ライフサイエンスコーパス: oral mucosa

1 sbronchial, 27 lymph node, two skin, and two oral mucosa).

2 ve wound-healing phenotypes seen in skin and oral mucosa.

3 lation in tumor samples compared with normal oral mucosa.

4 and applications of tissue-engineered human oral mucosa.

5 >/=1 case of ligneous disease involving the oral mucosa.

6 cosa is more permissive to invasion than the oral mucosa.

7 escence and impaired re-epithelialization of oral mucosa.

8 red oral mucosa closely resembles the normal oral mucosa.

9 and genitourinary tracts in addition to the oral mucosa.

10 but most patients experienced thickening of oral mucosa.

11 the C-terminal domain of DP in the skin and oral mucosa.

12 sible that recolonization may occur from the oral mucosa.

13 e organized similarly to the cells in native oral mucosa.

14 pared with endothelial cells from the normal oral mucosa.

15 chronic, painful, ulcerative lesions of the oral mucosa.

16 mal in the tonsil epithelium, in contrast to oral mucosa.

17 s an important opportunistic pathogen in the oral mucosa.

18 apical region of teeth or perforation of the oral mucosa.

19 anism of invasion of certain bacteria in the oral mucosa.

20 mal papillomaviruses that infect the skin or oral mucosa.

21 hat all of the fragments were present in the oral mucosa.

22 s known of the immune system that serves the oral mucosa.

23 were taken of both expanded and non-expanded oral mucosa.

24 ) and its receptor (uPAR) relative to normal oral mucosa.

25 a basic mechanism of immunoregulation in the oral mucosa.

26 rized by blisters and erosive lesions in the oral mucosa.

27 tobacco smoke on the expression of COX-2 in oral mucosa.

28 erifying the critical function of A20 in the oral mucosa.

29 nd angiogenesis might be observed in healing oral mucosa.

30 risk factors for SCC of the skin and of the oral mucosa.

31 ence assay directly in KCs comprising murine oral mucosa.

32 unoregulatory cytokines are generated in the oral mucosa.

33 Punch biopsies were taken of the expanded oral mucosa.

34 icles, in filiform and fungiform papillae of oral mucosa.

35 invasive squamous carcinomas of the skin and oral mucosa.

36 and histologically dysplastic lesions of the oral mucosa.

37 is a key feature of the privileged repair of oral mucosa.

38 ht into the mechanisms of EBV persistence in oral mucosa.

39 and characterized its expression in skin and oral mucosa.

40 rable early wound closure characteristics of oral mucosa.

41 th pathogenic SIVmac251 administered through oral mucosa.

42 activation in both oral SCC cells and intact oral mucosa.

43 dogs from high-dose viral infection of their oral mucosa.

44 ation, pain, and bleeding of the gingiva and oral mucosa.

45 ns is both a commensal and a pathogen at the oral mucosa.

46 and a different keratinization pattern than oral mucosa.

47 nsformation of epithelial tissues, including oral mucosa.

48 nd to be expressed throughout the airway and oral mucosa.

49 ial for short-term colonization of the mouse oral mucosa.

50 alter the persistence of streptococci on the oral mucosa.

51 factors (i.e., mechanical irritation) in the oral mucosa.

52 specificity tested against bovine and human oral mucosa.

53 esentation of these diseases on the skin and oral mucosa.

54 immunoinflammatory diseases of the skin and oral mucosa.

55 ustained release to controlled depths in the oral mucosa.

56 st common malignant tumor that occurs in the oral mucosa.

57 than the amount of compound adsorbed by the oral mucosa.

58 ion against virus infection initiated at the oral mucosa.

59 the periodontal ligament, the cementum, and oral mucosa.

60 ion in full-thickness surgical wounds on rat oral mucosa.

61 antly impaired neutrophil recruitment to the oral mucosa.

62 acent tissues such as the salivary gland and oral mucosa.

63 ected are presumably epithelial cells of the oral mucosa.

64 olves the study of cells exfoliated from the oral mucosa.

65 in gag sequences derived from the blood and oral mucosa.

66 d three-dimensional (3D) models of the human oral mucosa.

