戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 ells were killed as a result of necrosis and oncosis.
2 form of stress-activated cell death known as oncosis.
3 ent apoptosis, while higher CPE doses induce oncosis.
4  and that morphologically resembles cellular oncosis.
5 ent Shigella infection of HMDM and is termed oncosis.
6 lable to the cells induced cell death due to oncosis.
7 apoptosis, others induce cell starvation, or oncosis.
8 death, apoptosis, autophagy, pyroptosis, and oncosis.
9 optosis but is similar to that described for oncosis, a form of cell death accompanied by the membran
10 c relevance, these results suggest that both oncosis and apoptosis may occur in the intestines during
11           These features bore resemblance to oncosis and are in striking contrast to previously descr
12  This strain caused rapid host cell death by oncosis, and few bacterial CFU were recovered after incu
13 l death occurred rapidly as a consequence of oncosis, characterized by loss of cytoplasmic volume con
14 ath differentially mediated by apoptosis and oncosis effectors, with IVIg protecting target cells by
15 n this study for demonstrating apoptosis and oncosis have pathophysiologic relevance, these results s
16 CDEC strains appears to be critical for both oncosis in HMDM and apoptosis in J774 cells.
17                                Cell death by oncosis in human macrophages was confirmed by lactate de
18  death through two mechanisms: apoptosis and oncosis in MDA-MB-468 cells.
19 by distinct pathways including apoptosis and oncosis in response to various death signals.
20                                          The oncosis inhibitor glycine, but not a broad-spectrum casp
21 d morphologically by cell shrinkage, whereas oncosis is a form of cell death induced by energy deplet
22 eptor inducing membrane injury) mAb mediates oncosis-like cell death in Jurkat cells.
23                 Mechanistic studies revealed oncosis-like cell death mediated by reactive oxygen spec
24 e cytoplasm of HeLa cancer cells and induced oncosis-like cell death.
25 r these conditions neurons rapidly underwent oncosis (necrosis) with a complete loss in viability occ
26  categorized into three subtypes: apoptosis, oncosis (necrosis), and cell death with autophagy.
27 e expression, membrane damage, apoptosis and oncosis of living sensing organisms can provide insight
28 sis, the sensitivity of spectral analysis to oncosis or ischemic cell death had not previously been s
29 as morphologic features more consistent with oncosis/primary necrosis than apoptosis.
30                            Anti-Porimin (Pro-oncosis receptor inducing membrane injury) mAb mediates
31 antibodies, designated anti-Porimin (for pro-oncosis receptor inducing membrane injury), was found to
32 crosis is depicted by a gain in cell volume (oncosis), swelling of organelles, plasma membrane leakag
33 roduces a mixed form of cell death with both oncosis (swollen cells and nuclei) and apoptosis.
34 osis was associated with PARP-1 cleavage and oncosis was associated with a rapid decline in intracell
35 by reperfusion leads to necrotic cell death (oncosis), which often occurs within minutes of reperfusi
36 edominant form of cell death in all lines is oncosis, which is driven by the loss of plasma membrane