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1  observe a requirement for hydrolysis of any nucleotide triphosphate.
2 alance between binding and hydrolysis of the nucleotide triphosphate.
3 g site decreased the binding affinity of the nucleotide triphosphate.
4 dT) paired with an incoming dNTP or modified nucleotide triphosphate.
5  with the equivalent fluorescent dye-labeled nucleotide triphosphate.
6 groups that bind the beta phosphate group of nucleotide triphosphate.
7 f nucleotides and a dimer in the presence of nucleotide triphosphate.
8  the beta- and gamma-phosphate groups of the nucleotide triphosphate.
9 ay be capable of generating a broad range of nucleotide triphosphates.
10  dependent on the concentration of competing nucleotide triphosphates.
11 sis but probably the removal of noncanonical nucleotide triphosphates.
12 ted by the addition of ATP and several other nucleotide triphosphates.
13 ivity for binding to the naturally occurring nucleotide triphosphates.
14 dal in the presence of low concentrations of nucleotide triphosphates.
15 nhibited by micromolar concentrations of all nucleotide triphosphates.
16 y was supported by ATP or dATP but not other nucleotide triphosphates.
17 e the affinity of ARF for activating guanine nucleotide triphosphates.
18 on step and discrimination between different nucleotide triphosphates.
19 AD dependence of repressor activity required nucleotide triphosphates.
20 in kinase inhibitor (PKI), or they may mimic nucleotide triphosphates.
21 purine-labeled DNA substrates, 2-aminopurine nucleotide triphosphate, a nonhydrolyzable nucleotide an
22 st on the order of minutes in the absence of nucleotide triphosphates, although TFIIE remains unexpec
23 nger RNA half-lives, and the availability of nucleotide triphosphates, amino acids, RNA polymerase, a
24 ating incremental truncation libraries using nucleotide triphosphate analogs.
25 ervable effect, but ATPgammaS and GTPgammaS, nucleotide triphosphate analogues resistant to Hsp70 hyd
26                                     By using nucleotide triphosphate analogues, the reaction is subst
27  is followed by the reversible hydrolysis of nucleotide triphosphate and subsequent conformational tr
28 s strongly dependent on both the nature of a nucleotide triphosphate and that of a divalent metal.
29 and adenosine triphosphate or analogs of the nucleotide triphosphate and then analyzed by matrix-assi
30    The new assay principle uses only natural nucleotide triphosphates and avoids a labour-intensive f
31            Moreover, this step required both nucleotide triphosphates and cytosol.
32 al pathways by which 8-oxodG is converted to nucleotide triphosphates and incorporated into both DNA
33 cle intermediates, but they strongly deplete nucleotide triphosphates and may impede nucleotide synth
34 d contains small pores that permit influx of nucleotide triphosphates and metabolites of nucleoside a
35 ese advantages, diene- and cyclohexene-based nucleotide triphosphates are expected to find wider use
36                               The respective nucleotide triphosphates are substrates for some DNA pol
37                                NS3h utilizes nucleotide triphosphate (ATP) for hydrolysis energy to t
38 esis of the hrpA regions predicted to encode nucleotide triphosphate binding and hydrolysis functions
39                      Here we demonstrate how nucleotide triphosphate binding free energy can rectify
40                        HsORC4 has a putative nucleotide triphosphate binding motif that is not seen i
41 hanges in conformation that subtly alter the nucleotide triphosphate binding site such that ddNTPs be
42         Mutations were made in the predicted nucleotide triphosphate-binding domain, confirming the a
43 the first four AAA domains contain consensus nucleotide triphosphate-binding motifs, or P-loops.
44  chicken mitochondrial CK indicates that its nucleotide triphosphate-binding site indeed contains the
45 porate one or, at most, a few biotin-labeled nucleotide triphosphates (biotin-NTPs) into the 3' end o
46 irectly examine G protein conformations with nucleotide triphosphates bound, we synthesized several n
47 ational targeting and translocation requires nucleotide triphosphates but not cytosolic proteins.
