ライフサイエンスコーパス: odontogenic

1 nal (58%), intravascular catheter (35%), and odontogenic (15%).

2 e analyzed patient-matched dental plaque and odontogenic abscess clinical specimens and examined whet

3 Despite the high incidence of odontogenic abscesses in pet rabbits, published data on

4 ia, with Fn. polymorphum dominating, whereas odontogenic abscesses were exceptionally biased for the

5 1 and Tbx2 in turn antagonistically regulate odontogenic activity that leads to EK formation and to m

6 amelysin (MMP-20), kallikrein-4 (KLK-4), and odontogenic ameloblast-associated protein (ODAM) using q

7 in early expressed enamel matrix components (Odontogenic ameloblast-associated protein) and dentin dy

8 Osteo/odontogenic and adipogenic differentiation were analyzed

9 had the ability to differentiate into osteo/odontogenic and adipogenic lineages.

10 d the ability to be differentiated to osteo-/odontogenic and adipogenic pathways, resembling mesenchy

11 of CD146+ pericytes are likely controlled by odontogenic and Bmp2-dependent VegfA production in odont

12 racts with Notch signaling and promotes both odontogenic and osteogenic differentiation of SCAPs.

13 ithelium are required for the development of odontogenic and skeletogenic mesenchyme cells.

14 Odontogenic-associated genes and proteins were detected

15 fections and that of odontogenic infections, odontogenic bacteremia, and community-acquired brain abs

16 alence of, and sensitivity for, detection of odontogenic bacteremia.

17 n inducible in Aves and suggest that loss of odontogenic Bmp4 expression may be responsible for the e

18 In conclusion, P(i) enhanced osteo/odontogenic but inhibited adipogenic differentiation in

19 tion of cranial neural crest is specified as odontogenic by Dlx-1 and Dlx-2 genes.

20 This case report describes an unusual odontogenic carcinoma, which was detected during routine

21 olecular evidence of cftr mRNA expression by odontogenic cells strongly suggest that CFTR plays an im

22 me cells of the mandibular arch give rise to odontogenic cells, while more caudal cells form the dist

23 APC staining was not seen in any other odontogenic cells.

24 with potent capacities to differentiate into odontogenic cells.

25 uctive mesenchyme is capable of inducing the odontogenic commitment of both dental and non-dental epi

26 eem to have evolved through the extension of odontogenic competence from the external dermis to inter

27 ermal armor that consisted of osteogenic and odontogenic components.

28 most cases CT study in patients suspected of odontogenic craniofacial infection revealed presence of

29 The odontogenic keratocyst is a common odontogenic cyst which accounts for 10% of all oral cyst

30 val cyst of the adult are both developmental odontogenic cysts located predominantly in the mandibula

31 ific and closely resemble other inflammatory odontogenic cysts.

32 It represents 3% to 12% of all odontogenic cysts.

33 d collagen 1 (Col1) after 7, 14, and 21 d of odontogenic differentiation (P < 0.05).

34 hibition of Ca(2+) influx and suppression of odontogenic differentiation and mineralization as demons

35 ortantly, Phen-treated DPSCs showed enhanced odontogenic differentiation and mineralization in vivo.

36 evaluated the role of ORAI1 in mediating the odontogenic differentiation and mineralization of dental

37 howed that ORAI1 plays critical roles in the odontogenic differentiation and mineralization of DPSCs

38 The gene expression for odontogenic differentiation markers such as osteocalcin,

39 During the odontogenic differentiation of DPSCs, the expression of

40 ciated with the odontogenic region, promotes odontogenic differentiation of MSCs.

41 t BMP signaling activity is required for the odontogenic differentiation of MSCs.

42 activation in DPSCs rapidly increase during odontogenic differentiation, further amplified by inflam

43 uggest planar cell polarity signaling during odontogenic differentiation.

44 tivation of Wnt target genes responsible for odontogenic differentiation.

