ライフサイエンスコーパス: oligodendrocyte precursor

1 ted migration of SVZ-derived progenitors and oligodendrocyte precursors.

2 elin-specific genes and increased numbers of oligodendrocyte precursors.

3 favored in more lineage-restricted neuronal-oligodendrocyte precursors.

4 ation and the generation of motor neuron and oligodendrocyte precursors.

5 n cfy embryos due to an apparent decrease in oligodendrocyte precursors.

6 crophage accumulation, and the appearance of oligodendrocyte precursors.

7 halic ventricular zone subsequently generate oligodendrocyte precursors.

8 ogenitor cell cultures promotes formation of oligodendrocyte precursors.

9 ately prior to, and during the appearance of oligodendrocyte precursors.

10 ikely to influence the initial appearance of oligodendrocyte precursors.

11 differentiation defects of miR-219-deficient oligodendrocyte precursors.

12 onto shared loci within oligodendrocytes and oligodendrocyte precursors.

13 n the developing brain include maturation of oligodendrocyte precursors and genetically programmed ch

14 oxidative cell death in primary cultures of oligodendrocyte precursors and immature fetal cortical n

15 MRP2/KLHL1 is expressed in oligodendrocyte precursors and mature OLGs, and its expr

16 receptors in ischaemia leads to the death of oligodendrocyte precursors and the loss of myelin.

17 or both the initial dispersal of spinal cord oligodendrocyte precursors and their subsequent developm

18 During development, the earliest oligodendrocyte precursors appear in the metencephalic v

19 Most remarkably, a normal number of oligodendrocyte precursors are formed at day 12 of mouse

20 Oligodendrocyte precursors are found only at low density

21 s a sequential model in which motoneuron and oligodendrocyte precursors are sequentially generated in

22 In the chick metencephalon, oligodendrocyte precursors arise in distinct domains of

23 Spinal cord oligodendrocyte precursors arise in the ventral ventricu

24 s required for the appearance of spinal cord oligodendrocyte precursors as neutralization of Shh sign

25 receptor 2 is localized in oligodendrocytes, oligodendrocyte precursors, astrocytes and macrophages/m

26 early 60% loss in astrocytes and 50% loss in oligodendrocyte precursors at birth.

27 quired for initiating the differentiation of oligodendrocyte precursors but has to be down-regulated

28 inoic acid and artificial polysialylation of oligodendrocyte precursors by a bacterial polysialyltran

29 recursors, a function that is antagonized in oligodendrocyte precursors by Olig2.

30 t-mediated activation of Notch1 receptors on oligodendrocyte precursors by the ligand Jagged1 induces

31 a (PDGFRalpha), the earliest known marker of oligodendrocyte precursors, by several days.

32 Ectopic oligodendrocyte precursors can be induced by sonic hedge

33 The oligodendrocyte precursor cell (OPC) arises from the sub

34 the proliferation and differentiation of the oligodendrocyte precursor cell (OPC) as well as the spat

35 Temple and Raff previously showed that an oligodendrocyte precursor cell (OPC) can divide a maximu

36 ceptor induced excitotoxicity contributes to oligodendrocyte precursor cell (OPC) damage and hypomyel

37 M), bazedoxifene (BZA), as a potent agent of oligodendrocyte precursor cell (OPC) differentiation and

38 functional states of Wnt activity determine oligodendrocyte precursor cell (OPC) differentiation and

39 or GABAergic interneuron production, repress oligodendrocyte precursor cell (OPC) formation by acting

40 ons contain hyaluronan deposits that inhibit oligodendrocyte precursor cell (OPC) maturation.

41 te numbers and CNS hypomyelination, although oligodendrocyte precursor cell (OPC) numbers are normal.

42 ein (BMP) signaling, such as Noggin, promote oligodendrocyte precursor cell (OPC) production after hy

43 Gpr56-knockout mice manifest with decreased oligodendrocyte precursor cell (OPC) proliferation and d

44 ndrocyte generation by negatively regulating oligodendrocyte precursor cell (OPC) proliferation.