67 ysis done on oral cancers showed that normal oral mucosa (100%, 12 of 12) and 69.1% (47 of 68) of dys

68 Aome) as compared to their low expression in oral mucosa (2.87%).

69 forearm skin (64.4%), eyebrow hairs (60.9%), oral mucosa (35.6%), and anal mucosa (33.3%).

70 n) than across the 2 mucosal sites (anal and oral mucosa, 6.9%).

71 The main adverse effects were thickening of oral mucosa (72% in the palifermin group vs. 31% in the

72 ), HPV-12 (beta-1) in forearm skin (23%) and oral mucosa (9.2%), and HPV-76 (beta-3) in anal mucosa (

73 for host defense against C. albicans at the oral mucosa, a recent immunohistochemical evaluation of

74 ed fifty-three biopsies of the supra-implant oral mucosa adjacent to the cover screw of submerged den

75 tudy is to evaluate tissue response of human oral mucosa adjacent to titanium cover screws.

76 or tight adhesion of the whole system to the oral mucosa after application.

77 Most patients with hyperpigmentation of the oral mucosa also had melanonychia.

78 only in superficial epithelial cells of the oral mucosa, although latent proviruses are found in mos

79 al inflammation and ulceration of the caudal oral mucosa, alveolar and buccal mucosa, and varying deg

80 We have systematically examined oral mucosa among cGVHD patients and determined that the

81 on abiotic surfaces or on the surface of an oral mucosa analogue.

82 vations were extended to an engineered human oral mucosa and an in vivo rat model of catheter-associa

83 eated or glacial acetic acid-treated porcine oral mucosa and analyzed via CNN to determine if this me

84 and neck squamous cell carcinoma and normal oral mucosa and annotated gene expression levels to spec

85 Immunofluorescence analysis of oral mucosa and atherosclerotic plaques demonstrate infi

86 based on polymerase chain reaction from his oral mucosa and cutaneous vesicle fluid.

87 es that were consistently upregulated in the oral mucosa and demonstrated that induction of one of th

88 y of new alpha-defensins from rhesus macaque oral mucosa and determine the first alpha-defensin struc

89 nd other strategies to treat diseases of the oral mucosa and digestive tract.

90 We apply this technique to the biome of the oral mucosa and find that greater than 25% of recovered

91 ium implant was inserted into the engineered oral mucosa and further cultured to establish epithelial

92 unique population of MSCs derived from human oral mucosa and gingiva, especially their immunomodulato

93 ch might allow profilin penetration into the oral mucosa and induction of local inflammation.

94 e of different dendritic cell subsets in the oral mucosa and its diseases.

95 regulated in OSCC tissues compared to normal oral mucosa and low levels of SGPL1 mRNA correlated with

96 ophils are essential for host defense at the oral mucosa and neutropenia or functional neutrophil def

97 unique features of cellular structure in the oral mucosa and palatine tonsils, the high rate of oral

98 ulted in elevated levels of cytokines in the oral mucosa and plasma of the SIV-infected macaques.

99 ivo, thereby preserving the integrity of the oral mucosa and protecting from radiation-induced mucosi

100 g ex vivo experiments using a novel model of oral mucosa and saliva as well as in vivo dynamic instru

101 the virus to invade epithelial cells of the oral mucosa and salivary gland ducts via ACE2 receptors.

102 ing to intercellular adhesion defects in the oral mucosa and skin.

103 e production in wounds of equivalent size in oral mucosa and skin.

104 omedullin may serve a different functions in oral mucosa and skin.

105 several stratified epithelia, including the oral mucosa and skin.

106 tes and may have an immunoregulatory role in oral mucosa and skin.

107 for murine regenerative wound healing in the oral mucosa and sufficient to limit facial skin fibrosis

108 Yet, the function of A20 in the oral mucosa and the biological pathways in which A20 mit

109 le and in vitro adhesive properties, both to oral mucosa and to teeth surface, were obtained with a b

110 CO2 laser irradiation on biopsies of porcine oral mucosa and underlying bone under conditions that si

111 es from postcapillary venules in the in situ oral mucosa and, if so, whether bradykinin mediated this

112 affect oral hygiene, diagnosis, gingival and oral mucosa, and alveolar bone.

