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1 y factor for protection against cerebral and ocular toxoplasmosis.
2 s and might contribute to the development of ocular toxoplasmosis.
3 lls plays a central role in the pathology of ocular toxoplasmosis.
4 sefulness of blood samples for genotyping in ocular toxoplasmosis.
5 directly from blood samples of patients with ocular toxoplasmosis.
6 the regulation of inflammation during acute ocular toxoplasmosis.
7 he epidemiology and efficacious treatment of ocular toxoplasmosis.
8 likely to be involved in the pathogenesis of ocular toxoplasmosis.
9 NA sequence in samples from 12 patients with ocular toxoplasmosis, 1 sample from a patient with conge
11 urrence of certain forms of uveitis (such as ocular toxoplasmosis) after surgery, suggesting that per
14 med in 46 serum specimens from patients with ocular toxoplasmosis and in 28 serum specimens from pati
15 lf from CD4 T-cell-mediated immune damage in ocular toxoplasmosis and other types of retinal immune r
19 matopoietic cells and can cause cerebral and ocular toxoplasmosis, as a result of either congenital o
20 otide sequences from Colombian patients with ocular toxoplasmosis belonged to the group of mouse-viru
21 n of 12 sequences (83.3%) from patients with ocular toxoplasmosis clustered with those of mouse-virul
22 th an increased risk of being diagnosed with ocular toxoplasmosis, corroborating findings from a prio
25 retinochoroiditis is a major complication of ocular toxoplasmosis in infants and immunocompetent indi
28 sL interaction in a murine model of acquired ocular toxoplasmosis induced by intracameral inoculation
33 s of patients with Behcet uveitis (n = 259), ocular toxoplasmosis (n = 120), and multiple sclerosis (
37 us fluid of patients with severe or atypical ocular toxoplasmosis, polymerase chain reaction (PCR) re
38 ibships and 89 parent/case trios of presumed ocular toxoplasmosis (POT) to evaluate associations with
39 We tested all samples from patients with ocular toxoplasmosis sent to the Palo Alto Medical Found
40 eater odds of PPI/H2 blocker use in cases of ocular toxoplasmosis than matched controls (95% confiden
41 ole of CD4(+) T cells in the pathogenesis of ocular toxoplasmosis was investigated in murine models u
42 In a prospective clinical study of acute ocular toxoplasmosis, we assessed the cytokine pattern i
43 rstand the role of interleukin-10 (IL-10) in ocular toxoplasmosis, we compared C57BL/6 (B6) and BALB/
46 on regarding all pregnancies and episodes of ocular toxoplasmosis, whether or not episodes were obser