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1 ssion from Sox2(+) 'early' to Sox2(-) 'late' oligodendrocyte progenitor.
2 sm involving crosstalk between microglia and oligodendrocyte progenitors.
3 ick and mouse models and in vitro culture of oligodendrocyte progenitors.
4 uted population of glial cells that serve as oligodendrocyte progenitors.
5 f4, plays an important role in maturation of oligodendrocyte progenitors.
6 ntiated mitosis in primary cultures of mouse oligodendrocyte progenitors.
7 , and CC1-, indicated a close resemblance to oligodendrocyte progenitors.
8 presence of a susceptible population of late oligodendrocyte progenitors.
9 els, but we could detect the alpha(1A)-AR in oligodendrocyte progenitors.
10 sed markers for astrocytes and for neural or oligodendrocyte progenitors.
11  cerebral white matter and targeted death of oligodendrocyte progenitors.
12 es the expression of myelin basic protein in oligodendrocyte progenitors.
13 s that the ectopic astrocytes originate from oligodendrocyte progenitors.
14 dendrocyte development was delayed such that oligodendrocyte progenitors accumulated inappropriately
15 results showing defective differentiation of oligodendrocyte progenitors after silencing specific HDA
16 evel, lack of yy1 arrests differentiation of oligodendrocyte progenitors after they exit from the cel
17                             Furthermore, the oligodendrocyte progenitors also become progressively de
18                                 Depletion of oligodendrocyte progenitors and demyelination are major
19 , revealed that it is similarly expressed in oligodendrocyte progenitors and not oligodendrocytes.
20 sis using gene expression profiling of A2B5+ oligodendrocyte progenitors and O4+ oligodendrocytes.
21 ndently inhibited the maturation of purified oligodendrocyte progenitors, and pharmacological inhibit
22 Immunohistochemical studies showed that late oligodendrocyte progenitors appear at gestational age 22
23 n of neural progenitors and specification of oligodendrocyte progenitors are completed with the forma
24 ignificance statement: It is recognized that oligodendrocyte progenitors are mechanosensitive cells.
25 hibition, we demonstrate that specified NG2+ oligodendrocyte progenitors are plastic cells, whose dec
26 ed in apoptosis and reduced proliferation of oligodendrocyte progenitors, as well as arrested maturat
27  cytotoxic effects on actively proliferating oligodendrocyte progenitors but much less on immature ol
28                         NFIA is expressed in oligodendrocyte progenitors, but not differentiated olig
29                            Moreover, EGFR(+) oligodendrocyte progenitors, but not neuroblasts, expres
30                In the adult mammalian brain, oligodendrocyte progenitors can differentiate into matur
31                  Generating patient-specific oligodendrocyte progenitors capable of repairing myelina
32                          Recently, we showed oligodendrocyte progenitor cell (OPC) accumulation and r
33  Ca(2+) channels and receptors that regulate oligodendrocyte progenitor cell (OPC) and oligodendrocyt
34                                           In oligodendrocyte progenitor cell (OPC) cultures, IL-11 re
35 d Ca(2+) channels (L-VOCCs) are required for oligodendrocyte progenitor cell (OPC) development, we ge
36 n by acting at several critical steps during oligodendrocyte progenitor cell (OPC) development.
37                   The effect of olesoxime on oligodendrocyte progenitor cell (OPC) differentiation an
38 d increases both adult mouse and adult human oligodendrocyte progenitor cell (OPC) differentiation, i
39 s is enough to restrict them to an exclusive oligodendrocyte progenitor cell (OPC) fate during differ
40 he ventral telencephalon were generated, but oligodendrocyte progenitor cell (OPC) generation was sev
41 se (MAPK)-dependent pathway is implicated in oligodendrocyte progenitor cell (OPC) lineage progressio
42 re infants results in inflammation, arrested oligodendrocyte progenitor cell (OPC) maturation, and re
43  multiple sclerosis (PPMS) failed to promote oligodendrocyte progenitor cell (OPC) maturation, wherea
44 ibute to remyelination failure by perturbing oligodendrocyte progenitor cell (OPC) maturation.
