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1 ke (29-46% IA/g at 4 h in xenografted BALB/c nude mice).
2 heroid formation and tumorigenesis in Balb/c nude mice.
3 ess differentiated tumors when injected into nude mice.
4 nd diminishes tumorigenesis of xenografts in nude mice.
5 proliferation, migration and tumor growth in nude mice.
6 rived xenografts expressing SLC13A5-shRNA in nude mice.
7 -amplified neuroblastoma xenograft growth in nude mice.
8 s determined by xenograft studies in athymic nude mice.
9 KB-3-1 and COLO-205 tumor xenograft-bearing nude mice.
10 xenografts in the mammary fat pads of female nude mice.
11 to be nearly abolished in immunocompromised nude mice.
12 llomavirus has shown broad tissue tropism in nude mice.
13 CSC-like cells formed subcutaneous tumors in nude mice.
14 nd promotes the formation of fibrosarcoma in nude mice.
15 on and peritoneal tumor formation in athymic nude mice.
16 cells (A2780) implanted orthotopically into nude mice.
17 s ATF4 expression and its tumor formation on nude mice.
18 HV (TIVE-KSHV) into hyperglycemic and normal nude mice.
19 ly into NTR1-positive HT29 xenograft-bearing nude mice.
20 anted into calvarial defects created in CD-1 nude mice.
21 inhibition of HCC xenograft tumor growth in nude mice.
22 ctionally less effective at wound closure in nude mice.
23 l migration and abolished lung metastasis in nude mice.
24 bits proteasome function and tumor growth in nude mice.
25 cycle analysis and in vivo tumorigenesis in nude mice.
26 -expressing human lung cancer cell line into nude mice.
27 he multidrug resistant MCF-7/ADR xenografted nude mice.
28 plates, colonies in soft agar, and tumors in nude mice.
29 ses tumor growth and metastatic potential in nude mice.
30 rived orthotopic xenograft tumors in athymic nude mice.
31 ts and increases the median survival time of nude mice.
32 om MDA-MB-231 BC cells in bones and lungs of nude mice.
33 ancer cell proliferation and tumor growth in nude mice.
34 dard in vivo ectopic osteoinduction assay in nude mice.
35 on and a matrigel plug angiogenesis assay in nude mice.
36 -231 cells) growing in the brains of athymic nude mice.
37 blot assays, or injected subcutaneously into nude mice.
38 bited their tumorigenicity when engrafted in nude mice.
39 Calif) and injected in both flanks of eight nude mice.
40 a A375 human melanoma tumor model in athymic nude mice.
41 ion of breast cancer cell line xenografts in nude mice.
42 tocin-induced 8- to 10-week-old male athymic nude mice.
43 doses of a (90)Y-labeled GRPr antagonist in nude mice.
44 d into the distal posterior rectum of BALB/c-nude mice.
45 cell line and in vivo in PC-3 tumor-bearing nude mice.
46 nospheres in soft agar and nodules/tumors in nude mice.
47 gnificantly suppressed by NOS2 inhibition in nude mice.
48 in culture and promotes xenograft growth in nude mice.
49 duced tumorigenesis in an allograft model of nude mice.
50 ther subcutaneously or intraperitoneally, in nude mice.
51 ells in vitro and induces tumor formation in nude mice.
52 tion with the capacity to form metastases in nude mice.
53 orsphere assays and were less tumorigenic in nude mice.
54 sitive neovasculature when transplanted into nude mice.
55 man pancreatic adenocarcinomas propagated in nude mice.
56 m planar hair-bearing skin when grafted onto nude mice.
57 and orthotopic xenografts of CL1-5 cells in nude mice.
58 rongly reduced the growth of ccRCC tumors in nude mice.
59 to enhanced lethality of tumor xenografts in nude mice.
60 tides targeting HE4 arrested tumor growth in nude mice.
61 d enhanced metastatic potential in allograft-nude mice.
62 eration in vitro and HCC xenograft growth in nude mice.
63 lations markedly inhibited tumorigenicity in nude mice.
64 oliferation in vitro and after grafting onto nude mice.
65 the AKT pathway in the CAM model, as well as nude mice.
66 cells that were subcutaneously implanted in nude mice.
67 n promotes tumor growth of AFP- HCC cells in nude mice.
68 nospheres in soft agar and nodules/tumors in nude mice.
69 1 in vitro but also enhanced tumor growth in nude mice.
70 nograft growth and invasion of CRC tumors in nude mice.
71 gar, and increased xenograft tumor growth in nude mice.
72 s and generated transplantable xenografts in nude mice.
73 ream of EN, and abolishes tumor formation in nude mice.