45 red and evaluated the pro-osteogenic and pro-odontogenic effects of 4 small bioactive compounds- phen

46 It is a benign aggressive tumor of odontogenic epithelial origin with a high rate of recurr

47 ocally invasive benign neoplasm derived from odontogenic epithelium and presents with diverse phenoty

48 In contrast, high levels of Shh in odontogenic epithelium arrests tooth development at the

49 through bud, cap, and bell stages, in which odontogenic epithelium continuously extends into the und

50 Shh expression specifies the odontogenic epithelium during initiation and is consiste

51 ental hard tissue interspersed in a field of odontogenic epithelium in a glandular configuration, con

52 restrict Wnt and downstream FGF signaling in odontogenic epithelium through facilitation of Shh signa

53 including the skin, vagina, forestomach, and odontogenic epithelium.

54 al maturation of both colonic epithelium and odontogenic epithelium.

55 al cystic neoplasm thought to arise from the odontogenic epithelium.

56 factors that are expressed in early Astyanax odontogenic epithelium.

57 active tumor of the gingiva that may have an odontogenic etiology, whose origin seems to be the remna

58 The controlled release of odontogenic exosomes resulted in a reparative dentin bri

59 ed by reduced cell proliferation and loss of odontogenic fate marker Pitx2 expression in the dental e

60 o to investigate the mechanism that sustains odontogenic fate of dental mesenchyme during tooth devel

61 ium, with Wnt signaling also controlling the odontogenic fate.

62 Expansion of odontogenic field in Osr2-deficient mice required Msx1,

63 nstream of Pax9 and patterns the mesenchymal odontogenic field through protein-protein interactions w

64 to their molars because of expansion of the odontogenic field.

65 ivation of Osr2, a negative regulator of the odontogenic function of the Bmp4-Msx1 signaling pathway,

66 rived from both hDPSCs and MDPCs upregulated odontogenic gene expression and increased mineralization

67 oral epithelial protein signal that directs odontogenic gene expression in mesenchyme cells of the d

68 nalysis indicating overrepresentation of key odontogenic genes.

69 ow-derived cells all responded by expressing odontogenic genes.

70 the genes envisaged as participating in an 'odontogenic homeobox gene code' by specifying molar deve

71 The molecular events of odontogenic induction are beginning to be elucidated, bu

72 lecule RhoA, and its overexpression prevents odontogenic induction.

73 ates tooth sensory innervation and maintains odontogenic inductive potential in dental mesenchyme, en

74 P.g.-odontogenic infection may promote the progression of neo

75 Thus, we aimed to elucidate whether P.g.-odontogenic infection promotes NASH-related HCC developm

76 P.g.-odontogenic infection significantly increased the mean n

77 ntibiotics are important in the treatment of odontogenic infections and the prevention of infection f

78 THIS STUDY WAS TO ASSESS PATHWAYS, BY WHICH ODONTOGENIC INFECTIONS SPREAD INTO RESPECTIVE DEEP HEAD

79 biology data from previous investigations on odontogenic infections, bacteremia after extraction of i

80 es appear to be risk factors for pain due to odontogenic infections, comparatively little is known ab

81 ology of such pleural infections and that of odontogenic infections, odontogenic bacteremia, and comm

82 teria for Gorlin syndrome, although none had odontogenic jaw keratocysts.

83 onal biopsy revealing contents typical of an odontogenic keratocyst (OKC).

84 d clinical knowledge base for the peripheral odontogenic keratocyst and assist clinicians in the iden

85 An odontogenic keratocyst can develop at virtually any site

86 The odontogenic keratocyst is a common odontogenic cyst whic

87 ribes the rare peripheral presentation of an odontogenic keratocyst localized to the maxillary anteri

88 ggressive clinical course for the peripheral odontogenic keratocyst.

