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1  at the optic disc through patterning of the optic stalk.
2 ng neural retina and ventricular zone of the optic stalk.
3  of the PAX6 domain from the retina into the optic stalk.
4 c epithelium and move toward and through the optic stalk.
5 th highest levels of expression in the nasal optic stalk.
6 as in wild type, but are unable to enter the optic stalk.
7 ure as it forms in the ventral optic cup and optic stalk.
8  neural crest cells to accumulate around the optic stalk, a source of the ligand Pdgfaa.
9             As Pitx2 is not expressed in the optic stalk, an essential function of PITX2 protein in n
10 f the vertebrate optic vesicle into proximal/optic stalk and distal/neural retina involves midline-de
11                     The RBG originate in the optic stalk and have been thought to migrate into the ey
12 t8b expression upregulated in the Foxg1(-/-) optic stalk and hypothesized that, similar to what is ob
13 ived factor-1 (SDF1) is expressed within the optic stalk and its receptor CXCR4 is expressed in retin
14 d fissure of the optic disk, the body of the optic stalk and nerve, the optic chiasm and ventral dien
15 ression was also noted within the developing optic stalk and optic disk.
16  the Pax2+ cells in the embryonic retina and optic stalk and the initial misrouting of the ganglion c
17 e optic disk (OD), the interface between the optic stalk and the neuroretina.
18 e that arise from precursors in the eye-disc/optic stalk and those that arise from precursors in the
19 ) signaling ventralize the eye, by expanding optic stalk and ventral retina, and repressing dorsal re
20 ing domains encompassing the ventral retina, optic stalks and preoptic area.
21 optic cup, including the pigment epithelium, optic stalk, and ciliary body.
22 ystem, slit2 is expressed in the eye, in the optic stalk, and in the ventral diencephalon.
23 the ventral portion of the retina and in the optic stalk, and the ligands and binding proteins locali
24 nal pigment epithelium (RPE), the optic disk/optic stalk, and the pecten oculi.
25 d expression signatures of early optic-disc, optic-stalk, and RGCs.
26 oma leading to the misdifferentiation of the optic stalk as retina, which becomes continuous with the
27           A frequently observed defect is an optic stalk coloboma leading to the misdifferentiation o
28               During normal development, the optic stalks constrict, decreasing in width and are grad
29  ganglion cell axons exit the eye, enter the optic stalk, cross the ventral midline at the optic chia
30 pression of PKI partially rescues somite and optic stalk defects in no tail and cyclops mutants that
31 ndependent of the effects of Hh signaling on optic stalk development.
32                               The defects in optic-stalk differentiation correlate with reduced sonic
33 ut later their axons are unable to enter the optic stalk en route to the brain and continue to projec
34 cts in optic stalk morphogenesis whereby the optic stalk extends into the retina and impedes the late
35  to medial instead of bilateral induction of optic stalks followed by a partial fusion of the eyes at
36 mus, and are implicated in the regulation of optic stalk formation, whereas loss of Fgf3 alone result
37 inding and show that Shh acts to pattern the optic stalk in zebrafish but does not guide RGC axons in
38 G) is a group of glia that migrates from the optic stalk into the third instar larval eye disc while
39 entral identities, neural retina and ventral optic stalk is also compromised.
40 tructure arising from the optic vesicle, the optic stalk, is missing and is replaced by an expanded r
41 elium-like tissue, and ectopic expression of optic stalk markers in the region of the ventral retina
42             Colobomas result from defects in optic stalk morphogenesis whereby the optic stalk extend
43 lial cells that line the choroid fissure and optic stalk/nerve to its junction with the optic tract.
44 ddition, we show that the attenuation of the optic stalk occurs in parallel with ganglion cell differ
45 ntify upregulated expression of Wnt8b in the optic stalk of Foxg1(-/-) mutants before OF closure init
46 sis of these fields generates telencephalon, optic-stalk, optic-disc, and neuroretina along a spatial
47 omprising concentric zones of telencephalic, optic-stalk, optic-disc, and neuroretinal tissues along
48                     The diencephalon-derived optic stalk (OS) and neural retina are also patterned in
49 igate across the retina, exit the eye to the optic stalk (OS), and cross the diencephalon midline at
50                                          The optic stalk represents the ventral-most region of the ea
51     (3) A few glia close to the entry of the optic stalk suffice to guide the axons into the stalk, s
52 e and abnormal differentiation of the dorsal optic stalk; the development of proximo-ventral identiti
53                                 While in the optic stalk, they grow immediately adjacent to cells exp
54 in the absence of photoreceptor axons in the optic stalk; they also migrate to ectopic patches of dif
55 naling from the pharyngeal arch endoderm and optic stalk to Cxcr4a expressing CNCCs is important for
56 ganglion cell axons within the retina to the optic stalk to exit the retina.
57 s on Wnt8b suppression by Foxg1 in the nasal optic stalk to maintain balanced apoptosis and Pax2 expr
58 x2+ cells in the posterior optic cup and the optic stalk undergo abnormal morphogenetic movements and
59 mic high RA levels cause an expansion in the optic stalk with an increased cell content and a patent