113 ifferentiating epithelial cells of the skin, oral mucosa, and gut, expression is consistently up-regu

114 ntial structural role for K6 isoforms in the oral mucosa, and implicate filiform papillae as being th

115 ctival hyperemia, hemorrhagic erosion of the oral mucosa, and multiple flat atypical targets were pre

116 artments represented by the salivary glands, oral mucosa, and palatine tonsils.

117 dystrophic lesions affecting nails, glands, oral mucosa, and palmar-plantar epidermis.

118 e from the basement membrane of conjunctiva, oral mucosa, and skin.

119 (32% of primary tumors) compared with normal oral mucosa, and that expression correlated significantl

120 al carcinoma-derived cell lines of the skin, oral mucosa, and urogenital tract.

121 of stable stem-cell-like phenotypes, render oral mucosa- and gingiva-derived MSCs a promising altern

122 be used for in vitro reconstruction of human oral mucosa are also discussed.

123 ater adherence of bacterial pathogens to the oral mucosa are associated with the greater frequency of

124 C, suggesting that these immune cells of the oral mucosa are likely to be important for CMV transmiss

125 ular immunity and innate immune functions in oral mucosa are maintained.

126 anti-Candida host defense mechanisms at the oral mucosa are poorly understood.

127 IL-23-dependent manner, and that ILCs in the oral mucosa are the main source for these cytokines.

128 ticles in cells exfoliated from peri-implant oral mucosa around titanium dental implants.

129 ontal parameters and inflammatory lesions of oral mucosa as a characteristic of CD.

130 roliferation and migration stimulator of the oral mucosa as a potential therapy to prevent radiation

131 reporter TOPGAL mice and have identified the oral mucosa as a target tissue for RSpo1.

132 These studies establish the oral mucosa as an important model system to study epithe

133 wed that primary epithelial cells from human oral mucosa, as well as an oral epithelial cell line, in

134 previously isolated from the respiratory and oral mucosa, as well as circulating phagocytic cells.

135 tion profiles of bacterial microbiota in the oral mucosa associated with RAS.

136 studied after exposure to in vitro models of oral mucosa, at equilibrium by Gas-Chromatography-Flame

137 tential complementary mechanism by which the oral mucosa barrier may be disrupted during HIV-1 infect

138 d three-dimensional (3D) system of the human oral mucosa based on an immortalized human oral keratino

139 ray analysis of primary cultures of 4 normal oral mucosa biopsies, 19 dysplasias, and 16 SCCs.

140 e of CD11b(+)Ly-6G(low)F4/80(-) cells to the oral mucosa but were nonetheless highly susceptible to O

141 ive mRNA expression in histologically normal oral mucosa but with lost or down-regulated expression i

142 s highly expressed in esophagus, tongue, and oral mucosa but, in contrast to cornifin alpha, is not d

143 skin site may be affected (and, rarely, the oral mucosa) but lichen sclerosus is most common in the

144 expressed in OSCC tissues compared to normal oral mucosa by real-time PCR.

145 Oral mucosa can be consistently and successfully expande

146 s (OPC) is an opportunistic infection of the oral mucosa caused by the commensal fungus Candida albic

147 ive conjunctivitis, erythema of the lips and oral mucosa, changes in the extremities, rash, and cervi

148 sistent, nonproductive, EBV infection of the oral mucosa, characterized by limited expression of repl

149 mistry analyses suggests that the engineered oral mucosa closely resembles the normal oral mucosa.

150 Oral mucosa contains a unique transcriptional network th

151 ter-odour, the adsorption of odorants by the oral mucosa could be important but has been little explo

152 teractions occur between aroma compounds and oral mucosa depending on aroma chemical structure.

153 To investigate the role of miRNAs in oral mucosa development, we examined mice with mesenchym

154 f the immune response, is upregulated in the oral mucosa during CP compared to its level during gingi

155 phosphorylated PKCzeta expression in normal oral mucosa, dysplasia, and carcinoma as well as SCCHN t

156 of 53 tissue-specific microRNAs for skin and oral mucosa epithelium were identified.