74 ration and tumor formation and metastasis in nude mice.
75 reased growth of tumor xenografts in athymic nude mice.
76 inhibition of HCC xenograft tumor growth in nude mice.
77 e diabetes after transplantation in diabetic nude mice.
78 ration, and/or growth of xenograft tumors in nude mice.
79 2Lp53-expressing cells were xenografted into nude mice.
80 G12C)-expressing human PDAC line, in athymic nude mice.
81 idated in intracranial glioma bearing BALB/c nude mice.
82 vitro and growth of MDA-MB-468 xenografts in nude mice.
83 on CRPC cells in culture and implanted into nude mice.
84 uced the growth of human tumor xenografts in nude mice.
85 and a lung cancer tumor xenograft (A549) in nude mice.
86 etastases in human pancreatic cancer-bearing nude mice.
87 nvasion, migration, and xenograft tumors, in nude mice.
88 ilage plugs were implanted subcutaneously in nude mice.
89 lation, Ki-67 expression and tumor growth in nude mice.
90 istant human ovarian cancer cells in athymic nude mice.
91 ggressive orthotopic tumor models in athymic nude mice: a human PC-3 M-luc-C6 prostate tumor and a hu
93 cells, and in both eight-patient bloods and nude mice administered with the labeled CTCs in comparis
94 K1/2 inhibitor given to TNF-alpha-pretreated nude mice after human SSRBC infusion and onset of vasooc
95 experiments were performed on tumor-bearing nude mice after subcutaneous injection of RIN-m5F cells.
98 B or C-I inhibited tumor xenograft growth in nude mice and decreased the expression of CSC-markers an
99 ctively) to grow tumor xenografts in athymic nude mice and demonstrated the molecular specificity and
100 ed engrafting of pancreatic CSC's tumours in nude mice and displayed an antigrowth effect toward init
101 (67)Cu-CuSarTATE is well tolerated in BALB/c nude mice and highly efficacious against AR42J tumors in
102 evels of estradiol in human breast cancer in nude mice and in murine breast cancer in immune-competen
103 hibited cancer cell growth and metastasis in nude mice and inhibited cancer cell migration, invasion,
104 iments were then carried out in both healthy nude mice and nude mice with subcutaneous tumor to valid
105 s were grown as orthotopic tumors in athymic nude mice and PAF1 knockdown was induced by administrati
106 tifying the biodistribution of antibodies in nude mice and provides an alternative to PET analysis in
107 was initially established subcutaneously in nude mice and the subcutaneous tumor tissue was then ort
108 ChA-1 cells were injected into the flanks of nude mice and treated with miR-24 inhibitor or inhibitor
110 ces hypoxia in FaDu and HCT116 xenografts in nude mice, and causes a significant tumour growth delay
111 lorectal cancer metastases were generated in nude mice, and epifluorescence imaging of ICG, as well a
113 and H1975) were subcutaneously implanted in nude mice, and growth was followed by caliper measuremen
115 matrix proteins, subcutaneous tumor size in nude mice, and invasive behavior, including bone marrow
116 nd cervical cancer cell xenograft in vivo in nude mice, and suppress cervical cancer cell migration a
117 nt growth inhibition of MOLM13 xenografts in nude mice, and the activity correlates with inhibition o
118 bodies and transferred into infected SCID or nude mice, and the animals received the same antibody ev
119 ulated into the left thyroid lobe of athymic nude mice, and the orthotopic tumor growth was monitored
120 2 were injected into the pancreas of athymic nude mice, and their local and distant spread was monito
121 ously and orthotopically implanted tumors in nude mice, and was accompanied by c-SRC downregulation.
131 dent SSM3 mouse mammary tumors, male athymic nude mice bearing androgen-dependent CWR22 prostate canc
132 et involving ultrasound images of 23 athymic nude mice bearing C26 mouse adenocarcinomas was assemble
133 purified reagents were injected into athymic nude mice bearing CD44-positive human tumors (MDA-MB-231
134 trigel suspension model was established with nude mice bearing cells equally infected with each repor
137 ncer xenografts, and male and female athymic nude mice bearing estrogen-independent MDA-MB-231 human
138 jected intravenously into the BALB/c athymic nude mice bearing folate receptor (FR)-overexpressing KB
140 nd biodistribution studies were performed in nude mice bearing HCC4006 and A549 xenograft tumors.
141 s of intravenously injected nanoparticles in nude mice bearing HCT116 tumors radiosensitization was e
143 icacy of PEG-GIRLRG peptide was evaluated in nude mice bearing heterotopic cervical (HT3), esophageal
144 rculation and evaluated the tumor binding in nude mice bearing heterotopic cervical (HT3), esophageal
146 ed with [(68)Ga]Ga-FAPI-04 were conducted in nude mice bearing HT1080hFAP tumors or U87MG xenografts.
