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1 lion cells with uncrossed projections at the optic chiasm.
2 mbryonic brain during formation of the mouse optic chiasm.
3 of cell groups that lie within the nerve and optic chiasm.
4 rve outgrowth, including path-finding at the optic chiasm.
5 commissure, which is located adjacent to the optic chiasm.
6  olfactory tract, mammillothalamic tract, or optic chiasm.
7 l axon divergence associated with the albino optic chiasm.
8  days after birth (P24) at the centre of the optic chiasm.
9 amage to the visual pathway posterior to the optic chiasm.
10 DF1) is required for axon segregation at the optic chiasm.
11 halmia and hypoplasia of the optic nerve and optic chiasm.
12 equired for efficient RGC decussation at the optic chiasm.
13 athfinding of the ganglion cell axons at the optic chiasm.
14 ultimately affects axonal growth through the optic chiasm.
15 s a missorting of RGC axons as they exit the optic chiasm.
16 ome biogenesis and axonal growth through the optic chiasm.
17 lmic disorders affecting the optic nerve and optic chiasm.
18 tes, and anomalous axonal pathfinding at the optic chiasm.
19  axial planes 1-mm thick and parallel to the optic chiasm.
20 aplastic haemangiopericytoma compressing the optic chiasm.
21 with the RGCs themselves, most likely at the optic chiasm.
22 nas still project axons to the brain via the optic chiasm.
23 ltered in the developing Foxg1-/- retina and optic chiasm.
24               Foxg1 is also expressed at the optic chiasm.
25 r contralateral targets, thereby forming the optic chiasm.
26 ral diencephalon during the formation of the optic chiasm.
27 ws from the ventrotemporal retina toward the optic chiasm.
28 ons either cross or avoid the midline at the optic chiasm.
29 ial manifestation of disorders involving the optic chiasm.
30 ons either cross or avoid the midline at the optic chiasm.
31 A/OPCs isolated from cortex, optic nerve and optic chiasm.
32 on for retinal axon growth in the developing optic chiasm.
33 pond by making axonal guidance errors at the optic chiasm.
34 knockout mice and analyzed their retinas and optic chiasms.
35 ecision to cross or avoid the midline at the optic chiasm, a critical guidance maneuver that establis
36                             Thus, within the optic chiasm, a sequence of positional transformations o
37 e retina and in the region of the developing optic chiasm, a ventral midline structure in which retin
38 erns of heparan sulfation on RGCs and at the optic chiasm and (2) this differential sulfation directs
39 estigated with reference to disorders of the optic chiasm and anophthalmia (absence of the eyes).
40 s RGC axons exhibited normal crossing at the optic chiasm and fasciculation of the optic nerve.
41 axon guidance factors, and the absence of an optic chiasm and forebrain commissures.
42  their predetermined crossing pattern in the optic chiasm and grew to the ipsilateral LGN.
43 rocesses, such as pathfinding of RGCs at the optic chiasm and hippocampal long-term potentiation and
44 th a focus on axon guidance signaling at the optic chiasm and ipsi- and contralateral axon organizati
45 (RGC) fibers affects the organization of the optic chiasm and lateral geniculate nuclei (LGN) in huma
46  of the optic nerves, chiasm and tracts, and optic chiasm and LGN volume compared with controls (P <
47  directly control RGC axon divergence at the optic chiasm and may additionally function as a general
48 etain-->and markedly reduced the size of the optic chiasm and optic nerves.
49 llele reduced axonal midline crossing at the optic chiasm and optic tract fasciculation.
50                     Between E30 and E35, the optic chiasm and optic tract remain acellular, but the l
51                                          The optic chiasm and splenium of the corpus callosum were tr
52                It is dorsally bounded by the optic chiasm and the alar hypothalamus, and caudally by
53                         We have examined the optic chiasm and the retina in albino and normally pigme
54 lateral and contralateral projections at the optic chiasm and the subsequent segregation of retinal i
55                     Axon organization in the optic chiasm and tract and RGC growth cone morphologies
56 , the body of the optic stalk and nerve, the optic chiasm and ventral diencephalon, and the anterior
57 sal and temporal retinal fibers cross at the optic chiasm, and (2) ocular dominance columns normally
58 ovided identical tracing of the optic nerve, optic chiasm, and optic tracts to the level of the later
59 ptic stalk, cross the ventral midline at the optic chiasm, and terminate in the optic tectum of the z
60 spinal cord, the hindbrain and midbrain, the optic chiasm, and the median eminence in the forebrain.
61 n in significant numbers and fail to form an optic chiasm; and (4) axons in multiple commissural trac
62 n, the mechanisms for axon divergence in the optic chiasm are discussed in the context of other popul
63 silateral and contralateral RGC axons at the optic chiasm, are natural candidates for contributing to
64 tricular and subventricular zones and in the optic chiasm, areas that are rich in oligodendrocyte (OL
65  in determining the relative position of the optic chiasm at the ventral midline of the developing hy
66                                       At the optic chiasm, axons from either eye meet and decide whet
67 e, loss of axonal staining progressed to the optic chiasm by 7 days and remained undetectable at 2 we
68 hen GAP-43-deficient axons are cultured with optic chiasm, cortical, or dorsal midbrain cells.
69 e and in the pattern of decussation at their optic chiasm, demonstrating that a melanin-related agent
70 nal ganglion cell (RGC) axons at the midline optic chiasm determines whether RGCs project to ipsilate
71 d the alternative CXCL12 receptor ACKR3, the optic chiasm developed normally in mice lacking ACKR3.