157 dynamic nature of the cell population on the oral mucosa equivalent may be beneficial for intra-oral

158 The oral mucosa equivalent was cultured at an air-liquid int

159 we successfully assembled, ex vivo, a human oral mucosa equivalent, consisting of epidermal and derm

160 wn impaired formation of 3D Ex Vivo Produced Oral Mucosa Equivalents (EVPOME) and closure of an in vi

161 Ex vivo-produced oral mucosa equivalents (EVPOMEs) were fabricated under

162 it skin and to basement membranes in cornea, oral mucosa, esophagus, intestine, kidney collecting duc

163 el system appears appropriate for the use in oral mucosa, especially for sublingual and buccal tissue

164 Oral mucosa expansion before ridge augmentation is a pro

165 PP and intestinal mucosa, but not tonsils or oral mucosa, express mucosal addressin cell adhesion mol

166 In the oral mucosa, fibroblasts have a unique phenotype and dem

167 of gross motor patterns, by self-sampling of oral mucosa for assessment of rhythmic expression of the

168 Cs) and other innate immune cells patrol the oral mucosa for infecting microbes.

169 eons face is a lack of sufficient autogenous oral mucosa for reconstruction of the oral cavity.

170 ree-dimensional (3D) reconstruction of human oral mucosa for various in vivo and in vitro application

171 ases of HNSCC but was undetectable in normal oral mucosa from healthy subjects.

172 -fold in HNSCC (n = 24) compared with normal oral mucosa from healthy volunteers (n = 17).

173 acent to HNSCC (n = 10) compared with normal oral mucosa from healthy volunteers.

174 de a high-resolution microendoscopy image of oral mucosa from one volunteer.

175 rst and second insertions resulted in linear oral mucosa gain of 8.13 mm, and 6.44 mm, respectively.

176 and pathology in the lymphoid tissues, skin, oral mucosa, gastrointestinal tract, reproductive system

177 mmunoenzyme labeling, we show that the human oral mucosa (gingiva) is infiltrated by large numbers of

178 oral mucosa; only cases metastasizing in the oral mucosa, gingiva, and periodontium were included.

179 tro than fibroblasts isolated from untreated oral mucosa/gingiva.

180 Split-thickness or oral mucosa grafts require more than one surgical proced

181 constitutive expression in the epithelia of oral mucosa, hair follicles, and nail beds.

182 ly diverse pet shop mice, we report that the oral mucosa harbors CD8+ CD103+ resident memory T cells

183 We found that neutrophils from the oral mucosa harvested from morning saliva had released n

184 Although the oral mucosa has long been appreciated as a critical barr

185 Tissue-engineered oral mucosa have been further optimized in recent years

186 These results can justify delayed oral mucosa healing in the presence of inflammatory reac

187 Oral mucosa heals faster than does skin, yet few studies

188 s (flaccid blisters and erosions on skin and oral mucosa), histology (epidermal acantholysis), and im

189 GRgpA elicits plasma exudation from in situ oral mucosa in a catalytic site-dependent fashion by ela

190 nsplantation of cultured epithelial cells of oral mucosa in corneal limbal stem cell deficiency was s

191 fection characterized by inflammation of the oral mucosa in direct contact with the denture and affec

192 the molecular and cellular repertoire of the oral mucosa in health and disease.

193 in situ hybridisation to characterise human oral mucosa in health and oral chronic inflammatory dise

194 mpared the microRNAome of uninjured skin and oral mucosa in mice.

195 oH3 produced basement membrane separation of oral mucosa in organ culture.

196 may enhance paracellular penetration through oral mucosa in part by targeting the expression of GRHL2

197 Despite excellent healing of the oral mucosa in PDT, a lack of robust enabling technology

198 fluence the interaction between odorants and oral mucosa in the oral cavity during a "wine intake-lik

199 aims at better understanding the role of the oral mucosa in this phenomenon.

200 cytes maintained their ability to regenerate oral mucosa in vitro after 15 weeks of culture.

201 less tobacco elicits plasma exudation in the oral mucosa in vivo in a specific fashion, and that this

202 EMD improves oral mucosa incisional wound healing by promoting format

203 A levels of six innate/effector genes in the oral mucosa indicated that slower disease progression wa

204 The strength of the aroma-oral mucosa interactions seems to explain these results

205 irst time, the impact of wine composition on oral-mucosa interactions under physiological conditions.

