ライフサイエンスコーパス: opiate dependence

1 reduction in connectivity strength following opiate dependence.

2 he severe withdrawal symptoms resulting from opiate dependence.

3 mechanisms may modulate opiate tolerance and opiate dependence.

4 as the extended amygdala, may play a role in opiate dependence.

5 ay represent an important neuroadaptation in opiate dependence.

6 mphetamine dependence, and 368 subjects with opiate dependence.

7 athway in the LC that contribute to physical opiate dependence.

8 in synaptic transmission thought to underlie opiate dependence.

9 minimizing, or reversing the development of opiate dependence.

10 mu-opioid receptor agonist used for treating opiate dependence.

11 disorder (ADHD), 20 subjects with alcohol or opiate dependence, 18 subjects with depression, and 31 c

12 treatment with the development of functional opiate dependence and appeared to arise at a step after

13 nocortin peptides are reported to antagonize opiate dependence and tolerance, but the neural substrat

14 Once opiate dependence and withdrawal has developed, a functi

15 ioral and neural adaptations associated with opiate dependence and withdrawal.

16 h; this treatment has been shown to produce opiate dependence and withdrawal.

17 treatment contributing to the expression of opiate dependence and withdrawal.

18 te for the LC's involvement in mechanisms of opiate dependence and withdrawal.

19 us accumbens (NAcc) may play a major role in opiate dependence, and central NMDA receptors are report

20 the thalamus to nucleus accumbens circuit to opiate dependence, and suggests that reprogramming this

21 ever, the mechanisms that lead to preventing opiate dependence are still poorly understood.

22 of the model as well as the hypothesis that opiate dependence at least partially requires protein sy

23 Consistent with our model of opiate dependence, beta-gal expression increased in the

24 vailable for treating nicotine, alcohol, and opiate dependence but not stimulant or marijuana depende

25 72.0% of the patients (and preceded onset of opiate dependence in 44.4%).

26 ecently compared the time course of onset of opiate dependence in laboratory animals, with the mathem

27 icantly more comorbidity with alcohol and/or opiate dependence, major depression, and physical disord

28 alterations in opioid peptide levels during opiate dependence may contribute to the observed hyperac

29 y and induces signs of cellular and physical opiate dependence that endure after the stress.

30 on with nicotine dependence and linkage with opiate dependence, these data support roles for NRXN3 ha

31 denosine A1 receptors and created a state of opiate dependence, was used in this current study.

32 f these adult DG NSCs has been implicated in opiate dependence, whether NSC neuronal differentiation

33 Chronic opiate use induces opiate dependence, which is characterized by extremely u