45              This work describes the role of oligodendrocyte progenitor cell (OPC) microRNAs (miRNAs)
46 two nf1 orthologs in zebrafish and show that oligodendrocyte progenitor cell (OPC) numbers are increa
47                    Oligodendrocyte death and oligodendrocyte progenitor cell (OPC) proliferation duri
48         We have previously demonstrated that oligodendrocyte progenitor cell (OPC) proliferation is c
49                                        Human oligodendrocyte progenitor cell (OPC) specification and
50                                         Both oligodendrocyte progenitor cell commitment and oligodend
51 strates a unique role for Olig1 in promoting oligodendrocyte progenitor cell commitment, differentiat
52 mportant roles for Sox17 in controlling both oligodendrocyte progenitor cell cycle exit and different
53 nal LB supplementation promoted neuronal and oligodendrocyte progenitor cell development.
54 elevant, FDA-approved compounds that promote oligodendrocyte progenitor cell differentiation and indu
55 CN3 is a matricellular protein that promotes oligodendrocyte progenitor cell differentiation and myel
56                       Treg directly promoted oligodendrocyte progenitor cell differentiation and myel
57 endrocyte lineage cells completely inhibited oligodendrocyte progenitor cell differentiation and myel
58 tory supernatants also resulted in decreased oligodendrocyte progenitor cell differentiation without
59  also explore the effect of these changes on oligodendrocyte progenitor cell differentiation, which i
60  myelin debris, which contains inhibitors of oligodendrocyte progenitor cell differentiation.
61 d neither a direct nor an indirect impact on oligodendrocyte progenitor cell differentiation.
62 enhancer (PIKE) expression by shRNA prevents oligodendrocyte progenitor cell differentiation.
63 ess and promotes precocious neurogenesis and oligodendrocyte progenitor cell elaboration.
64 process formation were also inhibited in the oligodendrocyte progenitor cell line CG-4 after suppress
65 of transplanted cells co-labeled for NG2, an oligodendrocyte progenitor cell marker, but not for neur
66 urons and astrocytes remains the same, early oligodendrocyte progenitor cell markers are decreased in
67                As shown recently, failure in oligodendrocyte progenitor cell maturation contributes t
68                                              Oligodendrocyte progenitor cell number decreased with ag
69 signaling increases neurogenesis and reduces oligodendrocyte progenitor cell proliferation (OPC) in t
70 infiltrates and demyelination, and increased oligodendrocyte progenitor cell proliferation and BDNF+
71                                              Oligodendrocyte progenitor cell proliferation was observ
72 gh combined doses of oral antibiotics impair oligodendrocyte progenitor cell responses during remyeli
73 /or indirectly (via astrocytes) impact human oligodendrocyte progenitor cell survival and differentia
74  enriched population of cells expressing the oligodendrocyte progenitor cell-specific marker NG2.
75 NS cells expressing A2B5, an early marker in oligodendrocytes progenitor cell differentiation as well
76 se embryonic and lung fibroblasts to induced oligodendrocyte progenitor cells (iOPCs) using sets of e
77 al and mesenchymal glioblastoma, relative to oligodendrocyte progenitor cells (Oli-Neu).
78 e found that AXIN2 was expressed in immature oligodendrocyte progenitor cells (OLPs) in white matter
79 erived growth factor receptor alpha-positive oligodendrocyte progenitor cells (OPC) located within th
80 ntiate human embryonic stem cells (hESCs) to oligodendrocyte progenitor cells (OPCs) according to dev
81 nsequences of disrupting Fth iron storage in oligodendrocyte progenitor cells (OPCs) after demyelinat
82 se genes was measured in primary neurons and oligodendrocyte progenitor cells (OPCs) after inflammato
83                       Microarray analysis in oligodendrocyte progenitor cells (OPCs) after Sox17 atte
84                 AMPA stimulation of cultured oligodendrocyte progenitor cells (OPCs) also caused an i
85 in vitro primary rat embryonic cell model of oligodendrocyte progenitor cells (OPCs) and a mouse N20.
86 hogenetic protein (BMP) signaling pathway in oligodendrocyte progenitor cells (OPCs) and suppresses r
87  expression in the developing CNS identifies oligodendrocyte progenitor cells (OPCs) and whose activa
88 , both the proliferation and total number of oligodendrocyte progenitor cells (OPCs) appeared normal
89 ination and why remyelination is absent when oligodendrocyte progenitor cells (OPCs) are present.