206 However, HIV transmission through the oral mucosa is a rare event.

207 However, little is known as to whether the oral mucosa is able to modulate the local concentration

208 The oral mucosa is an attractive site for mucosal vaccinatio

209 and rs2240308 p = 0.02), and leukoplakia of oral mucosa is associated with both AXIN2 (rs2240308 p =

210 Each type of oral mucosa is believed to develop through discrete mole

211 The oral mucosa is exposed to a high density and diversity o

212 Compared to skin, wound healing in oral mucosa is faster and produces less scarring, but th

213 eviously shown that the chronically infected oral mucosa is in a state of endotoxin tolerance, as evi

214 The oral mucosa is one of the most rapidly dividing tissues

215 Radiotherapy-induced damage in the oral mucosa is the result of the deleterious effects of

216 erious effects of radiation, not only on the oral mucosa itself but also on the adjacent salivary gla

217 he lingual mucosa to determine whether minor oral mucosa lesions may enhance susceptibility to CWD in

218 leads to skin blistering (acantholysis) and oral mucosa lesions.

219 ntedanib prevented vascular pathology in the oral mucosa, lungs, and liver of the BMP9/10ib mice, as

220 icial differentiated epithelial cells of the oral mucosa, many of which appear to be shedding from th

221 he quality and quantity of desmosomes in the oral mucosa measured in the tongue and buccal mucosa.

222 mmation, we aimed to investigate whether the oral mucosa microbiota was associated with the clinical

223 he reactions suggest that intake through the oral mucosa might constitute a relevant route of exposur

224 thin layer of salivary proteins covering the oral mucosa (mucosal pellicle).

225 munolocalized to the metanephric mesenchyme, oral mucosa, nasal and cranial cartilage, and brain.

226 r component of precancerous lesions of skin, oral mucosa, nasopharynx, prostate, gut, and lung.

227 Oral mucositis refers to lesions of the oral mucosa observed in patients with cancer being treat

228 that the percentage of residents with normal oral mucosa (odds ratio (OR)=1.81, P=0.027), no visible

229 in amount of COX-2 mRNA was observed in the oral mucosa of active smokers versus never smokers.

230 ion of both ligands was also detected in the oral mucosa of active smokers.

231 test this hypothesis, we applied DHA to the oral mucosa of dogs that had been challenged with the ca

232 perturbations of polyamine metabolism in the oral mucosa of HIV+ patients.

233 ort that the altered immune landscape of the oral mucosa of HIV-positive patients on therapy involves

234 Infection of the oral mucosa of human immunodeficiency virus type 1 (HIV-

235 the phosphorylation of EGFR and HER2 in the oral mucosa of mice, and treatment with a dual EGFR and

236 and had a decreased ability to colonize the oral mucosa of mice.

237 ivo situation, we studied gene expression in oral mucosa of neonatal alpha3+/+ and alpha3-/- litterma

238 to the elevated levels of COX-2 found in the oral mucosa of smokers.

239 lin, a ligand of the EGFR, were found in the oral mucosa of smokers.

240 distinct from controls was discovered in the oral mucosa of TB patients at the start of treatment, an

241 1 microbial species could be identified from oral mucosa of two subjects.

242 (SCC25) were compared with cells from normal oral mucosa (OKF/6) and pre-malignant oral keratinocytes

243 rched for cases of metastatic lesions to the oral mucosa; only cases metastasizing in the oral mucosa

244 No harm to the oral mucosa or systemic health occurred.

245 lakia of oral mucosa, unspecified lesions of oral mucosa, or other disturbances of oral epithelium.

246 less of gender, with respect to thickness of oral mucosa overlying the tuberosity.

247 The oral mucosa plays critical roles in protection, sensatio

248 toxin tolerance, as observed in the inflamed oral mucosa, potentiates the innate Ag capture activity

249 Oral mucosa progenitor/stem cells reside as a small-size

250 are detected in primary epithelial cells of oral mucosa, prostate and mammary glands.

251 ed epithelial tissues such as the epidermis, oral mucosa, prostate, and urogenital tract.

252 sclerosis when an onlay procedure utilizing oral mucosa provides the best results using either a one

253 Oral mucosa provides the first line of defense against a

254 eered devices has demonstrated potential for oral mucosa regeneration.

