

185 Positive nuclear staining was found in 8 patients, whereas 12 lac

186 Specific nuclear staining was found in epithelial cells of the sm

187 Nuclear staining was found in the majority of tissues st

188 Nuclear staining was frequently seen in neurons througho

189 ized, 'flecked' distribution to one in which nuclear staining was homogeneous and diffuse.

190 and cytoplasm of all 16 benign tissues, but nuclear staining was lost from a significant 19/20 malig

191 A strong nuclear staining was observed for p27(Kip1).

192 No change in p27kip1 levels or in nuclear staining was observed in the nonsilencing contro

193 nuclei from medulloblastoma, whereas little nuclear staining was observed with HEK293T, hepatoma, or

194 etectable in the TH positive neurons, but no nuclear staining was observed with this antibody.

195 cell carcinoma and OSSN cases, while diffuse nuclear staining was present in 1 of 12 (8%) basal cell

196 ng were present or as atypical if definitive nuclear staining was seen but significant nuclear enlarg

197 Focal nuclear staining was seen in a small number of basal cel

198 ning the nuclear localization signal, and no nuclear staining was seen in the case of heparan sulfate

199 Immunofluorescent RelA nuclear staining was strongly inhibited in Ad5 IkappaB-i

200 Nuclear staining was weak in nonmalignant tissues, more

201 while each tumor with strong, homogeneous RB nuclear staining were p16 negative, supporting our hypot

202 and ethidium bromide/acridine orange (EB/AO) nuclear staining were performed to detect cell death.

203 2221 DeltasagA were full of neutrophils with nuclear staining, whereas MGAS2221 DeltasagA infection w

204 osteosarcoma cells, 53BP1 exhibited diffuse nuclear staining; whereas, within 5-15 min after exposur

205 hile UL32 is predominantly cytoplasmic, some nuclear staining which colocalizes with the major DNA bi

206 yed coagulation, cell shrinkage, and loss of nuclear staining, which are indicators of irreversible t

207 ge foci to an intense peripheral and diffuse nuclear staining, which could be used as response biomar

208 Interphase cells showed a fine, speckled, nuclear staining, while mitotic cells had bright stainin

209 ith the wild-type phenotype result in strong nuclear staining, while, in contrast, constructs express

210 tion of microbubbles containing virus showed nuclear staining with 5-bromo-4-chloro-3-indolyl-beta-D-

211 lactate dehydrogenase (LDH), by fluorescence nuclear staining with a membrane-impermeant dye and by m

212 ts was imaged with NLM using rapid extrinsic nuclear staining with acridine orange and intrinsic seco

213 ported by electron microscopy, as well as by nuclear staining with Hoechst 33342 dye, and by examinat

214 ined cells and sub-G1 populations as well as nuclear staining with orange fluorescence of treated can

215 Because nuclear staining with the 2B4-1 antibody was previously

216 These data suggest that in normal tissues, nuclear staining with the DUT415 antibody represents the

217 Increased nuclear staining with the membrane impermeable dye propi

218 icroarrays from 129 patients revealed strong nuclear staining with the P-S319-Runx2 antibody in prima

219 Nuclear staining with TO-PRO-3 was also used in place of

220 benzimidazole) derivatives, known as Hoechst nuclear stains, with high potency against poxvirus infec

221 ated both plasma membrane and perinuclear or nuclear staining within the basal and spinous layers.