ライフサイエンスコーパス: open-angle glaucoma

1 ar diseases, such as keratoconus and primary open angle glaucoma.

2 ion (ECP) surgeries in patients with primary open angle glaucoma.

3 in different types of primary and secondary open angle glaucoma.

4 Hg and he was found to have advanced primary open angle glaucoma.

5 s were mostly pseudo-exfoliative and primary open angle glaucoma.

6 M may be possible in human eyes with primary open angle glaucoma.

7 atients with coexisting cataract and primary open angle glaucoma.

8 transgenic mouse model of hereditary primary open-angle glaucoma.

9 ce to AH outflow, is a major risk factor for open-angle glaucoma.

10 in the United States and had newly diagnosed open-angle glaucoma.

11 myocilin, a protein associated with primary open-angle glaucoma.

12 6 had ocular hypertension and 14 had primary open-angle glaucoma.

13 be a new risk factor to consider in primary open-angle glaucoma.

14 ular-pressure-lowering drug in patients with open-angle glaucoma.

15 in lowering intraocular pressure in primary open-angle glaucoma.

16 high prevalence of glaucoma which is largely open-angle glaucoma.

17 and 2012 and newly diagnosed and treated for open-angle glaucoma.

18 e the most common genetic factors of primary open-angle glaucoma.

19 set of the prevalent ocular disorder primary open-angle glaucoma.

20 have previously been associated with primary open-angle glaucoma.

21 etic retinopathy, uveoretinitis, and primary open-angle glaucoma.

22 (IOP)-lowering medications in patients with open-angle glaucoma.

23 to mutations in the MYOCILIN gene is primary open-angle glaucoma.

24 when selecting treatments for patients with open-angle glaucoma.

25 ay lead to juvenile- and adult-onset primary open-angle glaucoma.

26 a multiple topical drug regimen for primary open-angle glaucoma.

27 procedure for reducing IOP in patients with open-angle glaucoma.

28 ic CPAMD8 variants to childhood and juvenile open-angle glaucoma.

29 pseudophakic patients with mild to moderate open-angle glaucoma.

30 n ethnicity, higher intraocular pressure and open-angle glaucoma.

31 ma and 2.1% (2/96) of probands with juvenile open-angle glaucoma.

32 and 2121 control individuals without primary open-angle glaucoma.

33 Genetic variants associated with primary open-angle glaucoma.

34 ellular matrix and frequent cause of chronic open-angle glaucoma.

35 Presence of primary open-angle glaucoma.

36 ent of patients with ocular hypertension and open-angle glaucoma.

37 surgery was performed in adults with various open-angle glaucomas.

38 The 5 most prevalent diagnoses were open-angle glaucoma (1.61%), exfoliation glaucoma (0.20%

39 In 30 patients with open-angle glaucoma, 1 eye (median mean deviation [MD],

40 tients were pseudophakic, with mild/moderate open-angle glaucoma, 12-month follow-up, and medicated i

41 ed diabetic macular edema and no preexisting open-angle glaucoma, 260 were randomly assigned to recei

42 study included 122 eyes treated for primary open angle glaucoma, 50 eyes (study group) in which, aft

43 gnoses in descending prevalence were primary open-angle glaucoma (55.9%), chronic angle-closure glauc

44 ed 354 eyes in 180 subjects (97 with primary open-angle glaucoma, 83 with glaucoma suspicion) who had

45 ecular meshwork is a primary risk factor for open-angle glaucoma, a leading cause of blindness.

46 have identified novel loci for POAG (primary-open-angle glaucoma) (ABCA1, AFAP1, GMDS, PMM2, TGFBR3,

47 childhood glaucoma after CYP1B1 and juvenile open-angle glaucoma after MYOC.