45 Oligodendrocyte precursor cell (OPC)-specific TrkB delet

46 NG2 cells in the SVZ and RMS expressed the oligodendrocyte precursor cell antigen platelet-derived

47 hin the nervous system, including defects in oligodendrocyte precursor cell development and a partial

48 -gamma is a positive regulator of endogenous oligodendrocyte precursor cell differentiation and remye

49 y, we identified novel molecules involved in oligodendrocyte precursor cell differentiation and valid

50 Specifically, exogenous CCL19 abolished oligodendrocyte precursor cell differentiation observed

51 ligodendrogenesis, it subsequently increases oligodendrocyte precursor cell differentiation, oligoden

52 an enrichment of proliferative pathways and oligodendrocyte precursor cell gene expression profile i

53 regulated expression of GPR17 in Oli-neu, an oligodendrocyte precursor cell line, making these cells

54 We have identified an oligodendrocyte precursor cell line, termed G144, that s

55 velopment, may also be active in controlling oligodendrocyte precursor cell migration in MS, and henc

56 ple sclerosis lesions are thought to inhibit oligodendrocyte precursor cell migration, limiting their

57 xons or do intrinsic properties of different oligodendrocyte precursor cell populations affect length

58 tate-methyltransferase (Gamt) did not affect oligodendrocyte precursor cell recruitment, but resulted

59 This conditioned media also enhanced oligodendrocyte precursor cell survival, maturation and

60 signaling contributes to the decision of an oligodendrocyte precursor cell to differentiate-both dur

61 cultures by co-culturing with astrocytes and oligodendrocyte precursor cells (complex culture).

62 NG2-expressing oligodendrocyte precursor cells (NG2 cells) are exposed

63 Synaptic signaling to NG2-expressing oligodendrocyte precursor cells (NG2 cells) could be key

64 promotes myelination and differentiation of oligodendrocyte precursor cells (NG2(+) cells) in a deco

65 adult CNS contains an abundant population of oligodendrocyte precursor cells (NG2(+) cells) that gene

66 bipotential oligodendrocyte-type-2 astrocyte/oligodendrocyte precursor cells (O-2A/OPCs).

67 ansmembrane proteoglycan NG2 is expressed by oligodendrocyte precursor cells (OPC), which migrate to

68 in reminiscent of neural stem cells (NSC) or oligodendrocyte precursor cells (OPC).

69 bsequent validation in both murine and human oligodendrocyte precursor cells (OPCs) and coculture sys

70 ovide an additional source of human cortical oligodendrocyte precursor cells (OPCs) and define a line

71 igration of both rat Schwann cells (SCs) and oligodendrocyte precursor cells (OPCs) and explored the

72 yelin is dependent on the differentiation of oligodendrocyte precursor cells (OPCs) and oligodendrocy

73 type 1 to type 2 status, elevated numbers of oligodendrocyte precursor cells (OPCs) and oligodendrocy

74 d (RA) signaling promotes differentiation of oligodendrocyte precursor cells (OPCs) and remyelination

75 Oligodendrocyte precursor cells (OPCs) are abundant in t

76 Oligodendrocyte precursor cells (OPCs) are generated fro

77 Oligodendrocyte precursor cells (OPCs) are lineage-restr

78 Other studies showed that oligodendrocyte precursor cells (OPCs) are responsible f

79 ventral spinal cord, motor neurons (MNs) and oligodendrocyte precursor cells (OPCs) are sequentially

80 Oligodendrocyte precursor cells (OPCs) are the major sou

81 Oligodendrocyte precursor cells (OPCs) are thought to ma

82 We and others have shown that oligodendrocyte precursor cells (OPCs) can also be the c

83 , it was demonstrated that lineage-committed oligodendrocyte precursor cells (OPCs) can be converted

84 Oligodendrocyte precursor cells (OPCs) can be differenti

85 We showed previously that purified rat oligodendrocyte precursor cells (OPCs) can be induced by

86 We previously showed that oligodendrocyte precursor cells (OPCs) can be transforme

87 hereas induced expression of Nkx2.2 in early oligodendrocyte precursor cells (OPCs) causes precocious

