ライフサイエンスコーパス: oogenesis

1 tive migration that occurs during Stage 9 of oogenesis.

2 s and, thus, support the maturation phase of oogenesis.

3 distribution of 5862 mRNAs during Drosophila oogenesis.

4 tegrating egg chambers, indicating defective oogenesis.

5 the tight suppression of transposons during oogenesis.

6 iptome assembly at different stages of mouse oogenesis.

7 poral restriction of actin remodeling during oogenesis.

8 erm cells for their survival past stage 5 of oogenesis.

9 cell nuclei and germinal vesicle during mid-oogenesis.

10 ry assimilated from the maternal diet during oogenesis.

11 on by Egg is required for multiple stages of oogenesis.

12 First, KHC is temporally regulated during oogenesis.

13 that H3K9 methylation by Egg is required for oogenesis.

14 ferentiation, or other processes involved in oogenesis.

15 d microtubule organization during Drosophila oogenesis.

16 ty in D. sechellia due to maternal arrest of oogenesis.

17 a on the size of the mtDNA bottleneck during oogenesis.

18 norhabditis elegans germline development and oogenesis.

19 eraction in photoreceptor axon targeting and oogenesis.

20 xes Iml1/GATOR1 and GATOR2 during Drosophila oogenesis.

21 a loss-of-function mouse model for STAG3 in oogenesis.

22 al mRNAs and proteins that accumulate during oogenesis.

23 and the latter has yet to be established in oogenesis.

24 cells called border cells during Drosophila oogenesis.

25 ion between Spir and Capu is required during oogenesis.

26 dynein-mediated processes during Drosophila oogenesis.

27 ssed by, the Matrimony (Mtrm) protein during oogenesis.

28 ential for establishing an actin mesh during oogenesis.

29 licle cell differentiation during Drosophila oogenesis.

30 of key components of Notch signaling during oogenesis.

31 pmental gene amplification during Drosophila oogenesis.

32 tant targets of EGFR signaling in Drosophila oogenesis.

33 licle cells into the amplification stages of oogenesis.

34 ed genes enhance apoptosis during C. elegans oogenesis.

35 r development during Drosophila melanogaster oogenesis.

36 ls to adopt the sperm fate at the expense of oogenesis.

37 oposed to repress expression of genes during oogenesis.

38 the diversification of BMP signaling during oogenesis.

39 he promotion of membrane organization during oogenesis.

40 anterior localization of bicoid mRNA in late oogenesis.

41 enorhabditis briggsae, GLD-1 acts to promote oogenesis.

42 changes are not responsible for the block in oogenesis.

43 seh1 has a crucial germline function during oogenesis.

44 membrane biogenesis and organization during oogenesis.

45 er called the chromocenter during Drosophila oogenesis.

46 dy axes are established in the oocyte during oogenesis.

47 tructural nucleoporin Seh1 during Drosophila oogenesis.

48 CP190, these proteins are not essential for oogenesis.

49 nges in ovarian physiology that occur during oogenesis.

50 s reveal critical functions for this gene in oogenesis.

51 ecause they also function at other stages of oogenesis.

52 ssion of functional mtDNA through Drosophila oogenesis.

53 ffective at silencing gene expression during oogenesis.

54 assembly of an actin mesh during Drosophila oogenesis.

55 ngation from a sphere to an ellipsoid during oogenesis.

56 owed the analysis of baz function throughout oogenesis.

57 ed mechanism that controls key events during oogenesis.

58 erm-associated genes during normal postnatal oogenesis.

59 D-2-binding partner, GLD-3, to ensure normal oogenesis.

60 the cytoplasm of cells undergoing IC during oogenesis.

61 the posteriorly localized germ plasm during oogenesis.

62 e maternal provision of Nv-Hb protein during oogenesis.

63 m for activation of cell death in Drosophila oogenesis.

64 FMRP controls germline proliferation during oogenesis.

65 aintain their localized distributions during oogenesis.