48 ree eyes of 2 patients who developed chronic open-angle glaucoma after Nd:YAG vitreolysis for symptom

49 Treatments for open-angle glaucoma aim to prevent vision loss through l

50 Family and personal history of primary open-angle glaucoma also increases the risk of corticost

51 2 eyes, comprising 38 glaucomatous eyes with open angle glaucoma and 24 healthy controls, were includ

52 s a highly heritable risk factor for primary open angle glaucoma and currently the only target for gl

53 h Weill-Marchesani-like syndrome and primary open angle glaucoma and ectopia lentis in dogs.

54 e patient with choroidal infarction, primary open angle glaucoma and Flammer syndrome.

55 r the development and progression of primary open angle glaucoma and is due to trabecular meshwork (T

56 Primary open angle glaucoma and ocular hypertension are habitual

57 uld be offered as a first-line treatment for open angle glaucoma and ocular hypertension, supporting

58 ry data set of 2320 individuals with primary open-angle glaucoma and 2121 control individuals without

59 cts (48 male, 40 female) with a diagnosis of open-angle glaucoma and a median age of 67 years (interq

60 anuary 2000 and September 2013 on the topics open-angle glaucoma and angle-closure glaucoma.

61 otal of 609 patients (609 eyes) with primary open-angle glaucoma and cataract were included.

62 A 75-year-old pseudophakic man with open-angle glaucoma and diabetic retinopathy developed n

63 GN, SETTING, AND PARTICIPANTS: Patients with open-angle glaucoma and healthy controls were examined b

64 ntia, including Alzheimer's disease, primary open-angle glaucoma and Helicobacter pylori (H.pylori) i

65 Consecutive trabeculectomy cases with open-angle glaucoma and no previous incisional glaucoma

66 NTG when compared with patients with primary open-angle glaucoma and nonglaucomatous control subjects

67 All patients with newly diagnosed open-angle glaucoma and ocular hypertension were include

68 he diurnal, but not the nocturnal, period in open-angle glaucoma and ocular hypertension.

69 rine transporter inhibitor, in patients with open-angle glaucoma and ocular hypertension.

70 with an increased prevalence of all forms of open-angle glaucoma and OHTN, whereas hyperopia was asso

71 One eye per patient with primary open-angle glaucoma and stable intraocular pressure (IOP

72 y 24-2 and 10-2 VFs in patients with primary open-angle glaucoma and to test the hypothesis that pati

73 aucoma Associates of Texas with uncontrolled open-angle glaucoma and underwent gonioscopy-assisted tr

74 Patients with open-angle glaucoma and VFs obtained from reliable exami

75 is and pars plana vitrectomy, and 1 juvenile open-angle glaucoma) and 21 of 70 eyes with nonglaucomat

76 A total of 234 patients (104 with primary open angle glaucoma, and 130 control subjects without an

77 ntil death; 203 patients (65.7%) had primary open-angle glaucoma, and 106 patients (34.2%) had exfoli

78 The inherited form of open angle glaucoma arises due to a toxic gain-of-functi

79 le have a 2.5-fold increased risk of primary open-angle glaucoma as compared with those in the lowest

80 To determine the stage of primary open angle glaucoma at presentation at a tertiary eye un

81 l, we enrolled patients with newly diagnosed open-angle glaucoma at ten UK centres (tertiary referral

82 trated differential association with primary open-angle glaucoma by ancestry.

83 osterior sclera in a canine model of primary open-angle glaucoma caused by the G661R missense mutatio

84 s with a minimum 2-year diagnosis of primary open-angle glaucoma, chronic primary angle-closure glauc

85 In an institutional setting, a patient with open-angle glaucoma consented to be the recipient of the

86 with many ocular problems, such as secondary open angle glaucoma, corneal dysfunction, cataract, and

87 Myocilin, a causative gene for open angle glaucoma, encodes a secreted glycoprotein wit

88 ERIA: participants with primary or secondary open-angle glaucoma (excluding uveitic) who had undergon

89 h patients diagnosed with moderate to severe open-angle glaucoma, focusing on outcomes they prioritiz

90 vitreous status and the presence of primary open-angle glaucoma for statistical analyses.

91 hundred seven patients with newly diagnosed open-angle glaucoma from the CIGTS.