88 We report that oligodendrocyte precursor cells (OPCs) contact sprouting

89 The appearance of oligodendrocyte precursor cells (OPCs) correlates with t

90 and transplantation of adult rat spinal cord oligodendrocyte precursor cells (OPCs) could enhance rem

91 Remyelination may fail because oligodendrocyte precursor cells (OPCs) do not completely

92 ll-intrinsic timer helps control when rodent oligodendrocyte precursor cells (OPCs) exit the cell cyc

93 In the mammalian CNS, oligodendrocyte precursor cells (OPCs) express most neur

94 Oligodendrocyte precursor cells (OPCs) express NMDA rece

95 Oligodendrocyte precursor cells (OPCs) express receptors

96 During differentiation, oligodendrocyte precursor cells (OPCs) extend a network

97 ansplantation of neural stem cells (NSCs) or oligodendrocyte precursor cells (OPCs) has been used to

98 Oligodendrocyte precursor cells (OPCs) have extraordinar

99 We find perivascular clustering of oligodendrocyte precursor cells (OPCs) in certain active

100 t hypoxia activates the ISR in primary mouse oligodendrocyte precursor cells (OPCs) in vitro and that

101 lly identified as a proliferative signal for oligodendrocyte precursor cells (OPCs) in vitro.

102 Jagged signalling via Notch receptors on oligodendrocyte precursor cells (OPCs) inhibits their di

103 Transplantation of oligodendrocyte precursor cells (OPCs) is a promising po

104 Adult oligodendrocyte precursor cells (OPCs) make up around 5-

105 rocytes are initially specified, after which oligodendrocyte precursor cells (OPCs) migrate and proli

106 cultured and exposed to media conditioned by oligodendrocyte precursor cells (OPCs) or differentiated

107 Oligodendrocyte precursor cells (OPCs) persist in substa

108 Addition of anacardic acid to cultured oligodendrocyte precursor cells (OPCs) rapidly increased

109 They differentiate from oligodendrocyte precursor cells (OPCs) that are produced

110 the central nervous system and develop from oligodendrocyte precursor cells (OPCs) that must first m

111 le explanation is the inability of recruited oligodendrocyte precursor cells (OPCs) to complete remye

112 f which represent a continuum from Pdgfra(+) oligodendrocyte precursor cells (OPCs) to distinct matur

113 Sox8 is known to be expressed in oligodendrocyte precursor cells (OPCs) together with oth

114 udy, we found that, in injured optic nerves, oligodendrocyte precursor cells (OPCs) undergo transient

115 e absence of Gsx2 expression, an increase in oligodendrocyte precursor cells (OPCs) with a concomitan

116 Oligodendrocyte precursor cells (OPCs), a major glial ce

117 yeloid cells, meningeal cells, proliferating oligodendrocyte precursor cells (OPCs), and a dense extr

118 n deep layer excitatory neurons and immature oligodendrocyte precursor cells (OPCs), and these contri

119 aberrant growth prior to malignancy only in oligodendrocyte precursor cells (OPCs), but not in any o

120 Neurons form bona fide synapses with oligodendrocyte precursor cells (OPCs), but the circuit

121 ive AMPARs by recording from rat optic nerve oligodendrocyte precursor cells (OPCs), known to express

122 endogenous remyelination, driven by resident oligodendrocyte precursor cells (OPCs), might partially

123 They develop from oligodendrocyte precursor cells (OPCs), most of which di

124 us examined the functional roles of CSPGs on oligodendrocyte precursor cells (OPCs), oligodendrocytes

125 xclusively expressed in oligodendrocytes and oligodendrocyte precursor cells (OPCs), which migrate co

126 nhibited the differentiation of purified rat oligodendrocyte precursor cells (OPCs).

127 itors sequentially produce motor neurons and oligodendrocyte precursor cells (OPCs).

128 wild-type controls, as was proliferation of oligodendrocyte precursor cells (OPCs).

