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1 8 for interaction of treatment with baseline orthostatic hypotension).
2 Midodrine is an alpha1-agonist approved for orthostatic hypotension.
3 classic orthostatic hypotension, and delayed orthostatic hypotension.
4 mostly influenced by age and the severity of orthostatic hypotension.
5 pheral resistance giving rise to symptomatic orthostatic hypotension.
6 st 30 beats-per-minute upon standing without orthostatic hypotension.
7 c reaction at OFC are common and can include orthostatic hypotension.
8 r other causes, such as vasovagal syncope or orthostatic hypotension.
9 harmacological approach to the management of orthostatic hypotension.
10 oglycerin, 3 neurocardiogenic syncope, and 1 orthostatic hypotension.
11 after having lost these abilities because of orthostatic hypotension.
12 One subject had evidence of orthostatic hypotension.
13 eral vascular resistance, often resulting in orthostatic hypotension.
14 ents were peripheral oedema, hypotension, or orthostatic hypotension.
15 oreceptor-cardiac reflex function and causes orthostatic hypotension.
16 m serve to protect against the occurrence of orthostatic hypotension.
17 Five of the LBD cases and all MSA cases had orthostatic hypotension.
18 action can be beneficial in the treatment of orthostatic hypotension.
19 ls, anemia, fatigue, nausea or vomiting, and orthostatic hypotension.
20 tive heart failure, cardiac arrhythmias, and orthostatic hypotension.
21 with standing, in the absence of significant orthostatic hypotension.
22 hort over 4 years; of those who died, 52 had orthostatic hypotension.
23 ious and safe in the treatment of neurogenic orthostatic hypotension.
25 bile Ehlers-Danlos syndrome, 213; neurogenic orthostatic hypotension, 86; diabetes type II, 71, mast
26 It might be possible to improve treatment of orthostatic hypotension acutely with water imbibation an
28 gns of sympathetic neurocirculatory failure (orthostatic hypotension and abnormal blood-pressure resp
29 Screening autonomic function tests indicated orthostatic hypotension and confirmed chronic autonomic
30 ide an explanation for the predisposition to orthostatic hypotension and intolerance in well-trained
34 betes increases the risk of hypertension and orthostatic hypotension and raises the risk of cardiovas
36 comes in patients with coexistent neurogenic orthostatic hypotension and supine hypertension, clinici
38 erapy in patients with coexistent neurogenic orthostatic hypotension and supine hypertension; and the
40 mediate benefits of treatment for neurogenic orthostatic hypotension and the long-term risks of supin
43 es, head trauma, hypertension in midlife and orthostatic hypotension) and 9 with Level B weaker evide
44 sed a significant increase in heart rate and orthostatic hypotension, and 20% of the nortriptyline-tr
45 with medications, structural heart disease, orthostatic hypotension, and arrhythmias, as well as wit
46 on, delayed blood pressure recovery, classic orthostatic hypotension, and delayed orthostatic hypoten
47 age, sex, race, chronic kidney disease, SBP, orthostatic hypotension, and frailty, with the exception
48 imates of impaired BP stabilization, initial orthostatic hypotension, and orthostatic hypotension are
49 c blood pressure (BP) stabilization, initial orthostatic hypotension, and orthostatic hypotension bas
50 Patients with constitutional hypotension, orthostatic hypotension, and predominant cardioinhibitio
51 re likely to have mild cognitive impairment, orthostatic hypotension, and RBD at baseline, and at pro
52 ients with PD for mild cognitive impairment, orthostatic hypotension, and RBD even at baseline visits
53 to model responses to haemorrhage, to assess orthostatic hypotension, and to evaluate haemodynamic an
56 zation, initial orthostatic hypotension, and orthostatic hypotension based on beat-to-beat blood pres
57 thesis that short-term alcohol intake causes orthostatic hypotension because of an impairment in the
59 There was no difference in the incidence of orthostatic hypotension between the treatment (11.4% [5
62 e develop autonomic dysfunction, featured by orthostatic hypotension, constipation, hypohidrosis and
63 have uncovered four major subtypes: initial orthostatic hypotension, delayed blood pressure recovery
66 tions and can capture clinically significant orthostatic hypotension during activities of daily livin
70 and all with Parkinson's disease-associated orthostatic hypotension, have a loss of cardiac sympathe
71 cause mortality among those without baseline orthostatic hypotension (hazard ratio [HR], 0.81; 95% CI
72 0.76-0.86) similarly to those with baseline orthostatic hypotension (HR, 0.83; 95% CI, 0.70-1.00; P
77 use, with the main distinction being whether orthostatic hypotension is neurogenic or non-neurogenic.