92 Patients with established open-angle glaucoma from the Macular Assessment and Prog

93 Individuals with primary open-angle glaucoma had open iridocorneal angles and dis

94 ined IOP reduction for patients with primary open-angle glaucoma (hazard ratio [HR], 1.19; 95% confid

95 eculoplasty (LTP) is routinely used to treat open-angle glaucoma; hence, understanding variations in

96 ncluding glaucoma suspect [GS], high-tension open-angle glaucoma [HTG], and normal-tension glaucoma [

97 tments for patients presenting with advanced open angle glaucoma in a publicly funded health service.

98 eyes of 72 participants with a diagnosis of open angle glaucoma in one or both eyes and a visual acu

99 e was being followed and treated for primary open angle glaucoma in our tertiary referral center.

100 benefits and harms of screening for primary open-angle glaucoma in adults.

101 al success, the pathophysiologic features of open-angle glaucoma in younger patients may be more loca

102 Patients with open-angle glaucoma including pigmentary dispersion synd

103 1 single-nucleotide polymorphism and primary open angle glaucoma is found.

104 Secondary open-angle glaucoma is a complication of Nd:YAG vitreoly

105 ar meshwork (TM) and the elevation of IOP in open-angle glaucoma is associated with dysfunction and l

106 y congenital glaucoma (PCG n = 22), juvenile open angle glaucoma (JOAG n = 16), glaucoma following ca

107 14 Normal Tension Glaucoma (NTG), 5 Juvenile Open Angle Glaucoma (JOAG) and 6 (OHT).

108 We report a case of advanced juvenile open-angle glaucoma (JOAG) in which peripapillary capill

109 Juvenile open-angle glaucoma (JOAG) is a severe neurodegenerative

110 llowing cataract surgery (GFCS) and juvenile open-angle glaucoma (JOAG) is variable and with relative

111 Medical and surgical treatments for open-angle glaucoma lower intraocular pressure and reduc

112 women) and 2121 individuals without primary open-angle glaucoma (mean [interquartile range] age, 63.

113 A total of 2320 individuals with primary open-angle glaucoma (mean [interquartile range] age, 64.

114 < 0.001) in both eyes with incident primary open-angle glaucoma (mean, 10.6%; standard deviation, 22

115 iris; MPS type IV patients are vulnerable to open-angle glaucoma; MPS type VI patients have narrow an

116 (MetS) with intraocular pressure and primary open angle glaucoma (OAG) have been reported.

117 present study, we investigated the risk for open angle glaucoma (OAG) in migraineurs using a 10-year

118 imary lesion site in the age-related disease open angle glaucoma (OAG).

119 However, the risk of open-angle glaucoma (OAG) among patients with OSA remain

120 aluate the longitudinal relationship between open-angle glaucoma (OAG) and incident dementia.

121 Consecutive patients with open-angle glaucoma (OAG) and no previous filtering surg

122 Coded open-angle glaucoma (OAG) and OAG suspects accounted for

123 beculoplasty (SLT) on medically uncontrolled open-angle glaucoma (OAG) and to evaluate the effects of

124 d (VF) deterioration events in patients with open-angle glaucoma (OAG) by 50% over a 2-year period.

125 personalized forecasts of how patients with open-angle glaucoma (OAG) experience disease progression

126 surgery (medical group), while 32 eyes with open-angle glaucoma (OAG) had previously undergone glauc

127 This study determined the risk factors for open-angle glaucoma (OAG) in adults examined in the Nige

128 ice-daily timolol eye drops in patients with open-angle glaucoma (OAG) or ocular hypertension (OHT) t

129 In randomized, controlled trials of open-angle glaucoma (OAG) or ocular hypertension (OHT),

130 oprost ophthalmic solution, in patients with open-angle glaucoma (OAG) or ocular hypertension (OHT).

131 intraocular pressure (IOP) in patients with open-angle glaucoma (OAG) or ocular hypertension (OHT).