66 ermatogenesis, whereas GLD-2/RNP-8 specifies oogenesis.

67 ent and follicle formation during Drosophila oogenesis.

68 n and microtubule dynamics during Drosophila oogenesis.

69 and Peg3, which all become methylated during oogenesis.

70 egg and future embryo are established during oogenesis.

71 germ line stem cell signaling and abrogated oogenesis.

72 ies with either synchronous and asynchronous oogenesis.

73 recocious transition from spermatogenesis to oogenesis.

74 Drosophila SAGA are differently required in oogenesis.

75 r follicles during mouse fetal and perinatal oogenesis.

76 ally occurs during either spermatogenesis or oogenesis.

77 tyltransferase with an essential function in oogenesis.

78 that barbed-end binding is not necessary for oogenesis.

79 lication and mitochondrial biogenesis during oogenesis.

80 to regulate ovarian germline stem cells and oogenesis.

81 mutational mechanisms in spermatogenesis and oogenesis.

82 clear actin must be tightly regulated during oogenesis.

83 icle formation during early, postnatal mouse oogenesis.

84 erning to the posterior of the oocyte during oogenesis.

85 of germ-line-derived nurse cells during late oogenesis.

86 targets and influencing transcription during oogenesis.

87 are maintained at a constant diameter before oogenesis.

88 jire as a cofactor of Notch signaling during oogenesis.

89 amounts of excess histones generated during oogenesis [9], and the maternal supplies of core histone

90 During oogenesis a maternal factor is localized to the vegetal

91 During Drosophila melanogaster oogenesis, a germline stem cell divides forming a cyst o

92 During Drosophila oogenesis, a gradient of EGFR activation patterns the fo

93 During Drosophila oogenesis, a prominent metabolic checkpoint occurs at th

94 e, cells undergoing spermatogenesis, but not oogenesis, activate transposons.

95 During Drosophila oogenesis, activation of Notch signaling in the follicul

96 iched with Me(199)Hg, spanning the period of oogenesis, allowing us to differentiate between mercury

97 trapment-mediated localization of nos during oogenesis, also function in da neurons for formation and

98 omotes translation of Cyclin A, beginning in oogenesis, an earlier onset than previously recognized.

99 hey are ineffective in gene knockdown during oogenesis, an important model system for the study of ma

100 t to an interplay between unique features of oogenesis and a host of endogenous and exogenous factors

101 tion allows females to assess the demands of oogenesis and alter their behavior and metabolic state t

102 re depend on centrosome positioning early in oogenesis and are independent of anterior-posterior axis

103 the animal-vegetal axis is determined during oogenesis and at ovulation, the egg is radially symmetri

104 genes in a cell-type specific manner during oogenesis and begin to reveal the relatedness in express

105 ly identified endoribonuclease that promotes oogenesis and contains a number of RNA binding domains,

106 n the FMR1 gene that appears to occur during oogenesis and during early embryogenesis.

107 rather than asymmetrically, localized during oogenesis and early embryogenesis.

108 xpression in the posterior germ plasm during oogenesis and early embryogenesis.

109 s of transcription in heterochromatin during oogenesis and early embryonic development.

110 ffect genes, these factors accumulate during oogenesis and enable the activation of the embryonic gen

111 is important for dorso-ventral patterning in oogenesis and for anterior-posterior pattern formation d

112 in is critical for normal endocytosis during oogenesis and for embryogenesis in the sea star and that

113 between Spire and Cappuccino is required for oogenesis and for in vitro synergistic actin assembly.

114 erized the metabolic mechanisms that support oogenesis and found that mitochondria in mature Drosophi

115 wth factor-like signaling in regulating both oogenesis and immune system activity, and propose a sign

116 Centrioles are lost during oogenesis and inherited from the sperm at fertilization.

117 nd nucleoplasmin methylation appears late in oogenesis and is most abundant in the laid egg.

