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1 sociated with increasing body mass index and nulliparity.
2 onset ovarian cancer, except for obesity and nulliparity.
3 breast or ovarian cancer, endometriosis, and nulliparity.
4 5, 0.96) after adjustment for age, race, and nulliparity.
5 ctin, but obstetrical measures that included nulliparity (0.171 +/- 0.058; P = 0.004), longer duratio
6 chronic hypertension (high-risk factors) and nulliparity, a body mass index greater than 30, African
9 significant interactions (P < 0.05) between nulliparity and PMD/NDA, age at menarche and area of den
10 ed risk; current use of oral contraceptives, nulliparity, and age 30 years or older at first birth we
11 nstrual cycle length, lower body mass index, nulliparity, and congenital obstructive mullerian anomal
12 ng age, race and ethnicity, body mass index, nulliparity, and factors associated with increased risk
15 udy suggest that irregular menstrual cycles, nulliparity, and multiparity were associated with higher
18 closely in terms of oral contraceptive use, nulliparity, and religion and differed from the cases on
19 odds ratio [AOR], 1.09; 95% CI, 1.05-1.12), nulliparity (AOR, 1.84; 95% CI, 1.31-2.60), private insu
20 cating that the effects of breastfeeding and nulliparity are modified by HLA DR4 status, suggesting a
21 viewed, HD risk was related to parity versus nulliparity but only among never nursers (odds ratio (OR
22 nt association for each of these factors and nulliparity combined compared with first birth before ag
24 years, first birth at age 20 years or later/nulliparity, family history of breast cancer, and histor
26 before 30 years of age (HR 1.87, P = 0.04), nulliparity (HR 2.14, P = 0.01), and unilateral oophorec
28 .6, 95% confidence interval (CI): 1.0, 2.4), nulliparity/late age at first full-term pregnancy (OR =
29 ancers) women to examine the hypothesis that nulliparity may increase susceptibility to established p
30 risk factors for endometrial cancer included nulliparity, never using oral contraceptives, fertility
31 (except PFNA), less educational attainment, nulliparity, no history of breastfeeding and higher prep
32 .5-25), history of benign breast biopsy, and nulliparity or age at first birth (>/=30 years vs <30 ye
33 obesity; a family history of pre-eclampsia, nulliparity or multiple pregnancies; and previous pre-ec
34 thweight (OR(adj) 2.22, 95% CI [1.79-2.75]), nulliparity (OR(adj) 1.81, 95% CI [1.60-2.05]), underwei
36 ty, family history of breast/ovarian cancer, nulliparity, oral contraceptive use, and tubal ligation)
37 able VL included multiparity (16.4% vs. 8.0% nulliparity; P = 0.002), black ethnicity (17.6% vs. 6.6%
38 highest completed educational qualification, nulliparity, polycystic ovary syndrome, physical activit
39 (hormone use, alcohol consumption, obesity, nulliparity) present a relatively modest relative risk f
41 did not were younger and had lower rates of nulliparity, twin pregnancy, hypertension, and gestation
42 ociated with higher risk of incident HF, and nulliparity was associated with higher risk for incident
44 sk factors but without any high risk factor, nulliparity was associated with significantly increased
45 Conversely, early age at first pregnancy and nulliparity were significantly associated with incident
47 equal to the increased risk associated with nulliparity, when compared to exposure in the first quar
48 lbirth (6.7% stillbirths, 1.4% live births); nulliparity with (10.5% stillbirths, 5.2% live births) a