79 pertensive crises, hypotensive episodes, and orthostatic hypotension, making it the most difficult fo
83 The best cluster solution found was based on orthostatic hypotension, mild cognitive impairment, rapi
85 nt (ABPM) as a screening tool for neurogenic orthostatic hypotension (nOH), postprandial hypotension
89 Patients with Parkinson's disease (PD) and orthostatic hypotension (OH) (PD+OH) or with pure autono
90 arkinsonism and non-motor features including orthostatic hypotension (OH) and cognitive impairment.
94 rivastigmine is affected by the presence of orthostatic hypotension (OH) in patients with Parkinson'
102 sed on HUT results, we divided patients into orthostatic hypotension (OH), postural tachycardia syndr
105 n the heart and other organs, manifesting as orthostatic hypotension (OH; also known as postural hypo
106 available concerning the predictive value of orthostatic hypotension on mortality in ambulatory elder
108 itation on return, no astronauts experienced orthostatic hypotension or intolerance during routine (f
111 ve supranuclear palsy if a patient developed orthostatic hypotension or urinary incontinence with the
112 cterised by autonomic failure, manifested as orthostatic hypotension or urogenital dysfunction, with
114 measured in patients with PD with or without orthostatic hypotension (PD+OH, PD-No-OH); in patients w
116 h vibration perception thresholds (VPTs) and orthostatic hypotension (pre-PT and 6 mo, 2-3, 5-6, and
117 tic intolerance symptoms was assessed by the Orthostatic Hypotension Questionnaire (OHQ), consisting
119 or autonomic failure in Parkinson disease), orthostatic hypotension reflects sympathetic neurocircul
120 Establishing whether symptoms are due to orthostatic hypotension requires careful history taking,
121 r adults include gait and balance disorders, orthostatic hypotension, sensory impairment, medications
122 uncomplicated faint, situational syncope, or orthostatic hypotension should receive electrocardiograp
123 lower handgrip strength, hearing impairment, orthostatic hypotension, stroke, diabetes, heart disease
124 n, upper gastrointestinal tract dysfunction, orthostatic hypotension, sweating abnormalities, or erec
125 gene encoding alpha-synuclein, also features orthostatic hypotension, sympathetic neurocirculatory fa
126 llenges, including changes in posture (e.g., orthostatic hypotension, syncope) and physical activity.
128 apeutic requirements for managing neurogenic orthostatic hypotension that manifests with falls or cog
129 standing blood pressure assessments, 9% had orthostatic hypotension (that is, a drop in blood pressu
130 supine hypertension when treating neurogenic orthostatic hypotension; the effectiveness of nocturnal
131 usion from cardiac arrest, heart failure, or orthostatic hypotension to AD pathology, while atheroscl
132 e symptomatic autonomic failure (symptomatic orthostatic hypotension, urinary incontinence, or both)
133 perechogenicity, olfactory loss, depression, orthostatic hypotension, urinary/erectile dysfunction, P
134 There was no effect by center, severity of orthostatic hypotension, use of fludrocortisone or compr
135 sk factors were identified across 6 domains: orthostatic hypotension, visual impairment, impairment o
138 t stroke, coronary heart disease and cancer, orthostatic hypotension was a significant independent pr
139 Program's fourth examination (1991 to 1993), orthostatic hypotension was assessed in relation to subs
143 ed mortality rates in those with and without orthostatic hypotension were 56.6 and 38.6 per 1000 pers
144 ous events, two of which (hallucinations and orthostatic hypotension) were deemed related to study dr
146 migraine and chronic vascular issues (e.g., orthostatic hypotension) were often misclassified, impac
148 of CVD or all-cause mortality regardless of orthostatic hypotension without evidence for different e