72 ription factor known for its role in eye and optic chiasm development, causes the rostral oral ectode
73           Here, we review recent findings on optic chiasm development, highlighting the specific tran
74                Electrical stimulation of the optic chiasm elicits reduced calcium transients and impa
75 ), and cross the diencephalon midline at the optic chiasm en route to their brain targets.
76 e on the ventral surface of the brain at the optic chiasm for sorting into the optic tracts.
77 nal axon growth and/or reorganization during optic chiasm formation.
78 he developing ventral diencephalon where the optic chiasm forms.
79 e site in the ventral diencephalon where the optic chiasm forms.
80 is absent from the ventral midline where the optic chiasm forms.
81 the midline, thus forming the bodily crossed optic chiasm found in fish.
82                     Optic nerve from EAE and optic chiasm from MS also showed decreased cholesterol s
83 due to the close anatomical proximity to the optic chiasm, hypothalamus and pituitary gland.
84 al axons and the cellular composition of the optic chiasm in albino mice are similar to those of norm
85 Vema is localized to the floor plate and the optic chiasm, intermediate targets located at the ventra
86 nal ganglion cell (RGC) axon growth from the optic chiasm into the optic tract are unknown.
87 GC) axons fail to progress normally from the optic chiasm into the optic tracts.
88                                    The mouse optic chiasm is a model for axon guidance at the midline
89                                          The optic chiasm is an important choice point at which retin
90             Interestingly, expression in the optic chiasm is high at postnatal day 6, but decreases w
91                     In affected animals, the optic chiasm is missing, and each retina projects entire
92 iculate nucleus (dLGN), when crossing at the optic chiasm midline is altered.
93 axons away from its ligand, ephrinB2, at the optic chiasm midline, and a transcription factor Zic2, t
94 cell (RGC) axons from nasal retina cross the optic chiasm midline, whereas temporal retina axons do n
95 o regulate RGC axon repulsion by cues at the optic chiasm midline.
96 at directs the ipsilateral projection at the optic chiasm, misrouted RGCs target the appropriate reti
97 ation associated with axonal behavior at the optic chiasm must affect ganglion cells in a cell-extrin
98 in the diencephalic preoptic area, where the optic chiasm normally forms.
99 ordinates for the optic nerve head (ONH) and optic chiasm (OC) ends of the optic nerve were recorded
100 (WM) tracts and in the corpus callosum (CC), optic chiasm (Och), and internal capsule.
101                                       In the optic chiasm of mammals, axons either cross the midline
102                                          The optic chiasm of marsupials differs from that of the euth
103 Brn3b(-/-) mice but missing were entirely in optic chiasms of Brn3b/Brn3c double knockout mice, sugge
104 Retinal axons cross the neuraxis to form the optic chiasm on the hypothalamus in a position defined b
105 lved both in determining the position of the optic chiasm on the ventral diencephalon (presumptive hy
106 ) project axons along the optic nerve to the optic chiasm on the ventral surface of the hypothalamus.
107                The core is located above the optic chiasm, receives primary and secondary visual affe
108 on of Vema in the developing spinal cord and optic chiasm resembles the expression patterns of a vari
109                                       At the optic chiasm, retinal ganglion cell (RGC) axons make the
110                                       At the optic chiasm, retinal ganglion cell axons from each eye
111                                       At the optic chiasm, retinal ganglion cells (RGCs) project ipsi
112 sm, and patients with tumors compressing the optic chiasm should be referred to an ophthalmologist fo
113         We found that glutamate agonists and optic chiasm stimulation inhibit serotonergic phase adva
114 ents this increase, abolishes glutamate- and optic chiasm stimulation-induced phase delays of the SCN
115 al hypothalamus, and in a site dorsal to the optic chiasm that included the suprachiasmatic nucleus.
116 nd therapeutic interventions that damage the optic chiasm, the pituitary stalk and the hypothalamic a
117                       The second site is the optic chiasm, the site of retinal axon divergence.
118 abnormal retinal decussation patterns at the optic chiasm: their uncrossed projections are smaller an
119              RGC axons decussate to form the optic chiasm, then grow to targets in the thalamus and m
120 ficient mice initially fail to grow from the optic chiasm to form optic tracts and are delayed tempor
121 ganglion cell (RGC) axons diverge within the optic chiasm to project to opposite sides of the brain.
122 whether to cross or avoid the midline at the optic chiasm to project to targets on both sides of the
123 s with ephrin-B2 on radial glia cells at the optic chiasm to repulse VT axons away from the midline a
124  agonists, and electrical stimulation of the optic chiasm to SCN brain slices to determine the effect
125  of retinal ganglion cell (RGC) axons at the optic chiasm to the appropriate hemisphere, a pattern cr
126 ding from the area immediately caudal to the optic chiasm to the level of the posterior hypothalamus.
127 sion that retinal ganglion cells make at the optic chiasm, to either cross or avoid the midline.
128                  Morphologic analysis of the optic chiasm was based on manual measurement of regions
129 ct retinal axon growth and divergence at the optic chiasm, we cocultured mouse retinal and chiasm exp
130 Ipsilateral and misrouted projections at the optic chiasm were overproduced in Brn3b(-/-) mice but mi
131 ls, some axons from each retina cross at the optic chiasm, whereas others do not.
132           RGC axon segregation occurs at the optic chiasm, which forms at the ventral diencephalon mi
133 eview, we compare guidance mechanisms at the optic chiasm with those in other midline models and high
134 rgence of retinal ganglion cell axons at the optic chiasm, with strictly controlled numbers projectin
135  nerve regeneration, with axons reaching the optic chiasm within 3 wk.
136 embryos deficient in GAP-43 have an enlarged optic chiasm within which RGC axons were reportedly stal

 
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