48 op activity was observed in the absence of a nucleotide triphosphate cofactor, indicating that the co
49  by single-stranded DNA in the presence of a nucleotide triphosphate cofactor, it mediates cleavage o
50 doxorubicin are potent (low-microM) DNA- and nucleotide triphosphate-competitive priming inhibitors t
51 ures capturing the active polymerase and its nucleotide triphosphate complexes in four distinct state
52  due to different forms of Fe-citrate and Fe-nucleotide triphosphate complexes.
53                                          The nucleotide triphosphate concentrations required at the T
54 n which the radioactive gamma-phosphate of a nucleotide triphosphate could transfer to a photoactivat
55 binding free energies for a free deoxyribose nucleotide triphosphate, dATP or dGTP, to Pol eta comple
56 ting sequencing platform that uses step-wise nucleotide triphosphate (dNTP) release, capture and dete
57 ic degradation of canonical and noncanonical nucleotide triphosphates (dNTPs).
58 sition of the RNA template and the substrate nucleotide triphosphates during initiation and elongatio
59 ficity and concentration requirements of the nucleotide triphosphate effect suggests a P2X(7) recepto
60 ed a strong positive correlation with TUNEL; nucleotide triphosphate/EPP showed a strong negative cor
61  brain (31)P magnetic resonance spectroscopy nucleotide triphosphate/exchangeable phosphate pool.
62 entration of CTP, the predominant initiating nucleotide triphosphate for this promoter.
63                       When Fapy*dG is in its nucleotide triphosphate form, Fapy*dGTP, it is inefficie
64                                 For example, nucleotide-triphosphates generate nucleotide-diphosphate
65 for organelle maintenance due to its role in nucleotide triphosphate generation.
66                            The small guanine nucleotide triphosphate (GTP)-binding protein RhoA stimu
67 nducibility compared with GTP, whereas other nucleotide triphosphates had no effect.
68                                          For nucleotide triphosphates, however, mixtures of acidic (0
69 t the first two folds, the P-loop containing nucleotide triphosphate hydrolase and the NAD(P)-binding
70  fold structure typical of P-loop containing nucleotide triphosphate hydrolases.
71  limitation because ATP is the most abundant nucleotide triphosphate in the cell, and Mg(2+) is also
72              Stoichiometric binding of these nucleotide triphosphates in PRK's substrate site is obse
73 uantified the levels of serum uridine and of nucleotide triphosphates in the liver, spleen, and lymph
74 sence of magnesium, but becomes specific for nucleotide triphosphates in the presence of manganese.
75 sphoenolpyruvate which together maintain the nucleotide triphosphates in the reaction mixture.
76               NMPylation can utilize diverse nucleotide triphosphates, including remdesivir triphosph
77 movement of the bridge helix that helps load nucleotide triphosphates into the active site.
78  products by substitution of the appropriate nucleotide triphosphates into the reaction.
79                  In addition, the respective nucleotide triphosphate is accepted as a substrate by th
80 ells, and the augmented synthesis and use of nucleotide triphosphates is a critical and universal met
81 ally applicable to reactions involving other nucleotide triphosphates is described.
82                      Transphosphorylation of nucleotide triphosphates is the central reaction in DNA
83 hemical mechanism of transphosphorylation of nucleotide triphosphates is, in most cases, unknown.
84    Oligomers could be converted to dimers by nucleotide triphosphate-Mg, and nucleotide release from
85           Motifs "A" and "B" are involved in nucleotide triphosphate (NTP) binding and hydrolysis, wh
86 f promoter open complexes and the effects of nucleotide triphosphate (NTP) concentration on the effic
87                                       At low nucleotide triphosphate (NTP) concentrations, we observe
88  the RTC requires recognition of the correct nucleotide triphosphate (NTP) for incorporation into the
89                            Helicases utilize nucleotide triphosphate (NTP) hydrolysis to translocate
90                                Elevated dNTP/nucleotide triphosphate (NTP) ratios in Deltalon cells p
91  coil to a helical hairpin that contacts the nucleotide triphosphate (NTP) substrate to allow rapid n
92  for elongation and apparent K(m) values for nucleotide triphosphate (NTP) use.
