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1 read electrotonically to the soma, promoting orthodromic action potential initiation.
2       Thus, the present results suggest that orthodromic action potentials initiate in the axon beyon
3 cordings from the soma and IS confirmed that orthodromic action potentials initiated in the axon and
4 ette-microwire recording, and antidromic and orthodromic activation from the ventral tegmental area a
5 usly described interneurons by their shorter orthodromic activation latencies and higher spontaneous
6              (6) Twenty Hertz activity after orthodromic activation of field CA3 was distributed in t
7 mine cell activity is mediated indirectly by orthodromic activation of putative GABAergic neurons in
8 sociated with both excitatory and inhibitory orthodromic activation of the contralateral posterior in
9 rval (S-Au) >75% TCL and was consistent with orthodromic activation of the upstream site despite its
10 eptogenesis, the cells respond to repetitive orthodromic activation with prolonged after-depolarizati
11 rated reciprocal interinsular antidromic and orthodromic activation, elicited with similar median ons
12  Neurons were identified as NTS or DMN using orthodromic and antidromic activation, respectively, fol
13 to the stimulation site and distally through orthodromic and antidromic mechanisms for several stimul
14  at sites within the carotid body from which orthodromic APs could be evoked at low threshold current
15 n potentials during and outside ripples were orthodromic, arguing against ectopic spike generation, w
16 mature atrial and ventricular complexes, and orthodromic atrioventricular reentrant tachycardia.
17 timulation and substantiated by induction of orthodromic AV reentrant tachycardia.
18                       In 7 dogs, the line of orthodromic block and the pathway of orthodromic propaga
19 t effective at baseline, which was caused by orthodromic block in the tricuspid annulus-eustachian ri
20 initiation of VT was secondary to functional orthodromic block of V2, propagation of V2 around the li
21  When pacing continued for two stimuli after orthodromic block, a second episode of block could rever
22 nd stopping pacing within one stimulus after orthodromic block.
23  contribute to neurogenic inflammation while orthodromic (centripetal) conduction could contribute to
24 ckade of AMPA receptors nearly abolished the orthodromic component of the response; subsequent antago
25                     In the other 5 dogs, the orthodromic conduction pathway of V2 around the line of
26                                              Orthodromic conduction time around the line to its dista
27 ateral posterior insula, confirming that the orthodromic electrical stimulation was not solely due to
28 the motor layers in a manner consistent with orthodromic excitation.
29 nfluence on buccal events because of the low orthodromic firing rate.
30 ed by nodulus/ventral uvula inhibition using orthodromic identification from the caudal vermis.
31 tential alternans contributed to block of an orthodromic impulse during rapid pacing.
32 d is attributable to unfiltering of multiple orthodromic impulses due to unidirectional conduction fa
33 behaviors characteristic of pain, reflecting orthodromic input to the spinal cord.
34 s in the mPFC that received monosynaptic and orthodromic inputs from the BLA demonstrated strong asso
35 iate between different types of stimulation: orthodromic inputs that engage synaptic transmission are
36 n of the reuniens evoked both antidromic and orthodromic intracellular mPFC responses, consistent wit
37                                              Orthodromic latencies to trigeminal ganglion shocks and
38   Third, the use of photostimulation ensures orthodromic neuronal activation and permits the rapid in
39                          Evoked responses to orthodromic paired-pulse stimulation were examined in th
40 line of orthodromic block and the pathway of orthodromic propagation were similar for different V1V2
41 iated influences were present throughout the orthodromic pyramidal cell EPSP/EPSC.
42 5/11 [45%] versus 0/15 [0%]; P<0.01), slower orthodromic reciprocating tachycardia (176+/-44 beats pe
43  inducible atrioventricular nodal reentry or orthodromic reciprocating tachycardia (n = 32).
44 atrioventricular nodal reentry (n = 102), 2) orthodromic reciprocating tachycardia (n = 43), 3) atria
45 lar node re-entrant tachycardia (AVNRT) from orthodromic reciprocating tachycardia (ORT) using a sept
46 ander nodofascicular [NF] accessory pathway, orthodromic reciprocating tachycardia [ORT] using a decr
47 syncope or atrial fibrillation) or low risk (orthodromic reciprocating tachycardia alone).
48 ases of atrioventricular nodal reentrant and orthodromic reciprocating tachycardia.
49 eral insula indicated that the most frequent orthodromic response was inhibitory, either alone or as
50 ur wash-out of the inhibitors antidromic and orthodromic responses were still blocked but hypoxic SD
51 nduce somatic polarization, but did modulate orthodromic responses, indicating an effect on afferents
52                Whether and how they modulate orthodromic signaling to postsynaptic targets is poorly
53 lied to the left sciatic nerve to block both orthodromic signals and antidromic DRRs without affectin
54 the somatic threshold membrane potential for orthodromic spikes.
55  examined the membrane potential response to orthodromic stimulation and intracellular current inject
56  measured extracellularly using homosynaptic orthodromic stimulation at an interval of 10 ms, was sig
57 le cell layer (GCL) that can be activated by orthodromic stimulation of AF axons in the PC.
58 occur spontaneously and are evoked by either orthodromic stimulation of the olfactory nerve or antidr
59                                We found that orthodromic stimulation of the Schaffer collaterals for
60  a normal (>/=10 mV) CA1 population spike by orthodromic stimulation of the Schaffer collaterals.
61 he thresholds of action potentials evoked by orthodromic stimulation, and shifted their initiation si
62 tion spikes (PS) in response to paired-pulse orthodromic stimulation.
63 e dentate gyrus field-potential responses to orthodromic stimulation.
64 sponse when drug applications were paired to orthodromic stimulation.
65  Bursting cells responded to brief trains of orthodromic stimuli (2-10 pulses, 5-10 ms interstimulus
66 -bursting cells responded to brief trains of orthodromic stimuli with repetitive firing (< or = 1 spi
67 ociated with only slight changes in the mean orthodromic stimulus threshold, but with a significant i
68 ardia characteristic that was diagnostic for orthodromic tachycardia, but it occurred in only 7% of a
69  in the segment activated first by the paced orthodromic wave front, and were mainly due to local pac