118 s to extensive glycogen accumulation late in oogenesis and is required for the developmental competen

119 teroplasmic flies was decreased, both during oogenesis and over multiple generations, at the restrict

120 effects of H3.3R26 and H3.3K27 in modulating oogenesis and partitioning cells to the inner cell mass

121 tand transcriptional regulation during human oogenesis and preimplantation development, we defined st

122 ese tightly regulated processes begin during oogenesis and proceed through gastrulation to establish

123 urs at the organelle level during Drosophila oogenesis and provides molecular entry points to test th

124 f oskar localization that occurs during late oogenesis and results in amplification of the germ plasm

125 , and females are sterile, showing disrupted oogenesis and severe defects in follicle cell differenti

126 ophila female germ line epigenome throughout oogenesis and show that the oocyte has a unique, dynamic

127 e dot chromosome during early embryogenesis, oogenesis and spermatogenesis resemble that of the curre

128 1 function interferes with the completion of oogenesis and spermatogenesis through sexually dimorphic

129 d migration--to the sex-specific pathways of oogenesis and spermatogenesis.

130 tabolism with the precursor l-DOPA, resuming oogenesis and stimulating egg production.

131 ss expression of maternal transcripts during oogenesis and suggest that interplay between OMA-1 and o

132 in germ cells throughout spermatogenesis and oogenesis and thus, focused further efforts on this fami

133 nimals, indole induces genes associated with oogenesis and, accordingly, extends fecundity and reprod

134 t may play important regulatory roles during oogenesis and/or early embryogenesis.

135 ght to determine whether ASUN plays roles in oogenesis and/or embryogenesis.

136 ce indicates that production of new oocytes (oogenesis) and their enclosure by somatic cells (follicu

137 maternal mRNAs, Zar2 was present throughout oogenesis, and endogenous Zar2 co-immunoprecipitated end

138 pecific DNA hypermethylation acquired during oogenesis, and its expression silencing was reversible o

139 versification of BMP signaling in Drosophila oogenesis, and they provide insight into a mechanism und

140 The piRNA pathway is essential in early oogenesis, and we find that nuclear localization of Zfrp

141 Oogenesis appeared to be more sensitive compared to sper

142 female tissues and the maternal diet during oogenesis are significant sources of Hg.

143 cells differentiated early during Drosophila oogenesis--are arrested at G2 phase and can serve as a m

144 Here we use Drosophila oogenesis as an in vivo model system to determine the ex

145 migration of border cells during Drosophila oogenesis as our model system.

146 Such removal can occur during oogenesis, as in Drosophila, where departure of Polo kin

147 the disordered character of transport at mid-oogenesis, as revealed by streaming, is an important com

148 s of germ cell-specific markers and in vitro oogenesis, as well as the use of intraovarian transplant

149 g wdr-5.1/wdr-5.2 function fail to switch to oogenesis at 25 degrees C, resulting in a masculinizatio

150 66 proteins likely act uniquely during late oogenesis, because they are up-regulated at maturation a

151 an shRNA-depletion strategy to overcome the oogenesis block.

152 Null alleles in these genes result in an oogenesis block.

153 During oogenesis, border cells delaminate from the follicular e

154 uration are factors needed not only for late oogenesis but also completion of meiosis and early embry

155 fic SEX-LETHAL (SXL) protein is required for oogenesis, but how Sxl interfaces with the genetic circu

156 blished along the animal-vegetal axis during oogenesis, but the underlying molecular mechanisms are p

157 osophila is protected from DNA damage during oogenesis by a mechanism involving short RNAs.

158 ed appears to be negatively regulated during oogenesis by Abelson kinase.

159 ion of the mechanical stress associated with oogenesis by conferring stability and elasticity to germ

160 regulate actin remodeling during Drosophila oogenesis by controlling Ena localization/activity, such

161 on between egg and oocyst numbers; impairing oogenesis by multiple 20E manipulations decreases parasi

162 nterior-posterior axis is established during oogenesis by the localization of bicoid and oskar mRNAs

163 Gro insufficiency during oogenesis can be explained by its downregulation in Brk-

164 Stage 10 of Drosophila oogenesis can be subdivided into stages 10A and 10B base

165 silencing of MISO abolishes the increase in oogenesis caused by mating in blood-fed females, causes

166 the absence of dTBCB during later stages of oogenesis causes major defects in cell polarity.