151 In vivo antitumor efficacy was tested in nude mice bearing PSMA+ PC3 PIP or PSMA- PC3 flu flank x
152 tion and SPECT/CT imaging experiments, using nude mice bearing PSMA-positive PC-310 and PSMA-negative
157 cer biodistribution was determined in BALB/c nude mice bearing subcutaneous CHL-GLP-1R xenografts.
158 of tumor cure while being well tolerated by nude mice bearing subcutaneous GPA33-positive SW1222 xen
159 One-hour dynamic PET scans were performed on nude mice bearing subcutaneous human head and neck tumor
161 cs of (89)Zr-DFO-AC-10 was studied in BALB/c nude mice bearing subcutaneous human Karpas 299 tumors (
163 nd biodistribution studies were performed on nude mice bearing U87MG and MDA-MB-231 xenografted tumor
164 /kg MEK1/2 inhibitor to TNF-alpha-pretreated nude mice before human SSRBC infusion inhibited SSRBC ad
169 in tumor xenografts grown in female athymic nude mice by small-animal PET/CT imaging and tissue biod
171 scles were subcutaneously injected into CD-1 nude mice (CD-1 nude mice, Crl:CD1-Foxn1(nu); Charles Ri
172 117085 significantly reduces tumor growth in nude mice compared with control untreated mice or either
173 nhibited orthotopic prostate tumor growth in nude mice, compared with monotherapy, by reversing the e
175 taneously injected into CD-1 nude mice (CD-1 nude mice, Crl:CD1-Foxn1(nu); Charles River Laboratories
180 the OVCAR-3 and OVCAR-8 xenograft models in nude mice, demonstrating synergistic antitumor activity
182 cells were subcutaneously transplanted into nude mice, DPSC/CTL cells induced mineralized tissue for
184 man breast cancer (MCF-7 xenograft) model in nude mice, EO-33 blocked 90% of tumor growth induced by
186 utside the primary tumor microenvironment in nude mice, exhibited signatures of immune evasion, incre
187 vivo an orthotopic liver injection model in nude mice further demonstrated that knockdown of RhoE en
188 ntal pulp on poly-l-lactic acid scaffolds in nude mice gave rise to perfect heterotopic ossicles in v
189 V1-induced cSCC from back skin, into athymic nude mice gave rise to secondary cSCCs, which lacked vir
192 ild cardiac hypertrophy was also observed in nude mice implanted with IDH2(R140Q)-expressing xenograf
195 uding HUVEC-mediated trophoblast invasion in nude mice, in vitro three-dimensional capillary tube for
196 tants strongly decreased tumor metastases in nude mice, indicating the requirement of PTTG for STAT3-
197 of these mutant-expressing cells in athymic nude mice induced rapid tumor development, showing their
198 el) into groups of 4-week-old athymic female nude mice (induced with subcutaneous triple negative xen
199 ties, SPECT and CT scans of HT29-xenografted nude mice injected with (177)Lu-3BP-227 were acquired, a
200 tically attenuates tumor growth in xenograft nude mice injected with human K562 leukemia cells and ce
201 Subcutaneous injection of TRIM24 iHMECs in nude mice led to growth of intermediate to high-grade tu
202 id gland scaffolds into the renal capsule of nude mice led to the differentiation of transplanted hDF
203 rs and xenografts established in NOD-SCID or nude mice, low MCPIP1 levels correlated strongly with in
204 next evaluated in a validated orthotopic GBM nude mice model, studying the tumor growth over time by
212 in human T2D M2MPhis transplanted to athymic nude mice (NMRI-Foxn1(nu)/Foxn1(nu) ) or systemic inhibi
214 ce fulfilled criteria for the model but only nude mice offered sufficient availability for large ther
216 ast cancer were examined using female BALB/c nude mice orthotopically implanted with human breast car
217 implantation of LOX-treated neocartilage in nude mice promoted further maturation of the neotissue,
218 uman prostate xenograft model established in nude mice, RAD001 alone or in combination with docetaxel
219 se model, after tumor establishment, athymic nude mice received treatment with progesterone or vehicl
222 ansplantation of hERG1-expressing cells into nude mice resulted in an increased incidence of tumors.