132 Sequential surgical cases of juvenile open-angle glaucoma (OAG) or primary congenital glaucoma

133 Nine patients had previous diagnoses of open-angle glaucoma (OAG) or suspected glaucoma.

134 lective laser trabeculoplasty (SLT) in early open-angle glaucoma (OAG) patients.

135 iciary person-years and per 10,000 diagnosed open-angle glaucoma (OAG) person-years.

136 iagnosis; 20.0% (n = 330) were identified as open-angle glaucoma (OAG) suspects, 9.2% (n = 151) were

137 or visual field progression in patients with open-angle glaucoma (OAG) were eligible.

138 ty affect receipt of common tests to monitor open-angle glaucoma (OAG).

139 tect against the development or worsening of open-angle glaucoma (OAG).

140 coherence tomography (OCT) volume scans for open-angle glaucoma (OAG).

141 plasty (SLT) on iStent inject(R) outcomes in open-angle glaucoma (OAG).

142 is an alternative to topical medications for open-angle glaucoma (OAG).

143 F)-preserving effect of medical treatment in open-angle glaucoma (OAG).

144 a major risk factor for the deterioration of open-angle glaucoma (OAG); medical IOP reduction is the

145 eroid use, myopia, socioeconomic status, and open-angle glaucoma (odds ratio [OR], 0.89; 95% confiden

146 2; chromosome 4, rs59892895T>C) with primary open-angle glaucoma (odds ratio [OR], 1.32 [95% CI, 1.20

147 cifically HiFU in patients with uncontrolled open-angle glaucoma on maximum tolerated medical therapy

148 ntrolled trial treatment-naive patients with open angle glaucoma or ocular hypertension and no ocular

149 ular hypertension, and patients with primary open angle glaucoma or primary angle closure glaucoma.

150 Patients with open-angle glaucoma or ocular hypertension (unmedicated

151 Adults with open-angle glaucoma or ocular hypertension (unmedicated

152 Total of 233 patients with open-angle glaucoma or ocular hypertension and with mean

153 tive cohort study of untreated patients with open-angle glaucoma or ocular hypertension at a hospital

154 l of 660 adults with a clinical diagnosis of open-angle glaucoma or ocular hypertension from a referr

155 Forty subjects with open-angle glaucoma or ocular hypertension were admitted

156 A total of 560 patients with primary open-angle glaucoma or ocular hypertension who had insuf

157 eserved with PQ reduced IOP in patients with open-angle glaucoma or ocular hypertension who were into

158 Patients with open-angle glaucoma or ocular hypertension who were into

159 In patients with open-angle glaucoma or ocular hypertension, polyquaterni

160 y and effectively lower IOP in patients with open-angle glaucoma or ocular hypertension, showing sign

161 Eligible patients were adults diagnosed with open-angle glaucoma or ocular hypertension.

162 Adult patients with open-angle glaucoma or ocular hypertension.

163 rz/brim/tim in Mexican patients with primary open-angle glaucoma or ocular hypertension.

164 during late-day time points in patients with open-angle glaucoma or ocular hypertension.

165 ts of glaucoma, but also in individuals with open-angle glaucoma or ocular hypertension.

166 intraocular pressure (IOP) in patients with open-angle glaucoma or ocular hypertension; however, the

167 r results suggest that patients with primary open-angle glaucoma or with narrow angles or chronic ang

168 e elevated in surgical patients with primary open-angle glaucoma (OR, 1.48; 95% CI, 1.08-2.01).

169 I, 2.09-1.11;P = 0.02), higher prevalence of open-angle glaucoma (OR, 2.40; 95% CI, 1.36-4.23;P = 0.0

170 I, 1.31-10.13; P = .01), presence of primary open-angle glaucoma (OR, 3.82; 95% CI, 1.60-9.14; P = .0

171 beculectomy surgery in patients with primary open angle glaucoma over a 3-year period of follow-up.