129 -length LINGO-1 inhibited differentiation of oligodendrocyte precursor cells (OPCs).

130 ression and function of REST in neonatal rat oligodendrocyte precursor cells (OPCs).

131 e a novel role in white matter by modulating oligodendrocyte precursor cells (OPCs).

132 demyelination, as were numbers of CXCR4+NG2+ oligodendrocyte precursor cells (OPCs).

133 upport the survival and proliferation of rat oligodendrocyte precursor cells (OPCs).

134 ation program in existing or newly recruited oligodendrocyte precursor cells (OPCs).

135 ta receptors may mediate the effect of TH on oligodendrocyte precursor cells (OPCs).

136 lso expressed by some glial cells, including oligodendrocyte precursor cells (OPCs).

137 ial lineage selection, expanding the pool of oligodendrocyte precursor cells (OPCs).

138 re postmitotic and derive from proliferative oligodendrocyte precursor cells (OPCs).

139 of functionally mature oligodendrocytes from oligodendrocyte precursor cells (OPCs).

140 factor-2, a guidance factor for migration of oligodendrocyte precursor cells (OPCs).

141 al differentiation-promoting effect of TH on oligodendrocyte precursor cells (OPCs): unlike wild-type

142 Oligodendrocyte precursor cells (OPCs; PDGFRalpha+) prod

143 ral spinal OLIG2-expressing progenitors, pre-oligodendrocyte precursor cells (pre-OPCs) and OPCs from

144 c MS lesions and that Notch1 is activated in oligodendrocyte precursor cells (see the related article

145 1 was significantly decreased in spinal cord oligodendrocyte precursor cells after onset of EAE, and

146 reduced remyelination, and increased loss of oligodendrocyte precursor cells and mature oligodendrocy

147 ances in our understanding of the biology of oligodendrocyte precursor cells and of the stage-depende

148 mbination with MCSF, increased the number of oligodendrocyte precursor cells and promoted remyelinati

149 In vitro and in vivo experiments in oligodendrocyte precursor cells and zebrafish were perfo

150 ation vulnerable to PVWMI and P5 when rodent oligodendrocyte precursor cells are more vulnerable to e

151 Oligodendrocyte precursor cells are the primary source o

152 al targets and cellular process expansion by oligodendrocyte precursor cells as well as expression an

153 s indicated 4-AP stabilization of myelin and oligodendrocyte precursor cells associated with increase

154 termine a window of opportunity during which oligodendrocyte precursor cells can successfully differe

155 pression of the helix-loop-helix gene Id4 in oligodendrocyte precursor cells decreases in vivo and in

156 Oligodendrocyte precursor cells differentiate into matur

157 We also show that oligodendrocyte precursor cells display sensitivity to t

158 -1, a secreted protein that repels migrating oligodendrocyte precursor cells during neural developmen

159 In mice that lacked RXR-gamma, adult oligodendrocyte precursor cells efficiently repopulated

160 Motor neuron and oligodendrocyte precursor cells express Olig genes, whic

161 hibiting myelination by deletion of Olig2 in oligodendrocyte precursor cells impairs spatial memory i

162 The alpha1B-AR is also expressed in NG2 oligodendrocyte precursor cells in both neonatal cell cu

163 Gold nanocrystal treatment of oligodendrocyte precursor cells in culture resulted in o

164 ) and retinoic acid (RA) induce purified rat oligodendrocyte precursor cells in culture to stop divis

165 nterestingly, despite a normal production of oligodendrocyte precursor cells in the double mutants, o

166 ast to multiple observations indicating that oligodendrocyte precursor cells in the embryonic day 14

167 ne fumarate can stimulate differentiation of oligodendrocyte precursor cells in vitro, in animal mode

168 usly differentiating OLs generated from pure oligodendrocyte precursor cells in vitro.