223 5alpha-depleted pancreatic cancer cells into nude mice resulted in markedly reduced tumorigenicity (P
224 KD SCC1 cells into the floor of the mouth in nude mice resulted in the formation of significantly sma
225 human gliomas (U87) grown orthotopically in nude mice resulting in a more than a doubling of median
226 ne was sufficient to induce tumorigenesis in nude mice resulting in short lifespan irrespective of wh
227 D25(+)IL7Ralpha(+)) after grafting recipient nude mice revealed that MPP3 cells were the most effecti
228 ografts of MKN45/5FU cells in the stomach of nude mice revealed that these cells had a high potential
230 lonization of melanoma cells in the lungs of nude mice, suggesting an increase in metastatic potentia
232 pressed DNAJB6a formed tumors more slowly in nude mice than control cells or cells that expressed a m
233 ive MDA-MB-231 tumors to a greater extent in nude mice than in NSG mice, pointing to the potential ro
235 expressing versican siRNA were injected into nude mice, the resulting tumors displayed significantly
236 n PMP tissue was i.p. grafted and grown into nude mice, then constituted into reliable and reproducib
238 hyme were grafted under the renal capsule of nude mice to generate prostatelike tissues, and mice wer
239 s computed from ultrasound images of athymic nude mice to predict tumor response to treatment at an e
240 and AGS, which do and do not form tumors in nude mice, to identify their genomic differences relevan
243 an ovarian SKOV3 tumors cells into 14 female nude mice, treatment with vehicle or pazopanib (2.5 mg p
244 e than WT cybrids, however, when injected in nude mice, tRNAmut cybrids produced larger tumours and s
247 ry cholangiocarcinoma (CCA) models in BALB/c nude mice using minimally invasive ultrasound-guided int
248 ia conditions, were then assessed in healthy nude mice using the left kidney and spleen as reference
249 he tracer in the pancreatic islets of BALB/c nude mice was examined using fluorescence microscopy.
250 of WT HBx-expressing cells to form tumors in nude mice was significantly higher than that of mutant H
254 ard this end, orthotopic xenografts grown in nude mice were exposed to a fractionated radiation proto
260 and preliminary in vivo xenograft studies in nude mice were performed to evaluate target binding.
261 1-CCK2R/A431-mock xenografted athymic BALB/c nude mice were used for biodistribution studies and smal
262 on in PCa cells and induces larger tumors in nude mice, whereas its silencing decreased proliferation
263 sulted in marked regression of xenografts in nude mice, whereas the delivery of an miR-125a inhibitor
264 g in aggressive tumor formation in xenograft nude mice, which could be suppressed by combined treatme
266 ive in suppressing xenograft tumor growth in nude mice, which underlines the translational potential
267 ous cell carcinoma upon inoculation into the nude mice, while parental HaCaT cells remain non-tumorig
268 avenously administered every 3 d for 16 d to nude mice with AR42J tumor xenografts that were approxim
269 OIS and enabled tumor transplants to grow in nude mice with characteristic cell morphology of anaplas
270 In vivo treatment of A375 xenograft-bearing nude mice with cryptolepine (10 mg/Kg body weight, i.p.)
271 t overexpress MFSD2A were transferred to CD1 nude mice with dextran sodium sulfate-induced colitis, w
273 of (177)Lu-octreotate was examined in BALB/c nude mice with GOT2 tumors 1-168 h after injection with
278 ribution, and fluorescence imaging on BALB/c nude mice with orthotopically transplanted PC346C tumors
279 26 nmol/mouse, 8-9 MBq/mouse) in male BALB/c nude mice with PSMA-expressing subcutaneous LS174T-PSMA
280 uorescence imaging to detect ccRCC tumors in nude mice with RCC xenografts by using mAb girentuximab
282 ing product, (18)F-FVIIai, was injected into nude mice with subcutaneous human pancreatic xenograft t
283 11B6 uptake was performed on NMRI and BALB/c nude mice with subcutaneous LNCaP xenografts up to 14 d
284 ouse) was injected intravenously into BALB/c nude mice with subcutaneous PSMA-expressing LNCaP tumors
285 111)In-labeled ADCs were performed on BALB/c nude mice with subcutaneous PSMA-positive LS174T-PSMA xe
286 en carried out in both healthy nude mice and nude mice with subcutaneous tumor to validate the contra
288 Methods: Two groups of 6 adult female BALB/c nude mice with subcutaneously implanted tumors underwent
289 ary tumors, in an NFATc1-dependent manner in nude mice with T-cell deficiency, revealing an addiction
290 targeting potential and antitumor effects in nude mice with tumors that were sensitive or resistant t
294 ot R-lycosin-I inhibited tumor growth in the nude mice xenograft model without generating side effect
296 ble for small-animal PET studies in multiple nude mice xenografted with the A431 carcinoma cell line.