90                      In the postnatal brain, oligodendrocyte progenitor cells (OPCs) arise from the s
91 termination of proliferation determines when oligodendrocyte progenitor cells (OPCs) can initiate dif
92                                              Oligodendrocyte progenitor cells (OPCs) can repair demye
93                             Remyelination by oligodendrocyte progenitor cells (OPCs) can restore thes
94 ular mechanisms that drive the maturation of oligodendrocyte progenitor cells (OPCs) during the remye
95 l myelinating glial cells, centrally derived oligodendrocyte progenitor cells (OPCs) ectopically exit
96 fish, we observed that prior to myelination, oligodendrocyte progenitor cells (OPCs) extend processes
97 factor-1 (IGF-1) provides neuroprotection to oligodendrocyte progenitor cells (OPCs) following cerebr
98 y also be a radial component of migration of oligodendrocyte progenitor cells (OPCs) from a ventral s
99                                 Migration of oligodendrocyte progenitor cells (OPCs) from proliferati
100  mice also exhibited an increased density of oligodendrocyte progenitor cells (OPCs) in CNS white mat
101                                              Oligodendrocyte progenitor cells (OPCs) in demyelinated
102 e show that the expression of Sox2 occurs in oligodendrocyte progenitor cells (OPCs) in rodent models
103 fficulties in generating pure populations of oligodendrocyte progenitor cells (OPCs) in sufficient qu
104 ion, and may contribute to the production of oligodendrocyte progenitor cells (OPCs) in the dorsal co
105                                              Oligodendrocyte progenitor cells (OPCs) in the postnatal
106  of human embryonic stem cell (hESC)-derived oligodendrocyte progenitor cells (OPCs) into adult rat s
107                           Differentiation of oligodendrocyte progenitor cells (OPCs) into mature olig
108 n obligatory step for the differentiation of oligodendrocyte progenitor cells (OPCs) into myelinating
109 in disorders can be treated by transplanting oligodendrocyte progenitor cells (OPCs) into the affecte
110            Differentiation and maturation of oligodendrocyte progenitor cells (OPCs) involve the asse
111 ted failure to produce oligodendrocytes from oligodendrocyte progenitor cells (OPCs) is associated wi
112 cal changes, while in developing neurons and oligodendrocyte progenitor cells (OPCs) it induces cellu
113 lutamatergic synapses onto adult-born NG2(+) oligodendrocyte progenitor cells (OPCs) migrating from t
114 t oligodendrocytes, whether by transplanting oligodendrocyte progenitor cells (OPCs) or by mobilizing
115 n which Tsc1 is deleted by Cre expression in oligodendrocyte progenitor cells (OPCs) or in premyelina
116                      Neonatal engraftment by oligodendrocyte progenitor cells (OPCs) permits the myel
117                                              Oligodendrocyte progenitor cells (OPCs) persist in human
118             We show how Sox2 is expressed in oligodendrocyte progenitor cells (OPCs) preparing to und
119 ination of the central nervous system (CNS), oligodendrocyte progenitor cells (OPCs) proliferate and
120                      In experimental models, oligodendrocyte progenitor cells (OPCs) rather than prev
121                                              Oligodendrocyte progenitor cells (OPCs) recruited to dem
122                                              Oligodendrocyte progenitor cells (OPCs) that are positiv
123      White matter stroke stimulates adjacent oligodendrocyte progenitor cells (OPCs) to divide and mi
124 isoprenoid and cholesterol synthesis, causes oligodendrocyte progenitor cells (OPCs) to migrate past
125 er, direct injection of neural stem cells or oligodendrocyte progenitor cells (OPCs) to the lesion si
126  in which we targeted Notch1 inactivation to oligodendrocyte progenitor cells (OPCs) using Olig1Cre a
127 ation by blocking the differentiation of rat oligodendrocyte progenitor cells (OPCs) via modulation o
128   Many chronically demyelinated lesions have oligodendrocyte progenitor cells (OPCs) within their bor
129 system (CNS) multipotent stem cells known as oligodendrocyte progenitor cells (OPCs)(2).