167 nctions of three individual H3.3 residues in oogenesis, cleavage-stage embryogenesis and early develo

168 ation and in physiological demand control of oogenesis, coordinating these processes.

169 During Caenorhabditis elegans oogenesis, cytoplasmic RNPs can transition among diffuse

170 In mouse oogenesis, DNA methylation establishment occurs on a lar

171 ages in spermatogenesis and then switches to oogenesis during late stages of larval development.

172 Oogenesis features an enormous increase in mitochondrial

173 emale reproductive structure and the site of oogenesis, fertilization, and maturation of the embryo a

174 d early embryos and plays vital roles during oogenesis, fertilization, and preimplantation developmen

175 A replication during Drosophila melanogaster oogenesis, finding that mtDNA replication commenced befo

176 During Drosophila oogenesis, follicle cells sequentially undergo three dis

177 In oogenesis, graded JAK activity stimulated by Upd specifi

178 extends through RCs and is important during oogenesis, had no effect on spermatogenesis or male fert

179 of egg-chamber elongation during Drosophila oogenesis has always been mysterious.

180 Although Drosophila oogenesis has been intensively studied, the genetic and

181 short hairpin RNAs (shRNAs) effective during oogenesis has provided an alternative to producing germl

182 In particular, research on Drosophila oogenesis has provided critical insights into piRNA biog

183 to-nuclear incompatibility during Drosophila oogenesis has severe consequences for egg production and

184 to the vegetal cortex during Xenopus laevis oogenesis have been reported to function in germ layer p

185 ents for the SMC5/6 complex during mammalian oogenesis have not previously been examined.

186 The spermatogenesis/oogenesis helix-loop-helix (SOHLH) proteins SOHLH1 and S

187 Additionally, preoogenesis Hg and during-oogenesis Hg were transferred proportionally to eggs, su

188 anism of centriole elimination during female oogenesis, highlighting a protective role for Polo kinas

189 During oogenesis, hundreds of maternal RNAs are selectively loc

190 Met and SRC regulate both vitellogenesis and oogenesis in C. lectularius.

191 cells in ovaries during the early stages of oogenesis in Drosophila melanogaster Inhibition of ceram

192 P107-associated protein SEH1 is required for oogenesis in Drosophila.

193 maintenance and egg chamber polarity during oogenesis in Drosophila.

194 es of Kinesin-dependent streaming during mid-oogenesis in Drosophila.

195 d, how filamin mechanosensing contributes to oogenesis in Drosophila.

196 ed the dysregulation of proteins involved in oogenesis in female livers.

197 In comparison to oogenesis in many organisms, spermatogenesis is particul

198 d to adversely affect two distinct stages of oogenesis in the developing ovary: the events of prophas

199 for early development is established during oogenesis in the form of the maternal transcriptome.

200 We previously established Drosophila oogenesis, in particular nurse cell dumping, as a new mo

201 to drive meiotic prophase I progression and oogenesis; in the absence of food, the resultant inactiv

202 pensable for the early MT-dependent steps of oogenesis, including cell division, and that dTBCB is no

203 ndent developmental events during Drosophila oogenesis, indicating ligand-independent Notch activity

204 eaks (ttk(twk)) reduces TTK69 levels late in oogenesis, inhibiting this expansion.

205 During Drosophila oogenesis, intercellular bridges (referred to as ring ca

206 Oogenesis is a complex developmental process that involv

207 follicular epithelium (FE) around stage 6 of oogenesis is essential for entry into the endocycle and

208 o downregulation of the RAS-ERK pathway, and oogenesis is stalled.

209 er, the function of Ago1 in other aspects of oogenesis is still elusive.

