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1 arded migration of the epithelium across the open wound.
2 is the migration of keratinocytes across the open wound.
3 ssure to rapidly stanch blood loss and close open wounds.
4 aling can lead to the development of chronic open wounds.
5 form involving the application of insects to open wounds.
6 ibuted similarly between the treatment arms (Open: wound 0.5%, liver 5.8%, lung 4.6%, other 8.4%; Lap
8 ncanonical members of the JNK pathway caused open wounds, as did several genes involved in actin cyto
9 Both groups had comparable readmissions for open wounds, but patients treated with CEA required more
13 ertension, dyspnea, diabetes, renal failure, open wounds/infection, or advanced American Society of A
14 al wounds healing by secondary intention (an open wound, <3 weeks' duration, resulting from surgery),
15 on of the eye and/or adnexa, open globes and open wounds of ocular adnexa, diplopia, superficial corn
17 ng of rectal swab samples, swab samples from open wounds or drainages, and urine samples from patient
21 litative morphological classification of the open wound phenotypes and evaluation of JNK activation s
22 om the nares, oropharynx or trachea, and any open wound routinely on admission to the intensive care
23 e and technically safe, with rates of breast open wounds similar to those reported with post-mastecto
24 onally, in the appropriate clinical setting, open wounds such as sinus tracts and ulcers, as well as
25 healing assays including time to closure of open wounds, tensile strength of healed incisional wound
26 The median time to wound closure for chronic open wounds was 14.6 vs. 8 weeks for recurrent wounds.
28 e surgical and medical care of contaminated, open wounds with immunization and immunoglobulin therapy