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1 xons is controlled regionally along axons by oligodendroglia.
2 critical for control of viral replication in oligodendroglia.
3 ive astrocytes but had morphology typical of oligodendroglia.
4 -immunopositive inclusions in neurons and in oligodendroglia.
5 and blood-derived immune cells destroy brain oligodendroglia.
6 3'-phosphodiesterase, a specific marker for oligodendroglia.
7 ase labeled astrocytes, but not microglia or oligodendroglia.
8 only along regions of the axon invested with oligodendroglia.
9 iving knockout mice still expressing Tns3 in oligodendroglia.
10 al dysregulation in unipolar brush cells and oligodendroglia.
11 x/flox) excision in neurons, astrocytes, and oligodendroglia.
12 erneurons but not in nuclei of astrocytes or oligodendroglia.
13 the complex interactions between neurons and oligodendroglia.
14 ion, proliferation, and myelin production by oligodendroglia.
15 to initiate radial expansion, astrocytes, or oligodendroglia.
16 esting the functional importance of MAP1B in oligodendroglia.
17 ons, MAP1B recently was found in myelinating oligodendroglia.
18 ng a dominant-negative IFN-gamma receptor on oligodendroglia.
19 by the selective RNA-binding protein QKI in oligodendroglia.
20 to be a common mechanism in both neurons and oligodendroglia.
21 tical for the control of JHMV replication in oligodendroglia.
22 pressed the infection of astrocytes, but not oligodendroglia.
23 nd therein, is abundant in both neurones and oligodendroglia.
24 te electron dense cytoplasm derived from the oligodendroglia.
25 ise to cortical interneurons, astroglia, and oligodendroglia.
26 d lesions, preferential differentiation into oligodendroglia accompanied by axonal preservation, and
27 n the injury site and hours to days later in oligodendroglia adjacent to and distant from the injury
28 ble to assess how timed ablation of Cxcr2 in oligodendroglia affected disease following intracranial
30 xons of retinal ganglion cells interact with oligodendroglia and become myelinated during normal mous
31 n (BMP) signaling inhibits the generation of oligodendroglia and enhances generation of astrocytes by
33 ese two genes sheds light on the ontogeny of oligodendroglia and genetic requirements for their devel
36 arge numbers of astrocytes, and in scattered oligodendroglia and monocytes/macrophages in both the in
38 he CNS.SIGNIFICANCE STATEMENT Replacement of oligodendroglia and myelin regeneration holds tremendous
39 axons and axon terminals in association with oligodendroglia and neuron loss and slowly progressive m
41 cate experience-driven circuit activation to oligodendroglia and precisely how changes in oligodendro
42 known as SLC16A1), is highly enriched within oligodendroglia and that disruption of this transporter
43 tients, Ndst1 is also found overexpressed in oligodendroglia and the number of Ndst1-expressing oligo
45 of IFN-gamma localized almost exclusively to oligodendroglia and was associated with increased CD8+ T
46 ositive inclusions that are predominantly in oligodendroglia and/or astroglia in the frontal, tempora
49 uced viral antigen in most cell types except oligodendroglia, and was associated with reduced demyeli
50 results suggest that MHC class II-expressing oligodendroglia are more prevalent with aging, which may
53 affect myelination, such as the function of oligodendroglia, are critical processes that could profo
56 mino-terminal region of Nogo-A, expressed by oligodendroglia, as necessary and sufficient to inhibit
57 sed approximately 2-fold each in endothelia, oligodendroglia, astrocytes, and axons of the optic nerv
61 ed glial cell numbers are most pronounced in oligodendroglia, but reduced astrocyte cell densities ha
63 t MAP1B mRNA is markedly up-regulated in the oligodendroglia cell line CG4 upon induced differentiati
64 cells (OPCs), immature oligodendrocytes and oligodendroglia cell lines, where it interacts with a su
65 perimental downregulation of EXOSC8 in human oligodendroglia cells and in zebrafish induce a specific
67 r results reveal dynamic regulation of human oligodendroglia circRNA landscapes during early differen
68 we identify developmentally programmed human oligodendroglia circRNA landscapes in the HOG oligodendr
71 nding EGF immediately after injury decreases oligodendroglia death, enhances generation of new oligod
72 Myelin basic protein expression and CC1(+) oligodendroglia decreased after hyperoxia at P8, but ret
73 myelinating coronavirus, B7-H1 expression on oligodendroglia delays virus control, but also dampens c
74 lities, and morphologic abnormalities in the oligodendroglia demonstrated in schizophrenic brains are
75 t the functional level, genetically blocking oligodendroglia-derived VEGF but not Wnt significantly d
76 strain of mouse hepatitis virus persists in oligodendroglia despite the presence of virus-specific C
79 monstrate that genetic silencing of CXCR2 on oligodendroglia did not affect clinical disease, neuroin