210 Oogenesis is the process by which ovarian germ cells und

211 orm germline cysts, but the role of cysts in oogenesis is unknown.

212 t traverses the Drosophila oocyte during mid-oogenesis, is essential for proper establishment of the

213 A is sequestered at the anterior pole during oogenesis, is not translated until fertilization, and pr

214 teroid signaling regulates multiple steps in oogenesis, it is not known whether it regulates Drosophi

215 y stages are determined by the female during oogenesis, it is unknown whether reproducibility is perp

216 TTK69 is required again later in oogenesis: it controls the volume of the dorsal-appendag

217 pecific PHD finger protein 7 (PHF7) disrupts oogenesis, leading to either an agametic or germ cell tu

218 letal organization and egg morphology during oogenesis, leading to female sterility.

219 not the preconception effect of diabetes on oogenesis, leads to fetal growth restriction and congeni

220 developmental progression through meiosis in oogenesis, little is known about Cyclin A.

221 During oogenesis, mammalian eggs accumulate proteins required f

222 on and a female Mating-Induced Stimulator of Oogenesis (MISO) protein.

223 We show that, during oogenesis, mitochondria accumulate at the oocyte posteri

224 Understanding the unique mechanisms of human oogenesis necessitates the development of an in vitro sy

225 genome integrity, and is not obligatory for oogenesis nor fertility.

226 Neo-oogenesis occurred early after transplantation, as evide

227 these mutations, and compare the effects on oogenesis of both strong loss-of-function and weak hypom

228 RNA system exist: somatic cells that support oogenesis only employ Piwi, whereas germ cells utilize a

229 females direct nutritional resources towards oogenesis only when inseminated.

230 e Drosophila oocyte anterior from stage 9 of oogenesis onwards to provide a local source for Bicoid p

231 During oogenesis, oocytes are arrested in prophase and resume m

232 ne-derived nurse cells during Drosophila mid-oogenesis or follicle development.

233 whether reproducibility is perpetuated from oogenesis or reacquired by the zygote.

234 demonstrates that vha-12 is not required for oogenesis or spermatogenesis in the adult germ line, but

235 female, or male) and type of gametogenesis (oogenesis or spermatogenesis).

236 fetal ovary or testis and hence to embark on oogenesis or spermatogenesis.

237 e eliminated during development, long before oogenesis or spermatogenesis.

238 tal life as they commit to, and prepare for, oogenesis or spermatogenesis.

239 Entry into the spermatogenesis or oogenesis pathway requires that the appropriate gene net

240 y exploiting the asymmetric cell division of oogenesis--present a potent selective pressure favoring

241 enzyme is a broad-spectrum regulator of the oogenesis program that acts within an RNA regulatory net

242 Drosophila oogenesis provides a developmental system with which to

243 During Drosophila oogenesis, RCs connect the maturing oocyte to nurse cell

244 faces with the genetic circuitry controlling oogenesis remains unknown.

245 icle cell (FC) at stages 8/9, 10 and >=11 of oogenesis, respectively.

246 d lead necessarily toward spermatogenesis or oogenesis, respectively.

247 uently, failure of oskar localization during oogenesis results in embryos lacking germ cells and abdo

248 At a specific time in oogenesis, somatic follicle cells transition from genomi

249 During Drosophila oogenesis, specialized actin-based structures called rin

250 ive repertoire of CSR-1 small RNAs targeting oogenesis-specific mRNAs.

251 During Drosophila melanogaster oogenesis, spindle assembly occurs without centrosomes a

252 ransition that occurs in the final stages of oogenesis (stage 13/14).

253 pression in all cells during early stages of oogenesis that diminishes in later stages.