130  and support cytocompatible encapsulation of oligodendrocyte progenitor cells (OPCs), as well as thei
131 mong three macroglial progenitor populations-oligodendrocyte progenitor cells (OPCs), astrocytes and
132  plaque-associated Olig2- and NG2-expressing oligodendrocyte progenitor cells (OPCs), but not astrocy
133 rect effects on the myelinating potential of oligodendrocyte progenitor cells (OPCs), in addition to
134                                           In oligodendrocyte progenitor cells (OPCs), Lrp1 is require
135                        By leveraging primary oligodendrocyte progenitor cells (OPCs), microglia-deple
136                                  In cultured oligodendrocyte progenitor cells (OPCs), Sox17 levels we
137 tly characterized by scarce undifferentiated oligodendrocyte progenitor cells (OPCs), suggesting the
138 gene expression profiling on purified murine oligodendrocyte progenitor cells (OPCs), the remyelinati
139 rve conduction, and the ectopic migration of oligodendrocyte progenitor cells (OPCs), the resident my
140 rotein fibronectin perturb the maturation of oligodendrocyte progenitor cells (OPCs), thereby impedin
141 oliferation is not altered in Seh1-deficient oligodendrocyte progenitor cells (OPCs), they fail to di
142        Since one role of NG2 glia is that of oligodendrocyte progenitor cells (OPCs), we investigated
143                                              Oligodendrocyte progenitor cells (OPCs), which different
144  cells produces first motor neurons and then oligodendrocyte progenitor cells (OPCs), which migrate,
145 and generate de novo synapses with recruited oligodendrocyte progenitor cells (OPCs), which, early af
146 ation, proliferation, and differentiation of oligodendrocyte progenitor cells (OPCs).
147  the autocrine Wnt/beta-catenin signaling in oligodendrocyte progenitor cells (OPCs).
148  most PLP-EGFP-expressing cells gave rise to oligodendrocyte progenitor cells (OPCs).
149  the generation of new oligodendrocytes from oligodendrocyte progenitor cells (OPCs).
150 ocytes and with a reduction in proliferating oligodendrocyte progenitor cells (OPCs).
151           CSPG4/NG2 is a hallmark protein of oligodendrocyte progenitor cells (OPCs).
152  modulator of intracellular Ca(2+) levels in oligodendrocyte progenitor cells (OPCs).
153 lay a fundamental role in the development of oligodendrocyte progenitor cells (OPCs).
154 glial-restricted progenitors cells, known as oligodendrocyte progenitor cells (OPCs).
155 cytes arise from migratory and proliferative oligodendrocyte progenitor cells (OPCs).
156 ult human forebrain contain large numbers of oligodendrocyte progenitor cells (OPCs).
157 h activity could promote formation of excess oligodendrocyte progenitor cells (OPCs).
158 emyelination by impairing differentiation of oligodendrocyte progenitor cells (OPCs).
159  myelin and myelin repair by differentiating oligodendrocyte progenitor cells (OPCs).
160 iated neuroinflammation, and an expansion of oligodendrocyte progenitor cells (OPCs).
161 liferation, migration and differentiation of oligodendrocyte progenitor cells (OPCs).
162 ig1-Cre-expressing cells reduces the pool of oligodendrocyte progenitor cells (OPCs).
163 endent on recruitment and differentiation of oligodendrocyte progenitor cells (OPCs).