172 ), more glaucoma suspects (p < 0.0001), more open angle glaucoma (p = 0.0006) and fewer other conditi

173 of glaucoma (P < .0001), and a diagnosis of open-angle glaucoma (P = .0003) were associated with inc

174 ithin different types of glaucoma, including open-angle glaucoma (P = 0.36), chronic angle closure gl

175 investigation of both childhood and juvenile open-angle glaucoma, particularly when associated with i

176 ve, observational cohort study that included open angle glaucoma patients with visually significant c

177 At baseline following washout, open-angle glaucoma patients (n = 75) were administered

178 Open-angle glaucoma patients may have increased odds of

179 olled and treated more strictly than primary open-angle glaucoma patients to avoid visual field deter

180 erived from non-glaucomatous donors and from open-angle glaucoma patients were used to determine the

181 Open-angle glaucoma patients without prior incisional gl

182 rgets after a first failed trabeculectomy in open-angle glaucoma patients.

183 d progression in exfoliation than in primary open-angle glaucoma patients.

184 in a dose-dependent IOP increase in treated open-angle glaucoma patients.

185 of TGF-beta2 has been found in many primary open-angle glaucoma patients.

186 gression in new-diagnosed exfoliation versus open-angle glaucoma patients.

187 The study included patients with primary open angle glaucoma (POAG group, n = 30) and controls (n

188 12 normal controls, 7 patients with primary open angle glaucoma (POAG) and 9 patients with normal te

189 OPTN) are linked to the pathology of primary open angle glaucoma (POAG) and amyotrophic lateral scler

190 is study was designed to evaluate if primary open angle glaucoma (POAG) and its severity are associat

191 les frequencies in primary glaucoma [primary open angle glaucoma (POAG) and primary angle closure gla

192 oci have been associated with either primary open angle glaucoma (POAG) or heritable ocular quantitat

193 eous humor as well as circulation of primary open angle glaucoma (POAG) patients.

194 s in a population of newly-diagnosed primary open angle glaucoma (POAG) patients.

195 Control TM and most primary open angle glaucoma (POAG) TM were collected from cadave

196 beagle model of autosomal recessive primary open angle glaucoma (POAG) to a 4-Mb interval on chromos

197 and combined screening for PACG and primary open angle glaucoma (POAG) to evaluate costs and benefit

198 of 30.8 months, including 28 (4.4%) primary open angle glaucoma (POAG), 27 (4.2%) primary angle clos

199 jects were glaucomatous: (67 PXG, 42 Primary Open Angle Glaucoma (POAG), 28 PACG, 14 Normal Tension G

200 Primary open angle glaucoma (POAG), a major cause of blindness w

201 defects also occur in patients with primary open angle glaucoma (POAG), in which there is specific R

202 ic or glaucoma faculty patients with primary open angle glaucoma (POAG), ocular hypertension (OHTN),

203 n the aqueous humor of patients with primary open angle glaucoma (POAG), pseudoexfoliation syndrome (

204 ied 61 patients (80 procedures) with primary open angle glaucoma (POAG).

205 essure (IOP) sensor in patients with primary open angle glaucoma (POAG).

206 cells is known to be associated with primary open angle glaucoma (POAG).

207 A total of 47 patients with primary open-angle glaucoma (POAG) and 36 normal participants we

208 yes of 75 patients with uncontrolled primary open-angle glaucoma (POAG) and cataract who underwent PK

209 med on 27 consecutive eyes with both primary open-angle glaucoma (POAG) and cataract; each eye was tr

210 (AD) and European descent (ED) with primary open-angle glaucoma (POAG) and control participants comp

211 ssociation of known loci for IOP and primary open-angle glaucoma (POAG) and identify four new IOP-ass

212 meshwork (TM) height differs between primary open-angle glaucoma (POAG) and primary angle-closure gla

213 ly examined the global prevalence of primary open-angle glaucoma (POAG) and primary angle-closure gla

214 s) endophthalmitis and risk of a new primary open-angle glaucoma (POAG) diagnosis within 365 days aft

215 ses (TIMPs) in the aqueous humour of primary open-angle glaucoma (POAG) eyes have been described.