93 hosphomonoesters, inorganic phosphate, gamma-nucleotide triphosphate (NTP), and beta-NTP were measure
94 ge proteins characterized by an NH2-terminal nucleotide triphosphate (NTP)-binding domain, two long s
95                                              Nucleotide triphosphate (NTP)-dependent molecular switch
96                                          The nucleotide triphosphate (NTP)-driven translocation hypot
97 vershoot (measured absolutely or relative to nucleotide triphosphate, NTP) following HI has been obse
98 ase of MANT ADP, where T, D, and Pi refer to nucleotide triphosphate, nucleotide diphosphate, and ino
99 ep Vent (exo(-)) DNA polymerase accepted the nucleotide triphosphate of C-nucleotide 6 as a substrate
100 hat the RecA-catalyzed hydrolysis of a given nucleotide triphosphate or analogue thereof is exquisite
101 etal muscle were observed in the presence of nucleotide triphosphates or diphosphates but not AMP, cA
102 ir 2-deoxy forms (collectively designated as nucleotide triphosphates or NTPs) as contractile substra
103                  A noticeable preference for nucleotide triphosphates over nucleotide diphosphates an
104        Reactive oxygen species (ROS) oxidize nucleotide triphosphate pools (e.g., 8-oxodGTP), which m
105                                        These nucleotide triphosphate preferences should have ramifica
106    Unlike mutT, a gene for another conserved nucleotide triphosphate pyrophosphohydrolase that functi
107 humb domain to create a tunnel through which nucleotide triphosphates reach the active site.
108 on of apyrase demonstrated a requirement for nucleotide triphosphate signalling.
109 nzymes are similar, their aminoglycoside and nucleotide triphosphate substrate profiles are distincti
110 esistance profile and the aminoglycoside and nucleotide triphosphate substrate profiles of four commo
111 d the other can utilize either ATP or GTP as nucleotide triphosphate substrate.
112 compromise blocked proliferation by limiting nucleotide triphosphate synthesis.
113       When trinitrophenyl-ATP, a fluorescent nucleotide triphosphate that functions as an alternative
114   In this work, we synthesized an artificial nucleotide triphosphate that is selectively inserted opp
115  Compared to those of the other hydrolyzable nucleotide triphosphates, the ATPase activity of Lon is
116                                        Among nucleotide triphosphates, the order is ATP > CTP approxi
117                           For binding to the nucleotide triphosphates, the order of binding affinity
118 KRas bound to its corresponding enantiomeric nucleotide triphosphate, this study sets the stage for f
119                  Some CYTH orthologs cyclize nucleotide triphosphates to 3',5'-cyclic nucleotides.
120 te of the active RecA protein using modified nucleotide triphosphates to discern key structural eleme
121 nsferases that couple sugar-1-phosphates and nucleotide triphosphates to form Leloir pathway glycosyl
122 e (10 units/ml), which rapidly hydrolyzes 5' nucleotide triphosphates to monosphophates, prevented th
123 er of the effectiveness of the corresponding nucleotide triphosphates to support force production in
124  kinase, which would transfer phosphate from nucleotide triphosphates to the GDP bound to Gk, produci
125 emical reaction, most commonly hydrolysis of nucleotide triphosphates, to their conformational change
126  we show that an exogenously expressed algal nucleotide triphosphate transporter efficiently imports
127                     The global regulation of nucleotide triphosphate turnover by intracellular Mg(2+)
128 he rate of transcription on concentration of nucleotide triphosphate, we infer that the combed DNA mo
129 e activity was specific for polynucleotides; nucleotide triphosphates were not hydrolyzed.
130                                 Radiolabeled nucleotide triphosphates were used to confirm the desire
131 us studies suggested that the interaction of nucleotide triphosphate with CFTR at ATP-binding site 2
132 sults also suggested that the interaction of nucleotide triphosphate with CFTR at ATP-binding site 2
133 ation with complete substitution of all four nucleotide triphosphates with phosphorothioates or the s

 
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