164                  Genetic deletion of TACE in oligodendrocyte progenitor cells (OPs) induces premature
165                                  Parenchymal oligodendrocyte progenitor cells (pOPCs) are considered
166  of betaT4-positive cells with A2B5-positive oligodendrocyte progenitor cells after transplantation (
167 peroxia showed a reduced capacity to protect oligodendrocyte progenitor cells against the toxic effec
168 e of the TUJ-1-positive cells, A2B5-positive oligodendrocyte progenitor cells and A2B5-negative cells
169  by an expression pattern resembling that of oligodendrocyte progenitor cells and carries a distincti
170                             Both parenchymal oligodendrocyte progenitor cells and endogenous adult ne
171                 Moreover, we showed that CNS oligodendrocyte progenitor cells are activated following
172         In the developing spinal cord, early oligodendrocyte progenitor cells are induced from the ve
173                                              Oligodendrocyte progenitor cells are stem cells in the c
174 ation that human embryonic stem cell-derived oligodendrocyte progenitor cells are susceptible to JC v
175 nical translation: first, transplantation of oligodendrocyte progenitor cells as a means of treating
176 vides direct evidence that targeting EGFR in oligodendrocyte progenitor cells at a specific time afte
177       However, myosin-1d was undetectable in oligodendrocyte progenitor cells at early and late time
178 pression of syntaxin 4 but not syntaxin 3 in oligodendrocyte progenitor cells but not immature oligod
179       PIKE-L expression is up-regulated when oligodendrocyte progenitor cells commit to differentiati
180 and constitutive ablation of NR1 in neonatal oligodendrocyte progenitor cells did not interrupt their
181 ous system (CNS) most often is the result of oligodendrocyte progenitor cells differentiating into my
182                           TACE deficiency in oligodendrocyte progenitor cells following demyelination
183 brinogen inhibits nerve repair by preventing oligodendrocyte progenitor cells from differentiating in
184 cal studies revealed that the recruitment of oligodendrocyte progenitor cells in response to demyelin
185 e fifth major cell population that serves as oligodendrocyte progenitor cells in the postnatal CNS.
186 eration of human oligodendrocytes from human oligodendrocyte progenitor cells in vitro.
187 ly tune axonal diameter, promote re-entry of oligodendrocyte progenitor cells into the cell cycle, or
188  this mild inflammatory environment promotes oligodendrocyte progenitor cells maturation and myelin r
189 on rather than uncontrolled proliferation of oligodendrocyte progenitor cells may have important impl
190 immature oligodendrocyte-lineage cells, with oligodendrocyte progenitor cells more vulnerable to inju
191 In the absence of ERK1/ERK2 signaling NG2(+) oligodendrocyte progenitor cells proliferated and differ
192                           Differentiation of oligodendrocyte progenitor cells requires activation of
193  noted because Nkx2.2 promotes maturation of oligodendrocyte progenitor cells specified by expression
194 w that miconazole and clobetasol function in oligodendrocyte progenitor cells through mitogen-activat
195 ibility of human embryonic stem cell-derived oligodendrocyte progenitor cells to infection with JC vi
196 y changes during the transition from A2B5(+) oligodendrocyte progenitor cells to premyelinating GalC(
197 man adult brain-derived oligodendrocytes and oligodendrocyte progenitor cells under conditions of met
198                 Whereas LINGO-1 expressed by oligodendrocyte progenitor cells was previously identifi
199                                              Oligodendrocyte progenitor cells were not decreased in d
200 rability of O4+ preoligodendrocytes, whereas oligodendrocyte progenitor cells were resistant to insul
201 o explore the mechanism of redistribution of oligodendrocyte progenitor cells with compensatory myeli
202 S immune milieu and concurrent activation of oligodendrocyte progenitor cells with subsequent remyeli
203 ng inflammatory injury, oligodendrocytes and oligodendrocyte progenitor cells within lesion sites are
204 r for cell proliferation), NG2 (a marker for oligodendrocyte progenitor cells) and brain-derived neur
205 56 regulates cortical lamination, whereas in oligodendrocyte progenitor cells, GPR56 controls develop
206 arative capabilities, and transplantation of oligodendrocyte progenitor cells, have generated substan
207 ed to the expansion of genetically wild-type oligodendrocyte progenitor cells, oligodendrocyte differ
208 uced myelin were examined for remyelination, oligodendrocyte progenitor cells, reactive astrocytes, a
209 sis, remyelination can fail despite abundant oligodendrocyte progenitor cells, suggesting impairment
210 d with a truncated proliferative response of oligodendrocyte progenitor cells, suggesting that deplet
211 ds for enhancing myelination from endogenous oligodendrocyte progenitor cells, we screened a library
212 ze the effects of BCNU on clonal cultures of oligodendrocyte progenitor cells-one of the best-charact
213 mouse pluripotent epiblast stem-cell-derived oligodendrocyte progenitor cells.