216 a genome-wide association study for primary open-angle glaucoma (POAG) in 1,007 cases with high-pres

217 culoplasty (SLT) as sole therapy for primary open-angle glaucoma (POAG) in an Afro-Caribbean populati

218 k, CA) for treating mild-to-moderate primary open-angle glaucoma (POAG) in patients undergoing catara

219 Primary open-angle glaucoma (POAG) is a blinding disease.

220 Primary open-angle glaucoma (POAG) is a highly prevalent conditi

221 Primary open-angle glaucoma (POAG) is a leading cause of blindne

222 Primary open-angle glaucoma (POAG) is a leading cause of irrever

223 Primary open-angle glaucoma (POAG) is a leading cause of irrever

224 Primary open-angle glaucoma (POAG) is a major cause of blindness

225 Primary open-angle glaucoma (POAG) is a major cause of irreversi

226 Primary open-angle glaucoma (POAG) is a progressive neurodegener

227 ract surgery on IOP in patients with primary open-angle glaucoma (POAG) is a subject of debate.

228 Primary open-angle glaucoma (POAG) is the most common form of gl

229 ctive cross-sectional study: 68 mild primary open-angle glaucoma (POAG) patients according to the Hod

230 venous pressure (RVP) in the eyes of primary open-angle glaucoma (POAG) patients and healthy subjects

231 difference in severity of disease in primary open-angle glaucoma (POAG) patients with a Myocilin (MYO

232 itary determination) was examined in primary open-angle glaucoma (POAG) patients with cataract and no

233 g can cause fibrosis of the TM as in primary open-angle glaucoma (POAG) patients, and is characterize

234 s in ocular hypertensive (OH) and in primary open-angle glaucoma (POAG) patients.

235 American Academy of Ophthalmology's Primary Open-angle Glaucoma (POAG) Preferred Practice Pattern (P

236 We studied 1 eye of 63 primary open-angle glaucoma (POAG), 30 ocular hypertension (OH),

237 egion previously was associated with primary open-angle glaucoma (POAG), although replication among i

238 the 4 forms of open-angle glaucoma: primary open-angle glaucoma (POAG), normal-tension glaucoma (NTG

239 stent was implanted in patients with primary open-angle glaucoma (POAG), normal-tension glaucoma (NTG

240 Primary open-angle glaucoma (POAG), the most common optic neurop

241 A prior clinic-based study of primary open-angle glaucoma (POAG)-related DH showed that they h

242 nction, which has been implicated in primary open-angle glaucoma (POAG).

243 low autoregulation are implicated in primary open-angle glaucoma (POAG).

244 a secondary glaucoma that resembles primary open-angle glaucoma (POAG).

245 at PMH use may decrease the risk for primary open-angle glaucoma (POAG).

246 TM, similar to those associated with primary open-angle glaucoma (POAG).

247 ry in subjects with mild to moderate primary open-angle glaucoma (POAG).

248 ODH) before and after development of primary open-angle glaucoma (POAG); determine the prognostic sig

249 ce interval, 3.3-5.4), consisting of primary open-angle glaucoma (POAG, 3.2%, including high-tension

250 -2013) to estimate effects of T2D on primary open-angle glaucoma (POAG; 3,554 cases).

251 [CRF], cup-to-disc ratio [CDR], and primary open-angle glaucoma [POAG]).

252 ELINES: Evidence-based update of the Primary Open-Angle Glaucoma Preferred Practice Pattern(R) (PPP)

253 PRIMARY OPEN-ANGLE GLAUCOMA PREFERRED PRACTICE PATTERN(R) GUIDEL

254 Participants were patients with open angle glaucoma presenting with advanced glaucoma in

255 Primary open-angle glaucoma presents with increased prevalence a

256 closure glaucoma (PACG); 1 of the 4 forms of open-angle glaucoma: primary open-angle glaucoma (POAG),

257 he OMNI system in pseudophakic patients with open-angle glaucoma provides effective IOP reduction or

258 glandin analog eye drops in treating primary open-angle glaucoma, published between December 2000 and