255 The oral mucosa represents a physical barrier and contains i

256 Wound healing within the oral mucosa results in minimal scar formation compared w

257 after administration but a higher uptake in oral mucosa, salivary glands, and thyroid for FAPI-21.

258 developed in squamous epithelia of the skin, oral mucosa, salivary glands, tongue, esophagus, foresto

259 ttle or no methylation was seen in 10 normal oral mucosa samples.

260 Wounds of the oral mucosa show faster closure with less scar formation

261 conjugated with a cell-permeable Tat tag to oral mucosa showed prophylactic and therapeutic effects

262 ces were also detected by PCR in lymph node, oral mucosa, skin, and peripheral blood mononuclear cell

263 The conditional (oral mucosa-specific) beta1 integrin knockout (KO) mice

264 epithelium and salivary glands) and diseased oral mucosa (squamous cell carcinoma and mucoepidermoid

265 diseases from the vaginal (staphylococci) or oral mucosa (streptococci) of the body.

266 Other oral mucosa substitutes, having different cell types and

267 the in vivo effects of EMD on healing of an oral mucosa surgical wound in rats.

268 He first developed oral mucosa symptoms and vomiting at 4 years and 10 mont

269 enal glucocorticoid system is present in the oral mucosa that may play an important role in disease.

270 During inflammation of the oral mucosa, the expression of immunoreactive calprotect

271 the primary carcinogen-activating enzymes in oral mucosa, the use of curcumin as an oral cavity chemo

272 ience mild, transient local reactions in the oral mucosa, these primary reactions rarely necessitate

273 The THCs of the three oral mucosa tissues are significantly different from eac

274 antify the tissue parameters of normal human oral mucosa tissues, including labial mucosa tissue, gin

275 Exposing the oral mucosa to antigen may stimulate immune tolerance.

276 n harboring and disseminating pathogens from oral mucosa to atherosclerosis plaques, which may provid

277 d that this may be one mechanism used in the oral mucosa to attempt to regulate local immune response

278 ranging in severity from mild itching of the oral mucosa to bronchospasm.

279 s cell tumors of the forestomach, esophagus, oral mucosa, tongue, and skin.

280 utively expressed (e.g. hair follicle, nail, oral mucosa, tongue, esophagus, forestomach).

281 cted, all squamous epithelia including skin, oral mucosa, trachea, vaginal epithelium, and the epithe

282 Intraepithelial dysplasia of the oral mucosa typically originates in the proliferative ce

283 Diagnosis of leukoplakia of oral mucosa, unspecified lesions of oral mucosa, or othe

284 Oral mucosa was probed because, like fetal skin, wound r

285 At that time, if GVHD only involves skin or oral mucosa we also decrease maintenance immunosuppressi

286 Using a three-dimensional model of the human oral mucosa, we found that E-cadherin was degraded in lo

287 sy of normal-appearing, perilesional skin or oral mucosa when possible.

288 DM) allergens are deposited at the nasal and oral mucosa, where IgA is produced abundantly.

289 replication, is restricted to tissues of the oral mucosa, where it is abundant.

290 n begins in the epidermis of the skin or the oral mucosa, where the virus infects keratinocytes, and

291 rveillance of the epidermis, intestinal, and oral mucosa, whereas the presence of pathogenic microorg

292 different routes of infection, including the oral mucosa which is the most common natural route of in

293 nfection of nonhuman primates (NHPs) via the oral mucosa, which is a probable portal of natural infec

294 nstitutive expression of pBD-1 in airway and oral mucosa, which is consistent with a lack of consensu

295 ttern of endotoxin tolerance occurs in human oral mucosa with chronic periodontitis (CP).

296 lticellular three-dimensional model of human oral mucosa with induced inflammation promoted MMP12 pro

297 ucturally integral elements in the fabric of oral mucosa with significant functional roles.

298 mice developed papillomas exclusively in the oral mucosa within 1 month after tamoxifen treatment.

299 A bilateral oral mucosa wound was created via a crestal incision in

300 ssociated with pathologic alterations of the oral mucosa, yet its direct effects on human keratinocyt