169 During development, oligodendrocyte precursor cells integrate environmental

170 this compound induces the differentiation of oligodendrocyte precursor cells into mature oligodendroc

171 cyte-derived TIMP-1 drove differentiation of oligodendrocyte precursor cells into mature oligodendroc

172 factors that inhibit the differentiation of oligodendrocyte precursor cells into myelinating oligode

173 Moreover, proliferation of oligodendrocyte precursor cells is altered by mutant hun

174 An intracellular timer in oligodendrocyte precursor cells is thought to help contr

175 udies demonstrated that the proliferation of oligodendrocyte precursor cells isolated from the develo

176 Oligodendrocyte precursor cells may differentiate into a

177 Seeding of oligodendrocyte precursor cells on these axons results i

178 NG2 cells, also known as oligodendrocyte precursor cells or polydendrocytes, whic

179 r glial fibrillary acidic protein (GFAP) and oligodendrocyte precursor cells positive for NG2 proteog

180 Here we show that most oligodendrocyte precursor cells purified from postnatal

181 nt mode of inheritance, in vitro analysis in oligodendrocyte precursor cells showed that mutant prote

182 art of the normal timer that determines when oligodendrocyte precursor cells stop dividing and differ

183 ived mediators influenced differentiation of oligodendrocyte precursor cells through a crosstalk with

184 glia/macrophage to lesions nor a failure for oligodendrocyte precursor cells to differentiate and rem

185 itical in regulating the transition of adult oligodendrocyte precursor cells to mature OLs that is es

186 e, we demonstrate that fear learning induces oligodendrocyte precursor cells to proliferate and diffe

187 hat certain extracellular signals can induce oligodendrocyte precursor cells to revert to multipotent

188 possible reason is the lack of migration of oligodendrocyte precursor cells to the lesion.

189 NLGN3 is cleaved from both neurons and oligodendrocyte precursor cells via the ADAM10 sheddase.

190 Migration and density of oligodendrocyte precursor cells were normal; however, a

191 ntracellular timer that helps determine when oligodendrocyte precursor cells withdraw from the cell c

192 efault of the resident stem/precursor cells (oligodendrocyte precursor cells) to differentiate into m

193 s system remyelination is mainly mediated by oligodendrocyte precursor cells, although subventricular

194 matter, apoptosis and arrested maturation of oligodendrocyte precursor cells, and hypomyelination.

195 ally infected neural stem cells, astrocytes, oligodendrocyte precursor cells, and microglia, whereas

196 ol cells, including normal human astrocytes, oligodendrocyte precursor cells, and primary explant cul

197 resses inflammation, attenuates apoptosis of oligodendrocyte precursor cells, and promotes myelinatio

198 Unlike oligodendrocyte precursor cells, APCs do not differentia

199 her obvious impairment in the recruitment of oligodendrocyte precursor cells, astrocytes, or reactive

200 domains VI and V of netrin-1 repel migrating oligodendrocyte precursor cells, but lack the chemoattra

201 We show that TNFR2 drives differentiation of oligodendrocyte precursor cells, but not proliferation o

202 nes, chemokines and growth factors, act upon oligodendrocyte precursor cells, causing their activatio

203 of origin for glioma, neural stem cells and oligodendrocyte precursor cells, exhibited a high glioma

204 y lethality, effects on myelination, loss of oligodendrocyte precursor cells, increased apoptosis in

205 changed the GABA-response characteristics in oligodendrocyte precursor cells, indicating their partic

206 In contrast to oligodendrocyte precursor cells, microglia formed only w

207 entative populations of neurons, astrocytes, oligodendrocyte precursor cells, newly formed oligodendr

208 g the muscarinic acetylcholine receptor 1 in oligodendrocyte precursor cells, or promoting oligodendr

209 G2(+) glia, also known as polydendrocytes or oligodendrocyte precursor cells, represent a new entity

210 eduction of mature oligodendrocytes, but not oligodendrocyte precursor cells, suggesting triglial dys

211 erating the correct numbers of WM but not GM oligodendrocyte precursor cells, whereas during astrocyt

212 ransformation following differentiation into oligodendrocyte precursor cells.

213 ls in the postnatal SVZ but are likely to be oligodendrocyte precursor cells.