89 This paper reviews odontogenic keratocysts and presents a case treated by c

90 Odontogenic keratocysts are believed to arise from remna

91 Odontogenic keratocysts are one component of the basal c

92 Odontogenic keratocysts in humans are aggressive, noninf

93 al cell nevus syndrome and all patients with odontogenic keratocysts should be evaluated for this syn

94 cell carcinomas, palmar and/or plantar pits, odontogenic keratocysts, skeletal and developmental anom

95 by multiple cutaneous basal cell carcinomas, odontogenic keratocysts, skeletal anomalies and facial d

96 ssues have been described to have osteogenic/odontogenic-like differentiation capacity, but little at

97 ferentiation capacity toward endothelial and odontogenic lineages were evaluated using live/dead assa

98 ol2a1, runx2a and chondromodulin, as well as odontogenic marker dlx2b.

99 n in the transcriptional regulation of early odontogenic markers that promote differentiation of DPSC

100 wth arrest-specific 1 (Gas1) is expressed in odontogenic mesenchyme and epithelium during multiple st

101 s directly involved in the activation of the odontogenic mesenchyme has not been characterized.

102 of a lateral signaling center over competent odontogenic mesenchyme led to loss of teeth in avians wh

103 for activin betaA is expressed in the early odontogenic mesenchyme of all murine teeth but mutant mi

104 tooth formation in the presumptive oral and odontogenic mesenchyme of the maxillary and mandibular p

105 that antagonizes Msx1-mediated activation of odontogenic mesenchyme, was significantly upregulated in

106 the management of ameloblastoma and related odontogenic neoplasms.

107 , 37 years; age range, 13-55 years), who had odontogenic orbital cellulitis underwent clinical evalua

108 Infections of odontogenic origin are the most common cause of inflamma

109 t (GCA) is an uncommon developmental cyst of odontogenic origin most frequently seen near mandibular

110 ence of a subperiosteal abscess suggested an odontogenic origin of orbital infection.

111 The odontogenic origin of the orbital infection was not clin

112 ival cysts in the adult are unusual cysts of odontogenic origin with less than 100 reported, includin

113 periodontal cyst (LPC) is an unusual cyst of odontogenic origin, most frequently encountered in the m

114 They are uncommon cysts of odontogenic origin, noted primarily in the fifth and six

115 ) cervicofacial infection were of mandibular odontogenic origin.

116 hown to be bioactive, capable of stimulating odontogenic/osteogenic differentiation as observed by ge

117 ike tissue formation in vivo and accelerated odontogenic/osteogenic differentiation both in vivo and

118 hat the ephrinB1-EphB2 interaction regulates odontogenic/osteogenic differentiation from dental pulp

119 lications for the mechanisms contributing to odontogenic pain and management strategies.

120 Odontogenic pain can be debilitating, and nonopioid anal

121 nt antagonist, is involved in regulating the odontogenic pathway downstream of Msx1.

122 1 plays a crucial role in the Msx1-dependent odontogenic pathway during early tooth morphogenesis.

123 view here the latest ideas on the control of odontogenic patterning and morphogenesis.

124 ess of the cranial neural crest cell-derived odontogenic populations.

125 al outer enamel epithelium, endowing it with odontogenic potential by the activation of several key t

126 The proposal that odontogenic potential extends into the trunk neural cres

127 aeal ectoderm and endoderm demonstrated that odontogenic potential extends not only further caudally

128 portion of the intermediate region, but that odontogenic potential extends to its most caudal limit.

129 synergizes with Msx1 to activate mesenchymal odontogenic potential for tooth morphogenesis and sequen

130 are known to be essential for the switch in odontogenic potential from the epithelium to the mesench

131 f tooth development depends on activation of odontogenic potential in the mesenchyme by signals from

132 n tooth development depends on activation of odontogenic potential in the presumptive dental mesenchy

133 The odontogenic potential of 12 individual single-colony-der

134 in vitro culture system was used to test the odontogenic potential of more caudal regions of neural c

135 ated to cyclic RGD peptides can increase the odontogenic potential of these cells.