259 0.77-0.80) and with increases in the odds of open-angle glaucoma ranging from 1.23 (95% CI, 1.20-1.26

260 involved diabetic macular edema and no prior open-angle glaucoma, repeated intravitreous injections o

261 PEX glaucoma is a common, aggressive form of open-angle glaucoma resulting from the deposition of fib

262 Patients with open-angle glaucoma (Shaffer Grade 3 and 4) and uncontro

263 and primary open-angle glaucoma vs secondary open-angle glaucoma (SOAG) (HR 2.51; 95% CI 1.01-6.23) r

264 In open-angle glaucoma, substantial RNFL thinning or struct

265 PRIMARY OPEN-ANGLE GLAUCOMA SUSPECT PREFERRED PRACTICE PATTERN(R

266 osis and management of patients with primary open-angle glaucoma suspect with detailed recommendation

267 h age, and particularly in eyes with primary open angle glaucoma, the number of cells residing within

268 In 57 patients with primary open-angle glaucoma, the IOP decreased by 7.7 mmHg (stan

269 Risk factors for primary open-angle glaucoma-the most common form of glaucoma-inc

270 or cataract surgery is 4500% and for medical open-angle glaucoma therapy is 4000%.

271 Open-angle glaucoma treatment costs and effects were pro

272 etrospective case series of 81 patients with open-angle glaucoma undergoing 110 SLT procedures from N

273 Lucia with medically treated primary open-angle glaucoma underwent a 30-day washout, followed

274 tio [HR] 2.51; 95% CI 1.12-5.65) and primary open-angle glaucoma vs secondary open-angle glaucoma (SO

275 The prevalence of primary open-angle glaucoma was 6.8% overall, increasing from 3.

276 mary glaucoma that had gonioscopy (n = 243), open-angle glaucoma was more common (86 %) than angle-cl

277 Primary open-angle glaucoma was the underlying diagnosis in 342

278 ation number, ISRCTN96423140), patients with open-angle glaucoma were allocated to receive latanopros

279 Exfoliation and open-angle glaucoma were defined based on the European G

280 tal of 607 participants with newly diagnosed open-angle glaucoma were enrolled at 14 clinical centers

281 s) with ocular hypertension or early primary open-angle glaucoma were enrolled.

282 Two hundred and six patients with open-angle glaucoma were examined at 4-month intervals w

283 Thirty-four eyes of 27 patients with open-angle glaucoma were included.

284 study, healthy individuals and patients with open-angle glaucoma were prospectively enrolled between

285 as associated with increased risk of primary open-angle glaucoma when all data were included in a met

286 itivity sometimes increases in patients with open-angle glaucoma when intraocular pressure (IOP) is d

287 ary 1, 2013, including patients with primary open-angle glaucoma who had a best-corrected visual acui

288 ial for patients with medically uncontrolled open-angle glaucoma who have previously received 360 deg

289 252 eyes from 198 adult patients with open-angle glaucoma who underwent SLT between July 2016

290 tudy of the Cypass micro-stent) with primary open-angle glaucoma who were using 0-4 classes of topica

291 otal of 603 patients (603 eyes) with primary open-angle glaucoma who were using up to 3 glaucoma medi

292 ophotographs from 1 eye of 125 patients with open-angle glaucoma with >/=8 reliable Swedish interacti

293 The second cohort included all stages of open-angle glaucoma with >=5 OCT retinal nerve fiber lay

294 analysis included 186 patients with primary open-angle glaucoma with a mean age of 59.1 years (range

295 g was performed in 117 patients with primary open-angle glaucoma with a minimum treatment duration of

296 osis and management of patients with primary open-angle glaucoma with an algorithm for patient manage

297 quality of life among patients with primary open-angle glaucoma with structural macular retinal gang

298 (XFS), the most common recognizable cause of open-angle glaucoma worldwide, is a systemic disorder wi

299 FS) is the most common recognizable cause of open-angle glaucoma worldwide.

300 as various ocular implications, most notably open angle glaucoma, zonular abnormalities, and cataract