214 ndent on the survival and differentiation of oligodendrocyte progenitor cells.
215  require technologies to generate functional oligodendrocyte progenitor cells.
216 lates cell cycle exit and differentiation in oligodendrocyte progenitor cells.
217 otential for recovery mediated by endogenous oligodendrocyte progenitor cells.
218 O mice revealed a specific deficit of NG2(+) oligodendrocyte progenitor cells.
219 ns and mice, between reactive astrocytes and oligodendrocyte progenitor cells.
220 ves differentiation of oligodendrocytes from oligodendrocyte progenitor cells.
221 mote production of new oligodendrocytes from oligodendrocyte progenitor cells.
222 tire CNS after they have differentiated from oligodendrocyte progenitor cells.
223 ouse for voltage-operated Ca(2+) channels in oligodendrocyte progenitor cells.
224  compression on the function and survival of oligodendrocyte progenitor cells/oligodendrocytes and ax
225 els to selectively delete TACE expression in oligodendrocyte progenitors cells (OPs), we found that T
226 alpha)-expressing stromal cells derived from oligodendrocytes progenitor cells (OPC) were discovered
227                   Solicitation of endogenous oligodendrocytes progenitor cells, the precursor of olig
228 te and determined whether transplanted adult oligodendrocyte progenitors could remyelinate the chroni
229 found that, in the absence of Schwann cells, oligodendrocyte progenitors cross ventral root transitio
230 tem inflammation as well as promoting neural/oligodendrocyte progenitor development in the offspring.
231 efects in PLP1 mRNA expression and splicing, oligodendrocyte progenitor development, and oligodendroc
232  treatment, and was associated with enhanced oligodendrocyte progenitor differentiation and epigeneti
233  effect of three mechanical stimuli on mouse oligodendrocyte progenitor differentiation and identify
234 est that NFIA participates in the control of oligodendrocyte progenitor differentiation and may contr
235 indicate that NFIA is sufficient to suppress oligodendrocyte progenitor differentiation during adult
236 scription factor nuclear factor IA (NFIA) in oligodendrocyte progenitor differentiation during develo
237 tral nervous system lesions are required for oligodendrocyte progenitor differentiation into remyelin
238  Yin Yang 1 (YY1) as a critical regulator of oligodendrocyte progenitor differentiation.
239        At the cellular level, astrocytes and oligodendrocyte progenitors displayed more differences i
240 l enlargement of the Olig2/Nkx2.2-expressing oligodendrocyte progenitor domain, whereas conditional H
241 so enhanced with precocious neurogenesis and oligodendrocyte progenitor elaboration.
242  Taken together, these data suggest that for oligodendrocyte progenitors, ErbB2 signaling plays a rol
243 ptional network regulating the transition of oligodendrocyte progenitors from cell cycle exit to diff
244 ides a favorable environment for SVZ-derived oligodendrocyte progenitor generation.
245 regions of proneural tumors were enriched in oligodendrocyte progenitor genes, whereas the NE regions
246              We also determined that A2B5(+) oligodendrocyte progenitors grown in conditioned media d
247 which to accelerate differentiation of human oligodendrocyte progenitors (hOPCs) directly, we used CD
248 genitors during brain tumorigenesis, wherein oligodendrocyte-progenitor intermediates are abundant, h
249 of neurons, and may regulate the function of oligodendrocyte progenitors, interneurons, GABA, and NMD
250          The last step of differentiation of oligodendrocyte progenitors into myelin-forming cells is
251 CNS laminin is to promote the development of oligodendrocyte progenitors into myelin-forming oligoden
252  is inhibited by impaired differentiation of oligodendrocyte progenitors into myelin-producing oligod
253                                 Although the oligodendrocyte progenitor is established as the major s
254 transition from multipotential precursors to oligodendrocyte progenitors is associated with the progr
255 genes is abolished, whereas the formation of oligodendrocyte progenitors is not affected.
256 nd Distalless-related homeobox (DLX), or the oligodendrocyte progenitor marker Nkx2.2.