214 proliferation and inhibits the migration of oligodendrocyte precursor cells.

215 eptor thus serves as a phenotypic marker for oligodendrocyte precursor cells.

216 e they likely exert their influence on early oligodendrocyte precursor cells.

217 y neurons as well as in oligodendrocytes and oligodendrocyte precursor cells.

218 , and decline of mature oligodendrocytes and oligodendrocyte precursor cells.

219 ues extends this active role by showing that oligodendrocyte precursors cells (OPCs) in the hippocamp

220 the proliferation of Sox2 stem cells and NG2 oligodendrocyte precursors cells originating in the SVZ

221 Human oligodendrocytes precursor cells (OPCs) were stimulated

222 on of in vitro analyses, we demonstrate that oligodendrocyte precursors closely regulate their number

223 n the affected brains may be process-bearing oligodendrocyte precursors containing unsulfated GC or a

224 tion causes a tumor phenotypic shift from an oligodendrocyte precursor-correlated proneural toward an

225 nally, blockage of these miRNA activities in oligodendrocyte precursor culture and knockdown of miR-2

226 bB2 is not necessary for the early stages of oligodendrocyte precursor development, but is essential

227 as specific to Schwann cells, as deletion in oligodendrocyte precursors did not impair myelin formati

228 This decrease may help control when oligodendrocyte precursors differentiate.

229 tes both cell proliferation and migration in oligodendrocyte precursors during development.

230 eath in mice, despite an initial increase of oligodendrocyte precursors during early development.

231 essed in zones of neuroepithelium from which oligodendrocyte precursors emerge, as well as in the pre

232 The inhibition of oligodendrocyte precursor emergence in the absence of Sh

233 essed at the ventral ventricular zone during oligodendrocyte precursors emigration, and, in vitro, ne

234 his density-dependent feedback inhibition of oligodendrocyte precursor expansion may play a primary r

235 Oligodendrocyte precursors express the netrin receptors

236 in the spinal cord of netrin-1 mutant mice, oligodendrocyte precursors failed to disperse from the v

237 loping chick neural tube, Zfp488 can promote oligodendrocyte precursor formation upon Notch activatio

238 of Cell Stem Cell, Piao et al. (2015) derive oligodendrocyte precursors from human embryonic stem cel

239 These data indicate the initial dispersal of oligodendrocyte precursors from their localized origin i

240 distinct types of morphologically identical oligodendrocyte precursor glial cells (OPCs) in situ in

241 tion of Shh signaling after the emergence of oligodendrocyte precursors had no effect on the appearan

242 While oligodendrocyte precursors have previously been localize

243 In contrast, Ascl1 expressing oligodendrocyte precursors in gray matter already coexpr

244 t a dose-dependent increase in the number of oligodendrocyte precursors in response to Shh.

245 Ascl1 also broadly marks oligodendrocyte precursors in subcortical gray and white

246 tion factor, transiently labels neuronal and oligodendrocyte precursors in the adult brain.

247 ping studies that cast NG2-glia as dedicated oligodendrocyte precursors in the healthy adult CNS-thou

248 These "NG2 cells" descend from oligodendrocyte precursors in the perinatal CNS and cont

249 lling evidence for a second dorsal origin of oligodendrocyte precursors in the spinal cord and hindbr

250 significant reduction in the number of p27+ oligodendrocyte precursors in the transgenic mice suppor

251 Myocilin also affects differentiation of oligodendrocyte precursors in vitro.

252 ded with global lower neuron densities, less oligodendrocyte precursors, increased apoptosis and less

253 ition to primary cultures of differentiating oligodendrocyte precursors increases levels of tested ma

254 Oligodendrocyte precursors initially arise in restricted

255 Oligodendrocyte precursors injected into presumptive whi

256 at population of presumptive white matter by oligodendrocyte precursors is dependent on localized exp

257 that spontaneous myelin repair by endogenous oligodendrocyte precursors is much more robust than prev