136 the maxillary molar epithelium has lost its odontogenic potential.

137 of the dental lamina that contains quiescent odontogenic progenitors that can be activated during phy

138 ator dental laminae, which contain quiescent odontogenic progenitors.

139 indicate that activation of the mesenchymal odontogenic program during early tooth development requi

140 n factor, Klf4, which is associated with the odontogenic region, promotes odontogenic differentiation

141 2, Barx1, and Pax9 defines a potential chick odontogenic region.

142 egrate Dlx1, Dlx2 and Fgf3 and Fgf8 into the odontogenic regulatory hierarchy along with Msx1, Msx2 a

143 e and embryonic oral epithelium stimulate an odontogenic response in the stem cells.

144 s were absent from the epithelium of the non-odontogenic second branchial arch.

145 hat Bmp4 is a key Msx1-dependent mesenchymal odontogenic signal for driving tooth morphogenesis throu

146 morphogenetic protein 4 (Bmp4), an essential odontogenic signal, in the developing tooth mesenchyme.

147 activin and Bmp4 act as crucial mesenchymal odontogenic signals during early tooth development.

148 ption factors are required for activation of odontogenic signals, including Bmp4 and Fgf3, in the ear

149 nk between these diseases, primarily through odontogenic sources, but comprehensive population-based

150 nt genes and molecular pathways using normal odontogenic tissue (human "dentome") for comparison.

151 and potential signaling pathways, as well as odontogenic tissue formation during dental pulp regenera

152 compare it with gene expression of different odontogenic tissues ("dentome").

153 e odontodes (dermal teeth) and an absence of odontogenic tissues in its spines.

154 ow that this population alters its fate from odontogenic to become chondrogenic.

155 paction-induced changes in cell shape induce odontogenic transcription factors (Pax9, Msx1) and a che

156 5.00 increased odds of having a keratocystic odontogenic tumor (95% CI, 2.22 to 11.3) and 2.79 increa

157 e occurrence and treatment of an adenomatoid odontogenic tumor (AOT) presenting as a periodontal intr

158 nship between ameloblastoma and keratocystic odontogenic tumor (KCOT) by means of a genome-wide expre

159 Keratocystic Odontogenic Tumor (KCOT) is a locally aggressive develop

160 The keratocystic odontogenic tumor (KCOT) is a relatively rare, benign ne

161 The squamous odontogenic tumor (SOT) is a rare, benign, locally infil

162 A primary human keratocystic odontogenic tumor cell population, KCOT-1, has been esta

163 The mutant mice developed an odontogenic tumor of dental epithelium origin.

164 s of a young woman with a giant keratocystic odontogenic tumor of the mandible.

165 Ameloblastoma (AB) is an odontogenic tumor that arises from ameloblast-lineage ce

166 AOT is a rare odontogenic tumor usually associated with unerupted teet

167 f ameloblastomas, the most common epithelial odontogenic tumor, and in this tumor's likely precursor

168 uding the WNT pathway, might be the cause of odontogenic tumorigenesis and maintenance.

169 Adenomatoid odontogenic tumors (AOT) make up 3% of odontogenic tumor

170 Keratocystic odontogenic tumors (KCOT) may occur sporadically or asso

171 Keratocystic odontogenic tumors (KCOTs) of the jaw affect more than 6

172 er neoplastic growths including keratocystic odontogenic tumors and ovarian fibromas.

173 structures similar to calcifying epithelial odontogenic tumors in humans.

174 r 80% of ameloblastomas, locally destructive odontogenic tumors of the jaw, by genomic analysis of ar

175 Odontogenic tumors originate from the remains of migrati

176 Ameloblastomas are odontogenic tumors that are rare in people but have a re

177 atoid odontogenic tumors (AOT) make up 3% of odontogenic tumors.

178 None had keratocystic odontogenic tumors.

179 sh a molecular signature for the usually non-odontogenic VL in mice and highlight several genes and s