254 mediator of lateral inhibition in zebrafish oogenesis that directs cell fate through mechanical cues

255 Therefore, in oogenesis, the activities of Upd and Upd3 both appear to

256 Our findings link two universal features of oogenesis, the Bb and the chromosomal bouquet, to oocyte

257 During Drosophila oogenesis, the egg chamber elongates along the anterior-

258 ation from stage 9 to stage 10 in Drosophila oogenesis, the egg chamber increases in length by approx

259 During stages 8 to 10 of oogenesis, the FC epithelium transitions between simple

260 During Drosophila oogenesis, the follicular epithelial cells undergo two m

261 Later in oogenesis, the GATOR2 components Mio and Seh1 are requir

262 Later in oogenesis, the gradient of EGFR activation is split into

263 egulates expression of maternal mRNAs during oogenesis, the oocyte to embryo transition, and early em

264 During oogenesis, the roles of patterning and morphogenesis by

265 During Drosophila oogenesis, the somatic follicle cells encasing oocytes u

266 We demonstrate that during Drosophila oogenesis, the two-domain expression pattern of Broad, a

267 follicle cells switch into endocycles during oogenesis they repress the apoptotic response to DNA dam

268 eld haplo-X oocytes, during XX hermaphrodite oogenesis they segregate to the first polar body to yiel

269 ndogenous gene during Caenorhabditis elegans oogenesis; this process is referred to as germ-line cosu

270 cumulates at the oocyte posterior during mid-oogenesis through a well-studied process involving kines

271 turbation of the oocyte's epigenome in early oogenesis, through depletion of the dKDM5 histone demeth

272 es that the Me31B interactome undergoes from oogenesis to early embryogenesis, we characterized the e

273 Mechanisms that couple later steps of oogenesis to environmental conditions remain largely und

274 We used oogenesis to explore developmental roles of two importan

275 dysferlin RNA and protein are expressed from oogenesis to gastrulation.

276 ity, is shown to be at play during zebrafish oogenesis to prevent polyspermy.

277 ates and inhibits Dicer during meiosis I for oogenesis to proceed normally in Caenorhabditis elegans

278 owing the canonical pattern during XX female oogenesis to yield haplo-X oocytes, during XX hermaphrod

279 of their development from stem cells to the oogenesis-to-ovulation transition.

280 manifests in the early stages of Drosophila oogenesis, triggered by reduction of the pro-fusion prot

281 During oogenesis, TspanC8 genes were up-regulated in border cel

282 ese hubs were analyzed further in Drosophila oogenesis, using targeted germline RNAi, and adhesion wa

283 ation are enriched in the germ plasm at late oogenesis via a diffusion and entrapment mechanism, the

284 induced in follicle cells in late stages of oogenesis via ecdysteroid signaling.

285 female 20E-interacting protein that promotes oogenesis via mechanisms also favoring Plasmodium surviv

286 We show that during C. elegans oogenesis WAPL-1 antagonizes binding of cohesin containi

287 ional analysis of the tail during Drosophila oogenesis we have gained an understanding of how KHC ach

288 During C. elegans oogenesis, we find that two groups of PUF RNA binding pr

289 Whereas Setd1a plays no role in oogenesis, we report that Setd1b deficiency causes femal

290 is essential for germ plasm assembly during oogenesis, we show that it is toxic to pole cells.

291 Because effects on both stages of oogenesis were elicited using doses that yield circulati

292 spheroid (Mys) is only required during early oogenesis when the pre-follicle cells form the FE.

293 echanism for such coordination in Drosophila oogenesis, when the expression of the transcription fact

294 restrict disulfide bond formation until late oogenesis, when the oocyte no longer experiences large v

295 lly wild-type fly, it is insufficient during oogenesis, where Brk must utilize CtBP and 3R to pattern

296 ical structures of embryos originates during oogenesis, which is when the expression of maternally pr

297 usually produced during spermatogenesis and oogenesis, which take place in the testis and the ovary,

298 tone acetyltransferase (HAT) activity blocks oogenesis, while loss of the H2B deubiquitinase (DUB) ac

299 mo, and thus are sufficient to sustain adult oogenesis without a source of renewal.

300 a number of biological processes, including oogenesis, wound healing, and cytokinesis.