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1 masses, urinary tract infection, trauma, and obstructive uropathy.
2 hat results from the development of in utero obstructive uropathy.
3 rosis and chronic renal failure secondary to obstructive uropathy.
4 nt females died at 4-6 months of age from an obstructive uropathy.
5 understanding the pathobiology of congenital obstructive uropathy.
6 which includes ischemic hypoxic insults, and obstructive uropathy.
7  the understanding of the pathophysiology of obstructive uropathy.
8 d increasing use of medications for treating obstructive uropathy.
9 tones, or renal insufficiency as a result of obstructive uropathy.
10 weight/obesity associated significantly with obstructive uropathy.
11 ovides valuable insight into a wide range of obstructive uropathies.
12 rovides unique insights into a wide range of obstructive uropathies and has been demonstrated to be u
13 ecessive disease characterized by congenital obstructive uropathy and abnormal facial expression.
14                                              Obstructive uropathy and dysplasia were the cause of CRI
15                 Urinary tract malformations, obstructive uropathy, and hypoplasia/dysplasia are extre
16 d risk factors, such as young recipient age, obstructive uropathy, and overall intensity of immunosup
17     Epithelial cell fate and nephron loss in obstructive uropathy are not fully understood.
18 ity was ischemic bowel (75%); the lowest was obstructive uropathy-associated urinary tract infection
19                                      Chronic obstructive uropathy (COU) created by unilateral ureteri
20                                   Congenital obstructive uropathy (COU) is a common cause of developm
21                                   Congenital obstructive uropathy (COU) is a prevalent human developm
22 tenatal intervention has been done for fetal obstructive uropathy for over a decade, yet little is kn
23 ing kidney in the fetus as it is affected by obstructive uropathy has had no significant advances in
24     In the current study, a model of chronic obstructive uropathy in the mouse is established and the
25 nital tract, abdominal pain, abdominal mass, obstructive uropathy, infertility, menstrual irregularit
26                                              Obstructive uropathy is the most common complication, al
27                                   In a mouse obstructive uropathy model of chronic kidney disease, in
28 e risk of acute or chronic renal conditions, obstructive uropathies, neoplasia, or infectious process
29 t age (OR, 1.1 per y younger; P < 0.001) and obstructive uropathy (OR, 12.4; P < 0.01) as primary ren
30 s, encompassing GD-CNVs and novel deletions; obstructive uropathy (OU) had a lower CNV burden and an
31 s in pediatric CKD based on diagnosis: FSGS, obstructive uropathy (OU), aplasia/dysplasia/hypoplasia
32 se models of postischemic renal fibrosis and obstructive uropathy, treatment with N-terminal Slit2 be
33 y Network criteria), exclusion criteria were obstructive uropathy, urothelial carcinoma, and metastat
34  those fetuses with the most severe forms of obstructive uropathy, usually associated with a fatal ne
35        Understanding the mechanisms of fetal obstructive uropathy will be essential for the specific