82 ion, the abundance of MHC class I-expressing oligodendroglia did not vary with age, whereas MHC class
85 tes, we reveal that deletion of Tsc1 affects oligodendroglia differently depending on the stage of th
86 e molecular level, HIFalpha stabilization in oligodendroglia does not perturb Wnt signaling but rathe
89 y affected in HD, and recent findings reveal oligodendroglia dysfunction as an early pathological eve
90 our data demonstrate that TrkB expression in oligodendroglia exerts a direct effect on oligodendrocyt
91 ions, but not normal appearing white matter, oligodendroglia exhibit enhanced expression of the immun
96 erging virus localized almost exclusively to oligodendroglia in B cell+ Ab-deficient mice, whereas it
99 ur finding of large numbers of proliferating oligodendroglia in MS brains expressing up-regulated PDG
100 part to widespread apoptosis of neurons and oligodendroglia in regions distant from and relatively u
102 tion was observed in a significant number of oligodendroglia in the dorsal and ventral funiculi, and
103 ased from neurons, astroglia, microglia, and oligodendroglia in the nervous system and their implicat
105 ve control levels during EAE, and numbers of oligodendroglia increased as well, but astrogenesis from
107 intercellular interactions between competing oligodendroglia influence the number and length of myeli
108 , our findings argue that CXCR2 signaling in oligodendroglia is dispensable with regard to contributi
109 ling through the chemokine receptor CXCR2 in oligodendroglia is important for developmental myelinati
110 endroglia and the number of Ndst1-expressing oligodendroglia is inversely correlated with lesion size
111 ivo genetic models, we show that HIFalpha in oligodendroglia is necessary and sufficient for angiogen
112 ceptor demonstrated that B7-H1 expression on oligodendroglia is predominantly regulated by IFN-gamma.
116 ompact myelin, as previously thought, but to oligodendroglia-like cells, whose processes converge to
118 that express Flag-QKI-6 specifically in the oligodendroglia lineage, driven by the proteolipid prote
121 heir 3D relationships with other astrocytes, oligodendroglia, neurons, and vasculature of the brain s
122 However, inhibition of viral replication in oligodendroglia occurs via a perforin-independent mechan
124 nt at moderate to high levels in neurons and oligodendroglia of the adult human brain, at a level clo
125 , including astrocytes (AS), microglia (MG), oligodendroglia (OG), neurons (NEU), and vascular cells
127 MS) is characterized by a targeted attack on oligodendroglia (OLG) and myelin by immune cells, which
128 lthough studies suggest a role for CXCL12 in oligodendroglia ontogeny in vitro, no studies have inves
129 t exhibit significantly different numbers of oligodendroglia or differentiated oligodendrocytes in th
130 at non-neuronal cells (microglia, astroglia, oligodendroglia) participate in the degenerative process
131 ly QKI-5, QKI-6, and QKI-7, are expressed in oligodendroglia progenitor cells (OPCs) prior to the ini
132 l expression, FMRP expression is detected in oligodendroglia progenitor cells (OPCs), immature oligod
133 se data indicate that IFN-gamma signaling by oligodendroglia reduces viral replication but affects bo
135 brain-derived Hp among brain cells and that oligodendroglia-released Hp plays protective roles again
137 Knockdown of Scribble expression in cultured oligodendroglia results in disrupted morphology and myel
140 3'UTR of MAP1B mRNA interacts with QKI, and oligodendroglia-specific QKI-deficiency in the quakingvi
145 ck of functional ASPA, an enzyme enriched in oligodendroglia that cleaves N-acetyl-l-aspartate (NAA)
146 CXCR2 is a chemokine receptor expressed on oligodendroglia that has been implicated in the pathogen
147 l cell lineages - astrocytes, microglia, and oligodendroglia - that are highly conserved between pati
148 A landscape and function in developing human oligodendroglia, the myelinating cells that govern neuro
149 nitor compartment produces new astroglia and oligodendroglia; the latter expand 10- to 18-fold postna
150 he many donor cells that differentiated into oligodendroglia-there appeared to be a shift in the fate
152 The functional consequence of excess MSP on oligodendroglia was determined in vitro by evaluating th
153 as mediator controlling JHMV replication in oligodendroglia was examined in mice deficient in IFN-ga
154 ction; however, although B7-H1 expression on oligodendroglia was prominent and sustained, it was sign
155 understand the biology of CXCR2 signaling on oligodendroglia, we generated transgenic mice in which C
157 ry with age, whereas MHC class II-expressing oligodendroglia were more abundant in aging mice than ne
159 d spinal cord, while MHC class II-expressing oligodendroglia were seen only in the spinal cord of agi
161 s more frequently observed in astrocytes and oligodendroglia, whereas NFIB expression was predominant
162 l nervous system, this plasticity stems from oligodendroglia, which form myelin sheaths to regulate t