258 What regulates this initial localization of oligodendrocyte precursors is unclear.

259 euroblasts, but DCX(+) cells coexpressed the oligodendrocyte precursor marker Olig2, suggesting cauti

260 During development oligodendrocyte precursors mature through a series of st

261 gram of cell proliferation during which many oligodendrocyte precursors, microglia, and some astrocyt

262 In the absence of viable axons, oligodendrocyte precursors migrated along the length of

263 Knockout mice show increased rates of oligodendrocyte precursor migration along the optic nerv

264 C antibody or netrin 1 dramatically inhibits oligodendrocyte precursor migration from the ventral ven

265 lice preparations, CXCL1 inhibited embryonic oligodendrocyte precursor migration, and widespread disp

266 erosis lesions, have the capacity to inhibit oligodendrocyte precursor migration, identifying netrin-

267 Signaling through CXCR2, CXCL1 inhibited oligodendrocyte precursor migration.

268 merits of the GRP cell vs. the motor neuron-oligodendrocyte precursor (MNOP) cell hypothesis.

269 h nondifferentiated and differentiated mouse oligodendrocyte precursor (mOP) cells in vitro.

270 e of demyelinated lesions is the presence of oligodendrocyte precursors (OLPs) blocked at a premyelin

271 They are descended from oligodendrocyte precursors (OLPs) in the perinatal CNS,

272 neurons (MNs), before switching abruptly to oligodendrocyte precursors (OLPs).

273 surface binding of IgG or IgM antibodies to oligodendrocyte precursor (OPC)-derived cell lines was s

274 y results in defects in the morphogenesis of oligodendrocyte precursors (OPCs) and CNS hypomyelinatio

275 During development, spinal cord oligodendrocyte precursors (OPCs) originate from the ven

276 expressed by motor neurons postnatally, and oligodendrocyte precursors (OPCs), as previously reporte

277 Oligodendrocyte precursors (OPs) continue to proliferate

278 sequentially generate motoneurons (MNs) and oligodendrocyte precursors (OPs).

279 mitotic oligodendrocytes but is not found in oligodendrocyte precursors or astrocytes.

280 In the spinal cord, oligodendrocyte precursors originate at the ventral midl

281 Neuronal (TUBB3) and oligodendrocyte precursor (PLP) markers were down-regula

282 Oligodendrocyte precursors (pre-OLs; O4(+)O1(-)) predomi

283 secretion of Inhibin A and downregulation of oligodendrocyte precursor production of Matrilin-2 limit

284 , supplemented cholesterol directly supports oligodendrocyte precursor proliferation and differentiat

285 hown previously to promote remyelination and oligodendrocyte precursor proliferation in a murine mode

286 n along the optic nerve and reduced rates of oligodendrocyte precursor proliferation in different reg

287 ne-alpha (GRO-alpha) is a potent promoter of oligodendrocyte precursor proliferation.

288 In purified cultures of oligodendrocyte precursors, Shh promotes cell survival a

289 The addition of large numbers of oligodendrocyte precursors substantially reduces precurs

290 differentiation of NCAM- or ST8SIA2-negative oligodendrocyte precursors suggested an underlying cell-

291 osum, Ascl1 defines a ventral layer of early oligodendrocyte precursors that do not yet express other

292 In spinal cord, oligodendrocyte precursors that give rise to myelin-form

293 Following Brg1 deletion in oligodendrocyte precursors, these cells showed normal su

294 responsible for the localized appearance of oligodendrocyte precursors throughout the CNS, irrespect

295 oliferative response of immature spinal cord oligodendrocyte precursors to their major mitogen, plate

296 For example, mammalian oligodendrocyte precursors typically proliferate for app

297 perimental animals the appearance of ectopic oligodendrocyte precursors was correlated with local flo

298 ing in the early appearance of metencephalic oligodendrocyte precursors, while in vitro studies sugge

299 dicate that NG2+ cells in the normal CNS are oligodendrocyte precursors with restricted lineage poten

300 nately controlled biophysical interaction of oligodendrocyte precursors within an axonal niche leadin