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1 males, resulting in persistent infection and oligospermia.
2 tected against testis infection, injury, and oligospermia.
3 nished testosterone and inhibin B levels and oligospermia.
4 spermia (OR 1.22, 95% CI 1.11-1.34), and for oligospermia (1.14, 1.04-1.25), but age at diagnosis and
6 rkably, several AR mutations associated with oligospermia and androgen insensitivity syndrome map to
7 old increases in risk for clinically defined oligospermia and azoospermia and improved prediction of
8 nk2a2 has been disrupted are infertile, with oligospermia and globozoospermia ('round-headed' spermat
9 with infertility secondary to her partner's oligospermia and her chronic anovulation presented 13 da
10 permia, 8480 mg/m(2) (4264) in patients with oligospermia, and 6626 mg/m(2) (3576) in patients with n
12 ity with survivors exhibiting hydrocephalus, oligospermia, and cerebellar hypoplasia, and variably ex
14 rcentage of males with azoospermia or severe oligospermia, and its homology with a Drosophila male in
16 ome infertile men with azoospermia or severe oligospermia have small deletions in regions of the Y ch
17 a was noted in 53 (25%) of 214 participants, oligospermia in 59 (28%), and normospermia (sperm concen
18 that include testicular and tubular atrophy, oligospermia, Leydig cell hyperproliferation and increas
19 hould be suspected in an infertile male with oligospermia or azoospermia with low ejaculate volume, n
22 the infertile men had azoospermia or severe oligospermia (sperm concentration, <5 million per millil
23 ration, <5 million per milliliter), four had oligospermia (sperm concentration, 5 million to <20 mill
24 child; in sperm DNA from a patient with mild oligospermia treated with eBEACOPP; and in caecal adenoc
25 h sperm DNA fragmentation, elevated ROS, and oligospermia were more prevalent in the study group comp