257 droitin sulfate proteoglycan-NG2(+) and late oligodendrocyte progenitor marker(+)), and terminal-diff
258 ssociation with myelin, without reexpressing oligodendrocyte progenitor markers or reentering the cel
259 ly rarely found to be immunoreactive against oligodendrocyte progenitor markers such as NG2 or PDGFRa
260      Myelin regeneration requires endogenous oligodendrocyte progenitor migration and activation of t
261  be a critical molecule in the regulation of oligodendrocyte progenitor migration and myelination.
262 ransfer glycolytic substrates to neurons and oligodendrocyte progenitors (NG2(+) cells) exhibit enhan
263 re population has long been considered to be oligodendrocyte progenitors, numerous NG2(+) cells are p
264  nick end labeling and a marked depletion of oligodendrocyte progenitors of 71 +/- 8%.
265 ng regions overlapping with ventral sites of oligodendrocyte progenitor (OLP) generation.
266                   Klf6 is rapidly induced in oligodendrocyte progenitors (OLP) by gp130 factors, and
267        In mouse neural tube, newly specified oligodendrocyte progenitors (OLPs) are first exposed to
268                             Specification of oligodendrocyte progenitors (OLPs) begins early in devel
269  the transcription factor OLIG2 and generate oligodendrocyte progenitors (OLPs) in culture.
270                  The developmental origin of oligodendrocyte progenitors (OLPs) in the forebrain has
271  (TGFbeta) signaling is crucial for allowing oligodendrocyte progenitor (OP) cell cycle withdrawal, a
272                                              Oligodendrocyte progenitors (OPCs) are electrically resp
273 ite matter progenitors respond to injury, as oligodendrocyte progenitors (OPCs) proliferate.
274  mice with sustained activation of ERK1/2 in oligodendrocyte progenitors (OPCs), oligodendrocytes, an
275 ccumulation in pluripotent-stem-cell-derived oligodendrocyte progenitors (OPCs), we demonstrate that
276                                              Oligodendrocyte progenitors (OPCs), which normally matur
277 nction between subcortical white matter NG2+ oligodendrocyte progenitors (OPs) and O4+ preoligodendro
278 ) expression and survival of differentiating oligodendrocyte progenitors (OPs) in proinflammatory cyt
279 ion of both postmitotic oligodendrocytes and oligodendrocyte progenitors (OPs) is the major hallmark
280 The abundance and widespread distribution of oligodendrocyte progenitors (OPs) within the adult CNS a
281  changes in the proliferation or survival of oligodendrocyte progenitor or mature cells.
282              NG2(+) glial cells (also called oligodendrocyte progenitors or polydendrocytes) also pro
283 rsely, overexpression of MRF within cultured oligodendrocyte progenitors or the chick spinal cord pro
284 ncluding radial glia (GFAP+, vimentin+), and oligodendrocyte progenitors (PDGFR-alpha+) were prolifer
285 e mPFC at P15, leading to a depletion of the oligodendrocyte progenitor pool in MS adults.
286  local proliferation and mobilization of the oligodendrocyte progenitor pool.
287 inating deficits despite an expansion of the oligodendrocyte progenitor pool.
288 ponses to acute death of premyelinating late oligodendrocyte progenitors (preOLs).
289            In response to preOL death, early oligodendrocyte progenitors rapidly proliferate and diff
290                                              Oligodendrocyte progenitors respond to biophysical or me
291 stration of rmTIMP-1 to A2B5(+) immunopanned oligodendrocyte progenitors significantly increased the
292                                       Unlike oligodendrocyte progenitors, SPNs displayed pronounced r
293                              Therefore, axon-oligodendrocyte progenitor synapse formation is a transi
294 and LINC complex, mediate nuclear changes in oligodendrocyte progenitors that favor a default pathway
295             We show here that the ability of oligodendrocyte progenitors to acquire the identity of m
296 ates were found to promote the transition of oligodendrocyte progenitors to newly formed oligodendroc
297 , the presence of susceptible populations of oligodendrocyte progenitors underlies regional predilect
298  as well as after transplantation of t(1;11) oligodendrocyte progenitors were significantly reduced w
299 erivation and prospective isolation of human oligodendrocyte progenitors, which, upon transplantation
300 ta was also detected in NG2 cells (potential oligodendrocyte progenitors) within the white matter and

 
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